What If Free We Increase Free IGF-1 Unlocked Forward Growth, Larger Skull& Extra Height? (28 Studies Theory)

[teddy101]

You may be wondering what the hell I'm suggesting with such a high-promise idea. Well, it's obesity... but hold up I'm not claiming obesity itself is the hero here. It's the chronically high insulin levels that often come with it.

This is completely theoretical. No personal experiments, no physical proof from me, just logical connecting of dots from existing peer-reviewed studies. Correlations, not proven causation. Obesity carries serious long-term health risks (insulin resistance, diabetes, inflammation, heart issues, etc.), and I'm not recommending anyone pursue weight gain or hormone hacking. Growth plates fuse after puberty, so any real-world height impact in adults is extremely limited at best. This is for discussion and curiosity only. If you're thinking of trying anything, talk to actual doctors don't be dumb.

I've pulled together 28 studies (full numbered list at the bottom). Anytime I make a claim, I'll reference the specific ones so you can check them yourself. Happy to fix mistakes if you spot them.

The Key Studies on Insulin, IGFBPs, and Free IGF-1​

Multiple papers show a consistent pattern in obesity: people often have lower growth hormone (GH) but still maintain decent or elevated free (bioavailable) IGF-1. Why? High insulin seems to play a big role by suppressing IGF-binding proteins (especially IGFBP-1 and IGFBP-2), which normally keep IGF-1 bound and less active.1

Studies like #1, #3, #4, #6, #8, #10, #11, #12, #13, #14, #15, #16, and #17 back this up strongly. Obese individuals show this "normal" growth factor activity despite the low GH, and the most plausible mechanism pointed to again and again is insulin-driven blockage/reduction of those binding proteins.

What the Research Shows on Height and Facial Growth​

Other studies (#20, #21, #22, #23, #24, #25, #26, #27, #28) look at actual body and face measurements in obese vs. normal-weight people (mostly adolescents, where growth is still active).

The pattern: Obese individuals tend to be bigger and wider skeletally overall longer mandibular length, maxillary length, greater face height, more bimaxillary prognathism (forward jaw positioning). They often have accelerated bone maturation, which can mean they "finish growing" earlier, leading to similar final height to peers in many cases. But the facial dimensions (mandible, maxilla, anterior face height) frequently end up larger. Some facial growth (especially certain sutures and condylar areas) can continue or be influenced longer than long-bone epiphyseal plates.2

The remaining studies in the list also support the obesity → higher free IGF-1 correlation.

My Logical Chain of Thought​

Putting it together step by step:

Obesity → Hyperinsulinemia (chronically elevated insulin) → Decreased IGFBP-1 and IGFBP-2 → Increased free (bioavailable) IGF-1 → Potential for accelerated skeletal maturation + larger craniofacial dimensions (mandibular length, maxillary length, face height).

That's the core hypothesis. IGF-1 is a major driver of bone and cartilage growth, so it makes biological sense on paper.

Speculative extension (even more theoretical): If we could somehow slow epiphyseal plate closure (e.g., with things like aromatase inhibitors/AIs that reduce estrogen's role in closure), combined with the above, maybe bigger height potential. But to offset the downsides of excess fat inflammation, negative hormone shifts, etc. the smarter approach might be bulking with a focus on muscle via HIT (high-intensity training/resistance work) rather than just getting fat. That could theoretically give some of the insulin effect while mitigating other problems. Again, pure logic, massive risks, not advice.

My Next Plan​

I want to research more and put together a proper theoretical guide on how someone might approach this carefully and "properly" (if such a thing even exists in speculation land). More reading on timing, mitigating sides, monitoring, etc.

While I'm online, I'll reply to comments. If you have counterpoints, questions, better studies, or spot holes/misconceptions in my reasoning, hit me with them. Suggestions on what to add to the guide would be awesome too.





What do you think of the overall logic? Does this track biologically? Any major red flags or things I'm missing? Should I emphasize certain risks more? Let's discuss.

