Low e2 sides are extremely real

[Deleted member 28722]

0.5mg of arimidex destroyed my estrogen. Sides are extremely real. Nipple sensitivity was extreme from the changing levels… why am i so sensitive to medication… … what the fuck. Even stimulants i have to microcode… I can’t even handle t3 or t4 at regular doses anymore it turns my TSH into below 0.

 

[Deleted member 28722]

My e2 is literally defficent levels. This is horrible. I’ve never felt this before I can’t even get up… Even on aromasin it’s never gotten that bad. I’m definitely going to an endocrinologist twmr. 16.5 Bone age i should be growing… but my growrh has been stagnant for years… yet my wrist BA has been frozen.. what the fuck.

 

[thereallegend]

I'm hopping back on Aromasin

 

[Deleted member 28722]

Jesus christ. Aromasin spiked my free test too 180, DHT to 77, and my test to 1200, and my e2 was only relatively low. But adex has the opposite effect where my dht isn’t spiked that much but my test is extremely high and my e2 is 0

 

[Deleted member 28722]

When will i be able to figure out how the fuck my body works bro. WHEN. The only thing i know is my family reacts extremely well to steorids. My dad was insanely huge for his height, 340lbs of muscle at 5’8 throwing a 480bench and was bending bars… my brother was on steroids in high school and became No 1 pitcher in the state with only a extremely small dose of a DHT derrivitive.

 

[Deleted member 28722]

I feel like i have some sort of disorder. My GP don’t move. My IGF1 is lower. I didn’t grow much with high quality growth hormone. Is my body fighting against the HGH? Do i need to inhibit something. @Osie i’m gonna blast ghrp 6 and alpha GPC. what are good doses? I have cjc dac and hgh. i remember i only got growth when i was on ghrp6, alpha gpc, and cjc dac, but nothing noticeable with Hgh In 6 months despite knee and wrist plates and SPINE being open and on aromasin.

 

[Deleted member 28722]

Am i really just swallowing the genetic pill. My grandpa is 6’4 and i’m the shortest male in my family. What The fuck.

 

[Deleted member 28722]

I think i’m a small portion of the population that has IGF-1 receptor issues. I think i’m genuinely one of them….

 

[enchanted_elixir]

I feel like i have some sort of disorder. My GP don’t move. My IGF1 is lower. I didn’t grow much with high quality growth hormone. Is my body fighting against the HGH? Do i need to inhibit something. @Osie i’m gonna blast ghrp 6 and alpha GPC. what are good doses? I have cjc dac and hgh. i remember i only got growth when i was on ghrp6, alpha gpc, and cjc dac, but nothing noticeable with Hgh In 6 months despite knee and wrist plates and SPINE being open and on aromasin.

"heIgHtMaXinG doEsNt wORk bRO" - If You're Doing Any Of These 5 Mistakes, Heightmaxing WON'T Work.

This all assumes your longitudinal bones (legs and/or spine) growth plates aren't closed. Footer Citations. Studies that have been consulted to make this thread (I used A.I. in a small section of this thread and I made it cite the studies that it used) Ma Y, Bertun P, Zheng W...

looksmax.org

 

[Deleted member 28722]

My bone age has been 16.5 in my wrists probably since I was 14 years fucking old im finding out.

 

[Deleted member 28722]

It’s like my body becomes more sensitive to oral medication the more i take it

 

[enchanted_elixir]

I think i’m a small portion of the population that has IGF-1 receptor issues. I think i’m genuinely one of them….

Please read this thread:

"heIgHtMaXinG doEsNt wORk bRO" - If You're Doing Any Of These 5 Mistakes, Heightmaxing WON'T Work.

This all assumes your longitudinal bones (legs and/or spine) growth plates aren't closed. Footer Citations. Studies that have been consulted to make this thread (I used A.I. in a small section of this thread and I made it cite the studies that it used) Ma Y, Bertun P, Zheng W...

looksmax.org

 

[Deleted member 28722]

Please read this thread:

"heIgHtMaXinG doEsNt wORk bRO" - If You're Doing Any Of These 5 Mistakes, Heightmaxing WON'T Work.

