THE COMPLETE GUIDE TO IQMAXXING (actual, tangible methods)

[kantlie]

The Complete Guide to IQMaxxing

​

Why does IQ matter?

So that people don't ask retarded questions like that. IQ is the glue that pulls together any potential form of human interaction with the reality in which we exist so long as one's goal in it is prosperity in a domain to any measurable extent; you will always rely on intelligence to some extent, and if you want to be successful to whatever that extent may be you need to make it such that the extent is invariant relative to your intelligence.

In this guide I'm going to go over the process of the maximization of intelligence through a few layers that contribute to intellectual development and accentuation of cognitive ability throughout one's lifetime:

1. The stabilization layer: this would be your supplements, basic nootropics, and whatever other substances that can be bought in stores or ordered online that will aid in long-term cognitive development.
2. The active layer: this is going to contain that with which neuroplasticity engulfs the necessary thinking models for you to actually comprehend, process, and make connections with information faster and more productively. This will include the most evidence-based methods we have to date for the increase of IQ (citations will be included),
3. The neurochemical alteration layer: this layer will serve as the means through which the training methods will actually be consolidated in the mind and engrained into one's cognitive apparatus such that it becomes second nature and utility from each respective training method can be maximized. This will be your experimental pharmaceuticals and research chems that fags fear-monger about.
4. The inhibition layer: this layer will address any potential on-going physiological problems that could be halting the full expression of your baseline intelligence, which you may not be aware of. I'm going to focus on breathing and its significance since it is often overlooked and is the very urelement of human cognition; if you can't breathe, you can't think. As a result this will be concentrating on sleep apnea and kinds of nasal obstruction that could be inhibiting your brain from ensuring maximum oxygen intake.
Clarifying the science

1. How the Brain Uses Neuroplasticity

Neuroplasticity is the brain’s ability to reorganize its structure and function in response to experience, learning, or injury. It happens at multiple levels:

• Synaptic plasticity: Strengthening or weakening of existing synapses (connections between neurons). Key mechanism: Long-Term Potentiation (LTP) — repeated co-activation of neurons makes the synapse stronger (“cells that fire together wire together”).
• Synaptogenesis: Formation of entirely new synapses.
• Dendritic spine changes: Neurons grow more spines (receptors) to receive inputs.
• Structural changes: New neurons (neurogenesis, mainly in hippocampus) and rewiring of networks.

This allows skills, knowledge, and habits to become encoded in neural circuits.

2. How the Brain Consolidates Information

Consolidation turns fragile short-term memory into stable long-term memory:

• Encoding (hippocampus + prefrontal cortex): New info creates temporary neural patterns during learning.
• System consolidation: Over hours to weeks (especially during deep sleep and quiet wakefulness), the hippocampus “replays” the activity patterns. This transfers the memory to the neocortex for permanent storage.
• Synaptic consolidation: Requires protein synthesis (e.g., via BDNF signaling) to stabilize new/enhanced synapses within hours of learning.
• Myelination and network integration make the skill faster/automatic (“second nature”).

Repeated practice + sleep + focused attention = stronger, more efficient circuits. Without consolidation, new info fades.

3. Role of Synaptogenic & BDNF-Upregulating Compounds

Compounds that boost BDNF (Brain-Derived Neurotrophic Factor) or promote synaptogenesis amplify these natural processes:

• BDNF is a key neurotrophin. It:
  
  • Enhances LTP (makes learning stickier).
  • Promotes growth of new dendrites/spines.
  • Supports neuron survival and neurogenesis.
  • Facilitates the protein synthesis needed for long-term memory.
• Synaptogenic agents (e.g., those acting on HGF/c-Met like Dihexa) directly increase synapse density and dendritic complexity, creating more “hardware” for new information to wire into.

Neurochemically:

• During/after training (RFT, Dual N-Back, logic, skills), these compounds raise BDNF signaling → stronger LTP at active synapses.
• They lower the threshold for new synapse formation, so repeated practice builds denser, more interconnected circuits faster.
• Enhanced hippocampal-cortical dialogue during sleep consolidates the changes more efficiently.
• Result: Faster transition from effortful (prefrontal-heavy) to automatic (basal ganglia + cortical networks) performance.

Example in Practice (Making Skills Second Nature):Learning mathematical logic or relational frames initially feels effortful. With elevated BDNF/synaptogenesis:

• More synapses form between relevant neurons during sessions.
• Consolidation during sleep engrains the patterns deeper.
• Over weeks, the neural pathway becomes highly myelinated and efficient → you intuitively “see” logical structures or relational patterns without conscious effort (second nature/automaticity).