---

Full list of studies referenced (numbered as I used them):
1. https://pubmed.ncbi.nlm.nih.gov/9152736/
2. https://www.nature.com/articles/0800412
3. https://academic.oup.com/jcem/article/110/3/e622/7658383
4. https://pubmed.ncbi.nlm.nih.gov/7476310/
5. https://www.sciencedirect.com/science/article/abs/pii/0026049595902198
6. https://pubmed.ncbi.nlm.nih.gov/19470623/
7. https://academic.oup.com/jcem/article/94/8/3093/2597545
8. https://pubmed.ncbi.nlm.nih.gov/11595780/
9. https://onlinelibrary.wiley.com/doi/full/10.1038/oby.2001.83
10. https://pubmed.ncbi.nlm.nih.gov/29679919/
11. https://pubmed.ncbi.nlm.nih.gov/9215275/
12. https://pmc.ncbi.nlm.nih.gov/articles/PMC3308317/
13. https://erc.bioscientifica.com/view/journals/erc/19/5/F27.xml
14. https://www.frontiersin.org/journals/endocrinology/articles/10.3389/fendo.2024.1456195/full
15. https://www.e-enm.org/journal/view.php?doi=10.3803/EnM.2024.101
16. https://www.tandfonline.com/doi/full/10.1080/21623945.2022.2148886
17. https://www.frontiersin.org/journals/endocrinology/articles/10.3389/fendo.2021.727004/full
18. https://www.nature.com/articles/s41598-022-23521-1
19. https://www.mdpi.com/1422-0067/25/7/3966
20. https://pubmed.ncbi.nlm.nih.gov/37932128/
21. https://pmc.ncbi.nlm.nih.gov/articles/PMC10783153/
22. https://academic.oup.com/ejo/article/46/1/cjad043/7325271
23. https://pubmed.ncbi.nlm.nih.gov/12222641/
24. https://pubmed.ncbi.nlm.nih.gov/16009667/
25. https://www.mdpi.com/2227-9067/12/3/377
26. https://pubmed.ncbi.nlm.nih.gov/40556469/
27. https://academic.oup.com/ejo/article-abstract/47/4/cjaf044/8173223
28. https://www.sciencedirect.com/science/article/abs/pii/S0003996920303423

 

[teddy101]

bump

 

[teddy101]

bump

 

[Zagro]

gpt

does blud know injecting insulin is also an option instead of trying to be obese

 

[teddy101]

gpt

i wrote the script and gave it for grammatical correction and better formatting every thing idea was written by me and i check most of the studies being shown

 

[Zagro]

i wrote the script and gave it for grammatical correction and better formatting every thing idea was written by me and i check most of the studies being shown

Obesity → Hyperinsulinemia (chronically elevated insulin) → Decreased IGFBP-1 and IGFBP-2 → Increased free (bioavailable) IGF-1 → Potential for accelerated skeletal maturation + larger craniofacial dimensions (mandibular length, maxillary length, face height).

“No bro i just used it for grammatical correction!”

 

[teddy101]

Obesity → Hyperinsulinemia (chronically elevated insulin) → Decreased IGFBP-1 and IGFBP-2 → Increased free (bioavailable) IGF-1 → Potential for accelerated skeletal maturation + larger craniofacial dimensions (mandibular length, maxillary length, face height).

“No bro i just used it for grammatical correction!”

omfg bro it aint that a big deal i did the research way before like months ago i was just lazy to post ts shi you can just skip it if you dont belive me

 

[Zagro]

omfg bro it aint that a big deal i did the research way before like months ago i was just lazy to post ts shi you can just skip it if you dont belive me

You didn’t do the research way before/months ago, you just found out about this through ChatGPT (https://looksmax.org/threads/nocturnal-kent-might-be-half-right.1849328/#post-26808015) and asked for more studies proof from the AI.

Don’t even try to lie you fucking retard you’re making it worse, you did not read a singular paper you provided aswell I’d bet on that, you just read the abstract at most which I don’t think you did

Your theory is also the most retarded thing ever, “just be obese for more free igf-1 bro!1!!” you also added that it would be better with androgens i cant even imagine how fucking estrogenic you’d be which you also wont be able to prevent fully

If you accepted your usage of AI i would’ve let it slide, do not make a guide that no one will read cause they can also just ask gpt and get the same answer yk

 

[teddy101]

Obesity → Hyperinsulinemia (chronically elevated insulin) → Decreased IGFBP-1 and IGFBP-2 → Increased free (bioavailable) IGF-1 → Potential for accelerated skeletal maturation + larger craniofacial dimensions (mandibular length, maxillary length, face height).