This all assumes your longitudinal bones (legs and/or spine) growth plates aren't closed. Footer Citations. Studies that have been consulted to make this thread (I used A.I. in a small section of this thread and I made it cite the studies that it used) Ma Y, Bertun P, Zheng W...

looksmax.org

Bro i did. I sleep, maybe i should eat more. It could be i have a dogshit liver, but idk how to fix that.

 

[Osie]

I feel like i have some sort of disorder. My GP don’t move. My IGF1 is lower. I didn’t grow much with high quality growth hormone. Is my body fighting against the HGH? Do i need to inhibit something. @Osie i’m gonna blast ghrp 6 and alpha GPC. what are good doses? I have cjc dac and hgh. i remember i only got growth when i was on ghrp6, alpha gpc, and cjc dac, but nothing noticeable with Hgh In 6 months despite knee and wrist plates and SPINE being open and on aromasin.

Hit pm's!

 

[Osie]

My e2 is literally defficent levels. This is horrible. I’ve never felt this before I can’t even get up… Even on aromasin it’s never gotten that bad. I’m definitely going to an endocrinologist twmr. 16.5 Bone age i should be growing… but my growrh has been stagnant for years… yet my wrist BA has been frozen.. what the fuck.

Well, what we've seen from Aromatase-deficient men is that they often grow very slow or none at all during their teenage years then get a growth spurt from 18+. We have to keep in mind when your E2 levels are that low, your serum IGF-1 levels are probably low as well, and adding on the fact that you were hypothyroid, I am not surprised that your growth rate has slowed drastically but your bone age has stayed the same. Aromatase-deficient men had IGF-1 levels in the 100s but grew on average taller than 183cm, so be patient if you are going to use AI without any HGH stimulant1

Low E2 = Low IGF-1 = Bone age not getting any older and freezing but growth velocity also decreasing.

 

[Osie]

I'm hopping back on Aromasin

Why?

 

[Osie]

Bro i did. I sleep, maybe i should eat more. It could be i have a dogshit liver, but idk how to fix that.

Well, make a checklist of everything you need to grow taller (Sleep, high protein, low cortisol, high IGF-1 levels, low e2 levels, high t4 and t3 levels , etc) and if you are fulfilling all of those, you should grow somewhat!

 

[Saint Casanova]

When will i be able to figure out how the fuck my body works bro. WHEN. The only thing i know is my family reacts extremely well to steorids. My dad was insanely huge for his height, 340lbs of muscle at 5’8 throwing a 480bench and was bending bars… my brother was on steroids in high school and became No 1 pitcher in the state with only a extremely small dose of a DHT derrivitive.

340lb is very obese at that height.

 

[Saint Casanova]

Nuking E makes you feel shit. I was so cold for a couple of days after taking half an aromasin tablet.

 

[looksmidder]

Well, make a checklist of everything you need to grow taller (Sleep, high protein, low cortisol, high IGF-1 levels, low e2 levels, high t4 and t3 levels , etc) and if you are fulfilling all of those, you should grow somewhat!

osie!! im sorry if this is oir of nowhere but i love u man!! u have given me so musch good info!! i lray everything goes well for u in life

 

[Deleted member 28722]

340lb is very obese at that height.

dude was jacked so idk what ur talking abt

 

[barnmatrix]

Low E2 = Low IGF-1 = Bone age not getting any older and freezing but growth velocity also decreasing.

Not if hes pinning HGH exogenously.

 

[barnmatrix]

340lb is very obese at that height.

Was about to say. I find that hard to believe. 340lbs of muscle at 5'8 is impossible in any human, maybe a silverback guerilla. Definitely an exaggeration / OP larping.

