mats.v
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Steroid users GTFIH
listed below are all the major and minor(which lead to major), and most common cardiovascular issues associated with ANABOLIC ANDROGENIC STEROID USE.
WHY?
THIS IS WHYYYY you niggers should be careful and stay alive when pinning or orally investing anabolics. Through understanding we can better avoid fatalities and dysfunction. Many have died suddenly from steroid use from cardiac events which show no symptoms. Getting labs alone doesn’t guarentee you won’t suddenly collapse (and die) from dilative cardiomyopathy enduced heart failure or arrhythmia..
how does the heart work?
The main map of spots to remember is below:
Blue deoxygenated blood enters the superior vena cava, to the right atrium, travels through the tricuspid valve.. through the right ventricle… and enters the pulmonary artery, to reach and receive oxygen from the respiratory(lungs) system. The red oxygenated blood then enters the pulmonary vain, enters the left atrium, and passes the mitral valve to enter the left ventricle. The left ventricle then flows blood to the aorta to be released into the blood vessels and travel through the body. Steroids affect this process.
If this process fails you run into highly fatal problems.
blood is the life of the body.
The heart is made of three tissues:
Endocadium(inner layer), myocardium, and pericardium(outer)…
Its functions consist of structural and electrical processes.
The filling of blood is referred to as diastole(DIE-Ass-Tow-Lee). Done in the right atrium and ventricle.
And the contraction/pumping of blood is systole(sis-tow-lee). Done after blood is oxygenated. from the left atrium and finally to release from the aorta.
Most steroid damage is done in the myocardium(mid, muscular) and eventually pericardium(outer) layers.
STRUCTURAL PROBLEMS
ISSUES IN THE MYOCARDIUM/Heart structure:
VASCULAR AND ISCHEMIC DISEASE
No 200mG of testosterone will not do this. But if your genetics are fucked it really can.. most people experience a risk of encountering these issues on higher androgen levels. Many of these problems like cardiomyopathy or arrhythmia develop by genetic factors alone and show no signs on blood markers, or blood pressure until it’s too late.
Stay in tune for the next thread follow up:
“How to avoid these cardiovascular issues and take steroids so you can mog and slay low tier bitches.. (‘Cause even muscles cannot defeat hypergamy).”
listed below are all the major and minor(which lead to major), and most common cardiovascular issues associated with ANABOLIC ANDROGENIC STEROID USE.
WHY?
THIS IS WHYYYY you niggers should be careful and stay alive when pinning or orally investing anabolics. Through understanding we can better avoid fatalities and dysfunction. Many have died suddenly from steroid use from cardiac events which show no symptoms. Getting labs alone doesn’t guarentee you won’t suddenly collapse (and die) from dilative cardiomyopathy enduced heart failure or arrhythmia..
how does the heart work?
The main map of spots to remember is below:
Blue deoxygenated blood enters the superior vena cava, to the right atrium, travels through the tricuspid valve.. through the right ventricle… and enters the pulmonary artery, to reach and receive oxygen from the respiratory(lungs) system. The red oxygenated blood then enters the pulmonary vain, enters the left atrium, and passes the mitral valve to enter the left ventricle. The left ventricle then flows blood to the aorta to be released into the blood vessels and travel through the body. Steroids affect this process.
If this process fails you run into highly fatal problems.
blood is the life of the body.
The heart is made of three tissues:
Endocadium(inner layer), myocardium, and pericardium(outer)…
Its functions consist of structural and electrical processes.
The filling of blood is referred to as diastole(DIE-Ass-Tow-Lee). Done in the right atrium and ventricle.
And the contraction/pumping of blood is systole(sis-tow-lee). Done after blood is oxygenated. from the left atrium and finally to release from the aorta.
Most steroid damage is done in the myocardium(mid, muscular) and eventually pericardium(outer) layers.
