How do mfs do all this research for heightmaxxing

[zackkoma]

those so much shit that goes into this heightmax shit it’s so annoying. I wish I can just take Hgh and be done with it bro

 

[ezranigger]

those so much shit that goes into this heightmax shit it’s so annoying. I wish I can just take Hgh and be done with it bro

just take erda and ur good

 

[Deleted member 341805]

those so much shit that goes into this heightmax shit it’s so annoying. I wish I can just take Hgh and be done with it bro

Well I can boil it down to you, high dose rhGH, nothing less than 10iu or you will stunt yourself. Take 2.5mg letrozole every day and you're already off to a good start and honestly most of the work you need.

Erdafitinib is a really good addition for releasing the brake on chondrocyte proliferation but aren't as necessary as the other 2 as they are staples. It makes you grow really fast but can have some seriously undesirable side effects if you don't know how to mitigate them.

Non aromatising anabolic androgenic steroids can really help with hypersensitising your IGF-1 receptors to IGF-1 which makes the work of GH much more potent. It also buffers igf-1 levels as a whole anyways.

Keeping testosterone on the lower side of normal is good too, to reduce intracrine aromatisation. Some people go as far as to nuking it but that's too much for me imo

Doing the things I said here will likely grant you a good 1-3 inches onto your fah.

There's literally no research that needs to be done beyond this. It's simply just modifying your hormones, But Erda is a different case I guess

 

[Zagro]

Dumbass the basics are still golden there's no niche gatekept compound that will make you gain 6 inches overnight you need to do hyper polypharmacy and use multiple compounds correctly

And also give up on learning it will just waste your time everything we need is already public even random fucking 13 year old fuckers are making slideshows on tiktok nowadays

 

[jj1yden]

Well I can boil it down to you, high dose rhGH, nothing less than 10iu or you will stunt yourself. Take 2.5mg letrozole every day and you're already off to a good start and honestly most of the work you need.

Erdafitinib is a really good addition for releasing the brake on chondrocyte proliferation but aren't as necessary as the other 2 as they are staples. It makes you grow really fast but can have some seriously undesirable side effects if you don't know how to mitigate them.

Non aromatising anabolic androgenic steroids can really help with hypersensitising your IGF-1 receptors to IGF-1 which makes the work of GH much more potent. It also buffers igf-1 levels as a whole anyways.

Keeping testosterone on the lower side of normal is good too, to reduce intracrine aromatisation. Some people go as far as to nuking it but that's too much for me imo

Doing the things I said here will likely grant you a good 1-3 inches onto your fah.

There's literally no research that needs to be done beyond this. It's simply just modifying your hormones, But Erda is a different case I guess

You do understand that in clinical studies the HV for GH + Letroze was lower than that of Anastrozole + GH? If you aren't using non aromatizing androgens genuinely letroze will rape you. Local E2 cannot be inhibited when you are rocketing your test 3 fold with 2.5 Letroze. Also if you run a stack like this without achillaries you will have a fucking cardiovascular event.

Erda will fucking rape you btw.

https://academic.oup.com/jcem/article/110/9/e2871/8125692

@Zagro wdyt?

 

[Zagro]

You do understand that in clinical studies the HV for GH + Letroze was lower than that of Anastrozole + GH? If you aren't using non aromatizing androgens genuinely letroze will rape you. Local E2 cannot be inhibited when you are rocketing your test 3 fold with 2.5 Letroze. Also if you run a stack like this without achillaries you will have a fucking cardiovascular event.

Erda will fucking rape you btw.

https://academic.oup.com/jcem/article/110/9/e2871/8125692

@Zagro wdyt?

All true you’ve got it figured out

Only one thing that isn’t certain is the reason for the letrozole cohorts FAH being lower than anastrozole cohort purely because of more endogenous androgens, but that just seems to be the most suitable hypothesis. It can also be caused by the more potent aromatase inhibition from letrozole fully nuking your estrogen levels, thus also its positive effects on pituitary growth hormone secretion.

Also about him telling people not to nuke estrogen, if you don’t nuke aromatase you’re risking hastened fusion greatly, as even trace levels left behind will be amplified significantly just because of the increased androgens, growth factors and the compensatory mechanisms the body has

 

[Deleted member 341805]

You do understand that in clinical studies the HV for GH + Letroze was lower than that of Anastrozole + GH? If you aren't using non aromatizing androgens genuinely letroze will rape you. Local E2 cannot be inhibited when you are rocketing your test 3 fold with 2.5 Letroze. Also if you run a stack like this without achillaries you will have a fucking cardiovascular event.