 

[Deleted member 28722]

Was about to say. I find that hard to believe. 340lbs of muscle at 5'8 is impossible in any human, maybe a silverback guerilla. Definitely an exaggeration / OP larping.

i exaggerated but i do have pics of him

 

[Deleted member 28722]

Was about to say. I find that hard to believe. 340lbs of muscle at 5'8 is impossible in any human, maybe a silverback guerilla. Definitely an exaggeration / OP larping.

also the dudes frame is literally wide asf and he has. 6’1 wingspan so definitely scales things a bit more

 

[barnmatrix]

0.5mg of arimidex destroyed my estrogen. Sides are extremely real. Nipple sensitivity was extreme from the changing levels… why am i so sensitive to medication… … what the fuck. Even stimulants i have to microcode… I can’t even handle t3 or t4 at regular doses anymore it turns my TSH into below 0.

Schitzo post, regardless will offer my advice.

1. Your claiming the AI destroyed your estrogen ( low estrogen ) yet you mention nipple sensitivity. Nipple sensitivity is commonly considered a side effect of high estrogen and prolactin, so id double check that your actually experiencing that. If you are really concerned, just get bloods done, but understand that tissue concentration is not the same as blood concentration so its also possible you have a high tissue concentration somewhere although I highly doubt it.

2. Your saying aromasin increased your dht whereas arimidex increased your testosterone, that doesnt really make sense either. High testosterone = high dht, and vise versa. There is no selectivity of either aromatase inhibitor to increase specifically DHT or Testosterone more than the other. By reducing aromatase it frees up more aromatization for 5ar, but this is entirely dose dependent, 0.5mg of adex does not have the same potency as 12.5mg of asin. Do some research if you want to see which one is more potent.

3. Your mentioning your IGF1 is low despite being on high quality growth hormone. Im not sure what dose your on but if its anything around whats most used on the forum ( 5-8 IUs) then your IGF1 should be out of range high if the GH is actually good quality. If its not out of range high then re evaluate your routine and protocol, make sure your fasting before injection, make sure your insulin / blood sugar isnt diabetic, test your liver uptake, consider the posibility your GH may not be good quality, and consider inhibiting somatostatin. Also make sure to test your actual blood concentration of growth hormone, not just IGF1. If your growth hormone is high but igf1 isnt, then you know theres a problem with your conversion. If your growth hormone is also low then its likely your source is trash.

4. You mention a bone age of 16.5 but also said you havent grown at all. Im not sure how long you've been taking this stuff for but id recommend getting an actual x-ray of your growth plates, not just a bone age x ray in the wrist, and see whether or not your plates are open in certain areas, you can check your knees and spine as those are the two areas your actually attempting to grow from.

 

[barnmatrix]

also the dudes frame is literally wide asf and he has. 6’1 wingspan so definitely scales things a bit more

Las Vegas bodybuilder transforms himself into a 350lbs giant

Craig Golias, who lives in Las Vegas, Nevada, spent 10 years training six days a week - for an hour and a half at a time - to transform his body after he 'didn't like what he saw in the mirror' as a child.

www.dailymail.co.uk

For reference, this is a 350lb man at 6'3. Your dad was probably 230-250 at most.

 

[Deleted member 28722]

Schitzo post, regardless will offer my advice.

1. Your claiming the AI destroyed your estrogen ( low estrogen ) yet you mention nipple sensitivity. Nipple sensitivity is commonly considered a side effect of high estrogen and prolactin, so id double check that your actually experiencing that. If you are really concerned, just get bloods done, but understand that tissue concentration is not the same as blood concentration so its also possible you have a high tissue concentration somewhere although I highly doubt it.

2. Your saying aromasin increased your dht whereas arimidex increased your testosterone, that doesnt really make sense either. High testosterone = high dht, and vise versa. There is no selectivity of either aromatase inhibitor to increase specifically DHT or Testosterone more than the other. By reducing aromatase it frees up more aromatization for 5ar, but this is entirely dose dependent, 0.5mg of adex does not have the same potency as 12.5mg of asin. Do some research if you want to see which one is more potent.

3. Your mentioning your IGF1 is low despite being on high quality growth hormone. Im not sure what dose your on but if its anything around whats most used on the forum ( 5-8 IUs) then your IGF1 should be out of range high if the GH is actually good quality. If its not out of range high then re evaluate your routine and protocol, make sure your fasting before injection, make sure your insulin / blood sugar isnt diabetic, test your liver uptake, consider the posibility your GH may not be good quality, and consider inhibiting somatostatin. Also make sure to test your actual blood concentration of growth hormone, not just IGF1. If your growth hormone is high but igf1 isnt, then you know theres a problem with your conversion. If your growth hormone is also low then its likely your source is trash.

