Roid Linear Growth

milkas

milkas

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I recently turned 15, and im starting a cycle this week.

The first 4 weeks will consist of following

Anavar 25mg daily
Proviron 25mg daily
(The only anchillaries i will be using here is 600mg NAC daily)



The next 10 weeks will consist of following
Anavar
25mg daily
Proviron 25mg daily
Test E 250mg weekly
Tren E 100mg weekly



Anchillaries i will be running alongside this
Telismartan
40mg daily
NAC 600mg daily
Accutane 20mg daily
Arimidex 0.5mg daily (Might bump up to 1mg, i already have nuked e2, so im kinda used to the mental sides)
Ezetimibe 10mg daily
Melatonin 3-6mg daily
Cabergoline 0.25mg weekly
Ketoconazole x2-3 a week

For Pct, i will be doing 20mg tamoxifen for 6 weeks (Hcg if tamoxifen isn't enough)
I may add Ru58841 or kx-826 if i have the budget, and if it is needed



My questions are

1.
Will this stack stunt my FAH, increase it, or remain the same? My e2 is nuked atm, and i plan on keeping it really low for a while now (May take a break after the cycle).

2. Will this age me horribly making me look like an uncanny goblin?

3. Would this be an unsafe cycle? I cant really take any bloodwork (Unless anyone knows a Diy company in the EU), so i want to minimize the risk of having to go to the hospital

Thanks for reading all ts, and appreciate it if you could rep or answer my questions:feelsautistic::heart:
 
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Reactions: juggingfent, TheSadAlbanian and Nerogen
I already know ts will get 0 replies and 5 views:feelswhy:
 
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Reactions: Nerogen
I recently turned 15, and im starting a cycle this week.

The first 4 weeks will consist of following

Anavar 25mg daily
Proviron 25mg daily
(The only anchillaries i will be using here is 600mg NAC daily)



The next 10 weeks will consist of following
Anavar
25mg daily
Proviron 25mg daily
Test E 250mg weekly
Tren E 100mg weekly



Anchillaries i will be running alongside this
Telismartan
40mg daily
NAC 600mg daily
Accutane 20mg daily
Arimidex 0.5mg daily (Might bump up to 1mg, i already have nuked e2, so im kinda used to the mental sides)
Ezetimibe 10mg daily
Melatonin 3-6mg daily
Cabergoline 0.25mg weekly
Ketoconazole x2-3 a week

For Pct, i will be doing 20mg tamoxifen for 6 weeks (Hcg if tamoxifen isn't enough)
I may add Ru58841 or kx-826 if i have the budget, and if it is needed



My questions are

1.
Will this stack stunt my FAH, increase it, or remain the same? My e2 is nuked atm, and i plan on keeping it really low for a while now (May take a break after the cycle).

2. Will this age me horribly making me look like an uncanny goblin?

3. Would this be an unsafe cycle? I cant really take any bloodwork (Unless anyone knows a Diy company in the EU), so i want to minimize the risk of having to go to the hospital

Thanks for reading all ts, and appreciate it if you could rep or answer my questions:feelsautistic::heart:
I dont kno much bout roids but why the First 4 weeks without test base
 
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Reactions: Nerogen
Take out var no need also higher tren and higher test and get on a ai
 
  • Hmm...
Reactions: cowmuncher26
Lower testosterone to max 70mg, otherwise you will stunt your FAH

manage your prolactin, also use Tamoxifen to degrade the estrogen receptor, paired with letrozole
 
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Test coming with the same order as the rest of the anchillaries and tren
 
Hold on I'm playing Roblox rn I'll explain why you shouldn't use high dose Test for height in a min
 
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So basically:

Estrogen accelerates growth plate senescense. High Test + an AI = nuked serum systemic e2, but not always nuked local e2 at the growth plates due to the androgen:estrogen ratio being lower in the growth plate tissue

What you'd say here is "but I'm using Aromasin to kill the aromatase enzyme!!!"

