neveroverbro
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This is for all the niggers who think basketball is cope for height
First lets start with the anatomy of a bone and with basketball we have the femur tibia and the fibula. All of these have two heads ( everyone knows this but since ur retarded ill still explain it ) and when someone plays basketball then the for es that work on it as said in wolffs law days that these forces apply pressure which makes it so everytime a player lands a jump the compressive forces send shockeaves through the skeletal system. this mechanical loading creates microscopic cracks in the mineralised matrix of the bone and this activates the BMU ( basic multicellular unit ) which responds with mechanotransduction. The bone matrix contains a network of osteo sites. These are embedded in the fluid filled channel called the lacunocanalicular network. Basically the bone sends slightly when landing and this changes the fluid shear stress. The osteoporosis sites sends the shift and flags the micro cracks anf the body recruits osteoclasts to the site they secrete enzymes and acids to clear out the site to form a small cavity . Then osteoblasts migrate into the cavity. They lay down ostioids. Which is a flexible unmineralised matrix made primarily of type 1 collagen, over several months collagen and sulfates deposit calcium and phosphAte into the osteoid to form hydroxyapatite crystals. Because the initial stimulus was a high load impact the osteoblasts over ompensation laying down more matrix then what there was original causing thicker and denser bones
The other mechanism involves actual microtears in the tendons. Think of the quadtriceps and patellar tendons pulling violently on the tibial tuberosity during a sprint and a jump.
The point where a tendon inserts into the bone is called the enthesis. Repetitive explosive jumping causes microtears in the tendon
fibers right at this junction
The localised inflammation and the shear physical traction pulling on the periosteum ( the bones outer membrane stimulates osteoblasts activity exactly at the insertion site which overtime calcifies the bone further into the tendon to strengthen the anchor point. )
This is why athletes often develop thicker bones where majoy bones attached.
First lets start with the anatomy of a bone and with basketball we have the femur tibia and the fibula. All of these have two heads ( everyone knows this but since ur retarded ill still explain it ) and when someone plays basketball then the for es that work on it as said in wolffs law days that these forces apply pressure which makes it so everytime a player lands a jump the compressive forces send shockeaves through the skeletal system. this mechanical loading creates microscopic cracks in the mineralised matrix of the bone and this activates the BMU ( basic multicellular unit ) which responds with mechanotransduction. The bone matrix contains a network of osteo sites. These are embedded in the fluid filled channel called the lacunocanalicular network. Basically the bone sends slightly when landing and this changes the fluid shear stress. The osteoporosis sites sends the shift and flags the micro cracks anf the body recruits osteoclasts to the site they secrete enzymes and acids to clear out the site to form a small cavity . Then osteoblasts migrate into the cavity. They lay down ostioids. Which is a flexible unmineralised matrix made primarily of type 1 collagen, over several months collagen and sulfates deposit calcium and phosphAte into the osteoid to form hydroxyapatite crystals. Because the initial stimulus was a high load impact the osteoblasts over ompensation laying down more matrix then what there was original causing thicker and denser bones
The other mechanism involves actual microtears in the tendons. Think of the quadtriceps and patellar tendons pulling violently on the tibial tuberosity during a sprint and a jump.
The point where a tendon inserts into the bone is called the enthesis. Repetitive explosive jumping causes microtears in the tendon
fibers right at this junction
The localised inflammation and the shear physical traction pulling on the periosteum ( the bones outer membrane stimulates osteoblasts activity exactly at the insertion site which overtime calcifies the bone further into the tendon to strengthen the anchor point. )
This is why athletes often develop thicker bones where majoy bones attached.