4. You mention a bone age of 16.5 but also said you havent grown at all. Im not sure how long you've been taking this stuff for but id recommend getting an actual x-ray of your growth plates, not just a bone age x ray in the wrist, and see whether or not your plates are open in certain areas, you can check your knees and spine as those are the two areas your actually attempting to grow from.

nipple sensitivity is literally also caused by the immense rapid change of hormones lmao. so what the fuck are you talking about.

 

[Deleted member 28722]

Schitzo post, regardless will offer my advice.

1. Your claiming the AI destroyed your estrogen ( low estrogen ) yet you mention nipple sensitivity. Nipple sensitivity is commonly considered a side effect of high estrogen and prolactin, so id double check that your actually experiencing that. If you are really concerned, just get bloods done, but understand that tissue concentration is not the same as blood concentration so its also possible you have a high tissue concentration somewhere although I highly doubt it.

2. Your saying aromasin increased your dht whereas arimidex increased your testosterone, that doesnt really make sense either. High testosterone = high dht, and vise versa. There is no selectivity of either aromatase inhibitor to increase specifically DHT or Testosterone more than the other. By reducing aromatase it frees up more aromatization for 5ar, but this is entirely dose dependent, 0.5mg of adex does not have the same potency as 12.5mg of asin. Do some research if you want to see which one is more potent.

3. Your mentioning your IGF1 is low despite being on high quality growth hormone. Im not sure what dose your on but if its anything around whats most used on the forum ( 5-8 IUs) then your IGF1 should be out of range high if the GH is actually good quality. If its not out of range high then re evaluate your routine and protocol, make sure your fasting before injection, make sure your insulin / blood sugar isnt diabetic, test your liver uptake, consider the posibility your GH may not be good quality, and consider inhibiting somatostatin. Also make sure to test your actual blood concentration of growth hormone, not just IGF1. If your growth hormone is high but igf1 isnt, then you know theres a problem with your conversion. If your growth hormone is also low then its likely your source is trash.

4. You mention a bone age of 16.5 but also said you havent grown at all. Im not sure how long you've been taking this stuff for but id recommend getting an actual x-ray of your growth plates, not just a bone age x ray in the wrist, and see whether or not your plates are open in certain areas, you can check your knees and spine as those are the two areas your actually attempting to grow from.

are you retarded. There’s many people who don’t get a dht increase from arimidex. Aromasin is different entirely it’s literally a DHT derivative, and it is highly androgenic so wtf are you also talking about.

 

[Deleted member 28722]

Schitzo post, regardless will offer my advice.

1. Your claiming the AI destroyed your estrogen ( low estrogen ) yet you mention nipple sensitivity. Nipple sensitivity is commonly considered a side effect of high estrogen and prolactin, so id double check that your actually experiencing that. If you are really concerned, just get bloods done, but understand that tissue concentration is not the same as blood concentration so its also possible you have a high tissue concentration somewhere although I highly doubt it.

2. Your saying aromasin increased your dht whereas arimidex increased your testosterone, that doesnt really make sense either. High testosterone = high dht, and vise versa. There is no selectivity of either aromatase inhibitor to increase specifically DHT or Testosterone more than the other. By reducing aromatase it frees up more aromatization for 5ar, but this is entirely dose dependent, 0.5mg of adex does not have the same potency as 12.5mg of asin. Do some research if you want to see which one is more potent.

3. Your mentioning your IGF1 is low despite being on high quality growth hormone. Im not sure what dose your on but if its anything around whats most used on the forum ( 5-8 IUs) then your IGF1 should be out of range high if the GH is actually good quality. If its not out of range high then re evaluate your routine and protocol, make sure your fasting before injection, make sure your insulin / blood sugar isnt diabetic, test your liver uptake, consider the posibility your GH may not be good quality, and consider inhibiting somatostatin. Also make sure to test your actual blood concentration of growth hormone, not just IGF1. If your growth hormone is high but igf1 isnt, then you know theres a problem with your conversion. If your growth hormone is also low then its likely your source is trash.

