GLP1 AGONISTS MOG DNP, GTFIH

[134applesauce456]

JFL if you choose DNP over GLP1 agonists tbh

inject tirz/sema and watch ur hunger disappear with literally no side effects . You will have no urge to eat and you will feel amazing. It also has other positive benefits too with no bad sides. These agonists improves insulin sensitivity, increases energy expenditure, enhances lipid metabolism, positively alters gut hormone levels, reduces fat accumulation, possesses anti-inflammatory properties, and improves glycemic control. Literally amazing and again, no bad sides

DNP on the other hand is basically hell simulator. Not even mentioning all the things that can go wrong like cataracts , peripheral neuropathy , literal death but simply the way DNP makes you feel (especially on moderate - higher doses) is hell. Some people have told me DNP is the worse pain they've felt in their life. You are going to sweat profusely and feel sick w headaches , not having the energy to do anything.

DNP is outdated, there is no reason to take it now GLP1 agonists exist. If you can buy a 2024 camry why buy a 1999 camry

A lot of users here like to promote DNP but trust me don't get it, look into a GLP1 agonists instead and thank me later.

pretty water thread tbh

 

[not__cel]

Fuck I’ve gotten probably 12 piercings but I don’t want to be near injectables

 

[134applesauce456]

Fuck I’ve gotten probably 12 piercings but I don’t want to be near injectables

low dose dnp + glp1 for appetite suppressant mogs

 

[craven]

Dnr + cope NIGGER
my dnp just arrived

 

[134applesauce456]

Dnr + cope NIGGER
my dnp just arrived

i have some too but glp1 MOGGS

 

[moodless]

GLP 1 agonists have way more sides lol its just new. wait 10 years and youll see its the devils drug

 

[134applesauce456]

GLP 1 agonists have way more sides lol its just new. wait 10 years and youll see its the devils drug

can i see some studies/proof suggesting that?

 

[moodless]

can i see some studies/proof suggesting that?

its not yet released but it shrinks the heart, destroys how ur stomach processes food for the rest of your life and most people once they get off it have utterly destroyed hormonal systems. Its only been in the market for 3 years, dnp was in the market for 30 years. Wait 7 years and glp 1 agonist will be withdrawn from market, although i think it may be safe if used for a month and then cold turkeyd.

 

[Alexanderr]

GLP 1 agonists have way more sides lol its just new. wait 10 years and youll see its the devils drug

They're not new. Some are, since the previous ones have been such a success there are a lot of new ones in the pipeline, but GLP-1s have been researched for decades, in fact. They just blew up recently.

 

[moodless]

They're not new. Some are, since the previous ones have been such a success there are a lot of new ones in the pipeline, but GLP-1s have been researched for decades, in fact. They just blew up recently.

wide scale human studies and in vitro studies are quite different. amphetamines were known for a long time before they were approved for treating obesity

 

[Alexanderr]

wide scale human studies and in vitro studies are quite different

Yes, and they've had wide-scale human studies? Are you sure you're not confusing them for something else? They'd never get released with just in-vitro studies to back them up.

 

[moodless]

Yes, and they've had wide-scale human studies? Are you sure you're not confusing them for something else? They'd never get released with just in-vitro studies to back them up.

dnp was out for around 10 years before it was withdrawn, it was known beforethat

 

[134applesauce456]

its not yet released but it shrinks the heart, destroys how ur stomach processes food for the rest of your life and most people once they get off it have utterly destroyed hormonal systems. Its only been in the market for 3 years, dnp was in the market for 30 years. Wait 7 years and glp 1 agonist will be withdrawn from market, although i think it may be safe if used for a month and then cold turkeyd.

interesting.. ur kinda making sense ngl but how did you know this ? so you dont have any research studies backing your stance up?

 

[Alexanderr]

View attachment 3374689

dnp was out for around 10 years before it was withdrawn, it was known beforethat

I don't think you properly understand the history behind GLP-1s and the process that underlies approval for widespread medical use.
You can't just claim there are nasty unknown side-effects that will only be seen '10 years from now' when there's no evidence showcasing that to be so, and there's been lots of human studies spanning decades already.
Tirzepatide is one of the more recent GLP-1s, but several earlier generations of GLP-1s have been approved for at least a decade. GLP-1s are not a new invention.

