U WONT GROW TALLER FROM HGH AI

I didn’t copy he did
Nigga I doubt you even know what you are talking about, I hate you graycel larpcels
Screenshot 2024 08 24 134008
 
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I've grown 1.5-2 inches after prevriously not growing after using hgh.
My igf1 was also at the lower percentiles which I think matters too
 
I've grown 1.5-2 inches after prevriously not growing after using hgh.
My igf1 was also at the lower percentiles which I think matters too
It’s natural
 
Cool thread. There isn't any harm in believing that heightmaxxing is bad and people should stop. However, there are a few problems with this thread. First of all, a lot of the points you make don't align with the evidence you use to back it up. For example, using a study done on rabbits as a point to prove that androgen receptors on chondrocytes promote ossification regardless of the use of AI isn't fair, since the study wasn't even done on humans or at least rats. Rabbit studies are most applicable to humans when it comes to research about immunology! Also, using a study that showcases that letrozole doesn't help increase adult height in prepubertal children is once again useless, as why would it? Letrozole's main purpose as a compound to increase height is to prolong height growth when you are getting closer to the end of puberty and your growth rate begins to falter, so using a study done on early puberty kids doesn't prove anything.

Another thing is that I think you are overcomplicating the height growth that comes from HGH usage. HGH isn't a miracle drug, you won't grow 6+ inches from it at the bone age of 18. The majority of people also know that as you keep aging throughout puberty, the more likely your growth rate will continue faltering. This isn't some fact that nobody knows about like you are seemingly trying to present here. However, when you take HGH while you have a bone age under 16.5 - 17, you give yourself a much better chance to grow taller than if you didn't! Let's take your analogy for example, would you not at least try to revive your dying farmland, before it's gone forever, especially, if there's a chance it can come back again? While people believe it can be a waste of time and resources, it is still possible that you can grow at older bone ages, as long as you have an open growth plate, at least in the knee growth plate!

Furthermore, you lack knowledge and understanding of how growth plates work in general! Because your birth age doesn't matter at all in regards to height growth. Height growth is very specific from person to person. Some people may stop growing at 17, others may stop growing at 15, and hundreds of thousands could stop growing at 19. On top of that, even though many people stop growing after a certain age, it doesn't even mean that their growth plates are completely closed. Even when your growth plates close, they close in multiple different phases, and it's not a straight-line process.

Lastly, there have been a few studies done on older children (15 - 16) with older bone ages, that had faltering growth rates and have grown from taking HGH and AI. While I don't feel like going into depth explaining them like I have many times before in previous posts, I'll list them below!:

A pilot study of growth hormone administration in boys with predicted adult short stature and near-ending growth

A randomized pilot trial of growth hormone with anastrozole versus growth hormone alone, starting at the very end of puberty in adolescents with idiopathic short stature


Both of the studies above showcase how growth hormone and AI can still work for even at the end of puberty in adolescents, however, it requires higher doses and more effects on the body.

In conclusion, I don't have any problem with people thinking that heightmaxxing is dangerous and not worth it for most people because, at the end of the day, the way that most people are going to have to go about is going to be dangerous. You are putting hormones and drugs in your body without medical supervision most of the time, there isn't anything safe about that. However, there's no point in making a point calling it cope while making the most general points and not even providing the proper evidence. Call it cope once multiple research studies have come out disproving the effects of HGH and AI, and saying that the conclusion previous studies have come to have been wrong and has been caused by something else in the human body. Call it cope because maybe the way height growth is defined in many of these HGH and AI studies is wrong and leads to inaccurate results.
 
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I:INTRO

This dumb forum promotes the utilisation of hgh and ai.
However, this claim overlooks crucial points
View attachment 3118801

II:CHONDROCYTES
Growth plates contain conrocyte that proliferate hypertrophy and undergo apoptosis
View attachment 3118800


These processes regulated by growth hormone IGF-1, Testosterone and Estrogen are crucial for height growth
View attachment 3118805

Endochondrial ossification replaced cartilage which can grow with bones which can’t grow
View attachment 3118806

Growth hormone (GH) and IGF-1 promote conrocyte division in the proliferative zone enlargement in the hypertrophic zone and bone replacement in the ossification zone.
View attachment 3118809