The Stabilization Layer (supplements)

BDNF / Neurotrophic Signaling​

Lion's Mane — stimulates NGF and BDNF synthesis directly; crosses BBB
Omega-3 (EPA/DHA) — DHA upregulates BDNF gene expression, reduces neuroinflammation
Curcumin — raises BDNF via histone acetylation (epigenetic upregulation)
Resveratrol — activates SIRT1 → downstream BDNF/CREB signaling
Exercise — strongest known driver of acute and chronic BDNF release (not a supplement, but the benchmark)

Synaptogenesis & Membrane / Cholinergic Support​

Citicoline — supplies choline + cytidine for phosphatidylcholine (synaptic membrane building block); boosts acetylcholine
Alpha-GPC — choline donor for acetylcholine synthesis and membrane phospholipids
Phosphatidylserine — structural phospholipid concentrated at synapses; supports membrane fluidity
DHA — incorporates into synaptic membranes, affecting fluidity and receptor/ion channel function
Bacopa monnieri — promotes hippocampal dendritic branching; modulates acetylcholinesterase

Neurogenesis​

Lion's Mane — NGF/BDNF pathway drives hippocampal neurogenesis (animal-confirmed)
Flavonoids (blueberry, cocoa, EGCG) — cross BBB, activate CREB/BDNF cascades tied to neurogenesis
Omega-3 — DHA supports neural stem cell membrane integrity during differentiation

Neuroprotection / Antioxidant​

NAC — precursor to glutathione, the brain's main endogenous antioxidant
Vitamin E — protects membrane fats (like DHA) from oxidative damage
Sulforaphane — activates Nrf2, the master regulator of antioxidant enzyme production
Alpha-lipoic acid — regenerates other antioxidants (C, E, glutathione); supports mitochondrial enzymes

Mitochondrial / Energy Support​

CoQ10 — core component of the mitochondrial electron transport chain (ATP production)
PQQ — stimulates mitochondrial biogenesis (new mitochondria formation)
Creatine — buffers ATP/phosphocreatine reserves for high cognitive load

Vascular / Neurovascular Support​

Ginkgo biloba — improves cerebral microcirculation; antioxidant/platelet effects
Nitrate/beetroot extract — converts to nitric oxide → vasodilation → more cerebral blood flow and oxygen deliver

The Active Layer (RFT, N-back, Image streaming)

Dual N-Back​

The task forces continuous updating of working memory contents — holding a stimulus, comparing it to one from N steps back, then discarding it as the next one arrives. You're not just holding items in your mind, you're actively overwriting and refreshing them under load. The dual-stream format (spatial position and auditory letter simultaneously) adds a divided-attention demand on top of the updating demand, recruiting both the dorsolateral prefrontal cortex (executive control, updating) and posterior parietal cortex (spatial/attentional buffer) more intensively than single-stream tasks. As you get used to a particular combinatorial setting, you can increase the difficulty and on some websites even add additional stimuli like photos, 3 dimensions, etc.


Evidence for memory increase:


Melby-Lervåg & Hulme's meta-analysis found solid transfer to verbal working memory (d ≈ 0.79) and spatial working memory (d ≈ 0.52) tasks — the strongest, most consistent finding in the whole n-back literature, well above the shaky far-transfer-to-IQ numbers.


A 2017 multi-level meta-analysis (33 RCTs, 203 effect sizes) found a medium-sized transfer effect to untrained n-back tasks specifically, with smaller effects for other working memory measures — gains are strongest on tasks structurally similar to n-back itself and taper off the further you get from that structure.


Several individual RCTs report improved performance on digit span and reading span, standard validated working-memory measures, following dual n-back training, consistent with the meta-analytic pattern above.


A 2021 study comparing dual n-back against the method of loci found dual n-back produced superior transfer effects on working memory measures like digit span and change detection, suggesting the updating-specific training does something a general mnemonic technique doesn't.


Overall, the N-back method protocol isn't for the consolidation of the methods by which your mind processes information and uses systems to make critical analyses of it (the theoretical foundation for RFT), so the point isn't IQ increase, something for which N-back doesn't have much evidence. However, it clearly has strong evidence for the improvement of one's short-term memory which is incredibly helpful in a wide variety of cognitive tasks and would not hurt to have in one's arsenal.

Note: If it matters at all, despite there not being conclusive scientific evidence for N-back resulting in IQ increase, many people have reported standard deviations worth of increase on their IQ tests before and after the administration of N-back training, so make of that what you wish.

