
FutureSlayer
Go back to sleep, Ellen
- Joined
- Oct 4, 2019
- Posts
- 8,948
- Reputation
- 10,326
nope . zero changeDidn’t even make it softer?
Follow along with the video below to see how to install our site as a web app on your home screen.
Note: this_feature_currently_requires_accessing_site_using_safari
nope . zero changeDidn’t even make it softer?
But dht inhibits elastin, you shouldve seen at leasr improvmentnope . zero change
only rat studiesBut dht inhibits elastin, you shouldve seen at leasr improvment
yea bro I’d like to but sadly zero change at all, I’ve been on 0.5mg/day for 2 years rnBut dht inhibits elastin, you shouldve seen at leasr improvment
depends on countryIs it prescription only? Sorry if this was already asked
finasteride yea, only take it if you are balding though the cons are greater than any of the other effects other than hairgrowthIs it prescription only? Sorry if this was already asked
eh dht is the culprit of hairloss but side effects of fin are not 1% id say closer to 50% maybe 5% severe side effects but i would say 50% of men have lower libido on finasteride if not moreanyone who is balding or has it in their family don’t listen to spergs who tell you that DHT isn’t the culprit or that’s it’s important. there’s so much fearmongering and bro science around a drug that has clearly demonstrated to improve acne, hair loss and prostate with side effects less than 1% more than placebo. don’t lose your hair because some retard has a hard on for dht. it can also be implicated in many other problems such as cardiovascular disease.
Nah sorry buddy but no study especially rigorous clinical trials show this. DHT is a paracrine not endocrine hormone and acts locally where it is converted!!!! If you are experiencing side effects i’m sorry you are but there is a much greater likelihood it is nocebo than it is from a change in hormones. There's no strong evidence linking natural DHT levels to improved libido, sexual function, mood, or cognitive function in adult men.eh dht is the culprit of hairloss but side effects of fin are not 1% id say closer to 50% maybe 5% severe side effects but i would say 50% of men have lower libido on finasteride if not more
I take finasteride and have lower libido and nipple sensitivity got bloods done and its not an estrodial issue
Fin is a tradeoff you trade hair for lower libido in most men. All my friends that take fin agree aswell.
How bad was your hairloss or did you just take it as a precautionyea bro I’d like to but sadly zero change at all, I’ve been on 0.5mg/day for 2 years rn
precautionHow bad was your hairloss or did you just take it as a precaution
bro I made this thread I am on finasteride lol im not fear mongering.Nah sorry buddy but no study especially rigorous clinical trials show this. DHT is a paracrine not endocrine hormone and acts locally where it is converted!!!! If you are experiencing side effects i’m sorry you are but there is a much greater likelihood it is nocebo than it is from a change in hormones. There's no strong evidence linking natural DHT levels to improved libido, sexual function, mood, or cognitive function in adult men.
I place little trust in guys like you who pull statistics out of their ass, as far as i’m concerned you’re in the same group that believes topical minox ruins their libido. The fearmongering is completely unwarranted.
dht is pretty good for sex drive though so its a tradeoff hopefully the cure to baldness will come soonIm happy for you mate. I swear DHT is pure cancer. Brings nothing but suffering
topical barely works itself, imagine fear mongering this much with sidesbro I made this thread I am on finasteride lol im not fear mongering.
Yea my swollen painful nipples on higher doses of topical are definitely a nocebo even in smaller doses I can still feel it a bit. You're also unable to prove that DHT has no effect on libido,mood,cognitive function because they are not measurable variables they are just patient reported.
There are so many men that come off finasteride and relize how much there libido was lowered and they didnt even know, i belive thats the case for majority of men on finasteride.
The merck studies are ass anyways they got sued and lost for hiding information about side effects, other studies show higher instidences of side effects and lower libido especcially.
Its naive to think that fin/dut wont cause any problems but my point is Hair>most of the minor side effects. If you can get it up still its worth it.
Also I forgot to mention higher doses of finasteride give me crazy insomina
you’re probably just really prone to gyno, lose bodyfat.bro I made this thread I am on finasteride lol im not fear mongering.
Yea my swollen painful nipples on higher doses of topical are definitely a nocebo even in smaller doses I can still feel it a bit. You're also unable to prove that DHT has no effect on libido,mood,cognitive function because they are not measurable variables they are just patient reported.
