H
heightmaxxer1133
Silver
- Joined
- Jun 13, 2024
- Posts
- 669
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- 310
I:INTRO
This dumb forum promotes the utilisation of hgh and ai.
However, this claim overlooks crucial points
II:CHONDROCYTES
Growth plates contain conrocyte that proliferate hypertrophy and undergo apoptosis
These processes regulated by growth hormone IGF-1, Testosterone and Estrogen are crucial for height growth
Endochondrial ossification replaced cartilage which can grow with bones which can’t grow
Growth hormone (GH) and IGF-1 promote conrocyte division in the proliferative zone enlargement in the hypertrophic zone and bone replacement in the ossification zone.
III:RESPONSIVENESS
As puberty progresses around ages 15 and 19 for developped countries and 12 and 15 for less developed countries because of faster telomere and shortening due to environmental and genetics factors.
converseite become less responsive to proliferative signals and more prone to sinescence and apoptosis leading to growth plates closure and the end of height increase
the JK stat and P I 3 K slash act pathways for growth and IGF-1 signaling are less responsive aging condracite
So blasting these condracite with growth hormone won’t do much like watering aging farmland
IV:HPG-Axis
Estrogen plays a key role in growth plates closure by binding to estrogen receptors on condrosite promoting their maturation and ossification
Using aromatase inhibitors to keep your growth plates open by reducing estrogen disrupts the HPG axis and increasing testosterone and dht. YOU MIGHT THINK THIS IS A GOOD THING ?
But both combine to androgen receptors on condrosite promoting ossification in conclusion
V:CONCLUSION
While letrozole might delay growth plates closure, it doesn’t mean that u can grow in that extra time
Mature contracyte won’t respond to increased growth hormone making additional height growth unlikely
You can blast as much growth hormone as you want at 17.
Your contracyte won’t respond to it and you will end up with insulin resistance.
SO IT’S COMPLETELY OVER FOR U JUST ROPE
This dumb forum promotes the utilisation of hgh and ai.
However, this claim overlooks crucial points
II:CHONDROCYTES
Growth plates contain conrocyte that proliferate hypertrophy and undergo apoptosis
These processes regulated by growth hormone IGF-1, Testosterone and Estrogen are crucial for height growth
Endochondrial ossification replaced cartilage which can grow with bones which can’t grow
Growth hormone (GH) and IGF-1 promote conrocyte division in the proliferative zone enlargement in the hypertrophic zone and bone replacement in the ossification zone.
III:RESPONSIVENESS
As puberty progresses around ages 15 and 19 for developped countries and 12 and 15 for less developed countries because of faster telomere and shortening due to environmental and genetics factors.
converseite become less responsive to proliferative signals and more prone to sinescence and apoptosis leading to growth plates closure and the end of height increase
the JK stat and P I 3 K slash act pathways for growth and IGF-1 signaling are less responsive aging condracite
So blasting these condracite with growth hormone won’t do much like watering aging farmland
IV:HPG-Axis
Estrogen plays a key role in growth plates closure by binding to estrogen receptors on condrosite promoting their maturation and ossification
Using aromatase inhibitors to keep your growth plates open by reducing estrogen disrupts the HPG axis and increasing testosterone and dht. YOU MIGHT THINK THIS IS A GOOD THING ?
But both combine to androgen receptors on condrosite promoting ossification in conclusion
V:CONCLUSION
While letrozole might delay growth plates closure, it doesn’t mean that u can grow in that extra time
Mature contracyte won’t respond to increased growth hormone making additional height growth unlikely
You can blast as much growth hormone as you want at 17.
Your contracyte won’t respond to it and you will end up with insulin resistance.
SO IT’S COMPLETELY OVER FOR U JUST ROPE