4. You mention a bone age of 16.5 but also said you havent grown at all. Im not sure how long you've been taking this stuff for but id recommend getting an actual x-ray of your growth plates, not just a bone age x ray in the wrist, and see whether or not your plates are open in certain areas, you can check your knees and spine as those are the two areas your actually attempting to grow from.

I have a growth plate xray, it was done by Bone Xpert

 

[Deleted member 28722]

Schitzo post, regardless will offer my advice.

1. Your claiming the AI destroyed your estrogen ( low estrogen ) yet you mention nipple sensitivity. Nipple sensitivity is commonly considered a side effect of high estrogen and prolactin, so id double check that your actually experiencing that. If you are really concerned, just get bloods done, but understand that tissue concentration is not the same as blood concentration so its also possible you have a high tissue concentration somewhere although I highly doubt it.

2. Your saying aromasin increased your dht whereas arimidex increased your testosterone, that doesnt really make sense either. High testosterone = high dht, and vise versa. There is no selectivity of either aromatase inhibitor to increase specifically DHT or Testosterone more than the other. By reducing aromatase it frees up more aromatization for 5ar, but this is entirely dose dependent, 0.5mg of adex does not have the same potency as 12.5mg of asin. Do some research if you want to see which one is more potent.

3. Your mentioning your IGF1 is low despite being on high quality growth hormone. Im not sure what dose your on but if its anything around whats most used on the forum ( 5-8 IUs) then your IGF1 should be out of range high if the GH is actually good quality. If its not out of range high then re evaluate your routine and protocol, make sure your fasting before injection, make sure your insulin / blood sugar isnt diabetic, test your liver uptake, consider the posibility your GH may not be good quality, and consider inhibiting somatostatin. Also make sure to test your actual blood concentration of growth hormone, not just IGF1. If your growth hormone is high but igf1 isnt, then you know theres a problem with your conversion. If your growth hormone is also low then its likely your source is trash.

4. You mention a bone age of 16.5 but also said you havent grown at all. Im not sure how long you've been taking this stuff for but id recommend getting an actual x-ray of your growth plates, not just a bone age x ray in the wrist, and see whether or not your plates are open in certain areas, you can check your knees and spine as those are the two areas your actually attempting to grow from.

I said both increased my test, aromasin increased my dht way more than arimidex ever does. Which if you understood what the fuck aromasin actually is it makes perfect sense.

 

[Deleted member 28722]

Las Vegas bodybuilder transforms himself into a 350lbs giant

Craig Golias, who lives in Las Vegas, Nevada, spent 10 years training six days a week - for an hour and a half at a time - to transform his body after he 'didn't like what he saw in the mirror' as a child.

www.dailymail.co.uk

For reference, this is a 350lb man at 6'3. Your dad was probably 230-250 at most.

My dad was literally a professional body builder, whatever his size was he was definitely huge because his weight right now is still over 210-230

 

[barnmatrix]

are you retarded. There’s many people who don’t get a dht increase from arimidex. Aromasin is different entirely it’s literally a DHT derivative, and it is highly androgenic so wtf are you also talking about.

Thats hilarious man. Spent a good ten minutes trying to give you advice and you write some disrespectful shit. I can promise you dont know more than me about these subjects but if you disagree with my opinion at least be respectful in doing so. Nothing you said in that comment is true btw. I suggest you do some basic research before making ridiculous claims on this forum. You obviously have no idea what your talking about and there is zero basis of evidence behind the following claim

here’s many people who don’t get a dht increase from arimidex

You probably dont even know what an HPTA axis is. This is why some people shouldnt mess with their hormones because they think they know more than they do, regardless ill still take my time to educate you since thats what people did for me when I started off. Testosterone converts into DHT via 5 alpha reductase enzyme. Aromatase inhibitors, ( arimidex, aromasin, letrozole, doesnt matter which one) all block aromatase enzyme and in doing so increase free testosterone which is no longer bound to aromatase ( the enzyme that converts testosterone to estrogen ), the blocking of aromatase frees up more testosterone which is now able to bind to more 5 alpha reductase enzymes thus increasing both testosterone and DHT.


