OsteoForgeNZ
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Thread song:
Mandibular recession is a structural hard-stop for facial aesthetics. You either cut bone, or you cope indefinitely.
The current space is drowning in soft-tissue copers operating under the staggering delusion that mastic gum and myofunctional therapy will reverse a decade of maxillo-mandibular downward growth and genetic retrognathia. Let’s establish biological reality: soft-tissue tension does not remodel fused cortical bone.
If you have cleared Tanner Stage 4 and your Pogonion remains vertically eclipsed by your Nasion along the Frankfort Horizontal Plane, you are not a "late bloomer." You are a skeletal Class II invalid.
This thread is a systematic dismantling of lower-third projection mechanics. I will break down the exact biological parameters of chin and jaw development, destroying the orthotropic cope. Expect a clinical deep-dive into:
You cannot fix a defect if you do not measure the defect. You must obtain a lateral cephalogram. Relying on an iPhone selfie taken at 50mm focal length to assess your facial thirds is low-IQ behavior due to lens distortion.
Extract the following points from your radiograph: Sella (S), Nasion (N), A-Point (A), B-Point (B), and Pogonion (Pog).
If your jaw is recessed, you either have microgenia (an underdeveloped symphysis menti) or retrognathia (an underdeveloped and posteriorly rotated mandible).
A weak chin forces the mentalis muscle to hyper-contract just to achieve lip seal (lip incompetence). This chronic contraction creates a dimpled, golf-ball appearance on the chin pad and actively pulls the soft tissue backward, visually worsening the recession.
Absolute garbage. Wolff’s Law dictates that bone remodels under mechanical stress, but masticatory forces act primarily on the gonial angle, the ramus, and the zygomatic arches where the masseter and temporalis attach. The anterior symphysis menti (chin point) has zero direct masticatory muscle attachments that exert a forward, appositional force. Hypertrophying your masseters while ignoring an SNB of 76 just turns you into a bloat-faced rectangle with a recessed profile.
MCC is secondary cartilage. Unlike the primary growth plates in your tibia, it lacks a true epiphysis and remains adaptive to mechanical loading, but only under a highly specific systemic cascade: Growth Hormone (GH) and bioavailable Insulin-like Growth Factor-1 (IGF-1). (Note: IGF-1 bioavailability is modulated by IGFBP-3, not SHBG. Anyone claiming SHBG binds IGF-1 does not understand basic endocrinology).
The catastrophic error early adolescents make is allowing premature systemic estrogen spikes to hijack this window. While heavy aromatization (T to E2) directly causes epiphyseal closure in your long bones, the condylar mechanism is different. The condyle doesn't strictly "fuse" like a femur, but premature E2 exposure accelerates global skeletal maturation, permanently crashing the GH/IGF-1 somatic growth phase. If you aromatize heavily during early puberty, your forward growth vectors flatline, locking your jaw into a permanently recessed, underdeveloped state.
If you are within this window, the protocol is aggressive aromatase inhibition combined with systemic GH elevation to maximize chondrogenesis in the temporomandibular joint.
Crashed E2 destroys bone mineral density (BMD) over time and induces severe cognitive lethargy and joint desiccation. Drop the Letrozole immediately if you experience joint clicking in the TMJ, as synovial fluid depletion will actively grind down the condyle you are trying to grow. Monitor standing height and bi-iliac width; once vertical growth ceases for 6 months, the AI is useless and actively harmful to remaining aesthetic development.
You must alter the bone.
Surgical Mechanics:
The cut must be made a minimum of 5.0mm below the mental foramen to preserve the inferior alveolar nerve (IAN) bundle.
Advancement maxes out around 8.0mm to 10.0mm. Beyond 10.0mm, you create a "step-off" deformity at the inferior border of the mandible, where the advanced segment disconnects wildly from the mandibular body, visible as an ugly notch through the skin.
Fixation requires 2.0mm titanium plates and monocortical screws.
You require a BSSO. The mandible is split bilaterally down the ramus. The entire tooth-bearing segment is slid forward.
The Force Multiplier: Counter-Clockwise (CCW) Rotation.
Downswung maxillas (steep mandibular plane angle) are the primary cause of poor chin projection in mouth breathers. Moving the jaw straight forward (linear advancement) leaves the face looking long and horsey.
You must combine Le Fort I impaction with BSSO CCW rotation. The posterior maxilla is impacted (moved up into the skull), and the mandible rotates upward and forward on a hinge. This flattens the occlusal plane, dramatically projects the Pogonion, and shortens the vertical midface. This is the absolute S-Tier of structural modification.
