P
psl6by2028
Iron
- Joined
- Jul 24, 2025
- Posts
- 40
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- 14
As you maybe seen from my previous post, I have subhuman hair genetics, causing recession and thinning at just age 18.
Now this is a bad situation, but I have a plan to ascend to cl in around 2/3 years and obviously hair is life/law, so I need to sort this out.
Currently I’ve been running topical finasteride (0.05%) and minoxidil (5%) 1ml twice a day for the past 7 months, and recently added 50mg RU58841 daily 3 weeks ago. I am currently losing/shedding around 100-200 hairs a day, so I need a better solution.
Now here’s the interesting part, I saw a post on here recently about using 2.5mg dutasteride to completely nuke DHT levels in the blood. That would likely halt my hair loss and even allow regrowth, but I personally believe DHT is still important post-puberty for mentality and general health.
To avoid these side effects, the post mentioned the use of proviron/mesterolone to effectively supplement the DHT. The key note is mesterolone has much lower binding affinity to androgen receptors than pure DHT, meaning RU58841 can be applied locally to the scalp and hopefully prevent mesterolone from damaging follicles, while still maintaining the mental/physical health benefits of DHT elsewhere in the body.
Of course, interfering with my body’s hormones is super risky, which is why I’m asking y’all for advice on whether this is a good idea and any potential sides?
I’ll need a method to gradually switch from my current stack to this new stack:
- continue current stack
- add in 0.5mg oral fin daily
- switch to 1mg oral fin daily
- add in 0.5mg oral dut every 3 days
- gradually increase to 0.5mg oral dut every 2 days then daily
- remove oral fin after 3 months
- start supplementing proviron/mesterolone and increase dut to 1.5mg daily
- gradually increase dut to 2.5mg daily
If anyone could review this transition plan and let me know how it could be improved that would be ideal. I’m trying to minimise the whiplashing of hormones as I introduce DHT blockers and proviron simultaneously.
Could the increased free test by preventing test -> DHT conversion lead to increased estrogen, in which case should I also take aromatase inhibitors ?
Now this is a bad situation, but I have a plan to ascend to cl in around 2/3 years and obviously hair is life/law, so I need to sort this out.
Currently I’ve been running topical finasteride (0.05%) and minoxidil (5%) 1ml twice a day for the past 7 months, and recently added 50mg RU58841 daily 3 weeks ago. I am currently losing/shedding around 100-200 hairs a day, so I need a better solution.
Now here’s the interesting part, I saw a post on here recently about using 2.5mg dutasteride to completely nuke DHT levels in the blood. That would likely halt my hair loss and even allow regrowth, but I personally believe DHT is still important post-puberty for mentality and general health.
To avoid these side effects, the post mentioned the use of proviron/mesterolone to effectively supplement the DHT. The key note is mesterolone has much lower binding affinity to androgen receptors than pure DHT, meaning RU58841 can be applied locally to the scalp and hopefully prevent mesterolone from damaging follicles, while still maintaining the mental/physical health benefits of DHT elsewhere in the body.
Of course, interfering with my body’s hormones is super risky, which is why I’m asking y’all for advice on whether this is a good idea and any potential sides?
I’ll need a method to gradually switch from my current stack to this new stack:
- continue current stack
- add in 0.5mg oral fin daily
- switch to 1mg oral fin daily
- add in 0.5mg oral dut every 3 days
- gradually increase to 0.5mg oral dut every 2 days then daily
- remove oral fin after 3 months
- start supplementing proviron/mesterolone and increase dut to 1.5mg daily
- gradually increase dut to 2.5mg daily
If anyone could review this transition plan and let me know how it could be improved that would be ideal. I’m trying to minimise the whiplashing of hormones as I introduce DHT blockers and proviron simultaneously.
Could the increased free test by preventing test -> DHT conversion lead to increased estrogen, in which case should I also take aromatase inhibitors ?