I suggest you do actual research on pubmed instead of basing all your knowledge off random claims you heard from obscure bodybuilding forums or other uneducated kids.

 

[Deleted member 28722]

Thats hilarious man. Spent a good ten minutes trying to give you advice and you write some disrespectful shit. I can promise you dont know more than me about these subjects but if you disagree with my opinion at least be respectful in doing so. Nothing you said in that comment is true btw. I suggest you do some basic research before making ridiculous claims on this forum. You obviously have no idea what your talking about and there is zero basis of evidence behind the following claim

You probably dont even know what an HPTA axis is. This is why some people shouldnt mess with their hormones because they think they know more than they do, regardless ill still take my time to educate you since thats what people did for me when I started off. Testosterone converts into DHT via 5 alpha reductase enzyme. Aromatase inhibitors, ( arimidex, aromasin, letrozole, doesnt matter which one) all block aromatase enzyme and in doing so increase free testosterone which is no longer bound to aromatase ( the enzyme that converts testosterone to estrogen ), the blocking of aromatase frees up more testosterone which is now able to bind to more 5 alpha reductase enzymes thus increasing both testosterone and DHT.


I suggest you do actual research on pubmed instead of basing all your knowledge off random claims you heard from obscure bodybuilding forums or other uneducated kids.

You litterally tried getting niggas to inject IGF1 Des into their knee caps lmao

 

[Deleted member 28722]

Thats hilarious man. Spent a good ten minutes trying to give you advice and you write some disrespectful shit. I can promise you dont know more than me about these subjects but if you disagree with my opinion at least be respectful in doing so. Nothing you said in that comment is true btw. I suggest you do some basic research before making ridiculous claims on this forum. You obviously have no idea what your talking about and there is zero basis of evidence behind the following claim

You probably dont even know what an HPTA axis is. This is why some people shouldnt mess with their hormones because they think they know more than they do, regardless ill still take my time to educate you since thats what people did for me when I started off. Testosterone converts into DHT via 5 alpha reductase enzyme. Aromatase inhibitors, ( arimidex, aromasin, letrozole, doesnt matter which one) all block aromatase enzyme and in doing so increase free testosterone which is no longer bound to aromatase ( the enzyme that converts testosterone to estrogen ), the blocking of aromatase frees up more testosterone which is now able to bind to more 5 alpha reductase enzymes thus increasing both testosterone and DHT.


I suggest you do actual research on pubmed instead of basing all your knowledge off random claims you heard from obscure bodybuilding forums or other uneducated kids.

I literally have blood tests your actually a fucking retard if you think every drug acts a specific way in a persons body. As I said 0.5 mg of arimidex did not increase my dht above the average range while Aromasin did. And no, 0.5mg of arimidex is more effective then 12.5mg of asin in terms of reducing estrodial. So by your logic the DHT increase yielded from arimidex in clinical trials should be more right? Expect it fucking isn’t and it also didn’t increase total test levels as much as aromasin

 

[barnmatrix]

I said both increased my test, aromasin increased my dht way more than arimidex ever does. Which if you understood what the fuck aromasin actually is it makes perfect sense.

Its actually hilarious to me that you think you know more than you do. Your the one typing up a schitzo post begging for help on stuff that doesnt even make sense, I tried to help you now you think you can school me on something I know more than you about. Whatever bro I have nothing to prove thats why Im a greycel I only post on here when I actually have value to add instead of just larping random nonsense.

 

[barnmatrix]

I literally have blood tests your actually a fucking retard if you think every drug acts a specific way in a persons body. As I said 0.5 mg of arimidex did not increase my dht above the average range while Aromasin did. And no, 0.5mg of arimidex is more effective then 12.5mg of asin in terms of reducing estrodial. So by your logic the DHT increase yielded from arimidex in clinical trials should be more right? Expect it fucking isn’t and it also didn’t increase total test levels as much as aromasin

You didnt even read my comment lmao. Maybe if you actually read and used your brain before acting then you wouldnt be begging for help on here.