“No bro i just used it for grammatical correction!”

here is my orginal prompt turn this keep it human and my ideas that same
(first this is completley therocitical and no physical proof just logical thougth based well cited and peer reviewed on studies)you may be wondering what could i possibly be sugesting for such a high promise well its obesity, but wait a minute its not the obesity which i am claiming to boost it but the chronicaly high insulin, but lets not just make claims first let me provie studies and a baseline sincetifci agrement and the grey area and complete myth 1.studies used 1. https://pubmed.ncbi.nlm.nih.gov/9152736/ 2. https://www.nature.com/articles/0800412 3. https://academic.oup.com/jcem/article/110/3/e622/7658383 4. https://pubmed.ncbi.nlm.nih.gov/7476310/ 5. https://www.sciencedirect.com/science/article/abs/pii/0026049595902198 6. https://pubmed.ncbi.nlm.nih.gov/19470623/ 7. https://academic.oup.com/jcem/article/94/8/3093/2597545 8. https://pubmed.ncbi.nlm.nih.gov/11595780/ 9. https://onlinelibrary.wiley.com/doi/full/10.1038/oby.2001.83 10. https://pubmed.ncbi.nlm.nih.gov/29679919/ 11. https://pubmed.ncbi.nlm.nih.gov/9215275/ 12. https://pmc.ncbi.nlm.nih.gov/articles/PMC3308317/ 13. https://erc.bioscientifica.com/view/journals/erc/19/5/F27.xml 14. https://www.frontiersin.org/journals/endocrinology/articles/10.3389/fendo.2024.1456195/full 15. https://www.e-enm.org/journal/view.php?doi=10.3803/EnM.2024.101 16. https://www.tandfonline.com/doi/full/10.1080/21623945.2022.2148886 17. https://www.frontiersin.org/journals/endocrinology/articles/10.3389/fendo.2021.727004/full 18. https://www.nature.com/articles/s41598-022-23521-1 19. https://www.mdpi.com/1422-0067/25/7/3966 20. https://pubmed.ncbi.nlm.nih.gov/37932128/ 21. https://pmc.ncbi.nlm.nih.gov/articles/PMC10783153/ 22. https://academic.oup.com/ejo/article/46/1/cjad043/7325271 23. https://pubmed.ncbi.nlm.nih.gov/12222641/ 24. https://pubmed.ncbi.nlm.nih.gov/16009667/ 25. https://www.mdpi.com/2227-9067/12/3/377 26. https://pubmed.ncbi.nlm.nih.gov/40556469/ 27. https://academic.oup.com/ejo/article-abstract/47/4/cjaf044/8173223 28. https://www.sciencedirect.com/science/article/abs/pii/S0003996920303423any time i make a claim i prove it with these studies and i have numbered them so you can come and check for your self i made mistakes i am happy to fix2. the theory from studies(1) (3) (4) (6) (8) (10) (11) (12) (13) (14) (15) (16) (17) its clear there is correlation between obesity and free igf-1 its impressive obesse poepl have normal amounts of growth desipte low gh levels and most porbable conclution is its because of igfbp blockage caused by high insulin whic is a know phenomenon in biology so we have showed that its corrlated and pointed fingers at the most porbalbe cause next 3.what the studies show about heigth and facial growth (20) (21) (22) (23) (24) (25) (26) (27) (28)these studies show obesse ppl have are generaly bigger and wider skeletaly that their peers but that advantage goes away because their bones mature fast so they reach similar final heigth but facial dimention(mandibular,maxillary,face hiegth) whcih contiune to grow even in adult hood(expet for maxillary which is hard to predict but still could be) show higher final dimenions 3what the other studies are they are also studies that corrlate obesity with free igf-1 4my logcial train of thougth is Obesity → Hyperinsulinemia → ↓IGFBP-1/2 → ↑Free IGF-1 → Accelerated skeletal maturation and larger craniofacial dimensions (mandibular length, maxillary length, face height) also think with pharmas like AI we could slow down plates closure for very large hiegth gain but to combat inflamation fat and other side effect and things that lower igf-1 gh and hormones using HIT exersie bulking up rather than becoming just fat COULD yield results 5my next plani will create a guide on how to do it properly but i need to research more and would like to hear your thougths and clear up misconeptions fix my mistakes etc as long as i am online i will reply to your comment if you have a counter point or have a question or suddgestion what gif or pic or anthign i should add also

