Why Bonesmashing Doesn't Work.

[polarpop]

LONG IN DEPTH READ, IF YOU KNOW BONESMAHSING IS BULLSHIT YOU DONT NEED TO READ

1. The “mechanostat” rule (this is what people misunderstand)​

Bone adaptation is governed by the mechanostat theory (Harold Frost).

There are 3 key strain zones:
Low strain to bone resorption
Physiological strain to bone maintenance
High strain (controlled andrepetitive) to bone formation

Critical detail: Bone growth only occurs when strain is within a narrow adaptive window AND repeated thousands of times

Bonesmashing:

too high magnitude
too low frequency
too irregular
This puts it in the damage zone, not the adaptation zone

2. Mechanotransduction requires REPETITION, not impact​

Bone cells respond to:

cyclic loading (so walking, chewing, lifting)
fluid shear stress in canaliculi (tiny channels in bone)

This requires:

requency (hundreds, thousands of cycles)
consistent strain direction

Bonesmashing:

single impacts
random direction
no cumulative signal
No meaningful osteocyte signaling so noo growth stimulus

3. What actually happens when you hit bone​

Impact to sequence:

1. Microdamage
2. Osteocyte apoptosis (cell death)
3. Osteoclast recruitment (bone removal)
4. Osteoblast repair (restore original structure)

Important: The body prioritizes restoring integrity, not enhancing shape

Net effect:

zero net outward growth
sometimes net loss if damage exceeds repair

4. Periosteal apposition why bonesmashing doesn’t trigger it
Periosteal apposition = adding bone to outer surface.

Requires:
sustained mechanical tension
hormonal environment (IGF-1, GH, testosterone)
long-term loading patterns

Bonesmashing provides:

acute compression
inflammation
no sustained tension signal

You are NOT activating periosteal osteoblasts in a meaningful way

5. Load-bearing vs non load bearin bones (this is a big difference)​

Tibia (shin):​

carries bodyweight
experiences axial loading every step
adapted for mechanical stress
high mechanosensitivity

Facial bones:​

NOT load bearing
minimal strain environment
low mechanostat threshold activation

the face is biologically protected from mechanical remodeling

​

6. Why shin conditioning “works”​

Fighters condition shins through:

repeated sub-threshold impacts
gradual loading over YEARS

This results in:

increased bone mineral density
slight cortical thickening

NOT:

large size increase
reshaping

​

7. Direction of force matters more than people think​

Bone adapts to:

predictable, directional stress

Examples:

tibia: vertical compression
humerus bending + torsion

Bonesmashing:

chaotic force vectors
no consistent loading axis
Bone cannot “learn” or adapt structurally

8. Craniofacial bones are fundamentally different​

Facial bones:

formed via intramembranous ossification
remodel primarily through:
growth (youth)
dental forces (orthodontics)
They are NOT designed for:

mechanical hypertrophy
load-driven expansion

Also:
utures fuse after adolescence growth potential drops sharply

​

9. Orthodontics proves the opposite of bonesmashing​

To move bone:
you need constant low force for months

Process:

pressure side to resorption
tension side to formation

This is:

slow
controlled
directional
Bonesmashing is:

fast
uncontrolled
compressive only
completely different biological pathway

10. The UFC fighter or fighter myth in general​

People say:
UFC fighters have good bone structure impacts must grow bone

This is fucking bullshit.

Reality:​

(A) Selection bias​

Fighters already have:

strong jaws
favorable bone structure
high testosterone profiles
They’re selected FOR these traits.

(B) Survivorship bias​

Fighters with weak bones get injured early
You only see the durable ones

(C) Training ≠ facial impact​

Most adaptation comes from:

lifting
grappling
overall skeletal loading
NOT getting punched.

(D) Facial trauma effects​

Repeated hits cause:

fractures
nasal deviation
orbital damage
asymmetry
NOT:
aesthetic enhancement

If impacts improved bone:
fighters would look more symmetrical over time

They clearly fucking don’t.