Erda will fucking rape you btw.

https://academic.oup.com/jcem/article/110/9/e2871/8125692

@Zagro wdyt?

I literally said to use NAAAS for one, secondly I also said to keep test low for the mitigation of the letrozole test increase. We already know AI's will trigger negative feedback loops that increase test. That's why you use an NAAAS to shut off the gonads. Can also use a really really low dosage of test like I'm talking no more than 30mg weekly.

No shit you will need ancillaries

 

[Deleted member 341805]

Also about him telling people not to nuke estrogen, if you don’t nuke aromatase you’re risking hastened fusion greatly, as even trace levels left behind will be amplified significantly just because of the increased androgens, growth factors and the compensatory mechanisms the body has

By "him" are you referring to me? I said to nuke e2 with 2.5 letro daily. I already know about compensatory mechanisms for trace amounts of e2 and aromatase. The androgens would be nuked from running an NAAAS for shutting off the gonads.

What's the issue with this?

 

[Zagro]

By "him" are you referring to me? I said to nuke e2 with 2.5 letro daily. I already know about compensatory mechanisms for trace amounts of e2 and aromatase. The androgens would be nuked from running an NAAAS for shutting off the gonads.

What's the issue with this?

“Keeping testosterone on the lower side of normal is good too, to reduce intracrine aromatisation. Some people go as far as to nuking it but that's too much for me imo”

How would you plan to shut them off on exogenous test

 

[zanenenene]

“Keeping testosterone on the lower side of normal is good too, to reduce intracrine aromatisation. Some people go as far as to nuking it but that's too much for me imo”

How would you plan to shut them off on exogenous test

So anastrozole is the ideal because of its estrogen suppression while preserving enough estrogen activities to maintain other stuff based on this "debate"

 

[DoNotResusciate]

those so much shit that goes into this heightmax shit it’s so annoying. I wish I can just take Hgh and be done with it bro

HGH wont do shit brah, stick to the good old 677

 

[Azie555]

“Keeping testosterone on the lower side of normal is good too, to reduce intracrine aromatisation. Some people go as far as to nuking it but that's too much for me imo”

How would you plan to shut them off on exogenous test

Please chrck dms

 

[Zagro]

So anastrozole is the ideal because of its estrogen suppression while preserving enough estrogen activities to maintain other stuff based on this "debate"

If you read everything with your ass yeah

 

[Deleted member 341805]

“Keeping testosterone on the lower side of normal is good too, to reduce intracrine aromatisation. Some people go as far as to nuking it but that's too much for me imo”

How would you plan to shut them off on exogenous test

If you were to run NAAAS the endocrine system would already shut off, I wasn't talking about shutting off test in your body, was talking about the endocrine

If you were to use a low dose of exogenous test like really low dosage it can't be that bad right? No shot a dht derivative is the only way you can gain pubertal development without stunting yourself with 0 test on this stack. Still has to produce some results

 

[Deleted member 341805]

So anastrozole is the ideal because of its estrogen suppression while preserving enough estrogen activities to maintain other stuff based on this "debate"

How on earth did you get to this conclusion from what we're talking about

 

[zackkoma]

Dumbass the basics are still golden there's no niche gatekept compound that will make you gain 6 inches overnight you need to do hyper polypharmacy and use multiple compounds correctly

And also give up on learning it will just waste your time everything we need is already public even random fucking 13 year old fuckers are making slideshows on tiktok nowadays

i know everything is public information I’m just saying there’s so much information that contradicts each other it’s exhausting to do hella research figuring out what goods and what’s not

 

[bluebandz]

those so much shit that goes into this heightmax shit it’s so annoying. I wish I can just take Hgh and be done with it bro

You can just take hgh and ai what

 

[zackkoma]

Dumbass the basics are still golden there's no niche gatekept compound that will make you gain 6 inches overnight you need to do hyper polypharmacy and use multiple compounds correctly

And also give up on learning it will just waste your time everything we need is already public even random fucking 13 year old fuckers are making slideshows on tiktok nowadays

What’s compounds are most effective

 

[zackkoma]

You can just take hgh and ai what

Nah

 

[bluebandz]

Nah

You can
im not recommending it tho or saying its the most affective method but depending on bone age and stuff it def works

 

[sq_blr]

Nah

Tf ym nah

 

[zanenenene]

How on earth did you get to this conclusion from what we're talking about

The dude said that letrozole nuking e2 could have negative effects on the growth hormone secretion, the other guy who linked the study, showed that the gh + anastrozole had better results then gh + Letrozole which could be due to the two possibilities that Zagro mentioned

just read again don't really know if I missed anything but if I'm wrong please enlighten me and what would the ideal be then?

