Why Non-Aromitizing Androgenic Anabolic Steroids Are KEY for Height-Maxxing

CalebIQ

CalebIQ

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NAAAS (Non-Aromatizing Androgenic Anabolic Steroids) are the second most important part of heightmaxxing.
Growth plates SOLELY close because of estrogen. People think androgens close growth plates, when they simply make them multiply more. But this only advances cells towards the hayflick limit. However, if someone reached the hayflick limit of chondrocytes without estrogen, they would be over 12 feet tall. Effectively, androgens do not close growth plates. The biggest piece of evidence in support of this is aromatase deficient men, growing into 20s and 30s despite higher testosterone.

The reason WHY estrogen closes growth plates is through 2 things:
1. Upregulating VEGF (Vascular Endothelial Growth Factor) which makes blood vessels invade the growth plate, causing it to turn into bone
2. Rapidly cycling chondrocytes through the hayflick limit WITHOUT letting them proliferate.. AND it inhibits the final hypertrophied size.

Androgens do neither of these.

Androgens enhance proliferation rate, effectively giving a permanent growth spurt in the absence of estrogen. Normally, androgens aromatize into estrogen, which is why steroids have the reputation of stunting growth.

This is where NAAAS come in. NAAAS (Such as Masteron, Trenbolone, Anavar, Primobolan, Halotestin, etc) are incapable of being converted into estrogen. This means they solely give the growth boost. On TOP of that, external steroid use shuts down your HPTA (Hypothalamic-Pituitary-Testicular Axis), which is what we want in this situation. Shutting off your own hormones with NAAAS guarantees zero estrogen (except for small amounts from adrenal glands, so an aromatase inhibitor can be helpful)

Anavar at an extremely low dose (2.5mg) was used in children with growth delay and it DOUBLED rate of height growth. Even at such a low dose it helped.

Overall, NAAAS are better than AIs at inhibiting estrogen and give a strong growth signal. Proliferation AND the final size of hypertrophied chondrocytes is increased. Hypertrophied chondrocytes are what actually add to your height. Many content creators say that androgens advance bone age, which is partially true, but only up to a certain point. If they really wanted to avoid estrogen and androgens, they would take puberty blockers. Yet in EVERY study, blocking androgens with puberty blockers vastly decreased growth rates.

Not all NAAAS are created equal, though. Anavar is mild and very hepatotoxic, Halotestin is extremely hepatotoxic, Tren can replicate progesterone and cause breast growth, and Primo is more anabolic than androgenic. You want androgenic signalling for facial and height growth. This is why Masteron is the best NAAAS available. Very androgenic, injectable, no major caveats. Something to note is that bone density is very impaired with low e2. Tamoxifen (a unique medicine that displays either estrogenic or anti estrogenic properties based on the type of tissue) can be used to improve bone density and lipid profiles because it is uniquely estrogenic in trabecular and cortical bone, and the liver, while being anti estrogenic in breast and growth plates. This improves bone density, lipid profiles, growth plates, and may help manage tren tits if you take tren.

Androgens are also synergistic with growth hormone, enhancing the receptors as well as modulating IGFBPs (IGF Binding Proteins) to make igf1 more effective. They cause paracrine (local) release of igf1 in chondrocytes. Androgens and gh will result in much higher proliferation and final hypertrophied size of chondrocytes, as well as expanding the resting zone. NAAAS are even stronger due to not being locally inhibited by estrogen within cells.

In conclusion, using NAAAS to suppress natural hormones and e2 would lead to growth plates that dont close for many, many years. With proper methods to increase growth velocity, you could basically choose any height you want with no e2.
 
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Hgh mogs
 
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DNR.
 
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NAAAS (Non-Aromatizing Androgenic Anabolic Steroids) are the second most important part of heightmaxxing.
Growth plates SOLELY close because of estrogen. People think androgens close growth plates, when they simply make them multiply more. But this only advances cells towards the hayflick limit. However, if someone reached the hayflick limit of chondrocytes without estrogen, they would be over 12 feet tall. Effectively, androgens do not close growth plates. The biggest piece of evidence in support of this is aromatase deficient men, growing into 20s and 30s despite higher testosterone.

The reason WHY estrogen closes growth plates is through 2 things:
1. Upregulating VEGF (Vascular Endothelial Growth Factor) which makes blood vessels invade the growth plate, causing it to turn into bone
2. Rapidly cycling chondrocytes through the hayflick limit WITHOUT letting them proliferate.. AND it inhibits the final hypertrophied size.

Androgens do neither of these.

Androgens enhance proliferation rate, effectively giving a permanent growth spurt in the absence of estrogen. Normally, androgens aromatize into estrogen, which is why steroids have the reputation of stunting growth.

This is where NAAAS come in. NAAAS (Such as Masteron, Trenbolone, Anavar, Primobolan, Halotestin, etc) are incapable of being converted into estrogen. This means they solely give the growth boost. On TOP of that, external steroid use shuts down your HPTA (Hypothalamic-Pituitary-Testicular Axis), which is what we want in this situation. Shutting off your own hormones with NAAAS guarantees zero estrogen (except for small amounts from adrenal glands, so an aromatase inhibitor can be helpful)

Anavar at an extremely low dose (2.5mg) was used in children with growth delay and it DOUBLED rate of height growth. Even at such a low dose it helped.