Intervention 3:
The Titanium Apex (Custom 3D-Printed Implants)
PEEK merely halts bone erosion. Custom 3D-printed titanium is the final escalation.
When your sagittal deficit aggressively eclipses the 10.0mm sliding genioplasty limit—that exact biomechanical threshold where advancing the mobilized segment creates a grotesque step-off deformity—you bypass native bone limitations entirely by bolting a mathematically perfect porous titanium matrix directly into the jaw. You extract a CBCT scan. An engineer dictates the precise geometric wrap-around.
The printed metal extends laterally along the mandibular body (masking the transition zone) and projects the symphysis forward with zero geometric restrictions.
Cortical bone cells grow directly into the porous metal. The implant becomes biologically indistinguishable from the skeleton.
You must mimic the viscoelasticity (G-prime) of actual bone. Injecting highly hydrophilic, soft hyaluronic acid (like Juvederm Ultra) into the chin creates a soft, doughy, undefined mass.
The Compound:
Radiesse (Calcium Hydroxylapatite - CaHA).
CaHA has the highest G-prime available. It consists of calcium microspheres suspended in a carboxymethylcellulose gel carrier. It does not draw in water. It mimics structural bone and induces neocollagenesis.
The Mechanical Execution:
Use a 27G 1/2" needle.
The injection must be a supraperiosteal bolus.
Enter at the exact anterior point of the Pogonion. Push the needle directly through the dermis, fat, and muscle until you tap the absolute floor of the bone (the periosteum). Aspirate for 3 seconds. If clear, deploy a bolus of 0.2ml to 0.4ml of CaHA directly onto the bone.
Mold it immediately before the carrier gel sets.
Surgical Botch: Permanent Lip Ptosis (Chimp-Lip)
During a genioplasty, the surgeon must strip the mentalis muscle off the bone to make the saw cut. If the surgeon fails to meticulously resuspend the mentalis muscle using non-resorbable sutures to the bone before closing the mucosa, the muscle retracts. The lower lip drops permanently. You expose your lower incisors at rest, drool, and adopt a vacant, low-IQ phenotype. This is nearly impossible to fix surgically once the muscle scars down in a retracted position.
Vascular Occlusion and Tissue Necrosis
If you botch a supraperiosteal Radiesse injection and deposit high G-prime CaHA into the submental artery, you cause immediate retrograde vascular occlusion. The skin on your chin will blanch white, then turn a mottled, bruised purple within 24 hours. Because CaHA is not reversible with Hyaluronidase, the tissue fed by that artery will literally rot, die, and slough off, leaving a permanent concave crater on your face.
The Witch Chin (Over-Projection)
Advancing a genioplasty beyond the STCA 0.0mm TVL limit creates a hyper-masculinized, crescent-moon profile. Combined with a deep labiomental fold, the soft tissue pad sags under its own projected weight.
If running the endocrine stack (Ages 13-16), cease all Somatropin and Letrozole the moment a hand-wrist radiograph confirms total fusion of the radial epiphysis. At this point, the receptors in the MCC are fully downregulated and ossified. Further GH administration will exclusively thicken the orbital ridges (acromegaly) and widen the nasal alar base without adding a single millimeter to your sagittal chin projection. Wash out the Letrozole slowly over 3 weeks to prevent severe estrogen rebound and subsequent gynecomastia.
If consulting for surgery (Post-Puberty), your absolute hard stop is the cephalometric norm. If your SNB is currently 79 degrees and your Pogonion is at -2.0mm, do not let a scalpel touch your face. You are within standard deviations of optimal. Chasing a +2.0mm Pogonion projection past the TVL moves you from masculine to grotesque.
Read the sticky before replying. JFL if you ask about biotin, tongue chewing, or bone mashing. I'm out.
For more information, and softmaxxing (On chin-projection): https://looksmax.org/threads/megath...g-skeletal-maintenance.2204704/#post-30239005
@nordsmog @buccalfatremoval @Topkra @AscendHQ @Jenson @Regret. @blinkers @fagg @HexumReincarnated @nigga123
Mandibular recession is a structural hard-stop for facial aesthetics. You either cut bone, or you cope indefinitely.
The current space is drowning in soft-tissue copers operating under the staggering delusion that mastic gum and myofunctional therapy will reverse a decade of maxillo-mandibular downward growth and genetic retrognathia. Let’s establish biological reality: soft-tissue tension does not remodel fused cortical bone.