 

[jj1yden]

All true you’ve got it figured out

Only one thing that isn’t certain is the reason for the letrozole cohorts FAH being lower than anastrozole cohort purely because of more endogenous androgens, but that just seems to be the most suitable hypothesis. It can also be caused by the more potent aromatase inhibition from letrozole fully nuking your estrogen levels, thus also its positive effects on pituitary growth hormone secretion.

Also about him telling people not to nuke estrogen, if you don’t nuke aromatase you’re risking hastened fusion greatly, as even trace levels left behind will be amplified significantly just because of the increased androgens, growth factors and the compensatory mechanisms the body has

You recommend halo as the androgen of choice right? How do you deal with low libido/no erecitons on 0 test and 0 e2 or do you just dnr it?

Do you think that erda is the only good FGFR3 inhibitor? infigratinib at low doses has almost 0 sides

 

[Deleted member 341805]

You recommend halo as the androgen of choice right? How do you deal with low libido/no erecitons on 0 test and 0 e2 or do you just dnr it?

Do you think that erda is the only good FGFR3 inhibitor? infigratinib at low doses has almost 0 sides

He dnrs the libido issues, not like you're using your dick anyways

I also think he believes in tren

 

[Deleted member 341805]

The dude said that letrozole nuking e2 could have negative effects on the growth hormone secretion, the other guy who linked the study, showed that the gh + anastrozole had better results then gh + Letrozole which could be due to the two possibilities that Zagro mentioned

just read again don't really know if I missed anything but if I'm wrong please enlighten me and what would the ideal be then?

Yeah but you don't get that from this convo, also heightmaxxing stacks always have rhGH. The igf-1 decrease isn't enough to make a difference if your'e running supraphysiological growth hormone.

Me and zagro were simply talking about nuking test or keeping it super low. And dealing with local estrogen increase through local test aromatisation.

 

[lmnopq]

those so much shit that goes into this heightmax shit it’s so annoying. I wish I can just take Hgh and be done with it bro

Read studies and use ai u lazy boy

 

[lmnopq]

You recommend halo as the androgen of choice right? How do you deal with low libido/no erecitons on 0 test and 0 e2 or do you just dnr it?

Do you think that erda is the only good FGFR3 inhibitor? infigratinib at low doses has almost 0 sides

U can still get hard and fuck and everything, just remember a kid and teenagers physiology is completely different than an adults

 

[zackkoma]

Read studies and use ai u lazy boy

Help me

 

[chudcell999]

the truth is there is no way to get taller without surgery after your growth plates close Don't you think they research this? For more than just lookism think of all the pro athletes that could have been if they were just 5 inches taller. Without surgery there's nothing you can do.

 

[bpkai]

just take erda and ur good

How to manage sides

just take erda and ur good

 

[ezranigger]

How to manage sides

rawdog it

 

[bpkai]

rawdog it

No like seriously. Ct scans just let you see how much fluid there is and saffron is a bit cope for it. Even if you do 2-3mg 3 weeks on 1 week off there’s still chance of it happening

 

[fraudster#1]

Yeah but you don't get that from this convo, also heightmaxxing stacks always have rhGH. The igf-1 decrease isn't enough to make a difference if your'e running supraphysiological growth hormone.

Me and zagro were simply talking about nuking test or keeping it super low. And dealing with local estrogen increase through local test aromatisation.

don't letrozole across this and block aromatization on the Gp aswell?

 

[zackkoma]

the truth is there is no way to get taller without surgery after your growth plates close Don't you think they research this? For more than just lookism think of all the pro athletes that could have been if they were just 5 inches taller. Without surgery there's nothing you can do.

Who said my growth plates were closed?

 

[zanenenene]

Yeah but you don't get that from this convo, also heightmaxxing stacks always have rhGH. The igf-1 decrease isn't enough to make a difference if your'e running supraphysiological growth hormone.

Me and zagro were simply talking about nuking test or keeping it super low. And dealing with local estrogen increase through local test aromatisation.

Ah alrigth that's my bad

I don't see the need in having a little test base, as it would still be a risk as u said, local test aromatisation and tell me if I'm wrong but couldn't other compounds somewhat replace test such as hcg ( which provides the minimal estrogen needed based on zagros two possibilities ), for the libido wouldn't dht derives do a way better job then test anyway?

 

[mt2]

just sleep boyo

 

[lmnopq]

Help me

Non aromatizable androgens+tamoxifen+fgfr3 inhibitor

 

[Deleted member 341805]

Ah alrigth that's my bad

I don't see the need in having a little test base, as it would still be a risk as u said, local test aromatisation and tell me if I'm wrong but couldn't other compounds somewhat replace test such as hcg ( which provides the minimal estrogen needed based on zagros two possibilities ), for the libido wouldn't dht derives do a way better job then test anyway?