 

[Zagro]

here is my orginal prompt turn this keep it human and my ideas that same
(first this is completley therocitical and no physical proof just logical thougth based well cited and peer reviewed on studies)you may be wondering what could i possibly be sugesting for such a high promise well its obesity, but wait a minute its not the obesity which i am claiming to boost it but the chronicaly high insulin, but lets not just make claims first let me provie studies and a baseline sincetifci agrement and the grey area and complete myth 1.studies used 1. https://pubmed.ncbi.nlm.nih.gov/9152736/ 2. https://www.nature.com/articles/0800412 3. https://academic.oup.com/jcem/article/110/3/e622/7658383 4. https://pubmed.ncbi.nlm.nih.gov/7476310/ 5. https://www.sciencedirect.com/science/article/abs/pii/0026049595902198 6. https://pubmed.ncbi.nlm.nih.gov/19470623/ 7. https://academic.oup.com/jcem/article/94/8/3093/2597545 8. https://pubmed.ncbi.nlm.nih.gov/11595780/ 9. https://onlinelibrary.wiley.com/doi/full/10.1038/oby.2001.83 10. https://pubmed.ncbi.nlm.nih.gov/29679919/ 11. https://pubmed.ncbi.nlm.nih.gov/9215275/ 12. https://pmc.ncbi.nlm.nih.gov/articles/PMC3308317/ 13. https://erc.bioscientifica.com/view/journals/erc/19/5/F27.xml 14. https://www.frontiersin.org/journals/endocrinology/articles/10.3389/fendo.2024.1456195/full 15. https://www.e-enm.org/journal/view.php?doi=10.3803/EnM.2024.101 16. https://www.tandfonline.com/doi/full/10.1080/21623945.2022.2148886 17. https://www.frontiersin.org/journals/endocrinology/articles/10.3389/fendo.2021.727004/full 18. https://www.nature.com/articles/s41598-022-23521-1 19. https://www.mdpi.com/1422-0067/25/7/3966 20. https://pubmed.ncbi.nlm.nih.gov/37932128/ 21. https://pmc.ncbi.nlm.nih.gov/articles/PMC10783153/ 22. https://academic.oup.com/ejo/article/46/1/cjad043/7325271 23. https://pubmed.ncbi.nlm.nih.gov/12222641/ 24. https://pubmed.ncbi.nlm.nih.gov/16009667/ 25. https://www.mdpi.com/2227-9067/12/3/377 26. https://pubmed.ncbi.nlm.nih.gov/40556469/ 27. https://academic.oup.com/ejo/article-abstract/47/4/cjaf044/8173223 28. https://www.sciencedirect.com/science/article/abs/pii/S0003996920303423any time i make a claim i prove it with these studies and i have numbered them so you can come and check for your self i made mistakes i am happy to fix2. the theory from studies(1) (3) (4) (6) (8) (10) (11) (12) (13) (14) (15) (16) (17) its clear there is correlation between obesity and free igf-1 its impressive obesse poepl have normal amounts of growth desipte low gh levels and most porbable conclution is its because of igfbp blockage caused by high insulin whic is a know phenomenon in biology so we have showed that its corrlated and pointed fingers at the most porbalbe cause next 3.what the studies show about heigth and facial growth (20) (21) (22) (23) (24) (25) (26) (27) (28)these studies show obesse ppl have are generaly bigger and wider skeletaly that their peers but that advantage goes away because their bones mature fast so they reach similar final heigth but facial dimention(mandibular,maxillary,face hiegth) whcih contiune to grow even in adult hood(expet for maxillary which is hard to predict but still could be) show higher final dimenions 3what the other studies are they are also studies that corrlate obesity with free igf-1 4my logcial train of thougth is Obesity → Hyperinsulinemia → ↓IGFBP-1/2 → ↑Free IGF-1 → Accelerated skeletal maturation and larger craniofacial dimensions (mandibular length, maxillary length, face height) also think with pharmas like AI we could slow down plates closure for very large hiegth gain but to combat inflamation fat and other side effect and things that lower igf-1 gh and hormones using HIT exersie bulking up rather than becoming just fat COULD yield results 5my next plani will create a guide on how to do it properly but i need to research more and would like to hear your thougths and clear up misconeptions fix my mistakes etc as long as i am online i will reply to your comment if you have a counter point or have a question or suddgestion what gif or pic or anthign i should add also