There are so many men that come off finasteride and relize how much there libido was lowered and they didnt even know, i belive thats the case for majority of men on finasteride.
The merck studies are ass anyways they got sued and lost for hiding information about side effects, other studies show higher instidences of side effects and lower libido especcially.
Its naive to think that fin/dut wont cause any problems but my point is Hair>most of the minor side effects. If you can get it up still its worth it.
Also I forgot to mention higher doses of finasteride give me crazy insomina
OH NO NOT THE CLEAR SEMENRINOS HOW WILL I COPE WITH MY CLEAR SEMENRINOS?????It started after two weeks on dut, after my semen turned clear and it never stopped
incredible deployment of the scientific method. Coomers don't have hair therefore???When was the last time you saw a coomer with a full head of hair? prolactin, which increases when you coom, is also involved in hairloss which is also increasd by high estrogen.
The root cause is contingent on AR polymorphisms. That's it. So fucking simplenot the root cause
You need to grow up. If you're still harping on about Jews past the age of 18 you've got developmental problemsis that what the white labcoats(jews) are telling you? better get the vax booter #164 as well while you're eating da bugz
Hence why some people need topical antiandrogens. The fact that in some individuals' hairloss worsens with dutasteride doesn't evidence your theory that hairloss occurs irrespective AR signalling, it's bolstering the fact that aberrant levels of androgens/androgen signalling is the cause. Testosterone skyrockets with dutasteride usage, that alone can cause shedding, sebaceous gland hypetrophy and every other telltale sign of high androgens.DHT blockers also don't work for everyone, typically when you take them your body will compensate for decreased serum dht by converting more testosterone in DHT (rebound)
This isn't untrue, fortunately PDE5 inhibitors exist and can be used longterm.DHT also enhances nitric oxide, which is why when people use fin/dut often get ED. DHT actually increases hair growth (usually on the body). How can something that increases circulation also "choke" hair folicles.
Peatfag spotted, imagine unironically falling for that utter garbage. People who hate estrogen look like haidutthey still prove my point that estrogen is not good for you
No it isn't. Realistically the first bout of negative feedback occurs because you've drastically decreased AR signalling with the introduction of duta/fin, then your body upregulates androgen synthesis to combat the lack of AR signalling, which then consequently increases estradiol, which then suppresses the HPTA. The body will reach a new homeostasis.this is also why some people continue to lose hair on fin/dut
Imagine fucking cope this hard. "Finasteride works, but it doesn't work through the mechanism BY WHICH WE KNOW IT WORKS"The main reason finasteride works is because it inhibits the conversion of progesterone into 5α-Dihydroprogesterone
Said it better than i’ve been able to, retard will probably still disagree with you tho. Based and high IQ anything else you’re knowledgeable asf on? I’m impressedOH NO NOT THE CLEAR SEMENRINOS HOW WILL I COPE WITH MY CLEAR SEMENRINOS?????
incredible deployment of the scientific method. Coomers don't have hair therefore???
The root cause is contingent on AR polymorphisms. That's it. So fucking simple
Stop the transcription from occurring at AR and you won't lose hair, it doesn't matter how you do this, just do it.
You need to grow up. If you're still harping on about Jews past the age of 18 you've got developmental problems
Hence why some people need topical antiandrogens. The fact that in some individuals' hairloss worsens with dutasteride doesn't evidence your theory that hairloss occurs irrespective AR signalling, it's bolstering the fact that aberrant levels of androgens/androgen signalling is the cause. Testosterone skyrockets with dutasteride usage, that alone can cause shedding, sebaceous gland hypetrophy and every other telltale sign of high androgens.
This isn't untrue, fortunately PDE5 inhibitors exist and can be used longterm.
Peatfag spotted, imagine unironically falling for that utter garbage. People who hate estrogen look like haidut
No it isn't. Realistically the first bout of negative feedback occurs because you've drastically decreased AR signalling with the introduction of duta/fin, then your body upregulates androgen synthesis to combat the lack of AR signalling, which then consequently increases estradiol, which then suppresses the HPTA. The body will reach a new homeostasis.
Imagine fucking cope this hard. "Finasteride works, but it doesn't work through the mechanism BY WHICH WE KNOW IT WORKS"
Take progesterone and see if your hair grows back.