 

[moodless]

I don't think you properly understand the history behind GLP-1s and the process that underlies approval for widespread medical use.
You can't just claim there are nasty unknown side-effects that will only be seen '10 years from now' when there's no evidence showcasing that to be so, and there's been lots of human studies spanning decades already.
Tirzepatide is one of the more recent GLP-1s, but several earlier generations of GLP-1s have been approved for at least a decade. GLP-1s are not a new invention.

GLP 1 agonists have been used in type 2 diabetics before it was started being prescribed off label for obesity. There are no large scale human trials or meta analyses spanning a decade or even tracking individual patients and their markers. There have been countless anecdotes of these drugs causing damage to bones, and it is well known by know it shrinks the heart and causes massive skeletal muscle loss. GLP1 agonist is present in even yerba matte, it does not mean that the doses used in something like wynlevi is physiologic.

DNP at low doses is anti-cancer and an uncoupler , in higher doses it leads to organ failure and being cooked from inside. The dose makes the poison and unless you;re drinking yerba matte comparing it to semaglutide has little relevance

 

[moodless]

interesting.. ur kinda making sense ngl but how did you know this ? so you dont have any research studies backing your stance up?

we have no large scale human trials to go on, all we have is anecdotes and individual users. thats why i would rather do low dose anavar since we have a wealth of data than something like sema

Weight Loss Drugs Like Ozempic Could Reduce Bone Density, Study Suggests

Other research suggests the increasingly popular weight loss drugs fail to preserve muscle.

www.forbes.com

https://www.sciencedirect.com/science/article/pii/S2452302X24002869

 

[Alexanderr]

GLP 1 agonists have been used in type 2 diabetics before it was started being prescribed off label for obesity. There are no large scale human trials or meta analyses spanning a decade or even tracking individual patients and their markers. There have been countless anecdotes of these drugs causing damage to bones, and it is well known by know it shrinks the heart and causes massive skeletal muscle loss. GLP1 agonist is present in even yerba matte, it does not mean that the doses used in something like wynlevi is physiologic.

DNP at low doses is anti-cancer and an uncoupler , in higher doses it leads to organ failure and being cooked from inside. The dose makes the poison and unless you;re drinking yerba matte comparing it to semaglutide has little relevance

GLP-1 agonists have been studied for over a decade, with long-term human trials and meta-analyses backing their safety and efficacy, especially in type 2 diabetes.

Claims like "shrinking the heart" or "massive muscle loss" are baseless unless you can provide peer-reviewed evidence. Anecdotes mean nothing here.

The yerba mate argument is a joke; dosage and pharmacodynamics matter. If you're making positive claims about "unknown side effects," the burden of proof is on you.

Show the peer-reviewed studies or stop spreading speculative nonsense.

 

[moodless]

GLP-1 agonists have been studied for over a decade, with long-term human trials and meta-analyses backing their safety and efficacy, especially in type 2 diabetes.

Claims like "shrinking the heart" or "massive muscle loss" are baseless unless you can provide peer-reviewed evidence. Anecdotes mean nothing here.

you're talking past me.

The yerba mate argument is a joke; dosage and pharmacodynamics matter.

literally what I said jfl. And I already linked two papers. Its clear you have a stake in this that I dont, so do what you wish.

 

[Alexanderr]

you're talking past me.

literally what I said jfl. And I already linked two papers. Its clear you have a stake in this that I dont, so do what you wish.

"Having a stake" doesn’t invalidate factual evidence. If you linked papers, cite the specific claims you’re referencing instead of vague assertions.
Seeing as the studies you cited don’t support your claims.

1. The JAMA study shows reduced bone density without exercise but notes this is mitigated by exercise. It doesn’t conclude that GLP-1s are inherently harmful.
2. The mouse study shows reduced cardiac and lean mass but explicitly states no functional impairments were observed and human relevance is speculative.

Neither proves severe harm or withdrawal risk. If you claim otherwise, provide human studies with direct, peer-reviewed evidence; not speculative interpretations.

Without direct evidence from those studies supporting your claims, they remain baseless.

 

[moodless]

The mouse study shows reduced cardiac and lean mass but explicitly states no functional impairments were observed and human relevance is speculative

you dont understand how drugs are released into the market, how they are tracked statistically and how mouse studies usually are of immense relevance in most cases to humans. Like I said, its obvious you have a stake in this I dont, so keep believing whatever it is you want to. By the same logic, SSRIs which have been around for 50 years are just now being recognised for how damaging they actually are.

 

[Alexanderr]

you dont understand how drugs are released into the market, how they are tracked statistically and how mouse studies usually are of immense relevance in most cases to humans. Like I said, its obvious you have a stake in this I dont, so keep believing whatever it is you want to.