11. Why impact does NOT equal growth (core principle)​

Bone responds to:
strain energy over time

NOT:
peak force

Example:

1 heavy hit ≠ 1000 light loads
Adaptation depends on:

total mechanical work
signal frequency
Bonesmashing fails both.

12. Why people THINK it works​

All illusions:

(A) Inflammation​

swelling = temporary “fullness”

(B) Fat loss over time​

sharper face misattributed to smashing

(C) Skin thickening​

repeated trauma = fibrosis
can create illusion of “structure”

(D) Psychological bias​

expectation = perceived result

​

13. When bone CAN actually change shape​

Only in these cases:

growth (before plates close)
orthodontics (controlled force)
surgery (osteotomy)
disease (acromegaly, etc.)

That’s it.

14. Final synthesis​

Bonesmashing fails because:

It produces damage, not adaptive strain
It lacks frequency, direction, and duration
It activates repair pathways, not growth pathways
Facial bones are non-load-bearing and tightly regulated
Periosteal apposition requires systemic + long-term signals, not impacts

 

[dhusc]

LONG IN DEPTH READ, IF YOU KNOW BONESMAHSING IS BULLSHIT YOU DONT NEED TO READ

1. The “mechanostat” rule (this is what people misunderstand)​

Bone adaptation is governed by the mechanostat theory (Harold Frost).

There are 3 key strain zones:
Low strain to bone resorption
Physiological strain to bone maintenance
High strain (controlled andrepetitive) to bone formation

Critical detail: Bone growth only occurs when strain is within a narrow adaptive window AND repeated thousands of times

Bonesmashing:

too high magnitude
too low frequency
too irregular
This puts it in the damage zone, not the adaptation zone

2. Mechanotransduction requires REPETITION, not impact​

Bone cells respond to:

cyclic loading (so walking, chewing, lifting)
fluid shear stress in canaliculi (tiny channels in bone)

This requires:

requency (hundreds, thousands of cycles)
consistent strain direction

Bonesmashing:

single impacts
random direction
no cumulative signal
No meaningful osteocyte signaling so noo growth stimulus

3. What actually happens when you hit bone​

Impact to sequence:

1. Microdamage
2. Osteocyte apoptosis (cell death)
3. Osteoclast recruitment (bone removal)
4. Osteoblast repair (restore original structure)

Important: The body prioritizes restoring integrity, not enhancing shape

Net effect:

zero net outward growth
sometimes net loss if damage exceeds repair

4. Periosteal apposition why bonesmashing doesn’t trigger it
Periosteal apposition = adding bone to outer surface.

Requires:
sustained mechanical tension
hormonal environment (IGF-1, GH, testosterone)
long-term loading patterns

Bonesmashing provides:

acute compression
inflammation
no sustained tension signal

You are NOT activating periosteal osteoblasts in a meaningful way

5. Load-bearing vs non load bearin bones (this is a big difference)​

Tibia (shin):​

carries bodyweight
experiences axial loading every step
adapted for mechanical stress
high mechanosensitivity

Facial bones:​

NOT load bearing
minimal strain environment
low mechanostat threshold activation

the face is biologically protected from mechanical remodeling

​

6. Why shin conditioning “works”​

Fighters condition shins through:

repeated sub-threshold impacts
gradual loading over YEARS

This results in:

increased bone mineral density
slight cortical thickening

NOT:

large size increase
reshaping

​

7. Direction of force matters more than people think​

Bone adapts to:

predictable, directional stress

Examples:

tibia: vertical compression
humerus bending + torsion

Bonesmashing:

chaotic force vectors
no consistent loading axis
Bone cannot “learn” or adapt structurally

8. Craniofacial bones are fundamentally different​

Facial bones:

formed via intramembranous ossification
remodel primarily through:
growth (youth)
dental forces (orthodontics)
They are NOT designed for:

mechanical hypertrophy
load-driven expansion

Also:
utures fuse after adolescence growth potential drops sharply

​

9. Orthodontics proves the opposite of bonesmashing​

To move bone:
you need constant low force for months

Process:

pressure side to resorption
tension side to formation

This is:

slow
controlled
directional
Bonesmashing is:

fast
uncontrolled
compressive only
completely different biological pathway

10. The UFC fighter or fighter myth in general​

People say:
UFC fighters have good bone structure impacts must grow bone

This is fucking bullshit.