---

Relational Frame Training (RFT)​

RFT's core claim is that derived relational responding — the ability to infer relationships you were never directly taught, such as if A=B and B=C then A=C, or if A is opposite to B and B is opposite to C then A=C — is the basic operant unit underlying verbal, analytical intelligence itself, not just a correlate of it. RFT drills this directly through multiple exemplar training: repeated practice deriving relations like same as, opposite of, more or less than, before or after, across many different stimulus sets, until the pattern of relational responding generalizes to entirely novel stimuli the person has never seen. This is mechanistically different from n-back: n-back trains a specific executive process and hopes it transfers, while RFT trains the actual operation that IQ test items like analogies, matrices, and syllogisms are built from. The theoretical distance between trained skill and tested skill is much shorter, which is the a priori reason to expect better transfer.


Foundational work by D. Barnes-Holmes and colleagues established that this kind of relational fluency is trainable and generalizable even in children who initially lacked it, meaning the underlying skill isn't fixed but is a learned repertoire.


Evidence for IQ increase:


Cassidy, Roche & Hayes's pilot study found children given stimulus equivalence training followed by relational frame training on SAME, OPPOSITE, MORE THAN, and LESS THAN showed significant full-scale WISC IQ gains after each stage, versus a no-treatment control.


Cassidy, Roche, Colbert & Grey, published in Learning and Individual Differences, found online SMART training produced average IQ increases of roughly 28 points, alongside gains in verbal, numerical, and educational aptitude, using the Wechsler scales as the outcome measure.


A correlational study using the Relational Abilities Index, a 55-item relational-puzzle assessment, found meaningful correlations between relational skill and full-scale IQ, the National Adult Reading Test, and other standard cognitive measures, supporting the theoretical link independent of the intervention effect.

This is the most promising means by which IQ and its underlying mechanistic foundations can be amplified and increased, and the one for which we have the most amount of evidence. However, the studies haven't been widely replicated and it's a very niche area of research, but nevertheless very promising. Also take into account that the younger you are the more effective this is going to be.

---

Image Streaming​

Wenger's model holds that closing your eyes and continuously narrating spontaneous mental imagery out loud forces simultaneous engagement of the visual/imagery system and the verbal articulation system, which he argues strengthens the neural connections between them, improving both visualization vividness and the speed and fluency of translating internal representations into language.
The idea is that continuous real-time narration under time pressure, describing the image as it's happening rather than after, trains rapid lexical retrieval and sentence construction, similar to how simultaneous interpretation trains fluency in interpreters.


Evidence for IQ increase:


Wenger's own claim, repeated across his self-published materials, is that roughly 25 hours of practice produces meaningful gains in IQ points.


I was unable to find any independent, peer-reviewed, controlled study measuring image streaming's effect on IQ, verbal fluency, or visualization ability. All available claims trace back to some niche, un-replicated studies with questionable protocols of research conduct and anecdotal or testimonial reports. Nevertheless I wanted to included because I've seen personal success with it in treating aphantasia (the inability to see in your mind's eye) and so have an entire dedicated community of people.


The Neurochemical Alteration Layer (research chems and experimental pharma)


This is the section dedicated to the compounds that alter your neurochemical architecture such that the absorption and maturation of information and the gains produced by the mentioned methods for cognitive development can be catalyzed and accelerated through drugs, shit that actually works. You can moral-fag and talk about the efficacy of these drugs but I'm not going to wait a life-time to get confirmation on whether shit that clearly functions is actually efficacious or not.

Neuroplasticity, synaptogenesis, BDNF/NGF signaling, neurogenesis, LTP, and neuroprotection. These compounds are designed to maximize the return on your RFT/SMART, Dual N-Back, mathematical logic, and Image Streaming training by making new neural connections denser, stronger, and more permanent. Compounds:

