Seven7Tokyo
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Welcome everybody to my official 2ND guide. (kinda nervous again)
In this guide I‘ll be explaing everything about AAS‘s a.k.a. Anabolic-Androgenic Steroids, let‘s get straight into it.
In this guide I‘ll be explaing everything about AAS‘s a.k.a. Anabolic-Androgenic Steroids, let‘s get straight into it.
This guide will be split into various sections:
1) Introduction to AAS
2) Common types of AAS
3) Functionality and Mechanisms + Benefits & Risks
4) Administration Protocols & Pharamcokinetics
5) Ancillary Drugs (The Support System)
6) Blood work and monitoring
7) Post-Cycle Therapy (PCT)
Coloured Text:
Yellow: Section
Purple: Sub-sections
Red: Important notes/Noted risks
Green: Noted benefits
1. Small Introduction to AAS (Anabolic-Androgenic Steroids)
Anabolic-Androgenic Steroids (AAS) are synthetic derivatives of the male hormone testosterone, designed to mimic its anabolic (muscle-building) and androgenic (masculinizing) effects.
These substances are widely used in sports and fitness to enhance physical performance and appearance.
2. Common Types of AAS
There are numerous types of AAS, each with unique properties and effects. Some of the well-known AAS include:
- Halotestin (Fluoxymesterone): Known for its strong androgenic effects, often used for cutting phases due to its ability to reduce body fat and increase muscle hardness.
- Masteron (Drostanolone): Popular for its ability to increase muscle density and strength, often used in the final stages of a cycle for a leaner physique.
- Winstrol (Stanozolol): Favored for its ability to enhance muscle definition and vascularity, often used in cutting phases.
- Dianabol (Methandrostenolone): One of the most popular AAS, known for its rapid muscle and strength gains, often used in bulking phases.
- Trenbolone: Known for its potent anabolic and androgenic effects, it enhances muscle growth, strength, and fat loss.
- Anavar (Oxandrolone): A mild AAS often used for cutting and re-composition due to its ability to increase strength without significant water retention.
3. Functionality and Mechanism of AAS + Benefits & Risks
AAS primarily function by binding to androgen receptors in the body, triggering various biological responses. The key functionalities include:
Benefits: AAS increase muscle protein synthesis rates, activating genes responsible for muscle growth, leading to increased lean body mass, strength, and endurance over time with proper training and nutrition.
They also stimulate bone density increase by promoting osteoblast activity while inhibiting osteoclasts, beneficial for preventing osteoporosis (=a condition that weakens your bones, making them fragile and more likely to break.)
Additionally, AAS promote erythropoiesis (red blood cell production), enhancing oxygen delivery during exercise; reduce adipose tissue through lipolytic effects on fat cells; and can modulate immune system functions temporarily while enhancing recovery post-injury or surgery.
Risks: Chronic abuse is associated with significant health risks including hypogonadism (suppressed natural testosterone production affecting libido, fertility, mood regulation)
Hair follicle miniaturization accelerating male pattern baldness via DHT conversion
Cardiovascular risks like hypertension
Atherosclerosis (arterial plaque buildup)
Cardiac hypertrophy potentially leading to arrhythmias or heart failure
Increased myocardial infarction risk
Other side effects include mood swings (roid rage), acne vulgaris due to sebaceous gland stimulation, liver damage from oral steroids causing elevated ALT/AST levels indicating hepatotoxicity and potentially irreversible changes like gynecomastia (male breast tissue growth) if not addressed early with anti-estrogens during cycles.
These can all be mitigated if used with correct ai‘s/ancillaries alongside and controlled doses.
4. Administration Protocols & Pharmacokinetics
Once you've established which drug is correct, only half of everything is understood, the steroids pharmacokinetics determine how and when it is administered.
By far the most important factor in this is the ester connected to the steroid molecule.
The Function of Esters: Natural testosterone has a very short half life (minutes). To allow it to be injected, the molecule has a fatty acid chain attached (estsers), which slows the delivery of the hormone from the site of injection into the blood.
Short Esters (eg Propionate): have a rapid release. Need to be administered more frequently (often daily or every other day) to keep blood levels steady.
Long Esters (e.g., Enanthate, Cypionate, Decanoate): Elapsed Time: will produce a slow release of the medication. They allow injections to be administered less frequently (once or twice per week).
Ultra-Long Esters ( Sustanon blends, Testosterone Undecanoate):Several weeks to months! Used in a maintenance cycle. Also used by advanced users who dislike frequent pinning.
Oral vs. Injectable
Injectables: Usually get around the "first pass metabolism" of the liver, thus they are less hepatotoxic. They are the ideal choice for bulk and strength gains, because of better bioavailability.
Orals: To reach the systemic circulation they must pass through the liver. The majority are 17-alpha-alkylated, a chemical modification that prevents breakdown but causes significant strain on the liver. As such it is common for orals to be used in short cycles (4-8 weeks) rather than long ones.
Common Cycle Archetypes
- Bulking: Focused on mass and strength. Usually based on a longer-ester Testosterone (Enanthate/Cypionate) along with either a short-acting mass-build Er (Dianabol) or a more heavy substance (Deca Durabolin).
- Cutting - Focused on maintaining as much muscle as possible while burning the excess fat. Usually a non-aromatizing compound such as Trenbolone, Masteron or Winstrol used with perhaps a low dose testosterone base.
