HGH and Aromasin

grumpygrape

grumpygrape

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I plan to do 6 ius of hgh for 8 months by itself and was wondering if aromasin is necessary to defy early growth plate closure and potentially surpass genetic potential cus i always see mixed answers, i am in between rn due to the side effects and need some help deciding. 16 btw my plates r open and my main goal is height and muscle with some hope of bonemass (i will get blood work). yes ik im a lil retarded but i want to learn.
 
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I plan to do 6 jus of hgh for 8 months and was wondering if aromasin is necessary to defy early growth plate closure and potentially surpass genetic potential cus i always see mixed answers, i am in between rn due to the side effects and need some help deciding. 16 btw my plates r open and my main goal is height and muscle with some hope of bonemass. yes ik im a lil retarded but i want to learn.
do more than 6, the optimal dosage range is 8-12ius ed so id reccomend u do 8. anything under its genuinely dumb.
furthermore aromatase inhibitors to keep plates open is genuinely cope, basically in ur body in general yes they can prevent secretion/absorption of aromatase, but locally in your growth plates it can not do this effectively and youd therefore need huge doses of ai to be able to slow down the closure of growth plates. just do the hgh, ai is only useful alongside androgens
 
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do more than 6, the optimal dosage range is 8-12ius ed so id reccomend u do 8. anything under its genuinely dumb.
furthermore aromatase inhibitors to keep plates open is genuinely cope, basically in ur body in general yes they can prevent secretion/absorption of aromatase, but locally in your growth plates it can not do this effectively and youd therefore need huge doses of ai to be able to slow down the closure of growth plates. just do the hgh, ai is only useful alongside androgens
alr thank you bro i appreciate it
 
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do more than 6, the optimal dosage range is 8-12ius ed so id reccomend u do 8. anything under its genuinely dumb.
furthermore aromatase inhibitors to keep plates open is genuinely cope, basically in ur body in general yes they can prevent secretion/absorption of aromatase, but locally in your growth plates it can not do this effectively and youd therefore need huge doses of ai to be able to slow down the closure of growth plates. just do the hgh, ai is only useful alongside androgens
This whole thing is retarded.

Firstly there is no “optimal” dose of hgh, and if there was, it would be more than fucking 8iu. You should be on however much you can afford, ideally 12iu+.

Secondly aromatase inhibitors aren’t cope. You know nothing about them. The Aromatase enzyme is present only in fat tissue, not in growth plates. AIs inhibit the aromatase enzyme, stopping the conversion of testosterone into estradiol (e2). E2 is primarily responsible for resting zone chondrocyte senescence, or simply growth plate fusion.

You are right, ais are commonly only required when on exogenous testosterone aswell, but they are still useful when not on test in a lower dose, to make sure e2 is low enough to extend the lifetime of your growth plates.

Don’t confidently give advice to people about things you know nothing about. It can be very harmful especially on topics like pharmacology
 
I plan to do 6 ius of hgh for 8 months by itself and was wondering if aromasin is necessary to defy early growth plate closure and potentially surpass genetic potential cus i always see mixed answers, i am in between rn due to the side effects and need some help deciding. 16 btw my plates r open and my main goal is height and muscle with some hope of bonemass (i will get blood work). yes ik im a lil retarded but i want to learn.
There are no side effects to Aromasin if you don’t crash your E2
 
This whole thing is retarded.

Firstly there is no “optimal” dose of hgh, and if there was, it would be more than fucking 8iu. You should be on however much you can afford, ideally 12iu+.

Secondly aromatase inhibitors aren’t cope. You know nothing about them. The Aromatase enzyme is present only in fat tissue, not in growth plates. AIs inhibit the aromatase enzyme, stopping the conversion of testosterone into estradiol (e2). E2 is primarily responsible for resting zone chondrocyte senescence, or simply growth plate fusion.

You are right, ais are commonly only required when on exogenous testosterone aswell, but they are still useful when not on test in a lower dose, to make sure e2 is low enough to extend the lifetime of your growth plates.

Don’t confidently give advice to people about things you know nothing about. It can be very harmful especially on topics like pharmacology
Bro u say not to be confidently wrong but that’s what ur doing, saying aromatase enzyme is only present in fat and not growth plates

When it literally exits in growth plate bone tissue so idk dnr the rest
 
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Bro u say not to be confidently wrong but that’s what ur doing, saying aromatase enzyme is only present in fat and not growth plates

When it literally exits in growth plate bone tissue so idk dnr the rest
I stand corrected. But even so, most of it is present in fat tissue, and also, Letrozole is very effective at binding to pretty much every aromatase enzyme in the body, way better than Aromasin or arimidex, which decreases intracellular e2 very intensely, which is what we need to avoid growth plate closure.

And if you read the rest you will see you are still retarded, even though I admit I was wrong about the fat tissue statement.
 

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