Reality:​

(A) Selection bias​

Fighters already have:

strong jaws
favorable bone structure
high testosterone profiles
They’re selected FOR these traits.

(B) Survivorship bias​

Fighters with weak bones get injured early
You only see the durable ones

(C) Training ≠ facial impact​

Most adaptation comes from:

lifting
grappling
overall skeletal loading
NOT getting punched.

(D) Facial trauma effects​

Repeated hits cause:

fractures
nasal deviation
orbital damage
asymmetry
NOT:
aesthetic enhancement

If impacts improved bone:
fighters would look more symmetrical over time

They clearly fucking don’t.

11. Why impact does NOT equal growth (core principle)​

Bone responds to:
strain energy over time

NOT:
peak force

Example:

1 heavy hit ≠ 1000 light loads
Adaptation depends on:

total mechanical work
signal frequency
Bonesmashing fails both.

12. Why people THINK it works​

All illusions:

(A) Inflammation​

swelling = temporary “fullness”

(B) Fat loss over time​

sharper face misattributed to smashing

(C) Skin thickening​

repeated trauma = fibrosis
can create illusion of “structure”

(D) Psychological bias​

expectation = perceived result

​

13. When bone CAN actually change shape​

Only in these cases:

growth (before plates close)
orthodontics (controlled force)
surgery (osteotomy)
disease (acromegaly, etc.)

That’s it.

14. Final synthesis (high level but precise)​

Bonesmashing fails because:

• It produces damage, not adaptive strain
• It lacks frequency, direction, and duration
• It activates repair pathways, not growth pathways
• Facial bones are non-load-bearing and tightly regulated
• Periosteal apposition requires systemic + long-term signals, not impacts

DNR NIGGER

 

[Finncelchud]

DNR, but will read later

 

[Jgns]

water

 

[Hernan]

it does work though, my source you may ask?

The Aromatic Revolution: Kekulé's Benzene Breakthrough

The history of chemistry is marked by moments of revelation, where the discovery of a fundamental concept reshapes our understanding of the molecular world. One such pivotal moment occurred in the mid-19th century when the structure of benzene, a compound known for its unique aromatic...

thecalculatedchemist.com

 

[slimester]

thanks for ur opinion lil bro

 

[polarpop]

water

not enough people know lmao but it is indeed water

 

[ImaASCENDsoon]

LONG IN DEPTH READ, IF YOU KNOW BONESMAHSING IS BULLSHIT YOU DONT NEED TO READ

1. The “mechanostat” rule (this is what people misunderstand)​

Bone adaptation is governed by the mechanostat theory (Harold Frost).

There are 3 key strain zones:
Low strain to bone resorption
Physiological strain to bone maintenance
High strain (controlled andrepetitive) to bone formation

Critical detail: Bone growth only occurs when strain is within a narrow adaptive window AND repeated thousands of times

Bonesmashing:

too high magnitude
too low frequency
too irregular
This puts it in the damage zone, not the adaptation zone

2. Mechanotransduction requires REPETITION, not impact​

Bone cells respond to:

cyclic loading (so walking, chewing, lifting)
fluid shear stress in canaliculi (tiny channels in bone)

This requires:

requency (hundreds, thousands of cycles)
consistent strain direction

Bonesmashing:

single impacts
random direction
no cumulative signal
No meaningful osteocyte signaling so noo growth stimulus

3. What actually happens when you hit bone​

Impact to sequence:

1. Microdamage
2. Osteocyte apoptosis (cell death)
3. Osteoclast recruitment (bone removal)
4. Osteoblast repair (restore original structure)