Bro, you’re deflecting. The studies you linked don’t support your claims, and instead of addressing that, you’re pivoting to vague generalities about drug tracking and mouse studies. Stick to the point: your evidence doesn’t prove the severe harm you’re claiming. If you have stronger evidence, present it. Otherwise, this is just baseless speculation.

 

[moodless]

Bro, you’re deflecting. The studies you linked don’t support your claims, and instead of addressing that, you’re pivoting to vague generalities about drug tracking and mouse studies. Stick to the point: your evidence doesn’t prove the severe harm you’re claiming. If you have stronger evidence, present it. Otherwise, this is just baseless speculation.

Read my comment on SSRIs on the top. The study I linked on bone density is on humans. I never claimed it causes severe harm, I even said it's probably safe if used for a month or so and then quit cold turkey. Usually most drugs have a higher efficacy/safety ratio or a goldilocks zone where you can get the maximum effectiveness with good damage control. True for Anavar, DNP , clen etc.

 

[Alexanderr]

Read my comment on SSRIs on the top. The study I linked on bone density is on humans. I never claimed it causes severe harm, I even said it's probably safe if used for a month or so and then quit cold turkey. Usually most drugs have a higher efficacy/safety ratio or a goldilocks zone where you can get the maximum effectiveness with good damage control. True for Anavar, DNP , clen etc.

@moodless, let’s stick to what you’ve claimed:

1. "Shrinks the heart and causes massive muscle loss" – The mouse study shows reduced cardiac mass but explicitly states no functional impairments, and it admits human relevance is speculative. The bone density study highlights risks mitigated by exercise and doesn’t conclude severe harm.
2. "Wait 7 years and GLP-1s will be withdrawn" – This is unsupported speculation. You’ve provided no evidence of market withdrawal or severe harm.
3. "I never claimed severe harm" – Yet you’ve said GLP-1s "shrink the heart," "destroy hormonal systems," and "ruin stomach processing," what does that imply if not significant harm?
4. "Goldilocks zone like Anavar, DNP, clen" – GLP-1s have decades of human data and vastly safer profiles compared to drugs like DNP or clen, which are notorious for toxicity. Apples and oranges.

My point is, and I'm not trying to be disrespectful here, your evidence doesn’t back your alarmist claims. If you’re just suggesting caution, fine, but stop overstating risks without solid data. Speculation ≠ evidence.

 

[moodless]

@moodless, let’s stick to what you’ve claimed:

1. "Shrinks the heart and causes massive muscle loss" – The mouse study shows reduced cardiac mass but explicitly states no functional impairments, and it admits human relevance is speculative. The bone density study highlights risks mitigated by exercise and doesn’t conclude severe harm.
2. "Wait 7 years and GLP-1s will be withdrawn" – This is unsupported speculation. You’ve provided no evidence of market withdrawal or severe harm.
3. "I never claimed severe harm" – Yet you’ve said GLP-1s "shrink the heart," "destroy hormonal systems," and "ruin stomach processing," what does that imply if not significant harm?
4. "Goldilocks zone like Anavar, DNP, clen" – GLP-1s have decades of human data and vastly safer profiles compared to drugs like DNP or clen, which are notorious for toxicity. Apples and oranges.

My point is, and I'm not trying to be disrespectful here, your evidence doesn’t back your alarmist claims. If you’re just suggesting caution, fine, but stop overstating risks without solid data. Speculation ≠ evidence.

glp1 agonists have never been as mainstream as in the last 6 years, and two the drug glp 1 agonists are modelled upon was only approved by the FDA in 2005. By contrast SSRIs have been around for 5 decades and only in the last years have we come to grips with how damaging they could be.

Functional impairments aren't caused even when some parts of the brain completely atrophy. Function is not equivalent to structure and vice versa.

Yes in a long enough time scale they COULD do those things, just like SSRIs could cause irrevocable damage in a long ENOUGH time scale.

" Exenatide's 2005 approval by the U.S. Food and Drug Administration was a landmark event which proved that targeting the GLP-1 receptor was a viable strategy and inspired other pharmaceutical companies to focus their research and development on that receptor."

 

[not__cel]

Dnr + cope NIGGER
my dnp just arrived

fuck you mine was supposed to arrive 9 days ago but still hasn't jfl

 

[benchcrab]

just do both you broke bitch

 

[Foreverbrad]

DNP is lovely, like a little fire inside to keep me warm through the cold winter. Might start using it every year!