• Dihexa — Extremely potent HGF/c-Met agonist. Preclinical data suggests massive synaptogenesis (claimed 10 million times more potent than BDNF). Strong memory rescue in impaired rodent models.
• Cerebrolysin — Porcine brain-derived neurotrophic mix (BDNF, NGF, GDNF etc.). Most clinically studied with multiple RCTs/meta-analyses in stroke, TBI, and dementia.
• Semax (including N-Acetyl Semax Amidate / NASA) — ACTH(4-10) analog. Potent BDNF/TrkB upregulation. Russian-approved for stroke and cognitive issues.
• Selank (including N-Acetyl Selank) — Tuftsin analog. Anxiolytic + BDNF modulation. Russian-approved.
• Cortexin — Russian peptide bioregulator used for neuroprotection and cognitive recovery.
• Noopept — Synthetic dipeptide. Increases NGF, BDNF, and supports memory consolidation.
• Pinealon — Synthetic tripeptide bioregulator targeting brain tissue.
• Cortagen — Synthetic tetrapeptide for cortical repair and neuroprotection.
• Humanin — Mitochondrial-derived peptide with neuroprotective and cognitive effects.
• NSI-189 — Hippocampal neurogenesis promoter. Early-phase depression/cognition trials.
• P21 / CNTF analog — Mimics ciliary neurotrophic factor for neurogenesis.

Racetams & Related Ampakines:

• Piracetam, Phenylpiracetam, Aniracetam, Oxiracetam, Pramiracetam, Fasoracetam, Coluracetam, Nefiracetam.

Cholinergics & Acetylcholinesterase Inhibitors:

• Donepezil, Galantamine, Rivastigmine, Alpha-GPC, Citicoline (CDP-Choline), Huperzine A.

Dopaminergic & Stimulant Agents:

• Bromantane (dopamine synthesis + BDNF upregulation).
• Selegiline (low-dose MAO-B inhibitor).
• Modafinil / Armodafinil.
• Adderall / Dexedrine / Methylphenidate (prescribed).
• 9-Me-BC (dopamine neuron regeneration).
• PRL-8-53 (memory-specific nootropic).
• Sulbutiamine, Benfotiamine.

Glutamatergic & Neuroprotective Modulators:

• Memantine (NMDA antagonist).
• ISRIB (Integrated Stress Response inhibitor — restores translation under stress).
• Minocycline (anti-inflammatory + neuroprotective).
• D-Cycloserine (NMDA co-agonist for extinction/learning enhancement).

Other Experimental / Emerging Compounds:

• Methylene Blue (low dose) — Mitochondrial enhancer + neuroprotectant.
• Rapamycin (low-dose, intermittent) — Autophagy inducer with mixed longevity/plasticity effects.
• NAD+ Precursors (NMN, NR, NMNH) — Boosts cellular energy and sirtuins.
• Lion’s Mane Mushroom (Hericeones + Erinacines) — Natural NGF inducer.
• Polygala Tenuifolia (sibiricose A5 etc.) — BDNF and neuroprotection.
• 7,8-DHF (7,8-Dihydroxyflavone) — BDNF receptor (TrkB) agonist.
• J147 — Curcumin derivative with potent neurotrophic effects.
• CN-19 or other synthetic BDNF mimetics (research stage)​

I can't be bothered to go into the chemical mechanisms by which each of these compounds produce cognitively beneficial change in the brain, but the idea is giving you the peripheral knowledge of them so you can do research and come to the realization of just how potent and useful they are for our stated purposes. In conjunction with the administration of the protocols previously mentioned, the usage of these compounds through intelligent dosing and cycling will provide the brain with the necessary chemically mobile environment to make accelerated changes, connections, and internalization of info. This shit works, that's it.

The Inhibition Layer (airway obstruction)

I'll be honest by now I'm tired as fuck so I'm going to keep it short. With no breathing there's no living, with dysfunctional breathing there's dysfunctional living. Sleep apnea has been shown to cause near a standard deviation decrease in one's IQ, making what one's intelligence should be not manifest and easily susceptible to one's equivocation of it with just "being dumb". You can check the sources for this one yourself idgaf.




WELP, that's it. Hope you got some use from this.

 

[Kingly]

inb4 "dnrd"

botb worthy

mirin effort and bookmarked

 

[duzxk1]

No TLDR?

 

[kantlie]

No TLDR?

I don't think that's possible in this context lol, the TLDR would be pretty long

 

[duzxk1]

I don't think that's possible in this context lol, the TLDR would be pretty long

Fairs

 

[hyperandrogenic]

Dnr but mirin effort

 

[Absurdist]

The Complete Guide to IQMaxxing

​

Why does IQ matter?

So that people don't ask retarded questions like that. IQ is the glue that pulls together any potential form of human interaction with the reality in which we exist so long as one's goal in it is prosperity in a domain to any measurable extent; you will always rely on intelligence to some extent, and if you want to be successful to whatever that extent may be you need to make it such that the extent is invariant relative to your intelligence.