Important: The body prioritizes restoring integrity, not enhancing shape

Net effect:

zero net outward growth
sometimes net loss if damage exceeds repair

4. Periosteal apposition why bonesmashing doesn’t trigger it
Periosteal apposition = adding bone to outer surface.

Requires:
sustained mechanical tension
hormonal environment (IGF-1, GH, testosterone)
long-term loading patterns

Bonesmashing provides:

acute compression
inflammation
no sustained tension signal

You are NOT activating periosteal osteoblasts in a meaningful way

5. Load-bearing vs non load bearin bones (this is a big difference)​

Tibia (shin):​

carries bodyweight
experiences axial loading every step
adapted for mechanical stress
high mechanosensitivity

Facial bones:​

NOT load bearing
minimal strain environment
low mechanostat threshold activation

the face is biologically protected from mechanical remodeling

​

6. Why shin conditioning “works”​

Fighters condition shins through:

repeated sub-threshold impacts
gradual loading over YEARS

This results in:

increased bone mineral density
slight cortical thickening

NOT:

large size increase
reshaping

​

7. Direction of force matters more than people think​

Bone adapts to:

predictable, directional stress

Examples:

tibia: vertical compression
humerus bending + torsion

Bonesmashing:

chaotic force vectors
no consistent loading axis
Bone cannot “learn” or adapt structurally

8. Craniofacial bones are fundamentally different​

Facial bones:

formed via intramembranous ossification
remodel primarily through:
growth (youth)
dental forces (orthodontics)
They are NOT designed for:

mechanical hypertrophy
load-driven expansion

Also:
utures fuse after adolescence growth potential drops sharply

​

9. Orthodontics proves the opposite of bonesmashing​

To move bone:
you need constant low force for months

Process:

pressure side to resorption
tension side to formation

This is:

slow
controlled
directional
Bonesmashing is:

fast
uncontrolled
compressive only
completely different biological pathway

10. The UFC fighter or fighter myth in general​

People say:
UFC fighters have good bone structure impacts must grow bone

This is fucking bullshit.

Reality:​

(A) Selection bias​

Fighters already have:

strong jaws
favorable bone structure
high testosterone profiles
They’re selected FOR these traits.

(B) Survivorship bias​

Fighters with weak bones get injured early
You only see the durable ones

(C) Training ≠ facial impact​

Most adaptation comes from:

lifting
grappling
overall skeletal loading
NOT getting punched.

(D) Facial trauma effects​

Repeated hits cause:

fractures
nasal deviation
orbital damage
asymmetry
NOT:
aesthetic enhancement

If impacts improved bone:
fighters would look more symmetrical over time

They clearly fucking don’t.

11. Why impact does NOT equal growth (core principle)​

Bone responds to:
strain energy over time

NOT:
peak force

Example:

1 heavy hit ≠ 1000 light loads
Adaptation depends on:

total mechanical work
signal frequency
Bonesmashing fails both.

12. Why people THINK it works​

All illusions:

(A) Inflammation​

swelling = temporary “fullness”

(B) Fat loss over time​

sharper face misattributed to smashing

(C) Skin thickening​

repeated trauma = fibrosis
can create illusion of “structure”

(D) Psychological bias​

expectation = perceived result

​

13. When bone CAN actually change shape​

Only in these cases:

growth (before plates close)
orthodontics (controlled force)
surgery (osteotomy)
disease (acromegaly, etc.)

That’s it.

14. Final synthesis​

Bonesmashing fails because:

It produces damage, not adaptive strain
It lacks frequency, direction, and duration
It activates repair pathways, not growth pathways
Facial bones are non-load-bearing and tightly regulated
Periosteal apposition requires systemic + long-term signals, not impacts

Not reading. It works and causes scar tissue which makes ur zygos mog.