I'm at 12% bf but trying to get to 10% anyway. I thought it was caused by test conversion into estrogen so I got blood done and everything was in midrange test/prolactin/estradiol ultra-sensitive. There are no studies that explain why gyno occurs with finasteride that I can find, but what makes sense is that dht antagonizes estrogen and the removal of it lets estrogen do its thing in certain tissues. I don't know but that's my theory based on my bloodwork results. Regardless I would not say it is a common symptom probably in around 5% get sensitivity.you’re probably just really prone to gyno, lose bodyfat.
as for all the other stuff you make some valid points, personally fin does nothing to me and n=1 but given how useless and cancerous it is im not surprised. dutasteride has also been known to increase libido through the extra increase of testosterone so who fucking knows. maybe your sides will go away bro just ride it out.
Absolute worst case solution that is guaranteed to work is to take 100mg of oxandrolone, grind it up, and homogenize it with eg. 75ml of CeraVe moisturizing cream (concentration 1.3mg/ml). Topical application to nipples will remove all chance of gyno development and immediately resolve puffiness.I'm at 12% bf but trying to get to 10% anyway. I thought it was caused by test conversion into estrogen so I got blood done and everything was in midrange test/prolactin/estradiol ultra-sensitive. There are no studies that explain why gyno occurs with finasteride that I can find, but what makes sense is that dht antagonizes estrogen and the removal of it lets estrogen do its thing in certain tissues. I don't know but that's my theory based on my bloodwork results. Regardless I would not say it is a common symptom probably in around 5% get sensitivity.
I plan to ride it out low doses help with the sensitivity and i don't belive I have chest growth over the past 5 months but I do get swelling from time to time.
Can’t you just surgerymax the nipples away?they will probably get huge and puffy in a few years and you will really regret it.
Big nipples is not worth more hair especially when there is rogaine
yea but there is a possibility of regrowth after surgeryCan’t you just surgerymax the nipples away?
Rogaine kills collagen which is a lot worse
Hmm, I've never heard of that ill look into it, usually people would recommend raloxifene or armidexAbsolute worst case solution that is guaranteed to work is to take 100mg of oxandrolone, grind it up, and homogenize it with eg. 75ml of CeraVe moisturizing cream (concentration 1.3mg/ml). Topical application to nipples will remove all chance of gyno development and immediately resolve puffiness.
Source : My black nigger assRogaine kills collagen which is a lot worse
there are studies in vitro and in rats usually using her doses that show collagen inhibition, pde2 is raised which also increases aging although they usually use way more minox than normal.Source : My black nigger ass
No, only the alcohol solvent in the topical is bad but there's no collagen rapeNot enough studies to prove it but there is a correlation.
the answer to this problem is there some evidence of collagen inhibition, its not concrete. There is nothing to disprove or approve of it.No, only the alcohol solvent in the topical is bad but there's no collagen rape
Yes bro let's totally ignore the burden of proof so you're rightthe answer to this problem is there some evidence of collagen inhibition, its not concrete. There is nothing to disprove or approve of it.
So basically we have no answer
Yeah this is more effective, know a guy on fin who did this exact thing and it worked completely. You won’t find much info about it as he invented it, smart MD so i trust him and the reasoning makes sense.Hmm, I've never heard of that ill look into it, usually people would recommend raloxifene or armidex
dude im 26 using collagen since 22Can’t you just surgerymax the nipples away?
Rogaine kills collagen which is a lot worse
how does that work foam is still suspended in alcohol and even worse ingredients than liquid it has butane and bht and stuff. The collagen worries are from the minoxidil molecule itself due to its potential to inhibit collagen.dude im 26 using collagen since 22
the collagen thing only happens when you use liquid minox
rogaine foam doesnt do as bad to collagen
Also you can just eat a lot of collage or mix it with water and inject it into your face like how they inject fillers
Having big nipples would be way weirder then getting lines on your face.