In this guide I'm going to go over the process of the maximization of intelligence through a few layers that contribute to intellectual development and accentuation of cognitive ability throughout one's lifetime:

1. The stabilization layer: this would be your supplements, basic nootropics, and whatever other substances that can be bought in stores or ordered online that will aid in long-term cognitive development.
2. The active layer: this is going to contain that with which neuroplasticity engulfs the necessary thinking models for you to actually comprehend, process, and make connections with information faster and more productively. This will include the most evidence-based methods we have to date for the increase of IQ (citations will be included),
3. The neurochemical alteration layer: this layer will serve as the means through which the training methods will actually be consolidated in the mind and engrained into one's cognitive apparatus such that it becomes second nature and utility from each respective training method can be maximized. This will be your experimental pharmaceuticals and research chems that fags fear-monger about.
4. The inhibition layer: this layer will address any potential on-going physiological problems that could be halting the full expression of your baseline intelligence, which you may not be aware of. I'm going to focus on breathing and its significance since it is often overlooked and is the very urelement of human cognition; if you can't breathe, you can't think. As a result this will be concentrating on sleep apnea and kinds of nasal obstruction that could be inhibiting your brain from ensuring maximum oxygen intake.
Clarifying the science

1. How the Brain Uses Neuroplasticity

Neuroplasticity is the brain’s ability to reorganize its structure and function in response to experience, learning, or injury. It happens at multiple levels:

• Synaptic plasticity: Strengthening or weakening of existing synapses (connections between neurons). Key mechanism: Long-Term Potentiation (LTP) — repeated co-activation of neurons makes the synapse stronger (“cells that fire together wire together”).
• Synaptogenesis: Formation of entirely new synapses.
• Dendritic spine changes: Neurons grow more spines (receptors) to receive inputs.
• Structural changes: New neurons (neurogenesis, mainly in hippocampus) and rewiring of networks.

This allows skills, knowledge, and habits to become encoded in neural circuits.

2. How the Brain Consolidates Information

Consolidation turns fragile short-term memory into stable long-term memory:

• Encoding (hippocampus + prefrontal cortex): New info creates temporary neural patterns during learning.
• System consolidation: Over hours to weeks (especially during deep sleep and quiet wakefulness), the hippocampus “replays” the activity patterns. This transfers the memory to the neocortex for permanent storage.
• Synaptic consolidation: Requires protein synthesis (e.g., via BDNF signaling) to stabilize new/enhanced synapses within hours of learning.
• Myelination and network integration make the skill faster/automatic (“second nature”).

Repeated practice + sleep + focused attention = stronger, more efficient circuits. Without consolidation, new info fades.

3. Role of Synaptogenic & BDNF-Upregulating Compounds

Compounds that boost BDNF (Brain-Derived Neurotrophic Factor) or promote synaptogenesis amplify these natural processes:

• BDNF is a key neurotrophin. It:
  
  • Enhances LTP (makes learning stickier).
  • Promotes growth of new dendrites/spines.
  • Supports neuron survival and neurogenesis.
  • Facilitates the protein synthesis needed for long-term memory.
• Synaptogenic agents (e.g., those acting on HGF/c-Met like Dihexa) directly increase synapse density and dendritic complexity, creating more “hardware” for new information to wire into.

Neurochemically:

• During/after training (RFT, Dual N-Back, logic, skills), these compounds raise BDNF signaling → stronger LTP at active synapses.
• They lower the threshold for new synapse formation, so repeated practice builds denser, more interconnected circuits faster.
• Enhanced hippocampal-cortical dialogue during sleep consolidates the changes more efficiently.
• Result: Faster transition from effortful (prefrontal-heavy) to automatic (basal ganglia + cortical networks) performance.

Example in Practice (Making Skills Second Nature):Learning mathematical logic or relational frames initially feels effortful. With elevated BDNF/synaptogenesis:

• More synapses form between relevant neurons during sessions.
• Consolidation during sleep engrains the patterns deeper.
• Over weeks, the neural pathway becomes highly myelinated and efficient → you intuitively “see” logical structures or relational patterns without conscious effort (second nature/automaticity).

