Stack for Height and Dimo - GTFIH

What's funny is edgy roiders call anything under like half a gram a "pussy dose" as if natty test isn't around 10x lower than that:lul::lul:

Yes test effects aren't linear but having even 5x more test has to have some noticeable roi. I don't know why people clown it
Most of them look like this.
1781870135074
 
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which ai do you prefer?
Letrozole ideally/theoretically

But I have not used it to give an anecdotal argument for it, though of course my lovely friends on here have
 
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:PepeLaugh::PepeLaugh::PepeLaugh:Roids are shit for the most part, gh will do most of your work

It's so funny seeing this new cope about rhGH "depleting" chondroctyes (they don't even know what that means nor does it make sense) but sometimes you'll have the more sophisticated retards that will specifically say resting zone chondrocytes get depleted.

Still retarded and don't know what they're talking about

There will always be some cope about rhGH because it's been the standard.. Humans like to go against the grain just as much as they like to fit in:EZclap:
Well said.
 
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Letrozole ideally/theoretically

But I have not used it to give an anecdotal argument for it, though of course my lovely friends on here have
Its fucking shit mentally.
 
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Letrozole ideally/theoretically

But I have not used it to give an anecdotal argument for it, though of course my lovely friends on here have
i will be using aromasin:Bruh: 350 test e 70 tren a 4iu hgh:Bruh::Bruh::Bruh::Bites:
 
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IGF-1/GH Axis:
rHGH - 16 ius ed

Androgens:
Test Base - 5 mg ed
Primo - 20 mg ed
Oxandrolone - 10 mg (morning/pre)
Fluoxymesterone - 2.5 mg ed (21 days on 7 off)
Tren - 30 mg pw microdose

E2:
Exemestane - 25 mg eod
Letrozole - 1.25 mg eod
Tamoxifen/Nolvadex - 20 mg ed

FGFR Axis:
Balversa - 3 mg ed (14 days on 7 off)

Skin and Hair:
RU58841 - 20-50 mg per day (5% solution)
Minox - 2.5 mg ed
Isotret - 20mg ed (incase of acne outbreak)
Asthaxanthin - 8 mg ed
GHK-cu - 2 mg ed

Ancillaries:
Empagliflozin
T3
T4
Sevelamer Carbonate
High EPA/DHA Fish Oil
Melatonin
Telmi
Cabergoline
P5P
Aspirin
NAC
TUDCA
Taurine
Lutein
Zeaxanthin
Plus Tons of other shit I will not mention

This will be my second blast as my current first cycle is a lot milder. Any advice is appreciated. :feelswhy:
Good stack. What's that balversa thing I've never heard of it. Is it another pan fgfr3 inhibitor?
 
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:PepeLaugh::PepeLaugh::PepeLaugh:Roids are shit for the most part, gh will do most of your work

It's so funny seeing this new cope about rhGH "depleting" chondroctyes (they don't even know what that means nor does it make sense) but sometimes you'll have the more sophisticated retards that will specifically say resting zone chondrocytes get depleted.

Still retarded and don't know what they're talking about

There will always be some cope about rhGH because it's been the standard.. Humans like to go against the grain just as much as they like to fit in:EZclap:
LMAO some retard on discord was just reiterating that hgh makes u shorter in higher doses bec is depletes resting zone and actiavtes cell sensence:feelskek::feelskek:

he later pulled a gpt screenshot aka undeniable proof!!! and understood it was natural selection:feelstastyman:
 
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Maybe its necessary for the roids not really into that but why are you taking two ais?
 
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LMAO some retard on discord was just reiterating that hgh makes u shorter in higher doses bec is depletes resting zone and actiavtes cell sensence:feelskek::feelskek:

he later pulled a gpt screenshot aka undeniable proof!!! and understood it was natural selection:feelstastyman:
LOL yeah this one is a new one I've started to come across in the last week, I had an extensive debate on here with someone about it

He never ended up responding after getting raped..:feelsbadman:
 
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Maybe its necessary for the roids not really into that but why are you taking two ais?
My family fucking loves to travel meaning we cross a lot of borders for some reason and before I started letro I used exemestane to permanently delete AEs (suicidal) so that I won't have a huge systemic rebound and I use letro bcs its just better.
 
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LOL yeah this one is a new one I've started to come across in the last week, I had an extensive debate on here with someone about it

He never ended up responding after getting raped..:feelsbadman:
That one nigha Ahmed smth
 
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That one nigha Ahmed smth
No I don't have the energy to argue with ahmed, I don't think he'd ever stop

He's been going back and fourth with zagro for months that's way too much time and energy

I can also admit when I've lost an intellectual discourse, so if I know I won I'm not gonna back down. Too much effort, just be mature and go your own ways lol
 
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My family fucking loves to cross borders for ome reason and before I started letro I used exemestane to permanently delete AEs (suicidal) so that I won't have a huge systemic rebound and I use letro bcs its just better.
bro added a fun fact:lul:
 
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No I don't have the energy to argue with ahmed, I don't think he'd ever stop

He's been going back and fourth with zagro for months that's way too much time and energy

I can also admit when I've lost an intellectual discourse, so if I know I won I'm not gonna back down. Too much effort, just be mature and go your own ways lol
I tried to argue with that nigha once I gave up after 2 replies :feelsohh:
 
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I tried to argue with that nigha once I gave up after 2 replies :feelsohh:
no point, he's too committed

Cool guy though, that's the homie
 
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L stack for dimo might asw pin estrogen and turn into a female
 
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Care to explain?
Sure. Eyesight side effects aren't really easy to prevent plus some patients died of a heart attack due Erda idrk the mechanism behind that but another thing is that the cartilage in tue growth plates won't turn into actual hard bone on erda because it inhibits fgfr1 which is needed for cartilage to turn into bone. Switch erda for something like aboloparatide bruh it's way better
 
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Sure. Eyesight side effects aren't really easy to prevent plus some patients died of a heart attack due Erda idrk the mechanism behind that but another thing is that the cartilage in tue growth plates won't turn into actual hard bone on erda because it inhibits fgfr1 which is needed for cartilage to turn into bone. Switch erda for something like aboloparatide bruh it's way better
Im thinking you mean Retinal fluid build up and then the retina detaching? This can be avoided through cycling mostly plus Zeaxanthin + Lutein. Heart attack is uncommon. You are reffering to the THOR study and BLC2001. Cardiac symptoms occurred in patients with advanced urothelial cancer, older and heavily pretreated patients. so some deaths could be related to the underlying disease instead of erda itself. Plus im willing to take the risk of dying my life sucks anyway. Where did you learn that the cartilage won't convert into bone due to FGFR1 Inhibiton?
 
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