Actually no one does it because no one thought about it. It's how they made fillers in the 1960show does that work foam is still suspended in alcohol and even worse ingredients than liquid it has butane and bht and stuff. The collagen worries are from the minoxidil molecule itself due to its potential to inhibit collagen.
is this ragebait? injecting collagen in your face that's not how it works bro if that as true everybody would be doing it eating collagen does nothing aswell
Idk sounds like cope havent heard of anyone doing that on this forum which is a place someone would try that, you might end up with a infection or something.Actually no one does it because no one thought about it. It's how they made fillers in the 1960s
so yea you can make your own fillers with a bovine collagen base and if you are really precise you can inject them like fillers![]()
The History of Dermal Fillers - Aesthetic Advancements Institute (AAI)
Dermal Fillers have been around for over 40 years, but have drastically changed over the years. In understanding any type of injection, it is important to look at its history for valuable lessons, in order for progress to be a success. And quite honestly, it’s very interesting to see where we...aestheticadvancements.com
Anyways the rogaine foam doesnt make me have rashes on my skin but the liquid minox irritates the fuck out of my skin. I dont look older than my age but also ive been injecting collagen fillers for like months now and i think it pretty much restored my collagen.
being impotent and not being able to father children - awesomeOH NO NOT THE CLEAR SEMENRINOS HOW WILL I COPE WITH MY CLEAR SEMENRINOS?????
Stereotypes are earned, cooming releases prolactin, which is also secreted by estrogen. Prolactin is also known to cause hairlossincredible deployment of the scientific method. Coomers don't have hair therefore???
There are multiple causes of hairloss - its not as simple as ARThe root cause is contingent on AR polymorphisms. That's it. So fucking simple
Stop the transcription from occurring at AR and you won't lose hair, it doesn't matter how you do this, just do it.
found the jew - seem's like you're shilling for big pharma, trying to poison the goys I presume. Hows the weather in Tel Aviv?You need to grow up. If you're still harping on about Jews past the age of 18 you've got developmental problems
There are countless pathways that all contribute to hairloss; wnt, tgf beta, vasodialation (NO), reactive oxygen species, IGF-1, anti-androgens (estrogen/prolactin), prostaglandins, inflamation, etc.Hence why some people need topical antiandrogens. The fact that in some individuals' hairloss worsens with dutasteride doesn't evidence your theory that hairloss occurs irrespective AR signalling, it's bolstering the fact that aberrant levels of androgens/androgen signalling is the cause. Testosterone skyrockets with dutasteride usage, that alone can cause shedding, sebaceous gland hypetrophy and every other telltale sign of high androgens.
There are countless studies showing DHT's effects on endothelial nitric oxide synthase, gene express modulation (DHT-AR transcription), ET-1. prostanoids, VSMCs proliferation etcThis isn't untrue, fortunately PDE5 inhibitors exist and can be used longterm.
Haidut is a PSL godPeatfag spotted, imagine unironically falling for that utter garbage. People who hate estrogen look like haidut
clearly not, there are hundreds of people on reddit/ internet forums that persistently shed on fin/dut, for over 2+ years (sometimes forever), more so on dut. The reason less people shed on fin (and tolerate it) is a clear indication that these drugs create persistent androgen dysfunction (PFS), the more androgens you suppress the more problems you have. simple as.No it isn't. Realistically the first bout of negative feedback occurs because you've drastically decreased AR signalling with the introduction of duta/fin, then your body upregulates androgen synthesis to combat the lack of AR signalling, which then consequently increases estradiol, which then suppresses the HPTA. The body will reach a new homeostasis.
Finasteride works because it increases progesterone, which in turn increases PGE1 PG12, PGE2, etc. There are also other mechanism wherby fin exerts effects such as cytokine modulation, inflamation, reducing fibrosis, VEGF modulation.Imagine fucking cope this hard. "Finasteride works, but it doesn't work through the mechanism BY WHICH WE KNOW IT WORKS"
Already doing that, good results so farTake progesterone and see if your hair grows back.
progesterone inhibits 5ar which is why you might see resultsbeing impotent and not being able to father children - awesome
unlike incels like u there are people who use their penis and want to impregnate bitches. for many people being able to have sex and father children is one of the most important aspects of their life. the sides from 5AR inhibitors are grosly underreported, everyone i know has had libido issues and clear semen.
Stereotypes are earned, cooming releases prolactin, which is also secreted by estrogen. Prolactin is also known to cause hairloss
There are multiple causes of hairloss - its not as simple as AR
found the jew - seem's like you're shilling for big pharma, trying to poison the goys I presume. Hows the weather in Tel Aviv?