The growth plates have their own steroidgenesis system and they can synthesise estrogen completely from scratch (through cholesterol, not the aromatase enzyme) directly in the chondrocytes

@Nerogen
 
Last edited:
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So basically:

Estrogen accelerates growth plate senescense. High Test + an AI = nuked serum systemic e2, but not always nuked local e2 at the growth plates due to the androgen:estrogen ratio being lower in the growth plate tissue

What you'd say here is "but I'm using Aromasin to kill the aromatase enzyme!!!"

The growth plates have their own steroidgenesis system and they can synthesise estrogen completely from scratch (through cholesterol, not the aromatase enzyme) directly in the chondrocytes

@Nerogen
alright, ill probably just do 70-100mg then. do you think that would that put me at baseline test?
 
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alright, ill probably just do 70-100mg then. do you think that would that put me at baseline test?
It would put you at around an above average mark, but honestly I'd go down to 50mg or less. Me personally I'm not even using Test, just Tren Mast and Winstrol
 
It would put you at around an above average mark, but honestly I'd go down to 50mg or less. Me personally I'm not even using Test, just Tren Mast and Winstrol
Was thinking about Mast, but i wanted to focus more on muscle growth, and couldnt really afford both lol
 
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2. Will this age me horribly making me look like an uncanny goblin?

3. Would this be an unsafe cycle? I cant really take any bloodwork (Unless anyone knows a Diy company in the EU), so i want to minimize the risk of having to go to the hospital
Any answer to these btw?
@cowmuncher26
( question 3 is retarded, ik)
 
how tall r u btw
Last time I measured I was 5'11, got out of my second spurt 2 months ago. Running Erdafitinib, Trenbolone, Masteron and Winstrol may be adding KY19382 later on. Wanna reach 6'1+
 
I recently turned 15, and im starting a cycle this week.

The first 4 weeks will consist of following

Anavar 25mg daily
Proviron 25mg daily
(The only anchillaries i will be using here is 600mg NAC daily)



The next 10 weeks will consist of following
Anavar
25mg daily
Proviron 25mg daily
Test E 250mg weekly
Tren E 100mg weekly



Anchillaries i will be running alongside this
Telismartan
40mg daily
NAC 600mg daily
Accutane 20mg daily
Arimidex 0.5mg daily (Might bump up to 1mg, i already have nuked e2, so im kinda used to the mental sides)
Ezetimibe 10mg daily
Melatonin 3-6mg daily
Cabergoline 0.25mg weekly
Ketoconazole x2-3 a week

For Pct, i will be doing 20mg tamoxifen for 6 weeks (Hcg if tamoxifen isn't enough)
I may add Ru58841 or kx-826 if i have the budget, and if it is needed



My questions are

1.
Will this stack stunt my FAH, increase it, or remain the same? My e2 is nuked atm, and i plan on keeping it really low for a while now (May take a break after the cycle).

2. Will this age me horribly making me look like an uncanny goblin?

3. Would this be an unsafe cycle? I cant really take any bloodwork (Unless anyone knows a Diy company in the EU), so i want to minimize the risk of having to go to the hospital

Thanks for reading all ts, and appreciate it if you could rep or answer my questions:feelsautistic::heart:
Why do you want to pct?
Unless there is something forcing you to pct I would recommend to cruise on trt.
 
Any answer to these btw?
@cowmuncher26
( question 3 is retarded, ik)
2. Possibly, everyone reacts differently

3. I am also not getting bloodwork and I'm running 200 Tren
 
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Last time I measured I was 5'11, got out of my second spurt 2 months ago. Running Erdafitinib, Trenbolone, Masteron and Winstrol may be adding KY19382 later on. Wanna reach 6'1+
Alr alr, not too shabby for 15
 
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Reactions: cowmuncher26
Estrogen accelerates growth plate senescense. High Test + an AI = nuked serum systemic e2, but not always nuked local e2 at the growth plates due to the androgen:estrogen ratio being lower in the growth plate tissue

What you'd say here is "but I'm using Aromasin to kill the aromatase enzyme!!!"

