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HOW TO SPEED UP BRAIN RECOVERY POST MDMA (MOLLY)(ECSTACY)*
Hello everyone. I hope after reading this you will gain some extra knowldge that will allow you to counter post molly effects effectively.
If you’ve ever experienced the crash a few days after using MDMA, you know it’s rough. You feel exhausted, flat, and just plain awful. It's a real biological effect. Molly forces your brain to dump a massive amount of its Serotonin (the key mood chemical) all at once. When it runs out, your system is in debt.
This thread will break down an advanced sciene backed method to help your brain heal faster and restore those depleted chemicals.
THE PROBLEM:
Serotonin debt and oxidative stressWhen you use molly, two things happen that cause the bad comedown:
Serotonin Overdraft: Your brain uses up its mood-boosting chemicals (5-HT) too quickly.
Cell Damage: The breakdown of the drug creates unstable, damaging molecules called Free Radicals. These can harm your brain cells (neurons) that store serotonin. This is the main concern we need to counter.
OUR GOAL:
Protect neurons (antioxidants) and give our brain the raw materials it needs to rebuild its serotonin supply later (replenishment).
SUPPLEMENTS:
This pair of supplements are the best for protecting brain cells and maximizing repair.
- Alpha-Lipoic Acid (ALA)
What it does:
ALA moves easily into the brain and cleans up those damaging Free Radicals (oxidative stress) right where they are causing trouble. Studies show it can completely prevent the serotonin drop and damage caused by MDMA in animals.
While you can take capsules, the injection route is superior because it delivers a massive, bioavailable dose straight into your system, bypassing digestion. This achieves much higher, faster concentrations for maximum defense.
Dose (Oral): 300 - 600mg immediately after, and then 100 - 150mg every 1-2 hours for the first 6-12 hours.
Injection Dose (recommended): A common dose would be 300 - 600mg in a single injection post-use.
- EGCG (Green Tea Extract)
What it does:
EGCG makes sure your serotonin building blocks go to the right place.It helps stop the breakdown of the 5-HT precursor outside the brain, ensuring more of it gets inside the brain where it can be converted into the 5-HT you need for mood.
Dose: 400 mg of EGCG, taken at the same time as 5-HTP (see below).
ADDITIONAL COMPONENTS
- 5-HTP* (recommended)
What it does:
5-HTP is the direct biological precursor to serotonin 5-HT. Your body takes 5-HTP and, using the cofactors like Vitamin B6, quickly converts it into the serotonin
Dose: 100-200 mg
- Vitamin B6 (P5P Form)
- ALCAR
ADDITIONAL COMPONENTS SUMMARY:
| Component | Dose Range | When to Take | Explanation |
| 5-HTP (The Serotonin Brick) | 100 - 200 mg | Starting Day 2 (24 hours AFTER the experience) | This is the direct building block your brain uses to make serotonin. SAFETY NOTE: NEVER take 5-HTP before or during the experience. |
| Magnesium (Glycinate/Threonate) | 400 mg | Before, During, and After | A necessary tool for serotonin creation. |
| Vitamin B6 (P5P Form) | 10 - 50 mg | With 5-HTP and EGCG | The other essential tool needed to convert 5-HTP into serotonin. The P5P form is the most active. |
| ALCAR | 500 - 1000 mg | Day 1 and Day 2 | Supports the cell's power factories (mitochondria), which are under stress after use. Helps with mental clarity. |
FINALLY - THE PLAN:
| Timing | Focus | Action |
| DURING | Protection | Hydrate with electrolytes, take Magnesium (400 mg). |
| Day 1 (Post-use) | Defense | ALA (Dose above), EGCG (400 mg), Magnesium, ALCAR. NO 5-HTP YET |
| Day 2-7 | Rebuild and Restore | 5-HTP (100 - 200mg every day) + EGCG (400mg every day), Vitamin B6. Continue Magnesium and good sleep. |
Sources + inspirations:
Acetyl-L-carnitine provides effective in vivo neuroprotection over 3,4-methylenedioximethamphetamine-induced mitochondrial neurotoxicity in the adolescent rat brain - PubMed
3,4-Methylenedioximethamphetamine (MDMA, ecstasy) is a worldwide abused stimulant drug, with persistent neurotoxic effects and high prevalence among adolescents. The massive release of 5-HT from pre-synaptic storage vesicles induced by MDMA followed by monoamine oxidase B (MAO-B) metabolism...
pubmed.ncbi.nlm.nih.gov
Alpha-lipoic acid prevents 3,4-methylenedioxy-methamphetamine (MDMA)-induced neurotoxicity - PubMed
A single administration of 3,4-methylenedioxymethamphetamine (MDMA, 20 mg/kg, i.p.), induced significant hyperthermia in rats and reduced 5-hydroxytryptamine (5-HT) content and [3H]paroxetine-labeled 5-HT transporter density in the frontal cortex, striatum and hippocampus by 40-60% 1 week later...
pubmed.ncbi.nlm.nih.gov
MDMA and 5-HT neurotoxicity: the empirical evidence for its adverse effects in humans – no need for translation - PMC
In this issue of the BJP, Green et al. suggest that animal data could not be used to predict the adverse effects of 3,4-methylenedioxymethamphetamine (MDMA) in humans and that MDMA did not produce 5-HT neurotoxicity in the human brain. This proposal ...
pmc.ncbi.nlm.nih.gov
Thanks for reading.
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