 

[polarpop]

Not reading. It works and causes scar tissue which makes ur zygos mog.

scar tissue is fibrotic soft tissue not bone it forms irregularly doesnt change your skeletal structure and typically worsens facial aesthetics through asymmetry and lumpiness ur not increasing zygomatic projection and it wont make your zygos mog lmao

 

[tomahawk]

I can't summon you... let me be your successor, @SpectrumAesthetics3

(PICS) Definitively documenting years of bonesmashing changes + Protocol | All denial ends here tbh

*This is a repost with permission – old one got deleted yesterday due to me having had other bonesmashing transformation posts in my history already. Shoutout to @Aryan Incel for getting them deleted so that I could repost this without violating any rules. Because those posts were more...

looksmax.org

 

[foidslayer92910]

LONG IN DEPTH READ, IF YOU KNOW BONESMAHSING IS BULLSHIT YOU DONT NEED TO READ

1. The “mechanostat” rule (this is what people misunderstand)​

Bone adaptation is governed by the mechanostat theory (Harold Frost).

There are 3 key strain zones:
Low strain to bone resorption
Physiological strain to bone maintenance
High strain (controlled andrepetitive) to bone formation

Critical detail: Bone growth only occurs when strain is within a narrow adaptive window AND repeated thousands of times

Bonesmashing:

too high magnitude
too low frequency
too irregular
This puts it in the damage zone, not the adaptation zone

2. Mechanotransduction requires REPETITION, not impact​

Bone cells respond to:

cyclic loading (so walking, chewing, lifting)
fluid shear stress in canaliculi (tiny channels in bone)

This requires:

requency (hundreds, thousands of cycles)
consistent strain direction

Bonesmashing:

single impacts
random direction
no cumulative signal
No meaningful osteocyte signaling so noo growth stimulus

3. What actually happens when you hit bone​

Impact to sequence:

1. Microdamage
2. Osteocyte apoptosis (cell death)
3. Osteoclast recruitment (bone removal)
4. Osteoblast repair (restore original structure)

Important: The body prioritizes restoring integrity, not enhancing shape

Net effect:

zero net outward growth
sometimes net loss if damage exceeds repair

4. Periosteal apposition why bonesmashing doesn’t trigger it
Periosteal apposition = adding bone to outer surface.

Requires:
sustained mechanical tension
hormonal environment (IGF-1, GH, testosterone)
long-term loading patterns

Bonesmashing provides:

acute compression
inflammation
no sustained tension signal

You are NOT activating periosteal osteoblasts in a meaningful way

5. Load-bearing vs non load bearin bones (this is a big difference)​

Tibia (shin):​

carries bodyweight
experiences axial loading every step
adapted for mechanical stress
high mechanosensitivity

Facial bones:​

NOT load bearing
minimal strain environment
low mechanostat threshold activation

the face is biologically protected from mechanical remodeling

​

6. Why shin conditioning “works”​

Fighters condition shins through:

repeated sub-threshold impacts
gradual loading over YEARS

This results in:

increased bone mineral density
slight cortical thickening

NOT:

large size increase
reshaping

​

7. Direction of force matters more than people think​

Bone adapts to:

predictable, directional stress

Examples:

tibia: vertical compression
humerus bending + torsion

Bonesmashing:

chaotic force vectors
no consistent loading axis
Bone cannot “learn” or adapt structurally

8. Craniofacial bones are fundamentally different​

Facial bones:

formed via intramembranous ossification
remodel primarily through:
growth (youth)
dental forces (orthodontics)
They are NOT designed for:

mechanical hypertrophy
load-driven expansion

Also:
utures fuse after adolescence growth potential drops sharply

​

9. Orthodontics proves the opposite of bonesmashing​

To move bone:
you need constant low force for months

Process:

pressure side to resorption
tension side to formation

This is:

slow
controlled
directional
Bonesmashing is:

fast
uncontrolled
compressive only
completely different biological pathway

10. The UFC fighter or fighter myth in general​

People say:
UFC fighters have good bone structure impacts must grow bone

This is fucking bullshit.

Reality:​

(A) Selection bias​

Fighters already have:

strong jaws
favorable bone structure
high testosterone profiles
They’re selected FOR these traits.

(B) Survivorship bias​

Fighters with weak bones get injured early
You only see the durable ones

(C) Training ≠ facial impact​

Most adaptation comes from:

lifting
grappling
overall skeletal loading
NOT getting punched.