STRUCTURAL PROBLEMS
ISSUES IN THE MYOCARDIUM/Heart structure:
- Cardiomyopathy. Weakening or largening of heart muscle(ventricle). Heart failure, arrhythmia(electrical dysfunction) as result.. sudden death likely. Those who carry genetic predisposition can develop dilative cardiomyopathy. This type of cardiomyopathy leads to the increased ability of diastole and reduced function of Systole..(meaning the heart can fill with blood well due to dilated left ventricle and atrium.. however pumping blood is significantly hindered due to reduced ability to pump blood
- Left ventricular hypertrophy. Androgen receptor stimulation in the left ventricle. The ventricle thickens in the heart cannot properly carry out systole function, nor diastole function. increases the risk of heart failure, arrhythmias (irregular heartbeats), ischemic heart disease, and sudden cardiac arrest.. myocardial fibrosis as well.
- Formation of scar tissue within the heart muscle, often occurring alongside left ventricular hypertrophy in chronic anabolic steroid users. This can disrupt electrical conduction and mechanical function, increasing the risk of arrhythmias, heart failure, and sudden cardiac death.
VASCULAR AND ISCHEMIC DISEASE
- Arteriosclerosis: the "hardening" or thickening of the arterial walls.. leads to plaque formation: atherosclerosis:
- Atherosclerosis. A type of arteriosclerosis. Example: coronary artery disease. Excess plaque buildup via increased LDL and reduced HDL in the bloodstream… also known as atherogenic dislipedemia.. the hepatic lipase enzyme is upregulated due to high testosterone levels, producing higher levels of low density lipoprotein, and reducing the level of high density Lipo proteins. High ldl in the bloodstream leads to plaque buildup in artery walls. LDL is filtered by HDL.. which is already decreased. Atherosclerosis is caused by blockage of plaque. results can be fatal due to blockage in arterial flow of blood.
- Myocardial infarction.. (heart attack).. due to atherosclerosis plaque rupture.. severe coronary artery spasm.. or sudden clot formation(also from atherosclerosis, or thick blood)… Clot formations are also known as thromboembolism(more on this later)
- Hypertension. Red blood cell hematocrit, increase..thicker volume leading to the increase of contraction forced to pump blood through already constricted vessels. Strain on the heart especially on the ventricle. The result is arteriosclerosis,atherosclerosis, left ventricular hypertrophy, & heart failure
- Endothelial dysfunction is damage to inner lining of vessels, also known as the endocardium. the result is hypertension, stiffness, vascular resistance, myocardial injury, and adverse remodeling.. can result in cardiomyopathy, heart failure from atherosclerosis, coronary disease,… thromboembolisms, and ischemia… myocardial infarction arrhythmias, and fibrosis of myocardium… arterial fibrosis also result of endothelial dysfunction
- Arterial fibrosis: thickening of artery walls leads to thromboembolisms and sudden cardiac death.. broader terminology; Vascular fibrosis due to increase scar tissue and thickening of blood vessels; thus vasoconstriction in an already stressed environment, such as polycythemia. Upregulation of testosterone induced androgen receptor activity in blood vessels enables oxidative stress and thus thickening of tissue.
- Thromboembolism: (blood clots) like deep vein thrombosis… thromboembolism can cause stroke. And spike aneurysm rupture.(nocturnalKent reference) or pulmonary embolisms… clots are usually caused by arterial fibrosis, polycythemia or atherosclerosis.
- Polycythemia: access erythropoietin production due to high testosterone levels erythropoietin directly stimulates red blood cell production in the bone marrow increase. High red blood cell count levels will lead to thicker blood leading.. to hypertension, increased thromboembolism risk , and thus fatal cardiac events..
- Arrhythmia. Electrical system dysfunction, life-threatening heartbeats, heartbeat irregularity. Leads to problems such as atrial fibrillation, ventricular fibrillation.. Ultimtaley resulting in increased risk of sudden cardiac death. Can be caused by cardiomyopathies. DNR my niggas
No 200mG of testosterone will not do this. But if your genetics are fucked it really can.. most people experience a risk of encountering these issues on higher androgen levels. Many of these problems like cardiomyopathy or arrhythmia develop by genetic factors alone and show no signs on blood markers, or blood pressure until it’s too late.
Stay in tune for the next thread follow up:
“How to avoid these cardiovascular issues and take steroids so you can mog and slay low tier bitches.. (‘Cause even muscles cannot defeat hypergamy).”
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