The Stabilization Layer (supplements)

BDNF / Neurotrophic Signaling​

Lion's Mane — stimulates NGF and BDNF synthesis directly; crosses BBB
Omega-3 (EPA/DHA) — DHA upregulates BDNF gene expression, reduces neuroinflammation
Curcumin — raises BDNF via histone acetylation (epigenetic upregulation)
Resveratrol — activates SIRT1 → downstream BDNF/CREB signaling
Exercise — strongest known driver of acute and chronic BDNF release (not a supplement, but the benchmark)

Synaptogenesis & Membrane / Cholinergic Support​

Citicoline — supplies choline + cytidine for phosphatidylcholine (synaptic membrane building block); boosts acetylcholine
Alpha-GPC — choline donor for acetylcholine synthesis and membrane phospholipids
Phosphatidylserine — structural phospholipid concentrated at synapses; supports membrane fluidity
DHA — incorporates into synaptic membranes, affecting fluidity and receptor/ion channel function
Bacopa monnieri — promotes hippocampal dendritic branching; modulates acetylcholinesterase

Neurogenesis​

Lion's Mane — NGF/BDNF pathway drives hippocampal neurogenesis (animal-confirmed)
Flavonoids (blueberry, cocoa, EGCG) — cross BBB, activate CREB/BDNF cascades tied to neurogenesis
Omega-3 — DHA supports neural stem cell membrane integrity during differentiation

Neuroprotection / Antioxidant​

NAC — precursor to glutathione, the brain's main endogenous antioxidant
Vitamin E — protects membrane fats (like DHA) from oxidative damage
Sulforaphane — activates Nrf2, the master regulator of antioxidant enzyme production
Alpha-lipoic acid — regenerates other antioxidants (C, E, glutathione); supports mitochondrial enzymes

Mitochondrial / Energy Support​

CoQ10 — core component of the mitochondrial electron transport chain (ATP production)
PQQ — stimulates mitochondrial biogenesis (new mitochondria formation)
Creatine — buffers ATP/phosphocreatine reserves for high cognitive load

Vascular / Neurovascular Support​

Ginkgo biloba — improves cerebral microcirculation; antioxidant/platelet effects
Nitrate/beetroot extract — converts to nitric oxide → vasodilation → more cerebral blood flow and oxygen deliver

The Active Layer (RFT, N-back, Image streaming)

Dual N-Back​

The task forces continuous updating of working memory contents — holding a stimulus, comparing it to one from N steps back, then discarding it as the next one arrives. You're not just holding items in your mind, you're actively overwriting and refreshing them under load. The dual-stream format (spatial position and auditory letter simultaneously) adds a divided-attention demand on top of the updating demand, recruiting both the dorsolateral prefrontal cortex (executive control, updating) and posterior parietal cortex (spatial/attentional buffer) more intensively than single-stream tasks. As you get used to a particular combinatorial setting, you can increase the difficulty and on some websites even add additional stimuli like photos, 3 dimensions, etc.


Evidence for memory increase:


Melby-Lervåg & Hulme's meta-analysis found solid transfer to verbal working memory (d ≈ 0.79) and spatial working memory (d ≈ 0.52) tasks — the strongest, most consistent finding in the whole n-back literature, well above the shaky far-transfer-to-IQ numbers.


A 2017 multi-level meta-analysis (33 RCTs, 203 effect sizes) found a medium-sized transfer effect to untrained n-back tasks specifically, with smaller effects for other working memory measures — gains are strongest on tasks structurally similar to n-back itself and taper off the further you get from that structure.


Several individual RCTs report improved performance on digit span and reading span, standard validated working-memory measures, following dual n-back training, consistent with the meta-analytic pattern above.


A 2021 study comparing dual n-back against the method of loci found dual n-back produced superior transfer effects on working memory measures like digit span and change detection, suggesting the updating-specific training does something a general mnemonic technique doesn't.


Overall, the N-back method protocol isn't for the consolidation of the methods by which your mind processes information and uses systems to make critical analyses of it (the theoretical foundation for RFT), so the point isn't IQ increase, something for which N-back doesn't have much evidence. However, it clearly has strong evidence for the improvement of one's short-term memory which is incredibly helpful in a wide variety of cognitive tasks and would not hurt to have in one's arsenal.

Note: If it matters at all, despite there not being conclusive scientific evidence for N-back resulting in IQ increase, many people have reported standard deviations worth of increase on their IQ tests before and after the administration of N-back training, so make of that what you wish.

---

Relational Frame Training (RFT)​

RFT's core claim is that derived relational responding — the ability to infer relationships you were never directly taught, such as if A=B and B=C then A=C, or if A is opposite to B and B is opposite to C then A=C — is the basic operant unit underlying verbal, analytical intelligence itself, not just a correlate of it. RFT drills this directly through multiple exemplar training: repeated practice deriving relations like same as, opposite of, more or less than, before or after, across many different stimulus sets, until the pattern of relational responding generalizes to entirely novel stimuli the person has never seen. This is mechanistically different from n-back: n-back trains a specific executive process and hopes it transfers, while RFT trains the actual operation that IQ test items like analogies, matrices, and syllogisms are built from. The theoretical distance between trained skill and tested skill is much shorter, which is the a priori reason to expect better transfer.