If you have cleared Tanner Stage 4 and your Pogonion remains vertically eclipsed by your Nasion along the Frankfort Horizontal Plane, you are not a "late bloomer." You are a skeletal Class II invalid.
This thread is a systematic dismantling of lower-third projection mechanics. I will break down the exact biological parameters of chin and jaw development, destroying the orthotropic cope. Expect a clinical deep-dive into:
- The Condylar Window: The exact endocrine and temporal parameters (Ages 13–16) for genuine natural apposition.
- The Orthotropic Ceiling: The hard biomechanical limits of Wolff’s Law and why tongue posture is fundamentally useless post-puberty.
- The Titanium Imperative: The absolute necessity of bimaxillary osteotomies (BSSO) and rigid titanium fixation once epiphyseal closure is finalized.
Code:
Acronym Clinical Definition
SNA Maxillary sagittal position relative to cranial base.
SNB Mandibular sagittal position; primary metric of recession.
ANB The sagittal delta between maxilla and mandible.
Pog Pogonion—most anterior point of the chin.
TVL True Vertical Line; aesthetic plumb line for projection assessment.
FH Frankfort Horizontal; the skull’s physiological level plane.
MCC Mandibular Condylar Cartilage—adaptive growth engine of the lower jaw.
BSSO Bilateral Sagittal Split Osteotomy; gold-standard surgical advancement.
1. The Mirin Audit and Geometric Baseline
Visual aesthetics are not subjective. They are bound to cephalometric mathematics.You cannot fix a defect if you do not measure the defect. You must obtain a lateral cephalogram. Relying on an iPhone selfie taken at 50mm focal length to assess your facial thirds is low-IQ behavior due to lens distortion.
Extract the following points from your radiograph: Sella (S), Nasion (N), A-Point (A), B-Point (B), and Pogonion (Pog).
If your jaw is recessed, you either have microgenia (an underdeveloped symphysis menti) or retrognathia (an underdeveloped and posteriorly rotated mandible).
Steiner Analysis defines the absolute coordinates of the human face.
True chin projection is dictated by the Pogonion (the most anterior point of the chin) relative to the Nasion (bridge of the nose) via the True Vertical Line (TVL). According to Arnett and MacLaughlin's STCA (Soft Tissue Cephalometric Analysis), the male soft tissue Pogonion should sit exactly -2.0mm to 0.0mm behind the TVL. If your Pogonion is at -6.0mm, you lack the skeletal foundation to support your lower lip, mentalis muscle, and submental skin.
- SNA Angle (Maxillary position): Norm is 82 degrees.
- SNB Angle (Mandibular position): Norm is 80 degrees.
- ANB Angle (The differential): Norm is 2 degrees.
True chin projection is dictated by the Pogonion (the most anterior point of the chin) relative to the Nasion (bridge of the nose) via the True Vertical Line (TVL). According to Arnett and MacLaughlin's STCA (Soft Tissue Cephalometric Analysis), the male soft tissue Pogonion should sit exactly -2.0mm to 0.0mm behind the TVL. If your Pogonion is at -6.0mm, you lack the skeletal foundation to support your lower lip, mentalis muscle, and submental skin.
A weak chin forces the mentalis muscle to hyper-contract just to achieve lip seal (lip incompetence). This chronic contraction creates a dimpled, golf-ball appearance on the chin pad and actively pulls the soft tissue backward, visually worsening the recession.
2. The Simulated Cope
Absolute garbage. Wolff’s Law dictates that bone remodels under mechanical stress, but masticatory forces act primarily on the gonial angle, the ramus, and the zygomatic arches where the masseter and temporalis attach. The anterior symphysis menti (chin point) has zero direct masticatory muscle attachments that exert a forward, appositional force. Hypertrophying your masseters while ignoring an SNB of 76 just turns you into a bloat-faced rectangle with a recessed profile.
3. Mechanistic Reality: The Endocrine Window (Ages 13-16)
Mandibular condylar cartilage (MCC) does not dictate the length of the mandibular body. The MCC governs ramus height and the downward-forward translation of the entire maxillo-mandibular complex. The mandibular body and symphysis (chin) project via intramembranous ossification—specifically periosteal apposition and resorption—driven by that initial condylar translation.MCC is secondary cartilage. Unlike the primary growth plates in your tibia, it lacks a true epiphysis and remains adaptive to mechanical loading, but only under a highly specific systemic cascade: Growth Hormone (GH) and bioavailable Insulin-like Growth Factor-1 (IGF-1). (Note: IGF-1 bioavailability is modulated by IGFBP-3, not SHBG. Anyone claiming SHBG binds IGF-1 does not understand basic endocrinology).