(D) Facial trauma effects​

Repeated hits cause:

fractures
nasal deviation
orbital damage
asymmetry
NOT:
aesthetic enhancement

If impacts improved bone:
fighters would look more symmetrical over time

They clearly fucking don’t.

11. Why impact does NOT equal growth (core principle)​

Bone responds to:
strain energy over time

NOT:
peak force

Example:

1 heavy hit ≠ 1000 light loads
Adaptation depends on:

total mechanical work
signal frequency
Bonesmashing fails both.

12. Why people THINK it works​

All illusions:

(A) Inflammation​

swelling = temporary “fullness”

(B) Fat loss over time​

sharper face misattributed to smashing

(C) Skin thickening​

repeated trauma = fibrosis
can create illusion of “structure”

(D) Psychological bias​

expectation = perceived result

​

13. When bone CAN actually change shape​

Only in these cases:

growth (before plates close)
orthodontics (controlled force)
surgery (osteotomy)
disease (acromegaly, etc.)

That’s it.

14. Final synthesis​

Bonesmashing fails because:

It produces damage, not adaptive strain
It lacks frequency, direction, and duration
It activates repair pathways, not growth pathways
Facial bones are non-load-bearing and tightly regulated
Periosteal apposition requires systemic + long-term signals, not impacts

Dnr

 

[polarpop]

I can't summon you... let me be your successor, @SpectrumAesthetics3

(PICS) Definitively documenting years of bonesmashing changes + Protocol | All denial ends here tbh

*This is a repost with permission – old one got deleted yesterday due to me having had other bonesmashing transformation posts in my history already. Shoutout to @Aryan Incel for getting them deleted so that I could repost this without violating any rules. Because those posts were more...

looksmax.org

Gonna respond to this when I’m home but looks like a decent thread

 

[wonderlen]

Not reading. It works and causes scar tissue which makes ur zygos mog.

You are stupid

 

[wonderlen]

LONG IN DEPTH READ, IF YOU KNOW BONESMAHSING IS BULLSHIT YOU DONT NEED TO READ

1. The “mechanostat” rule (this is what people misunderstand)​

Bone adaptation is governed by the mechanostat theory (Harold Frost).

There are 3 key strain zones:
Low strain to bone resorption
Physiological strain to bone maintenance
High strain (controlled andrepetitive) to bone formation

Critical detail: Bone growth only occurs when strain is within a narrow adaptive window AND repeated thousands of times

Bonesmashing:

too high magnitude
too low frequency
too irregular
This puts it in the damage zone, not the adaptation zone

2. Mechanotransduction requires REPETITION, not impact​

Bone cells respond to:

cyclic loading (so walking, chewing, lifting)
fluid shear stress in canaliculi (tiny channels in bone)

This requires:

requency (hundreds, thousands of cycles)
consistent strain direction

Bonesmashing:

single impacts
random direction
no cumulative signal
No meaningful osteocyte signaling so noo growth stimulus

3. What actually happens when you hit bone​

Impact to sequence:

1. Microdamage
2. Osteocyte apoptosis (cell death)
3. Osteoclast recruitment (bone removal)
4. Osteoblast repair (restore original structure)

Important: The body prioritizes restoring integrity, not enhancing shape

Net effect:

zero net outward growth
sometimes net loss if damage exceeds repair

4. Periosteal apposition why bonesmashing doesn’t trigger it
Periosteal apposition = adding bone to outer surface.

Requires:
sustained mechanical tension
hormonal environment (IGF-1, GH, testosterone)
long-term loading patterns

Bonesmashing provides:

acute compression
inflammation
no sustained tension signal

You are NOT activating periosteal osteoblasts in a meaningful way

5. Load-bearing vs non load bearin bones (this is a big difference)​

Tibia (shin):​

carries bodyweight
experiences axial loading every step
adapted for mechanical stress
high mechanosensitivity