III:RESPONSIVENESS
As puberty progresses around ages 15 and 19 for developped countries and 12 and 15 for less developed countries because of faster telomere and shortening due to environmental and genetics factors.
View attachment 3118812

converseite become less responsive to proliferative signals and more prone to sinescence and apoptosis leading to growth plates closure and the end of height increase
View attachment 3118814

the JK stat and P I 3 K slash act pathways for growth and IGF-1 signaling are less responsive aging condracite
View attachment 3118815
View attachment 3118825
View attachment 3118827
So blasting these condracite with growth hormone won’t do much like watering aging farmland
View attachment 3118828


IV:HPG-Axis
Estrogen plays a key role in growth plates closure by binding to estrogen receptors on condrosite promoting their maturation and ossification
View attachment 3118830

View attachment 3118836

Using aromatase inhibitors to keep your growth plates open by reducing estrogen disrupts the HPG axis and increasing testosterone and dht. YOU MIGHT THINK THIS IS A GOOD THING ?
But both combine to androgen receptors on condrosite promoting ossification in conclusion
View attachment 3118840
View attachment 3118842
V:CONCLUSION
While letrozole might delay growth plates closure, it doesn’t mean that u can grow in that extra time

View attachment 3118845
Mature contracyte won’t respond to increased growth hormone making additional height growth unlikely
View attachment 3118857
You can blast as much growth hormone as you want at 17.
Your contracyte won’t respond to it and you will end up with insulin resistance.

View attachment 3118866
SO IT’S COMPLETELY OVER FOR U JUST ROPE
Depending on how early you started
 
Average puberty starts before age 12 for boys in America you stupid ignorant fucking faggot. It ends at 16 for normal boys. Only low T faggots start puberty at 15. :blackpill::redpill::blackpill::redpill:
 
I:INTRO

This dumb forum promotes the utilisation of hgh and ai.
However, this claim overlooks crucial points
View attachment 3118801

II:CHONDROCYTES
Growth plates contain conrocyte that proliferate hypertrophy and undergo apoptosis
View attachment 3118800


These processes regulated by growth hormone IGF-1, Testosterone and Estrogen are crucial for height growth
View attachment 3118805

Endochondrial ossification replaced cartilage which can grow with bones which can’t grow
View attachment 3118806

Growth hormone (GH) and IGF-1 promote conrocyte division in the proliferative zone enlargement in the hypertrophic zone and bone replacement in the ossification zone.
View attachment 3118809

III:RESPONSIVENESS
As puberty progresses around ages 15 and 19 for developped countries and 12 and 15 for less developed countries because of faster telomere and shortening due to environmental and genetics factors.
View attachment 3118812

converseite become less responsive to proliferative signals and more prone to sinescence and apoptosis leading to growth plates closure and the end of height increase
View attachment 3118814

the JK stat and P I 3 K slash act pathways for growth and IGF-1 signaling are less responsive aging condracite
View attachment 3118815
View attachment 3118825
View attachment 3118827
So blasting these condracite with growth hormone won’t do much like watering aging farmland
View attachment 3118828


IV:HPG-Axis
Estrogen plays a key role in growth plates closure by binding to estrogen receptors on condrosite promoting their maturation and ossification
View attachment 3118830

View attachment 3118836

Using aromatase inhibitors to keep your growth plates open by reducing estrogen disrupts the HPG axis and increasing testosterone and dht. YOU MIGHT THINK THIS IS A GOOD THING ?
But both combine to androgen receptors on condrosite promoting ossification in conclusion
View attachment 3118840
View attachment 3118842
V:CONCLUSION
While letrozole might delay growth plates closure, it doesn’t mean that u can grow in that extra time

View attachment 3118845
Mature contracyte won’t respond to increased growth hormone making additional height growth unlikely
View attachment 3118857
You can blast as much growth hormone as you want at 17.
Your contracyte won’t respond to it and you will end up with insulin resistance.

View attachment 3118866
SO IT’S COMPLETELY OVER FOR U JUST ROPE
Read every molecule and you were just exaggerating the fact you can't grow once your plates are nearly closed, you can get a couple inches if you start a few years before that.
Also this guy on tiktok copied you:
 

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