The catastrophic error early adolescents make is allowing premature systemic estrogen spikes to hijack this window. While heavy aromatization (T to E2) directly causes epiphyseal closure in your long bones, the condylar mechanism is different. The condyle doesn't strictly "fuse" like a femur, but premature E2 exposure accelerates global skeletal maturation, permanently crashing the GH/IGF-1 somatic growth phase. If you aromatize heavily during early puberty, your forward growth vectors flatline, locking your jaw into a permanently recessed, underdeveloped state.
The synergistic effect of mechanical loading and IGF-1 on mandibular growth is documented literature, not forum theory.
Citation: Rabie AB, et al. "Systemic GH and local rhIGF-1 enhance mandibular condylar growth." Journal of Dental Research, 2003.
n-size: 45 in-vivo models.
p-value: p < 0.001.
Excerpt: "Systemic administration of Growth Hormone significantly upregulated local IGF-1 synthesis in the condylar cartilage, leading to a 34% increase in chondrocyte proliferation and endochondral ossification in the posterior condyle. This translated to a measurable increase in total mandibular length when combined with forward mandibular posturing devices."
Translation: HGH + mechanical forward posturing (functional appliances/orthotropics) creates actual skeletal length, provided the sutures are not fused by estradiol.
Citation: Rabie AB, et al. "Systemic GH and local rhIGF-1 enhance mandibular condylar growth." Journal of Dental Research, 2003.
n-size: 45 in-vivo models.
p-value: p < 0.001.
Excerpt: "Systemic administration of Growth Hormone significantly upregulated local IGF-1 synthesis in the condylar cartilage, leading to a 34% increase in chondrocyte proliferation and endochondral ossification in the posterior condyle. This translated to a measurable increase in total mandibular length when combined with forward mandibular posturing devices."
Translation: HGH + mechanical forward posturing (functional appliances/orthotropics) creates actual skeletal length, provided the sutures are not fused by estradiol.
If you are within this window, the protocol is aggressive aromatase inhibition combined with systemic GH elevation to maximize chondrogenesis in the temporomandibular joint.
4. Early-to-Mid Adolescence Pharmacological Stack
Do not touch this if you are past Tanner Stage 5. This is strictly for keeping the structural window open and forcing condylar length.| Compound | Dosage | Mechanism of Action | Half-Life |
| Letrozole (Non-steroidal AI) | 1.25mg twice weekly | Competitive, reversible inhibition of the aromatase enzyme system. Crashes systemic E2, delaying sutural and epiphyseal fusion. | 48-72 hours |
| Somatropin (rhGH) | 2.0 - 4.0 IU daily | Binds to somatotropic receptors, stimulating hepatic and local MCC IGF-1 synthesis. | 0.4 hours (Systemic) |
| MK-677 (Ibutamoren) | 12.5mg - 25.0mg daily | Ghrelin receptor agonist. Cheaper alternative to rhGH, forces endogenous pulsatile GH release. | 24 hours |
Injectable rhGH (Somatropin) demands absolute clinical sterility and flawless cold-chain execution. Mishandling the peptide fractures the molecular structure, leaving you injecting a biologically inert amino acid soup with zero IGF-1 conversion.
Execution must be flawless.
1. Reconstitution Mathematics
Execution must be flawless.
1. Reconstitution Mathematics
- The Substrate: 10 IU (3.33mg) lyophilized Somatropin powder.
- The Solvent: Bacteriostatic Water (0.9% Benzyl Alcohol). Do not use standard sterile water for multi-dose vials.
- The Volumetric Ratio: Inject exactly 1.0mL of BAC water into the vial.
- The Yield Concentration: 10 IU per 1.0mL.
- The Syringe Calibration: A standard 2.0 IU dose equates to exactly 0.2mL (the '20' tick mark) on a 1cc U-100 insulin syringe.
- Temperature Hard-Limits: Lyophilized powder must remain refrigerated between 2°C–8°C at all times.
- Dalton Structure Preservation: Once reconstituted, the high-molecular-weight peptide bonds become incredibly fragile. Do not shake or agitate the vial. Swirl it with agonizing slowness.