Facial bones:​

NOT load bearing
minimal strain environment
low mechanostat threshold activation

the face is biologically protected from mechanical remodeling

​

6. Why shin conditioning “works”​

Fighters condition shins through:

repeated sub-threshold impacts
gradual loading over YEARS

This results in:

increased bone mineral density
slight cortical thickening

NOT:

large size increase
reshaping

​

7. Direction of force matters more than people think​

Bone adapts to:

predictable, directional stress

Examples:

tibia: vertical compression
humerus bending + torsion

Bonesmashing:

chaotic force vectors
no consistent loading axis
Bone cannot “learn” or adapt structurally

8. Craniofacial bones are fundamentally different​

Facial bones:

formed via intramembranous ossification
remodel primarily through:
growth (youth)
dental forces (orthodontics)
They are NOT designed for:

mechanical hypertrophy
load-driven expansion

Also:
utures fuse after adolescence growth potential drops sharply

​

9. Orthodontics proves the opposite of bonesmashing​

To move bone:
you need constant low force for months

Process:

pressure side to resorption
tension side to formation

This is:

slow
controlled
directional
Bonesmashing is:

fast
uncontrolled
compressive only
completely different biological pathway

10. The UFC fighter or fighter myth in general​

People say:
UFC fighters have good bone structure impacts must grow bone

This is fucking bullshit.

Reality:​

(A) Selection bias​

Fighters already have:

strong jaws
favorable bone structure
high testosterone profiles
They’re selected FOR these traits.

(B) Survivorship bias​

Fighters with weak bones get injured early
You only see the durable ones

(C) Training ≠ facial impact​

Most adaptation comes from:

lifting
grappling
overall skeletal loading
NOT getting punched.

(D) Facial trauma effects​

Repeated hits cause:

fractures
nasal deviation
orbital damage
asymmetry
NOT:
aesthetic enhancement

If impacts improved bone:
fighters would look more symmetrical over time

They clearly fucking don’t.

11. Why impact does NOT equal growth (core principle)​

Bone responds to:
strain energy over time

NOT:
peak force

Example:

1 heavy hit ≠ 1000 light loads
Adaptation depends on:

total mechanical work
signal frequency
Bonesmashing fails both.

12. Why people THINK it works​

All illusions:

(A) Inflammation​

swelling = temporary “fullness”

(B) Fat loss over time​

sharper face misattributed to smashing

(C) Skin thickening​

repeated trauma = fibrosis
can create illusion of “structure”

(D) Psychological bias​

expectation = perceived result

​

13. When bone CAN actually change shape​

Only in these cases:

growth (before plates close)
orthodontics (controlled force)
surgery (osteotomy)
disease (acromegaly, etc.)

That’s it.

14. Final synthesis​

Bonesmashing fails because:

It produces damage, not adaptive strain
It lacks frequency, direction, and duration
It activates repair pathways, not growth pathways
Facial bones are non-load-bearing and tightly regulated
Periosteal apposition requires systemic + long-term signals, not impacts

How tf do people disagree

 

[polarpop]

How tf do people disagree

Dunno

 

[iwishicouldbediff]

Dnr tiktokcels are retarded anyways

 

[test monster]

LONG IN DEPTH READ, IF YOU KNOW BONESMAHSING IS BULLSHIT YOU DONT NEED TO READ

1. The “mechanostat” rule (this is what people misunderstand)​

Bone adaptation is governed by the mechanostat theory (Harold Frost).

There are 3 key strain zones:
Low strain to bone resorption
Physiological strain to bone maintenance
High strain (controlled andrepetitive) to bone formation

Critical detail: Bone growth only occurs when strain is within a narrow adaptive window AND repeated thousands of times

Bonesmashing:

too high magnitude
too low frequency
too irregular
This puts it in the damage zone, not the adaptation zone

2. Mechanotransduction requires REPETITION, not impact​

Bone cells respond to:

cyclic loading (so walking, chewing, lifting)
fluid shear stress in canaliculi (tiny channels in bone)

This requires:

requency (hundreds, thousands of cycles)
consistent strain direction

Bonesmashing:

single impacts
random direction
no cumulative signal
No meaningful osteocyte signaling so noo growth stimulus