Foundational work by D. Barnes-Holmes and colleagues established that this kind of relational fluency is trainable and generalizable even in children who initially lacked it, meaning the underlying skill isn't fixed but is a learned repertoire.


Evidence for IQ increase:


Cassidy, Roche & Hayes's pilot study found children given stimulus equivalence training followed by relational frame training on SAME, OPPOSITE, MORE THAN, and LESS THAN showed significant full-scale WISC IQ gains after each stage, versus a no-treatment control.


Cassidy, Roche, Colbert & Grey, published in Learning and Individual Differences, found online SMART training produced average IQ increases of roughly 28 points, alongside gains in verbal, numerical, and educational aptitude, using the Wechsler scales as the outcome measure.


A correlational study using the Relational Abilities Index, a 55-item relational-puzzle assessment, found meaningful correlations between relational skill and full-scale IQ, the National Adult Reading Test, and other standard cognitive measures, supporting the theoretical link independent of the intervention effect.

This is the most promising means by which IQ and its underlying mechanistic foundations can be amplified and increased, and the one for which we have the most amount of evidence. However, the studies haven't been widely replicated and it's a very niche area of research, but nevertheless very promising. Also take into account that the younger you are the more effective this is going to be.

---

Image Streaming​

Wenger's model holds that closing your eyes and continuously narrating spontaneous mental imagery out loud forces simultaneous engagement of the visual/imagery system and the verbal articulation system, which he argues strengthens the neural connections between them, improving both visualization vividness and the speed and fluency of translating internal representations into language.
The idea is that continuous real-time narration under time pressure, describing the image as it's happening rather than after, trains rapid lexical retrieval and sentence construction, similar to how simultaneous interpretation trains fluency in interpreters.


Evidence for IQ increase:


Wenger's own claim, repeated across his self-published materials, is that roughly 25 hours of practice produces meaningful gains in IQ points.


I was unable to find any independent, peer-reviewed, controlled study measuring image streaming's effect on IQ, verbal fluency, or visualization ability. All available claims trace back to some niche, un-replicated studies with questionable protocols of research conduct and anecdotal or testimonial reports. Nevertheless I wanted to included because I've seen personal success with it in treating aphantasia (the inability to see in your mind's eye) and so have an entire dedicated community of people.


The Neurochemical Alteration Layer (research chems and experimental pharma)


This is the section dedicated to the compounds that alter your neurochemical architecture such that the absorption and maturation of information and the gains produced by the mentioned methods for cognitive development can be catalyzed and accelerated through drugs, shit that actually works. You can moral-fag and talk about the efficacy of these drugs but I'm not going to wait a life-time to get confirmation on whether shit that clearly functions is actually efficacious or not.

Neuroplasticity, synaptogenesis, BDNF/NGF signaling, neurogenesis, LTP, and neuroprotection. These compounds are designed to maximize the return on your RFT/SMART, Dual N-Back, mathematical logic, and Image Streaming training by making new neural connections denser, stronger, and more permanent. Compounds:

• Dihexa — Extremely potent HGF/c-Met agonist. Preclinical data suggests massive synaptogenesis (claimed 10 million times more potent than BDNF). Strong memory rescue in impaired rodent models.
• Cerebrolysin — Porcine brain-derived neurotrophic mix (BDNF, NGF, GDNF etc.). Most clinically studied with multiple RCTs/meta-analyses in stroke, TBI, and dementia.
• Semax (including N-Acetyl Semax Amidate / NASA) — ACTH(4-10) analog. Potent BDNF/TrkB upregulation. Russian-approved for stroke and cognitive issues.
• Selank (including N-Acetyl Selank) — Tuftsin analog. Anxiolytic + BDNF modulation. Russian-approved.
• Cortexin — Russian peptide bioregulator used for neuroprotection and cognitive recovery.
• Noopept — Synthetic dipeptide. Increases NGF, BDNF, and supports memory consolidation.
• Pinealon — Synthetic tripeptide bioregulator targeting brain tissue.
• Cortagen — Synthetic tetrapeptide for cortical repair and neuroprotection.
• Humanin — Mitochondrial-derived peptide with neuroprotective and cognitive effects.
• NSI-189 — Hippocampal neurogenesis promoter. Early-phase depression/cognition trials.
• P21 / CNTF analog — Mimics ciliary neurotrophic factor for neurogenesis.

