prettyboypill
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fast low effort thread cause iam dumb and lazy, but i bet that u gon love it
I’ve been digging into the biomechanics behind facial changes from impacts, pressure, or “bonesmashing,” and I think a lot of people in these communities misunderstand what’s actually happening.
TL;DR: Most visible changes are fibrosis, not periosteal bone growth.
Here’s why:
After puberty, periosteal modeling slows down drastically.
Even under ideal mechanical and hormonal conditions, the max real bone gain is something like:
And that’s with controlled, low-force, high-frequency mechanical loading — not random impacts.
When you hit your face, you’re not giving the bone a clean mechanical strain signal.
You’re creating:
This creates fibrosis, which is thick, firm, and feels “bony” to the touch — but it’s not bone.
This is the part people underestimate.
Fibrosis can add:
Meanwhile bone growth is:
This is why fighters, boxers, and martial artists often look like their bones grew — in reality it’s thickened, fibrotic soft tissue over the bone.
Real bone would never grow 3–5 mm in a few months.
Fibrosis can.
And it’s permanent-ish.
So if someone sees quick changes from impact, pressure, chewing devices, whatever — it’s almost certainly connective-tissue thickening, not osteoblast activity.
Fibrosis can make the face look:
But it can also become:
It’s not a controlled process like bone modeling; it’s a scar-response.
btw this is just corrected by gpt but its my idea
Why Facial Bone “Growth” From Impact Is Almost Always Fibrosis — Not Real Bone Change
I’ve been digging into the biomechanics behind facial changes from impacts, pressure, or “bonesmashing,” and I think a lot of people in these communities misunderstand what’s actually happening.
TL;DR: Most visible changes are fibrosis, not periosteal bone growth.
Here’s why:
1. Bone modeling in adults is extremely limited
After puberty, periosteal modeling slows down drastically.
Even under ideal mechanical and hormonal conditions, the max real bone gain is something like:
- 0.1–2 mm per YEAR
And that’s with controlled, low-force, high-frequency mechanical loading — not random impacts.
2. Impacts don’t stimulate bone — they stimulate fibroblasts
When you hit your face, you’re not giving the bone a clean mechanical strain signal.
You’re creating:
- micro-hematomas
- low-grade inflammation
- TGF-β release
- fibroblast → myofibroblast conversion
- collagen deposition (scar-like tissue)
This creates fibrosis, which is thick, firm, and feels “bony” to the touch — but it’s not bone.
3. Fibrosis changes your look WAY more than bone ever could
This is the part people underestimate.
Fibrosis can add:
- 3–10 mm of thickness
- in just months
- visually widening cheekbones, jawline, temples, etc.
Meanwhile bone growth is:
- extremely slow
- extremely small
- barely visible without imaging
This is why fighters, boxers, and martial artists often look like their bones grew — in reality it’s thickened, fibrotic soft tissue over the bone.
4. Fibrosis is why changes from “impact techniques” happen so fast
Real bone would never grow 3–5 mm in a few months.
Fibrosis can.
And it’s permanent-ish.
So if someone sees quick changes from impact, pressure, chewing devices, whatever — it’s almost certainly connective-tissue thickening, not osteoblast activity.
5. This isn’t necessarily “good” or “bad” — just reality
Fibrosis can make the face look:
- harder
- wider
- more angular
But it can also become:
- uneven
- lumpy
- chronically inflamed
- stiff
It’s not a controlled process like bone modeling; it’s a scar-response.
btw this is just corrected by gpt but its my idea