There are countless pathways that all contribute to hairloss; wnt, tgf beta, vasodialation (NO), reactive oxygen species, IGF-1, anti-androgens (estrogen/prolactin), prostaglandins, inflamation, etc.
There are countless studies showing DHT's effects on endothelial nitric oxide synthase, gene express modulation (DHT-AR transcription), ET-1. prostanoids, VSMCs proliferation etc
Haidut is a PSL god
clearly not, there are hundreds of people on reddit/ internet forums that persistently shed on fin/dut, for over 2+ years (sometimes forever), more so on dut. The reason less people shed on fin (and tolerate it) is a clear indication that these drugs create persistent androgen dysfunction (PFS), the more androgens you suppress the more problems you have. simple as.
Finasteride works because it increases progesterone, which in turn increases PGE1 PG12, PGE2, etc. There are also other mechanism wherby fin exerts effects such as cytokine modulation, inflamation, reducing fibrosis, VEGF modulation.
Already doing that, good results so far)
Show me LITERALLY one piece of evidence that suggests 5ar inhibitors cause sterility. Fucking one.being impotent and not being able to father children - awesome
Massive cope you colossal faggot. I have sex MORE because my face isn't ridden with acne and my hair is intact.unlike incels like u there are people who use their penis and want to impregnate bitches
Again, incredible deployment of the scientific method. I don't give a literal fuck who you know has experienced sides, I could say the exact inverse and it'd still be just as invalid as the nonsense you're spewing.everyone i know has had libido issues and clear semen.
Found the 18 year old. Unironically grow up you moron, jesus christ. You'll be cringing at yourself in a couple years I guarantee youfound the jew
Who said there wasn't? What counts is the fact that androgens clearly play the most significant role by far. Just cage at the vasodialation holy shit you're irredeemably retarded.There are countless pathways that all contribute to hairloss; wnt, tgf beta, vasodialation (NO), reactive oxygen species, IGF-1, anti-androgens (estrogen/prolactin), prostaglandins, inflamation, etc.
I said it wasn't untrue. I still don't think anything that you just mentioned is worthy of losing your hair for. Imagine worshipping aThere are countless studies showing DHT's effects on endothelial nitric oxide synthase, gene express modulation (DHT-AR transcription), ET-1. prostanoids, VSMCs proliferation etc
Taking a stance that is so vehemently contrary to the widely accepted hypothesis on AGA is not SIMPLE. Pseudohemaphradites don't lose hair and have significantly lower levels of androgens. But I guess ummmmm..... *checks notes*.... uhmm *checks notes again* something about progesterone aahh protaglandins something something *checks notes again*simple as
Fantastic, just keep your retardation to yourself and let those who don't want a worthless subhuman hormone circulating their body actually do something about their hair/skinAlready doing that, good results so far)
In which case just use RU, which is infinitely better than dutasteride and finasterideIf dutasteride doesn't work you don't have androgenic alopecia or your genetics are just so bad your hair is sensitive to testosterone aswell.
RU isnt the safest reccomendation, it seems to really affect the heart and the creator dropped it with no explanation.In which case just use RU, which is infinitely better than dutasteride and finasteride
These peatfags get themseleves bred by haidut's VIRILE DHT-RIDDEN SEMEN on a daily basis
+upvoteComical how there’s always a strong link between DHT worshippers saying fin/dut are jew drugs and being anti vax.
For the joy of not being bald and depressed if you have androgenic alopecia don’t listen to Vista he is clearly traumatised and coping with his hair loss and angry with reality.
Amory JK, Amory DW, Bremner WJ. "Prospective study of the effects of low-dose finasteride on semen parameters in normal men." J Urol. 2007 May;177(5):2112-6Show me LITERALLY one piece of evidence that suggests 5ar inhibitors cause sterility. Fucking one.
"this is anti- semitism, the side effects don't exist goyim!"I swear you DHT worshipping faggots are all the same. Spewing absolute garbage about stuff that doesn't exist
you even sound like a castrated faggot lol, ur balls are probably the size of skittles, I'd be suprised if you even have an erection once a year roid tranny "respectable sex drive" - said like a true fagMassive cope you colossal faggot. I have sex MORE because my face isn't ridden with acne and my hair is intact.
