Sachlichkeit
Bronze
- Joined
- May 11, 2025
- Posts
- 375
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inject testosterone >>> T converts to E >> E give you man tits.
People run aromatase inhibitors to block the conversion of testosterone into estrogen during cycle to "normalize" their estrogen levels while keeping testosterone at steroid tier
AI's are also used to prevent growth plates from closing we already know this
estrogen is NECESSARY for normal function in like 10 separate significant ways we can't eradicate it from our body completely.
The selective estrogen receptor modulator RALOXIFENE is a drug intended for post menopausal women (I think) and has some unique properties.
First of all, in animal studies administration of raloxifene retarded longitudinal growth in animals with open growth plates. This is bad, but compared to estrogen which completely signals the ending of longitudinal growth its actually better than estrogen. I would go as far to say as its effects on plates are negligible when compared to estrogen
Low estrogen = cardiovascular risk
Low estrogen = Neurocognitive issues
Low estrogen = poor bone & joint health
Raloxifene acts on all these pathways. Is weaker than estrogen, but preferable if you plan on having very low E2 as a form of "synthetic estrogen," as it competes for estrogen receptors in breast tissue making it an anti-gynecomastia drug while simultaneously providing estrogenic benefits in the areas we want
People run aromatase inhibitors to block the conversion of testosterone into estrogen during cycle to "normalize" their estrogen levels while keeping testosterone at steroid tier
AI's are also used to prevent growth plates from closing we already know this
estrogen is NECESSARY for normal function in like 10 separate significant ways we can't eradicate it from our body completely.
The selective estrogen receptor modulator RALOXIFENE is a drug intended for post menopausal women (I think) and has some unique properties.
First of all, in animal studies administration of raloxifene retarded longitudinal growth in animals with open growth plates. This is bad, but compared to estrogen which completely signals the ending of longitudinal growth its actually better than estrogen. I would go as far to say as its effects on plates are negligible when compared to estrogen
Low estrogen = cardiovascular risk
Low estrogen = Neurocognitive issues
Low estrogen = poor bone & joint health
Raloxifene acts on all these pathways. Is weaker than estrogen, but preferable if you plan on having very low E2 as a form of "synthetic estrogen," as it competes for estrogen receptors in breast tissue making it an anti-gynecomastia drug while simultaneously providing estrogenic benefits in the areas we want