- Shear Stress Avoidance: Draw the required dose slowly. Rapid vacuum extraction introduces shear stress, literally cleaving the peptide bonds before the liquid even breaches the needle.
- Hardware: 31G 5/16" U-100 insulin syringe. Heavier gauges cause unnecessary micro-trauma and waste expensive compound in the needle hub (dead space).
- Site Topography: Subcutaneous umbilical adipose tissue. Maintain a strict 2-inch radial exclusion zone from the navel to avoid the umbilical fascia.
- Execution Vector: Pinch the lipid layer to isolate the fat pad. Insert the needle perpendicularly at a perfect 90-degree angle.
- Deployment: Aspirate the plunger briefly to confirm you haven't breached a capillary bed. Depress the plunger over a slow, deliberate 5-second count to prevent subcutaneous pooling and localized site inflammation.
Crashed E2 destroys bone mineral density (BMD) over time and induces severe cognitive lethargy and joint desiccation. Drop the Letrozole immediately if you experience joint clicking in the TMJ, as synovial fluid depletion will actively grind down the condyle you are trying to grow. Monitor standing height and bi-iliac width; once vertical growth ceases for 6 months, the AI is useless and actively harmful to remaining aesthetic development.
5. The Late Adolescence & Adult Protocol: Surgical Reality
Once you hit 18-20, the condylar cartilage is replaced by dense bone. Growth is finished. Your SNB angle is locked. Soft tissue manipulation (fillers) at this stage is a dermal camouflage that stretches the facial envelope and creates a bloated, bottom-heavy lower third.You must alter the bone.
Intervention 1: The Sliding Genioplasty (SG)
SG isolates the symphysis menti. The surgeon makes an intraoral incision, exposes the bone, and performs an osteotomy below the apices of the lower incisors and the mental foramen. The chin segment is mobilized, advanced anteriorly (and vertically if needed), and fixed with titanium.Surgical Mechanics:
The cut must be made a minimum of 5.0mm below the mental foramen to preserve the inferior alveolar nerve (IAN) bundle.
Advancement maxes out around 8.0mm to 10.0mm. Beyond 10.0mm, you create a "step-off" deformity at the inferior border of the mandible, where the advanced segment disconnects wildly from the mandibular body, visible as an ugly notch through the skin.
Fixation requires 2.0mm titanium plates and monocortical screws.
Intervention 2: Bilateral Sagittal Split Osteotomy (BSSO)
If your chin is recessed because your entire lower jaw is recessed (Class II malocclusion), a genioplasty alone is an aesthetic botch. It will result in a hyper-projected chin button attached to a weak mandibular body.You require a BSSO. The mandible is split bilaterally down the ramus. The entire tooth-bearing segment is slid forward.
The Force Multiplier: Counter-Clockwise (CCW) Rotation.
Downswung maxillas (steep mandibular plane angle) are the primary cause of poor chin projection in mouth breathers. Moving the jaw straight forward (linear advancement) leaves the face looking long and horsey.
You must combine Le Fort I impaction with BSSO CCW rotation. The posterior maxilla is impacted (moved up into the skull), and the mandible rotates upward and forward on a hinge. This flattens the occlusal plane, dramatically projects the Pogonion, and shortens the vertical midface. This is the absolute S-Tier of structural modification.
Surgeons will push off-the-shelf silicone chin implants. Decline them.
Silicone is structurally inferior. It does not osseointegrate. Under the chronic tension of the mentalis muscle, the implant is driven backward into the symphysis menti. This causes severe cortical bone resorption. Over a 5-year period, a silicone implant can erode 3.0mm into your native bone, leaving you more recessed than when you started if you ever remove it.
If you must use an alloplastic implant instead of a genioplasty, use PEEK (Polyether ether ketone).
Silicone is structurally inferior. It does not osseointegrate. Under the chronic tension of the mentalis muscle, the implant is driven backward into the symphysis menti. This causes severe cortical bone resorption. Over a 5-year period, a silicone implant can erode 3.0mm into your native bone, leaving you more recessed than when you started if you ever remove it.
If you must use an alloplastic implant instead of a genioplasty, use PEEK (Polyether ether ketone).
- Obtain a CBCT (Cone Beam Computed Tomography) scan.
- The implant is CNC-milled to the exact topography of your symphysis.
- Fixation is absolute via titanium screws. No encapsulation, no shifting, zero bone erosion.
Intervention 3:
The Titanium Apex (Custom 3D-Printed Implants)
PEEK merely halts bone erosion. Custom 3D-printed titanium is the final escalation.