I've been on and off steroids for years now and removing DHT has never negatively effected me, it only subtracts the hairloss and acne. Even when I've been natty I've maintained a respectable sex drive but w/o the poison that comes alongside DHT.
source??Again, incredible deployment of the scientific method. I don't give a literal fuck who you know has experienced sides, I could say the exact inverse and it'd still be just as invalid as the nonsense you're spewing.
I'm fairly confident I'm older than you, maybe in a couple of years you look in the mirror and realise how much of a fag u areFound the 18 year old. Unironically grow up you moron, jesus christ. You'll be cringing at yourself in a couple years I guarantee you
Its not a simple cause and effect relationship, high DHT is likely a side effect of some sort of metabolic dysfunction, have you ever thought to ask why folicles become DHT sensitive in the first place? why are more and more people balding?- there is always something beneath the surface, which is what I'm getting at with these biomarkers.Who said there wasn't? What counts is the fact that androgens clearly play the most significant role by far. Just cage at the vasodialation holy shit you're irredeemably retarded.
The thought of dying on the hill of DHT whilst doing everything you can to maximise/minimise the aforementioned biomarkers just to gain a fraction of what you could on 5ARis is unbelievably comical.
DHT is very anabolic (proviron, etc), I don't think you understand what you are doing to your body when u take 5ar blockers.I said it wasn't untrue. I still don't think anything that you just mentioned is worthy of losing your hair for. Imagine worshipping a
nonanabolic hormone. Tell me you look like haidut without telling me you look like haidut.
lol, how much is shlomo from J&J paying for each of these posts shill? Must be earning well over 100 sheckles for this quality lol.Taking a stance that is so vehemently contrary to the widely accepted hypothesis on AGA is not SIMPLE. Pseudohemaphradites don't lose hair and have significantly lower levels of androgens. But I guess ummmmm..... *checks notes*.... uhmm *checks notes again* something about progesterone aahh protaglandins something something *checks notes again*
You're a joke, cunt.
Fantastic, just keep your retardation to yourself and let those who don't want a worthless subhuman hormone circulating their body actually do something about their hair/skin
This has nothing to do with your original post you disingenuous cunt. Impotence is not the same as lowered sperm concentration, sperm motility and morphology. Speaking of which, those parameters don't seem to be effected at all on finasteride:Amory JK, Amory DW, Bremner WJ. "Prospective study of the effects of low-dose finasteride on semen parameters in normal men." J Urol. 2007 May;177(5):2112-6
Not a single soul said the side effects don't exist. What the fuck are you on about?"this is anti- semitism, the side effects don't exist goyim!"
Balls are huge because of HCGyou even sound like a castrated faggot lol, ur balls are probably the size of skittles
Respectable yes, unlike haidut-trannies, we value our looks as opposed to the marginal difference in sex drive that the deprivation of DHT causes.'d be suprised if you even have an erection once a year roid tranny "respectable sex drive" - said like a true fag
Low hanging fruit obviously because you're genuinely a fucking moron, but why are you even bothering with citing anything if this is your stance? Science is BAD except for when Haidut and the DHT-worshipping subhumans find some niche study that confirms our biases!!! Science GOOD!source??mainstream science is gay af, no journal/ scientists will admit there are side effects, people still continue to deny PFS lol. why would any scientist publish anything against the trillion dollar pharmacuetical industry.
If you're older it's fucking embarrassing and you need to blow your tiny skull to smithereens for even drawing breath.I'm fairly confident I'm older than you, maybe in a couple of years you look in the mirror and realise how much of a fag u are
Right we should just base everything off of what the Haidut-trannies say to one another whilst pumping each other with their poz loads. Awesome stuffscientific method is a relic of the past lol, science is overly politicized. you should eat the bugs and drink soy (the scientists say it's good for you)
AGA is the result of genetic polymorphisms at the AR. Removing potent androgens from the equation only alleviates the problem because you're depriving the overly sensitive AR of stimulation. This is why people bald regardless of their androgen indexIts not a simple cause and effect relationship, high DHT is likely a side effect of some sort of metabolic dysfunction, have you ever thought to ask why folicles become DHT sensitive in the first place?