3. What actually happens when you hit bone​

Impact to sequence:

1. Microdamage
2. Osteocyte apoptosis (cell death)
3. Osteoclast recruitment (bone removal)
4. Osteoblast repair (restore original structure)

Important: The body prioritizes restoring integrity, not enhancing shape

Net effect:

zero net outward growth
sometimes net loss if damage exceeds repair

4. Periosteal apposition why bonesmashing doesn’t trigger it
Periosteal apposition = adding bone to outer surface.

Requires:
sustained mechanical tension
hormonal environment (IGF-1, GH, testosterone)
long-term loading patterns

Bonesmashing provides:

acute compression
inflammation
no sustained tension signal

You are NOT activating periosteal osteoblasts in a meaningful way

5. Load-bearing vs non load bearin bones (this is a big difference)​

Tibia (shin):​

carries bodyweight
experiences axial loading every step
adapted for mechanical stress
high mechanosensitivity

Facial bones:​

NOT load bearing
minimal strain environment
low mechanostat threshold activation

the face is biologically protected from mechanical remodeling

​

6. Why shin conditioning “works”​

Fighters condition shins through:

repeated sub-threshold impacts
gradual loading over YEARS

This results in:

increased bone mineral density
slight cortical thickening

NOT:

large size increase
reshaping

​

7. Direction of force matters more than people think​

Bone adapts to:

predictable, directional stress

Examples:

tibia: vertical compression
humerus bending + torsion

Bonesmashing:

chaotic force vectors
no consistent loading axis
Bone cannot “learn” or adapt structurally

8. Craniofacial bones are fundamentally different​

Facial bones:

formed via intramembranous ossification
remodel primarily through:
growth (youth)
dental forces (orthodontics)
They are NOT designed for:

mechanical hypertrophy
load-driven expansion

Also:
utures fuse after adolescence growth potential drops sharply

​

9. Orthodontics proves the opposite of bonesmashing​

To move bone:
you need constant low force for months

Process:

pressure side to resorption
tension side to formation

This is:

slow
controlled
directional
Bonesmashing is:

fast
uncontrolled
compressive only
completely different biological pathway

10. The UFC fighter or fighter myth in general​

People say:
UFC fighters have good bone structure impacts must grow bone

This is fucking bullshit.

Reality:​

(A) Selection bias​

Fighters already have:

strong jaws
favorable bone structure
high testosterone profiles
They’re selected FOR these traits.

(B) Survivorship bias​

Fighters with weak bones get injured early
You only see the durable ones

(C) Training ≠ facial impact​

Most adaptation comes from:

lifting
grappling
overall skeletal loading
NOT getting punched.

(D) Facial trauma effects​

Repeated hits cause:

fractures
nasal deviation
orbital damage
asymmetry
NOT:
aesthetic enhancement

If impacts improved bone:
fighters would look more symmetrical over time

They clearly fucking don’t.

11. Why impact does NOT equal growth (core principle)​

Bone responds to:
strain energy over time

NOT:
peak force

Example:

1 heavy hit ≠ 1000 light loads
Adaptation depends on:

total mechanical work
signal frequency
Bonesmashing fails both.

12. Why people THINK it works​

All illusions:

(A) Inflammation​

swelling = temporary “fullness”

(B) Fat loss over time​

sharper face misattributed to smashing

(C) Skin thickening​

repeated trauma = fibrosis
can create illusion of “structure”

(D) Psychological bias​

expectation = perceived result

​

13. When bone CAN actually change shape​

Only in these cases:

growth (before plates close)
orthodontics (controlled force)
surgery (osteotomy)
disease (acromegaly, etc.)

That’s it.

14. Final synthesis​

Bonesmashing fails because:

It produces damage, not adaptive strain
It lacks frequency, direction, and duration
It activates repair pathways, not growth pathways
Facial bones are non-load-bearing and tightly regulated
Periosteal apposition requires systemic + long-term signals, not impacts

DNR, fucking idiot.