What the fuck is this slop of text going to prove, it still has fucking GPT arrows mate

 

[teddy101]

You didn’t do the research way before/months ago, you just found out about this through ChatGPT (https://looksmax.org/threads/nocturnal-kent-might-be-half-right.1849328/#post-26808015) and asked for more studies proof from the AI.

Don’t even try to lie you fucking retard you’re making it worse, you did not read a singular paper you provided aswell I’d bet on that, you just read the abstract at most which I don’t think you did

Your theory is also the most retarded thing ever, “just be obese for more free igf-1 bro!1!!” you also added that it would be better with androgens i cant even imagine how fucking estrogenic you’d be which you also wont be able to prevent fully

If you accepted your usage of AI i would’ve let it slide, do not make a guide that no one will read cause they can also just ask gpt and get the same answer yk

bro i cant prove it to you but i already have a a post way back that talked about this i have another 2 post talking about this i just tried to make it more understandable and professional you can check my account idk how to send it to you my first 2 post were about this

 

[BlackSilence]

dnr but repped for effort

 

[Zagro]

bro i cant prove it to you but i already have a a post way back that talked about this i have another 2 post talking about this i just tried to make it more understandable and professional you can check my account idk how to send it to you my first 2 post were about this

Okay still doesn’t change the fact that you did not read a single paper or do a molecule of research yourself

Gpt isn’t research, you doing it 2 months ago with gpt also still doesn’t make it research

It funny how you don’t fucking know the pathways behind it but surprisingly know that insulin increases free igf-1, this is like writing the answer on an exam without the explanation/how you came up with the answer

 

[teddy101]

https://looksmax.org/threads/nocturnal-kent-might-be-half-right.1849328/#post-26814382

What the fuck is this slop of text going to prove, it still has fucking GPT arrows mate

here is one of the post which is my very first post i joined org just to try and learn more about this

 

[Zagro]

https://looksmax.org/threads/nocturnal-kent-might-be-half-right.1849328/#post-26814382

here is one of the post which is my very first post i joined org just to try and learn more about this

I literally pasted this post of yours just now in my reply which you also haven’t read, nice one

 

[teddy101]

Okay still doesn’t change the fact that you did not read a single paper or do a molecule of research yourself

Gpt isn’t research, you doing it 2 months ago with gpt also still doesn’t make it research

It funny how you don’t fucking know the pathways behind it but surprisingly know that insulin increases free igf-1, this is like writing the answer on an exam without the explanation/how you came up with the answer

ik that high insulin blocks the production of igfbp which reduces the amount of proteins that bind with igf-1 this is basic shi i migth not be very smart but i 100% did my research the only ai i used was elict which i used to find studies because i was not find anything with google scholar

 

[teddy101]

I literally pasted this post of yours just now in my reply which you also haven’t read, nice one

mb but still what makes you think i did only ask ai you are doing a slippery slope logical fallacy just cause i used ai once doesnt meani have always used it

 

[Zagro]

ik that high insulin blocks the production of igfbp which reduces the amount of proteins that bind with igf-1 this is basic shi i migth not be very smart but i 100% did my research the only ai i used was elict which i used to find studies because i was not find anything with google scholar

Good now you do accept that you’ve used ai and learned it through there

This is water knowledge btw lantus insulin was popular begin 2025 for this reason

 