RU is by far the most efficaious treatment. Go on r/tressless and you'll see countless anecdotes of people adding in RU and seeing drastic changes in their regrowth/recession. With RU you're solving the root of the problem without causing systemic changes to your AR signalling elsewhere in the body (it does go slightly systemic obviously).perhaps only RU - which doesn't even work that well
Again, not a soul has said this. The side effects just aren't as dire as people make them out to be and are 100% worth it.side effects that fin/dut fanboys pretend don't exist
No one said androgens are bad. Dihydrotestosterone, however, is utterly worthless. Testosterone, contrarily, is actually a hormone you want in your body. It's anabolic and it's androgenic enough to maintain normal physiological function without being overbaringly so (acne, hairloss)You are attacking the problem from the top of the iceberg (le androgens bad) and complete fail to neglect anything below the surface.
Dude you don't know how hard I'm smiling right now. You've legitimately ousted yourself as a complete drooling imbecile. DHT is swiftly metabolised by 3-alpha-hydroxysteroid reductase in muscle tissue (non-androgenic tissue). It isn't anabolicDHT is very anabolic (proviron, etc), I don't think you understand what you are doing to your body when u take 5ar blockers.
Brother it's all good if you've managed to find a way to abate hairloss without touching DHT but that doesn't dissprove the fact that androgens are directly implicated in hairloss.You call me a retard but all you do is parrot the hivemind rhetoric of castrated "Pseudohemaphradite" soybois. You completely fail to neglect the complex endocrinological interplay between hormones and its extended effect on our body - "worthless subhuman hormone" lol.
It's so fucking hilarious to actually see you skinnyfat haidut peatcattle in the wild.Keep castrating yourself boyo, you're doing us all a favour by keeping your low iq genetics out of the pool
Prolactin causes hairloss, right? Why do we witness people employing nandrolone monotherapy achieve great success at reversing hairloss? Nandrolone greatly increases prolactin via progesterone signalling. It's almost as if DHN is a weaker androgenic metabolite than nandrolone. Ah, it must be some other cope then, you'll think of something.Stereotypes are earned, cooming releases prolactin, which is also secreted by estrogen. Prolactin is also known to cause hairloss
+upvoteThis has nothing to do with your original post you disingenuous cunt. Impotence is not the same as lowered sperm concentration, sperm motility and morphology. Speaking of which, those parameters don't seem to be effected at all on finasteride:
There were no significant effects of 1 mg. finasteride on sperm concentration, total sperm per ejaculate, sperm motility or morphology.
Not a single soul said the side effects don't exist. What the fuck are you on about?
Balls are huge because of HCG
Respectable yes, unlike haidut-trannies, we value our looks as opposed to the marginal difference in sex drive that the deprivation of DHT causes.
Low hanging fruit obviously because you're genuinely a fucking moron, but why are you even bothering with citing anything if this is your stance? Science is BAD except for when Haidut and the DHT-worshipping subhumans find some niche study that confirms our biases!!! Science GOOD!
If you're older it's fucking embarrassing and you need to blow your tiny skull to smithereens for even drawing breath.
Right we should just base everything off of what the Haidut-trannies say to one another whilst pumping each other with their poz loads. Awesome stuff
AGA is the result of genetic polymorphisms at the AR. Removing potent androgens from the equation only alleviates the problem because you're depriving the overly sensitive AR of stimulation. This is why people bald regardless of their androgen index
RU is by far the most efficaious treatment. Go on r/tressless and you'll see countless anecdotes of people adding in RU and seeing drastic changes in their regrowth/recession. With RU you're solving the root of the problem without causing systemic changes to your AR signalling elsewhere in the body (it does go slightly systemic obviously).
Again, not a soul has said this. The side effects just aren't as dire as people make them out to be and are 100% worth it.
No one said androgens are bad. Dihydrotestosterone, however, is utterly worthless. Testosterone, contrarily, is actually a hormone you want in your body. It's anabolic and it's androgenic enough to maintain normal physiological function without being overbaringly so (acne, hairloss)
Dude you don't know how hard I'm smiling right now. You've legitimately ousted yourself as a complete drooling imbecile. DHT is swiftly metabolised by 3-alpha-hydroxysteroid reductase in muscle tissue (non-androgenic tissue). It isn't anabolic
Mesterolone (proviron) isn't dihydrotestosterone, not even close. And before you run to google, yes, it's 1a-methyl-DHT but that doesn't mean it confers the same affinity nor potency at the AR as DHT.