When your sagittal deficit aggressively eclipses the 10.0mm sliding genioplasty limit—that exact biomechanical threshold where advancing the mobilized segment creates a grotesque step-off deformity—you bypass native bone limitations entirely by bolting a mathematically perfect porous titanium matrix directly into the jaw. You extract a CBCT scan. An engineer dictates the precise geometric wrap-around.
The printed metal extends laterally along the mandibular body (masking the transition zone) and projects the symphysis forward with zero geometric restrictions.
Cortical bone cells grow directly into the porous metal. The implant becomes biologically indistinguishable from the skeleton.
Code:
Protocol Hard Limit Failure State
Silicone 0.0mm Cortical erosion
Genioplasty 10.0mm Step-off notch
Custom Titanium Infinite Surgical botch6. Sub-Tier Soft Tissue Camouflage (The Injectable Route)
If you lack the IQ or capital for orthognathic surgery, you are relegated to dermal fillers. This is renting your face.You must mimic the viscoelasticity (G-prime) of actual bone. Injecting highly hydrophilic, soft hyaluronic acid (like Juvederm Ultra) into the chin creates a soft, doughy, undefined mass.
The Compound:
Radiesse (Calcium Hydroxylapatite - CaHA).
CaHA has the highest G-prime available. It consists of calcium microspheres suspended in a carboxymethylcellulose gel carrier. It does not draw in water. It mimics structural bone and induces neocollagenesis.
The Mechanical Execution:
Use a 27G 1/2" needle.
The injection must be a supraperiosteal bolus.
Enter at the exact anterior point of the Pogonion. Push the needle directly through the dermis, fat, and muscle until you tap the absolute floor of the bone (the periosteum). Aspirate for 3 seconds. If clear, deploy a bolus of 0.2ml to 0.4ml of CaHA directly onto the bone.
Mold it immediately before the carrier gel sets.
7. Aesthetic Risk and Irreversible Botches
Failure to execute these protocols flawlessly results in permanent facial degradation.Surgical Botch: Permanent Lip Ptosis (Chimp-Lip)
During a genioplasty, the surgeon must strip the mentalis muscle off the bone to make the saw cut. If the surgeon fails to meticulously resuspend the mentalis muscle using non-resorbable sutures to the bone before closing the mucosa, the muscle retracts. The lower lip drops permanently. You expose your lower incisors at rest, drool, and adopt a vacant, low-IQ phenotype. This is nearly impossible to fix surgically once the muscle scars down in a retracted position.
Vascular Occlusion and Tissue Necrosis
If you botch a supraperiosteal Radiesse injection and deposit high G-prime CaHA into the submental artery, you cause immediate retrograde vascular occlusion. The skin on your chin will blanch white, then turn a mottled, bruised purple within 24 hours. Because CaHA is not reversible with Hyaluronidase, the tissue fed by that artery will literally rot, die, and slough off, leaving a permanent concave crater on your face.
The Witch Chin (Over-Projection)
Advancing a genioplasty beyond the STCA 0.0mm TVL limit creates a hyper-masculinized, crescent-moon profile. Combined with a deep labiomental fold, the soft tissue pad sags under its own projected weight.
8. Hard Stop Metrics & Washout
You do not run these protocols blindly.If running the endocrine stack (Ages 13-16), cease all Somatropin and Letrozole the moment a hand-wrist radiograph confirms total fusion of the radial epiphysis. At this point, the receptors in the MCC are fully downregulated and ossified. Further GH administration will exclusively thicken the orbital ridges (acromegaly) and widen the nasal alar base without adding a single millimeter to your sagittal chin projection. Wash out the Letrozole slowly over 3 weeks to prevent severe estrogen rebound and subsequent gynecomastia.
If consulting for surgery (Post-Puberty), your absolute hard stop is the cephalometric norm. If your SNB is currently 79 degrees and your Pogonion is at -2.0mm, do not let a scalpel touch your face. You are within standard deviations of optimal. Chasing a +2.0mm Pogonion projection past the TVL moves you from masculine to grotesque.
Read the sticky before replying. JFL if you ask about biotin, tongue chewing, or bone mashing. I'm out.
For more information, and softmaxxing (On chin-projection): https://looksmax.org/threads/megath...g-skeletal-maintenance.2204704/#post-30239005
@nordsmog @buccalfatremoval @Topkra @AscendHQ @Jenson @Regret. @blinkers @fagg @HexumReincarnated @nigga123