[Zagro]

mb but still what makes you think i did only ask ai you are doing a slippery slope logical fallacy just cause i used ai once doesnt meani have always used it

You literally told me you found the studies with ai, corrected your mistakes with ai and corrected grammatica with ai and potentially also learnt the basics through ai

That covers just about everything

I just hate when people use ai and then rip their ass apart trying to convince me otherwise when it’s very obvious, I’m not necessarily against ai to learn about the mechanisms or if you’re having a hard time, finding studies with it is also practical but i just get triggered idk

 

[teddy101]

Good now you do accept that you’ve used ai and learned it through there

This is water knowledge btw lantus insulin was popular begin 2025 for this reason

yeah i used ai but i didnt just blindly follow it besides it used for finding studies i read countless studies your jus ragebaiting atp i research this for days and nights all i wanted is a decent counter point or constructive criticism but no one helped i kept posting again and again

 

[Zagro]

yeah i used ai but i didnt just blindly follow it besides it used for finding studies i read countless studies your jus ragebaiting atp i research this for days and nights all i wanted is a decent counter point or constructive criticism but no one helped i kept posting again and again

This is already known for most, and it’s legit

Your few days of research with ai still doesn’t make me care in the slightest, if i have to suffer and find studies individually and summarise them and compile etc i will not let shit like this slide

 

[teddy101]

wh

You literally told me you found the studies with ai, corrected your mistakes with ai and corrected grammatica with ai and potentially also learnt the basics through ai

That covers just about everything

I just hate when people use ai and then rip their ass apart trying to convince me otherwise when it’s very obvious, I’m not necessarily against ai to learn about the mechanisms or if you’re having a hard time to find studies with it is also practical but i just get triggered idk

valid but you have to make the distinction i used ai i will continue to use it i will never hide it but never will i ever blindly follow it if you looked in the post you may see the effort i put into it i did a lot bro a lot i i could trust the ai i always douted it will halucitnate i would use dif ais at the very least i read the summary or conclusion and mostly read the full studies when they are free to access

 

[teddy101]

This is already known for most, and it’s legit

Your few days of research with ai still doesn’t make me care in the slightest, if i have to suffer and find studies individually and summarise them and compile etc i will not let shit like this slide

i would be happy to know where to find studies very happy its just i cant find studies idk where to search besides using ai make you more effective if you attualy do your research stop being so anti ai may be try to use it use ai or not the conclusion will still be the same

 

[Zagro]

wh

valid but you have to make the distinction i used ai i will continue to use it i will never hide it but never will i ever blindly follow it if you looked in the post you may see the effort i put into it i did a lot bro a lot i i could trust the ai i always douted it will halucitnate i would use dif ais at the very least i read the summary or conclusion and mostly read the full studies when they are free to access

Okay you’re cool i like you now

Let me read the papers and give you a high effort response now that i know you aren’t a retard, btw the reason for me pressing you like this is also because of retards as the likes of Ahmed88

 

[teddy101]

Okay you’re cool i like you now

Let me read the papers and give you a high effort response now that i know you aren’t a retard, btw the reason for me pressing you like this is also because of retards as the likes of Ahmed88

thank you bro this all i wanted thanks so much

 

[Zagro]

i would be happy to know where to find studies very happy its just i cant find studies idk where to search besides using ai make you more effective if you attualy do your research stop being so anti ai may be try to use it use ai or not the conclusion will still be the same

I used ai when i first began aswell its practical but the thing is that it wont extrapolate/think outside the box it like connect the dots for you, it just answers whatever question you ask it

You can get him to agree on both sides of an argument easily, that’s why i stopped depending on it

 

[teddy101]

I used ai when i first began aswell its practical but the thing is that it wont extrapolate/think outside the box it like connect the dots for you, it just answers whatever question you ask it

You can get him to agree on both sides of an argument easily, that’s why i stopped depending on it

bro after you finish accessing my theory i wuold like to know what you use to research

 

[teddy101]

I used ai when i first began aswell its practical but the thing is that it wont extrapolate/think outside the box it like connect the dots for you, it just answers whatever question you ask it