Racetams & Related Ampakines:

• Piracetam, Phenylpiracetam, Aniracetam, Oxiracetam, Pramiracetam, Fasoracetam, Coluracetam, Nefiracetam.

Cholinergics & Acetylcholinesterase Inhibitors:

• Donepezil, Galantamine, Rivastigmine, Alpha-GPC, Citicoline (CDP-Choline), Huperzine A.

Dopaminergic & Stimulant Agents:

• Bromantane (dopamine synthesis + BDNF upregulation).
• Selegiline (low-dose MAO-B inhibitor).
• Modafinil / Armodafinil.
• Adderall / Dexedrine / Methylphenidate (prescribed).
• 9-Me-BC (dopamine neuron regeneration).
• PRL-8-53 (memory-specific nootropic).
• Sulbutiamine, Benfotiamine.

Glutamatergic & Neuroprotective Modulators:

• Memantine (NMDA antagonist).
• ISRIB (Integrated Stress Response inhibitor — restores translation under stress).
• Minocycline (anti-inflammatory + neuroprotective).
• D-Cycloserine (NMDA co-agonist for extinction/learning enhancement).

Other Experimental / Emerging Compounds:

• Methylene Blue (low dose) — Mitochondrial enhancer + neuroprotectant.
• Rapamycin (low-dose, intermittent) — Autophagy inducer with mixed longevity/plasticity effects.
• NAD+ Precursors (NMN, NR, NMNH) — Boosts cellular energy and sirtuins.
• Lion’s Mane Mushroom (Hericeones + Erinacines) — Natural NGF inducer.
• Polygala Tenuifolia (sibiricose A5 etc.) — BDNF and neuroprotection.
• 7,8-DHF (7,8-Dihydroxyflavone) — BDNF receptor (TrkB) agonist.
• J147 — Curcumin derivative with potent neurotrophic effects.
• CN-19 or other synthetic BDNF mimetics (research stage)​

I can't be bothered to go into the chemical mechanisms by which each of these compounds produce cognitively beneficial change in the brain, but the idea is giving you the peripheral knowledge of them so you can do research and come to the realization of just how potent and useful they are for our stated purposes. In conjunction with the administration of the protocols previously mentioned, the usage of these compounds through intelligent dosing and cycling will provide the brain with the necessary chemically mobile environment to make accelerated changes, connections, and internalization of info. This shit works, that's it.

The Inhibition Layer (airway obstruction)

I'll be honest by now I'm tired as fuck so I'm going to keep it short. With no breathing there's no living, with dysfunctional breathing there's dysfunctional living. Sleep apnea has been shown to cause near a standard deviation decrease in one's IQ, making what one's intelligence should be not manifest and easily susceptible to one's equivocation of it with just "being dumb". You can check the sources for this one yourself idgaf.




WELP, that's it. Hope you got some use from this.

Can you link sources? admirin effort tho.

 

[saintsyard]

tldr; do various cognitively demanding tasks, especially new ones

 

[kantlie]

Can you link sources? admirin effort tho.

for which part? I cited the studies for the training methods and their supposed efficacy. The list of the compounds is so long that were I to provide citations for each one it would be basically unreadable.

 

[kantlie]

tldr; do various cognitively demanding tasks, especially new ones

no

tldr; take bdnf upregulating and synaptogenic research chems while doing RFT, N-back, image streaming and taking supplements for long term neuroprotection

(i guess it is possible to do a tldr for this lol)

 

[saintsyard]

no

tldr; take bdnf upregulating and synaptogenic research chems while doing RFT, N-back, image streaming and taking supplements for long term neuroprotection

(i guess it is possible to do a tldr for this lol)

all of which are cognitively demanding tasks with the exception of boosting bdnf through pharma, i didnt mention that ur right

 

[kantlie]

all of which are cognitively demanding tasks with the exception of boosting bdnf through pharma, i didnt mention that ur right

yeah but you could be doing cognitively demanding tasks like studying calculus but that wouldn't raise ur IQ even if you were taking pharma. It's about which cognitively demanding tasks are being done and those would be the ones that functionally target the mechanisms required to increase IQ.

 

[cfshere]

Work on the formatting

 

[kantlie]

Work on the formatting

yeah srry about that

 

[kantlie]

bump

 

[kantlie]

spent a lot of fucking time on this post