I still think you're misunderstanding what zagro is saying, he is (and we all are) fully against estradiol. He isn't saying you need little estradiol, the possibility he brought up was because of letrozole raping your e2, the test increase is so substantial that it rapes the growth plates more than anastrozole does.

 

[Deleted member 341805]

i know everything is public information I’m just saying there’s so much information that contradicts each other it’s exhausting to do hella research figuring out what goods and what’s not

There's no contradicting info if you aren't double digit iq

 

[Boshitoshi]

All true you’ve got it figured out

Only one thing that isn’t certain is the reason for the letrozole cohorts FAH being lower than anastrozole cohort purely because of more endogenous androgens, but that just seems to be the most suitable hypothesis. It can also be caused by the more potent aromatase inhibition from letrozole fully nuking your estrogen levels, thus also its positive effects on pituitary growth hormone secretion.

Also about him telling people not to nuke estrogen, if you don’t nuke aromatase you’re risking hastened fusion greatly, as even trace levels left behind will be amplified significantly just because of the increased androgens, growth factors and the compensatory mechanisms the body has

smart guy over here

 

[Deleted member 341805]

What’s compounds are most effective

Bro it's so simple, keep estrogen low, keep gh super high.

 

[Deleted member 341805]

Non aromatizable androgens+tamoxifen+fgfr3 inhibitor

Tamoxifen is overrated

 

[Deleted member 341805]

don't letrozole across this and block aromatization on the Gp aswell?

It does but not enough. Trace amounts of estradiol are still locally synthesised. Which is why it doesn't halt fusion but it does dramatically slow it down and thats what it's used for in trials.

 

[lmnopq]

Tamoxifen is overrated

Nope

 

[zanenenene]

Nope

It is, it can increase estrogen, u should replace it with endoxifen

 

[Deleted member 341805]

Nope

It is, it's literally a pkc antagonist

 

[Fuldblod]

those so much shit that goes into this heightmax shit it’s so annoying. I wish I can just take Hgh and be done with it bro

its about drive it's about power we stay hungry we devour put in the work put in the hours and take whats ours

 

[thekey]

Well I can boil it down to you, high dose rhGH, nothing less than 10iu or you will stunt yourself. Take 2.5mg letrozole every day and you're already off to a good start and honestly most of the work you need.

Erdafitinib is a really good addition for releasing the brake on chondrocyte proliferation but aren't as necessary as the other 2 as they are staples. It makes you grow really fast but can have some seriously undesirable side effects if you don't know how to mitigate them.

Non aromatising anabolic androgenic steroids can really help with hypersensitising your IGF-1 receptors to IGF-1 which makes the work of GH much more potent. It also buffers igf-1 levels as a whole anyways.

Keeping testosterone on the lower side of normal is good too, to reduce intracrine aromatisation. Some people go as far as to nuking it but that's too much for me imo

Doing the things I said here will likely grant you a good 1-3 inches onto your fah.

There's literally no research that needs to be done beyond this. It's simply just modifying your hormones, But Erda is a different case I guess

How would it stunt you if you dose less then 10IUs?

 

[Deleted member 341805]

How would it stunt you if you dose less then 10IUs?

formula for kids is 0.08mg/kg daily of gh secreted which usually is 9-12iu. All dosages below this was shown to stunt in iss kids

 

[mirinmirinmirin]

Well I can boil it down to you, high dose rhGH, nothing less than 10iu or you will stunt yourself. Take 2.5mg letrozole every day and you're already off to a good start and honestly most of the work you need.

Erdafitinib is a really good addition for releasing the brake on chondrocyte proliferation but aren't as necessary as the other 2 as they are staples. It makes you grow really fast but can have some seriously undesirable side effects if you don't know how to mitigate them.

Non aromatising anabolic androgenic steroids can really help with hypersensitising your IGF-1 receptors to IGF-1 which makes the work of GH much more potent. It also buffers igf-1 levels as a whole anyways.

Keeping testosterone on the lower side of normal is good too, to reduce intracrine aromatisation. Some people go as far as to nuking it but that's too much for me imo

Doing the things I said here will likely grant you a good 1-3 inches onto your fah.

There's literally no research that needs to be done beyond this. It's simply just modifying your hormones, But Erda is a different case I guess

are you actually on 160mg of isotret daily? if so, can you explain why you take such a high dose, and why like 60 or even 80mgs daily isn't enough