The reason Mesterolone feels so androgenic is because it binds to SHBG at 440% the rate of DHT, which means it is essentialy binding all SHBG and freeing up more androgens in general. This is why people report acne and hairloss, because the androgen index skyrocketts due to the bound SHBG.
Brother it's all good if you've managed to find a way to abate hairloss without touching DHT but that doesn't dissprove the fact that androgens are directly implicated in hairloss.
It's so fucking hilarious to actually see you skinnyfat haidut peatcattle in the wild.
Prolactin causes hairloss, right? Why do we witness people employing nandrolone monotherapy achieve great success at reversing hairloss? Nandrolone greatly increases prolactin via progesterone signalling. It's almost as if DHN is a weaker androgenic metabolite than nandrolone. Ah, it must be some other cope then, you'll think of something.
You'll come around eventually, once you realise how futile your ridiculous pursuit is. The irony of this sitatuon is that trannies literally use progesterone as a form of HRT.
I’ve used both. Nasty dark circles from minSource : My black nigger ass
ok i've used both aswell and didn't get shit, what do we do nowI’ve used both. Nasty dark circles from min
dnrThis has nothing to do with your original post you disingenuous cunt. Impotence is not the same as lowered sperm concentration, sperm motility and morphology. Speaking of which, those parameters don't seem to be effected at all on finasteride:
There were no significant effects of 1 mg. finasteride on sperm concentration, total sperm per ejaculate, sperm motility or morphology.
Not a single soul said the side effects don't exist. What the fuck are you on about?
Balls are huge because of HCG
Respectable yes, unlike haidut-trannies, we value our looks as opposed to the marginal difference in sex drive that the deprivation of DHT causes.
Low hanging fruit obviously because you're genuinely a fucking moron, but why are you even bothering with citing anything if this is your stance? Science is BAD except for when Haidut and the DHT-worshipping subhumans find some niche study that confirms our biases!!! Science GOOD!
If you're older it's fucking embarrassing and you need to blow your tiny skull to smithereens for even drawing breath.
Right we should just base everything off of what the Haidut-trannies say to one another whilst pumping each other with their poz loads. Awesome stuff
AGA is the result of genetic polymorphisms at the AR. Removing potent androgens from the equation only alleviates the problem because you're depriving the overly sensitive AR of stimulation. This is why people bald regardless of their androgen index
RU is by far the most efficaious treatment. Go on r/tressless and you'll see countless anecdotes of people adding in RU and seeing drastic changes in their regrowth/recession. With RU you're solving the root of the problem without causing systemic changes to your AR signalling elsewhere in the body (it does go slightly systemic obviously).
Again, not a soul has said this. The side effects just aren't as dire as people make them out to be and are 100% worth it.
No one said androgens are bad. Dihydrotestosterone, however, is utterly worthless. Testosterone, contrarily, is actually a hormone you want in your body. It's anabolic and it's androgenic enough to maintain normal physiological function without being overbaringly so (acne, hairloss)
Dude you don't know how hard I'm smiling right now. You've legitimately ousted yourself as a complete drooling imbecile. DHT is swiftly metabolised by 3-alpha-hydroxysteroid reductase in muscle tissue (non-androgenic tissue). It isn't anabolic
Mesterolone (proviron) isn't dihydrotestosterone, not even close. And before you run to google, yes, it's 1a-methyl-DHT but that doesn't mean it confers the same affinity nor potency at the AR as DHT.
The reason Mesterolone feels so androgenic is because it binds to SHBG at 440% the rate of DHT, which means it is essentialy binding all SHBG and freeing up more androgens in general. This is why people report acne and hairloss, because the androgen index skyrocketts due to the bound SHBG.
Brother it's all good if you've managed to find a way to abate hairloss without touching DHT but that doesn't dissprove the fact that androgens are directly implicated in hairloss.
It's so fucking hilarious to actually see you skinnyfat haidut peatcattle in the wild.
Prolactin causes hairloss, right? Why do we witness people employing nandrolone monotherapy achieve great success at reversing hairloss? Nandrolone greatly increases prolactin via progesterone signalling. It's almost as if DHN is a weaker androgenic metabolite than nandrolone. Ah, it must be some other cope then, you'll think of something.
You'll come around eventually, once you realise how futile your ridiculous pursuit is. The irony of this sitatuon is that trannies literally use progesterone as a form of HRT.