 

[Deleted member 28722]

Its actually hilarious to me that you think you know more than you do. Your the one typing up a schitzo post begging for help on stuff that doesnt even make sense, I tried to help you now you think you can school me on something I know more than you about. Whatever bro I have nothing to prove thats why Im a greycel I only post on here when I actually have value to add instead of just larping random nonsense.

Cause your out here saying i’m wrong . I shared my experience on a forum. Your the dumb fuck whose trying to say it’s false. BRB tho. Here’s real bloods retard. JFL

 

[mogstars]

Am i really just swallowing the genetic pill. My grandpa is 6’4 and i’m the shortest male in my family. What The fuck.

If you're 5'10 nobody cares

 

[Deleted member 28722]

You didnt even read my comment lmao. Maybe if you actually read and used your brain before acting then you wouldnt be begging for help on here.

How about you re read your own comment instead of correcting mine. You literally stated 12.5 mg of asin was more potent than 0.5mg of adex. You never stated any context about that. Rather you meant the E2 lowering or the DHT increase. But by your logic, you fucking pig. Because adex lowers more E2 than aromasin, it should also increase more DHT and total test correct?

 

[barnmatrix]

Cause your out here saying i’m wrong . I shared my experience on a forum. Your the dumb fuck whose trying to say it’s false. BRB tho. Here’s real bloods retard. JFL

Dude im not arguing with your bloods. Let me dumb down my point for you because I guess you couldnt understand it.

All I was saying is that arimidex and aromasin have no specific affinity to increase dht more than testosterone or vise versa. Thats all I said. Im not arguing with your bloodwork.

 

[barnmatrix]

If you're 5'10 nobody cares

Dont even give this guy the light of day hes a schitzo.

 

[Deleted member 28722]

Dont even give this guy the light of day hes a schitzo.

“Yo bro, stick this needle of IGF1 des into your fucking knee caps real quick, i swear the results are good bro”

 

[Deleted member 28722]

Dude im not arguing with your bloods. Let me dumb down my point for you because I guess you couldnt understand it.

All I was saying is that arimidex and aromasin have no specific affinity to increase dht more than testosterone or vise versa. Thats all I said. Im not arguing with your bloodwork.

Exemestane and Its Active Metabolite 17-Hydroexemestane Induce UDP-Glucuronosyltransferase (UGT) 2B17 Expression in Breast Cancer Cells - PubMed

Exemestane (EXE) is an aromatase inhibitor indicated for endocrine therapy of breast cancer in postmenopausal women. The primary active metabolite of EXE, 17-hydroexemestane (17-HE), is inactivated via glucuronidation, mainly by UDP-glucuronosyltransferase 2B17 (UGT2B17). UGT2B17 also has a...

pubmed.ncbi.nlm.nih.gov

Exemestane metabolites: Synthesis, stereochemical elucidation, biochemical activity and anti-proliferative effects in a hormone-dependent breast cancer cell line - PubMed

Exemestane is a third-generation steroidal aromatase inhibitor that has been used in clinic for hormone-dependent breast cancer treatment in post-menopausal women. It is known that exemestane undergoes a complex metabolization, giving rise to some already identified metabolites, the...

pubmed.ncbi.nlm.nih.gov

How about you read both of these and realize there’s a reason why aromasin causes a higher up regulation in androgens seperate from it being a AI. Also my blood results are loading. These studies literally say it’s a DHT derivative. It is totally possible for someone to get more of a DHT increase from asin than adex no matter the dosage. I actually think Osie explained this to you

 

[barnmatrix]

“Yo bro, stick this needle of IGF1 des into your fucking knee caps real quick, i swear the results are good bro”

This you pal??