You can get him to agree on both sides of an argument easily, that’s why i stopped depending on it

don't use llm use consensus you cant make it agree on sum thing that doesn't work it purely analytical and shows what the general consensus is, it and elict is all i use after you tell me what you use to find your research i only use concensus but it only general and doesn't account for limitations of studies and methodology etc you have to manualy check every thing, you should only use it when your trying to see what the surface says and basic things not the actual deep truth i didnt use it for this tho i used it to find palatal matruation stage

 

[garfyld]

You need BMP2 for bigger facial bones bro

 

[teddy101]

You need BMP2 for bigger facial bones bro

thx bro let me research but what is the correlation

 

[teddy101]

BMP2

You need BMP2 for bigger facial bones bro

thank you so mcuh now i got another method for bone growth that was very help full but i need to research way more

 

[teddy101]

Okay you’re cool i like you now

Let me read the papers and give you a high effort response now that i know you aren’t a retard, btw the reason for me pressing you like this is also because of retards as the likes of Ahmed88

https://looksmax.org/threads/ahmed88-intellectually-disabled-or-trolling.1889868/ such a tard i hate when people mention genetic height limit jus cuz an ai told them they cant be taller than their height genes like it basic math and perfect simple blue print most of the propaganda was spread by tiktok and ai when people talked to them about roids but i hate them tards so much i understand where you are coming from i saw a tiktok just like that with the same "proof" i hate people are this dumb

 

[teddy101]

Okay you’re cool i like you now

Let me read the papers and give you a high effort response now that i know you aren’t a retard, btw the reason for me pressing you like this is also because of retards as the likes of Ahmed88

still waiting for the response bro n wat u use

 

[teddy101]

Okay you’re cool i like you now

Let me read the papers and give you a high effort response now that i know you aren’t a retard, btw the reason for me pressing you like this is also because of retards as the likes of Ahmed88

you died or sum?

 

[teddy101]

Okay you’re cool i like you now

Let me read the papers and give you a high effort response now that i know you aren’t a retard, btw the reason for me pressing you like this is also because of retards as the likes of Ahmed88

gng where my high effort response i am waiting

 

[MindOfBeni]

12IU HGH plus insulin?

 

[HubertSkeletrix]

dnr

 

[teddy101]

12IU HGH plus insulin?

injected insulin doesnt actually has worse effect for igf-1 there was a study that measured it i think besied it should be chronic not a spike a spike does nothing will provide with the study if you want but not that sure tho

 

[childishkillah]

Serious

 

[MindOfBeni]

injected insulin doesnt actually has worse effect for igf-1 there was a study that measured it i think besied it should be chronic not a spike a spike does nothing will provide with the study if you want but not that sure tho

only looked this thing up trough grok and he said it could work

 

[teddy101]

only looked this thing up trough grok and he said it could work

here is the study that shows acute insulin spike decrease free igf-1 https://sci-hub.africa/10.1210/jcem.82.7.4070 or the DOI https://doi.org/10.1210/jcem.82.7.4070 i didnt read it all but they gave them injected insulin and saw 20% decrease in free igf-1

 

[teddy101]

only looked this thing up trough grok and he said it could work

The time courses of free IGF-I, total IGF-I, IGFBP-1, andIGFBP-3 during the FSIGT are shown in Fig. 1. Total IGF-Ilevels remained constant throughout the experiment. FreeIGF-I declined from 0.89 6 0.21 mg/L (basal) to 0.71 6 0.17mg/L at 19 min and 0.59 6 0.15 mg/L at 40 min. Thereafter,free IGF-I levels did not change significantly until the end ofthe experiment at 180 min. After glucose injection, serumIGFBP-1 remained at basal levels for about 50 min. IGFBP-1fell steadily thereafter, resulting in an undershoot by 100 min,then sharply increased by about 3-fold by the end of theexperiment at 180 min. IGFBP-3 levels remained constant forabout 30 min, then they increased linearly thereafter to about20% above basal values. this from the study so injection doesnt work

 

[AtrophicPyra]

gpt

does blud know injecting insulin is also an option instead of trying to be obese

heh hepatic igf1 saaaar growth plate circadian rythm heh dark triad genius