The growth plates have their own steroidgenesis system and they can synthesise estrogen completely from scratch (through cholesterol, not the aromatase enzyme) directly in the chondrocytes
Thoguhts on this? @Anavarforthehuz69
 
So basically:

Estrogen accelerates growth plate senescense. High Test + an AI = nuked serum systemic e2, but not always nuked local e2 at the growth plates due to the androgen:estrogen ratio being lower in the growth plate tissue

What you'd say here is "but I'm using Aromasin to kill the aromatase enzyme!!!"

The growth plates have their own steroidgenesis system and they can synthesise estrogen completely from scratch (through cholesterol, not the aromatase enzyme) directly in the chondrocytes

@Nerogen
Here is what my fellow friend Grok had to say
(Please don’t dnr bhaiiii:Comfy:)


While it is true that growth plate chondrocytes express aromatase and can produce estrogen locally from androgen precursors, the claim that this local production is independent of the aromatase enzyme — or that growth plates can synthesize estrogen completely from scratch via cholesterol without aromatase — is not supported by the evidence.


The critical step in forming actual estrogen (estrone/estradiol) from C19 androgens is still the aromatase reaction. Systemic aromatase inhibitors significantly reduce both circulating and local estrogen signaling at the growth plate. This is precisely why aromatase inhibitors are used clinically in adolescent boys to slow bone age advancement and prolong growth potential.


In short: local estrogen production exists, but it is not “AI-proof.” Aggressively suppressing aromatase still meaningfully lowers estrogenic activity at the growth plates.


Supporting studies/references:


• Oz OK et al. (2001). Expression of aromatase in the human growth plate. Journal of Molecular Endocrinology.


• van der Eerden BCJ et al. (2002). Expression of estrogen receptors and enzymes involved in estrogen synthesis in the rat growth plate.


• Chagin AS et al. (2006). Locally produced estrogen promotes fetal rat metatarsal bone growth.


• Mauras N et al. studies on anastrozole in adolescent boys (showing delayed bone age progression with aromatase inhibition).


• Clinical reviews on aromatase inhibitors for height augmentation in adolescent males (demonstrating that systemic AIs affect growth plate maturation)
 
  • Hmm...
Reactions: cowmuncher26
Here is what my fellow friend Grok had to say
(Please don’t dnr bhaiiii:Comfy:)


While it is true that growth plate chondrocytes express aromatase and can produce estrogen locally from androgen precursors, the claim that this local production is independent of the aromatase enzyme — or that growth plates can synthesize estrogen completely from scratch via cholesterol without aromatase — is not supported by the evidence.


The critical step in forming actual estrogen (estrone/estradiol) from C19 androgens is still the aromatase reaction. Systemic aromatase inhibitors significantly reduce both circulating and local estrogen signaling at the growth plate. This is precisely why aromatase inhibitors are used clinically in adolescent boys to slow bone age advancement and prolong growth potential.


In short: local estrogen production exists, but it is not “AI-proof.” Aggressively suppressing aromatase still meaningfully lowers estrogenic activity at the growth plates.


Supporting studies/references:


• Oz OK et al. (2001). Expression of aromatase in the human growth plate. Journal of Molecular Endocrinology.


• van der Eerden BCJ et al. (2002). Expression of estrogen receptors and enzymes involved in estrogen synthesis in the rat growth plate.


• Chagin AS et al. (2006). Locally produced estrogen promotes fetal rat metatarsal bone growth.


• Mauras N et al. studies on anastrozole in adolescent boys (showing delayed bone age progression with aromatase inhibition).


• Clinical reviews on aromatase inhibitors for height augmentation in adolescent males (demonstrating that systemic AIs affect growth plate maturation)
@cowmuncher26
Any opinions abt this?
 
  • Hmm...
Reactions: cowmuncher26
@cowmuncher26
Any opinions abt this?
Bradar, answer my question saar. Just tell me if I’m a little faggot retard jfl

But seriously tho. Ik no Ai that stops 100% of the aromatization, but wouldn’t that apply to all doses of test?
 

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