- Beginner Stack: in general one compound (eg Testosterone Enanthate) should be used at the beginning to determine the tolerance for this substance, and then introduce additional drugs later.
5. Ancillary Drugs (The Support System)
AAS do not exist in a vacuum. They interfere with the hormonal balance of the body. Ancillaries are drugs taken during or after a cycle to deal with any potential side effects and help bring the hormonal balance back to normal.
Aromatase Inhibitors (AIs)
- Acquired: Arimidex (Anastrozole), Aromasin (Exemestane), Aromasin.
- Function: Injecting Testosterone leads to conversion of excess testosterone to Estradiol (Estrogen) because of an enzyme called aromatase. Excess Estrogen causes gynocomastia (puffy nipples), water retention and mood swings. AIs prevent this conversion.
Note: Over-suppression of estrogen with AIs is just as bad as having too much (joint pain, poor lipid profiles, low libido).
SERMs (Selective Estrogen Receptor Modulators)
- Specify drugs Clomid (Clomiphene), Nolvadex (Tamoxifen) etc.
- Function: They act as estrogen in some tissues and as anti-estrogen in others. PCT drugs (see Section 8) work in this way.
- They cause the hypothalamus to think that the body's estrogenic levels are below normal, and make the pituitary gland restart production of leutinising hormone (LH) and follicle stimulating hormone (FSH).
These cause the testes to restart production of testosterone.
Dopamine Agonists
- Examples: Cabergoline, Pramipexole.
- Function: some steroids, eg Trenbolone and Dianabol may increase Prolactin, which can lead to sex problems and more gyno.. Cabergoline can reduce prolactin in a matter of hours.
6. Blood Work & Monitoring
If you don't measure it, you can't manage it. A great number of side effects are silent until they damage you. Regular blood tests are important for optimizing health.
Key Markers to Monitor:
1. Lipids Profile (Cholesterol): AAS is notorious for crushing HDL (good) and elevating LDL (bad).
(This promotes atherosclerosis at an alarming rate. Regularly checking is crucial for all but especially so for oral users and Trenbolone Users.)
2. Liver Enzymes (ALT/AST): Any rises are indicative of liver stress, the main marker for oral steroids. If ALT/AST stay high for long periods of time then it means the liver is hepatotoxic.
3. Hematocrit & Hemoglobin: AAS stimulate red blood cell production (erythropoiesis). This improves oxygen transport to tissues but can cause thickening of blood (polycythemia) predisposing to clots and stroke. If hematocrit exceeds 50-54% phlebotomy may be necessary.
4. Kidney Function (Creatinine/BUN): Due to heavy lifting, high protein consumption and the use of AAS there is considerable strain placed on the kidneys.
5. Estradiol (Sensitive Assay): Total Testosterone levels indicate how much of the drug is in your system; Estradiol is an indicator of how much is converting. You will need a 'sensitive' test so that you can measure low-but-damaging levels of estrogen.
6. Hormonal Panel(LH/FSH): Low Lh/FSH tells us that your body is shutting down production of testosterone.
Timing:
- Baseline: If used during your cycle then you have to inject them depending on the amount of steroids you inject.
- Mid-Cycle: If you don‘t increase your intake of steroids, then you should stick to ur normal ancills amount.
- End-Cycle: Use your ancills even after your cycle ended for a little while just to really kickstart your natural hormones, and then hop off.
7. Post-Cycle Therapy (PCT)
When you add exogenous hormones, your body senses it and inhibits it's own production (negative feedback).
When you come off the drugs, your body's own production is shut down. PCT is the rail-road tracks that get your body's own testosterone production up and running before you hit your crash into hypogonadism.
When to Begin PCT PCT cannot begin until the exo steroid clears your system. With the use of Testosterone Enanthate (half-life about 5 days), you normally have to wait about 10-14 days after your last injection before you begin Clomid or Nolvadex.
Going in too early will mean the receptors are still being blocked by the exo testosterone and the PCT drugs are ineffective.
Common PCT Protocol
When you add exogenous hormones, your body senses it and inhibits it's own production (negative feedback).
When you come off the drugs, your body's own production is shut down. PCT is the rail-road tracks that get your body's own testosterone production up and running before you hit your crash into hypogonadism.
When to Begin PCT PCT cannot begin until the exo steroid clears your system. With the use of Testosterone Enanthate (half-life about 5 days), you normally have to wait about 10-14 days after your last injection before you begin Clomid or Nolvadex.
Going in too early will mean the receptors are still being blocked by the exo testosterone and the PCT drugs are ineffective.
Common PCT Protocol
- How long: Usually 4-6 weeks.
- Drugs: Clomid & Nolvadex are used together.
- Week 1-2: Higher doses of both.
- Weekly 3-4: Reduced doses.
- Week 5-6: Gradually reducing training volume and intensity.
Goal is to get the LH and FSH back to normal and have the testes start manufacturing their own testosterone.
No PCT or doing PCT badly often causes a "crash". This means that you can lose all your new muscle mass, have no libido at all and be hypogonadal for several months.
THANK y’all so much for reading this guide if y‘all got any questions LMK and also make me aware of any mistakes if there are.
Peace out
Peace out
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