 

[barnmatrix]

Exemestane and Its Active Metabolite 17-Hydroexemestane Induce UDP-Glucuronosyltransferase (UGT) 2B17 Expression in Breast Cancer Cells - PubMed

Exemestane (EXE) is an aromatase inhibitor indicated for endocrine therapy of breast cancer in postmenopausal women. The primary active metabolite of EXE, 17-hydroexemestane (17-HE), is inactivated via glucuronidation, mainly by UDP-glucuronosyltransferase 2B17 (UGT2B17). UGT2B17 also has a...

pubmed.ncbi.nlm.nih.gov

Exemestane metabolites: Synthesis, stereochemical elucidation, biochemical activity and anti-proliferative effects in a hormone-dependent breast cancer cell line - PubMed

Exemestane is a third-generation steroidal aromatase inhibitor that has been used in clinic for hormone-dependent breast cancer treatment in post-menopausal women. It is known that exemestane undergoes a complex metabolization, giving rise to some already identified metabolites, the...

pubmed.ncbi.nlm.nih.gov

How about you read both of these and realize there’s a reason why aromasin causes a higher up regulation in androgens seperate from it being a AI. Also my blood results are loading. These studies literally say it’s a DHT derivative. It is totally possible for someone to get more of a DHT increase from asin than adex no matter the dosage. I actually think Osie explained this to you

Bro pulled up two random unrelated studies. You cant even read the studies you send to others let alone the ones relevant to you. Any difference in testosterone or DHT increase exhibited by one AI vs another is only due to the difference in potency and efficacy in reducing aromatase of one AI or another. A relatively equal dose of different AIs will yield similar results of testosterone/dht levels assuming all other factors are the same.

Dont know why im even explaining this you dont even listen.

 

[Deleted member 28722]

This you pal??

Nah, I spoke to niggas who are in your Biobros group. It’s not like i was taking you seriously let’s be real nigga. Your entire basis for that theory was a shit Igf1-lr3 study that was used in children who had a reduced IGF1 expression from HGH. But it never said anything about using DES

 

[Deleted member 28722]

Bro pulled up two random unrelated studies. You cant even read the studies you send to others let alone the ones relevant to you. Any difference in testosterone or DHT increase exhibited by one AI vs another is only due to the difference in potency and efficacy in reducing aromatase of one AI or another. A relatively equal dose of different AIs will yield similar results of testosterone/dht levels assuming all other factors are the same.

Dont know why im even explaining this you dont even listen.

read the actual full study before saying it doesn’t correlate dumb fuck. Don’t just focus on the “metabolites”

 

[Saint Casanova]

Exemestane and Its Active Metabolite 17-Hydroexemestane Induce UDP-Glucuronosyltransferase (UGT) 2B17 Expression in Breast Cancer Cells - PubMed

Exemestane (EXE) is an aromatase inhibitor indicated for endocrine therapy of breast cancer in postmenopausal women. The primary active metabolite of EXE, 17-hydroexemestane (17-HE), is inactivated via glucuronidation, mainly by UDP-glucuronosyltransferase 2B17 (UGT2B17). UGT2B17 also has a...

pubmed.ncbi.nlm.nih.gov

Exemestane metabolites: Synthesis, stereochemical elucidation, biochemical activity and anti-proliferative effects in a hormone-dependent breast cancer cell line - PubMed

Exemestane is a third-generation steroidal aromatase inhibitor that has been used in clinic for hormone-dependent breast cancer treatment in post-menopausal women. It is known that exemestane undergoes a complex metabolization, giving rise to some already identified metabolites, the...

pubmed.ncbi.nlm.nih.gov

How about you read both of these and realize there’s a reason why aromasin causes a higher up regulation in androgens seperate from it being a AI. Also my blood results are loading. These studies literally say it’s a DHT derivative. It is totally possible for someone to get more of a DHT increase from asin than adex no matter the dosage. I actually think Osie explained this to you

Idiot, it is not a DHT derivative. The metabolite you’re talking about is structurally similar to EQ, which is a test derivative. Stop making it out like asin is masteron or primo.

 

[Saint Casanova]

read the actual full study before saying it doesn’t correlate dumb fuck. Don’t just focus on the “metabolites”

Bro stop taking any pharmaceuticals, you have no clue about anything and you’ll end up damaging yourself.