Overall, NAAAS are better than AIs at inhibiting estrogen and give a strong growth signal. Proliferation AND the final size of hypertrophied chondrocytes is increased. Hypertrophied chondrocytes are what actually add to your height. Many content creators say that androgens advance bone age, which is partially true, but only up to a certain point. If they really wanted to avoid estrogen and androgens, they would take puberty blockers. Yet in EVERY study, blocking androgens with puberty blockers vastly decreased growth rates.

Not all NAAAS are created equal, though. Anavar is mild and very hepatotoxic, Halotestin is extremely hepatotoxic, Tren can replicate progesterone and cause breast growth, and Primo is more anabolic than androgenic. You want androgenic signalling for facial and height growth. This is why Masteron is the best NAAAS available. Very androgenic, injectable, no major caveats. Something to note is that bone density is very impaired with low e2. Tamoxifen (a unique medicine that displays either estrogenic or anti estrogenic properties based on the type of tissue) can be used to improve bone density and lipid profiles because it is uniquely estrogenic in trabecular and cortical bone, and the liver, while being anti estrogenic in breast and growth plates. This improves bone density, lipid profiles, growth plates, and may help manage tren tits if you take tren.

Androgens are also synergistic with growth hormone, enhancing the receptors as well as modulating IGFBPs (IGF Binding Proteins) to make igf1 more effective. They cause paracrine (local) release of igf1 in chondrocytes. Androgens and gh will result in much higher proliferation and final hypertrophied size of chondrocytes, as well as expanding the resting zone. NAAAS are even stronger due to not being locally inhibited by estrogen within cells.

In conclusion, using NAAAS to suppress natural hormones and e2 would lead to growth plates that dont close for many, many years. With proper methods to increase growth velocity, you could basically choose any height you want with no e2.
whats ur current stack
 
10iu GH, aromasin, Test P 280 mg, Tamoxifen
i want to max every mm of height development during puberty. whats the perfect stack for me?
 
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bump
 
Last edited:
theres always a price to pay

low estrogen will fuck you up but aye anything to be 2m right
 
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I would run with no test base correct? At like 200mg weekly? I think imma run 200mg mast with an AI
 
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Rate mine,
8iu GH, Test E 500mg, Arimidex 0.5mg, Nolvadex 40mg (PCT)
v bad if goal is height, lower test to 100-150 max and add NAAS , arimidex 1 mg daily , nolvadex is awful too for height
 
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im using 50mg test and 50mg tren shuld I drop the test? or just lower it even more like 25mg?
 
NAAAS (Non-Aromatizing Androgenic Anabolic Steroids) are the second most important part of heightmaxxing.
Growth plates SOLELY close because of estrogen. People think androgens close growth plates, when they simply make them multiply more. But this only advances cells towards the hayflick limit. However, if someone reached the hayflick limit of chondrocytes without estrogen, they would be over 12 feet tall. Effectively, androgens do not close growth plates. The biggest piece of evidence in support of this is aromatase deficient men, growing into 20s and 30s despite higher testosterone.

The reason WHY estrogen closes growth plates is through 2 things:
1. Upregulating VEGF (Vascular Endothelial Growth Factor) which makes blood vessels invade the growth plate, causing it to turn into bone
2. Rapidly cycling chondrocytes through the hayflick limit WITHOUT letting them proliferate.. AND it inhibits the final hypertrophied size.

Androgens do neither of these.

Androgens enhance proliferation rate, effectively giving a permanent growth spurt in the absence of estrogen. Normally, androgens aromatize into estrogen, which is why steroids have the reputation of stunting growth.

This is where NAAAS come in. NAAAS (Such as Masteron, Trenbolone, Anavar, Primobolan, Halotestin, etc) are incapable of being converted into estrogen. This means they solely give the growth boost. On TOP of that, external steroid use shuts down your HPTA (Hypothalamic-Pituitary-Testicular Axis), which is what we want in this situation. Shutting off your own hormones with NAAAS guarantees zero estrogen (except for small amounts from adrenal glands, so an aromatase inhibitor can be helpful)

Anavar at an extremely low dose (2.5mg) was used in children with growth delay and it DOUBLED rate of height growth. Even at such a low dose it helped.

Overall, NAAAS are better than AIs at inhibiting estrogen and give a strong growth signal. Proliferation AND the final size of hypertrophied chondrocytes is increased. Hypertrophied chondrocytes are what actually add to your height. Many content creators say that androgens advance bone age, which is partially true, but only up to a certain point. If they really wanted to avoid estrogen and androgens, they would take puberty blockers. Yet in EVERY study, blocking androgens with puberty blockers vastly decreased growth rates.

Not all NAAAS are created equal, though. Anavar is mild and very hepatotoxic, Halotestin is extremely hepatotoxic, Tren can replicate progesterone and cause breast growth, and Primo is more anabolic than androgenic. You want androgenic signalling for facial and height growth. This is why Masteron is the best NAAAS available. Very androgenic, injectable, no major caveats. Something to note is that bone density is very impaired with low e2. Tamoxifen (a unique medicine that displays either estrogenic or anti estrogenic properties based on the type of tissue) can be used to improve bone density and lipid profiles because it is uniquely estrogenic in trabecular and cortical bone, and the liver, while being anti estrogenic in breast and growth plates. This improves bone density, lipid profiles, growth plates, and may help manage tren tits if you take tren.

Androgens are also synergistic with growth hormone, enhancing the receptors as well as modulating IGFBPs (IGF Binding Proteins) to make igf1 more effective. They cause paracrine (local) release of igf1 in chondrocytes. Androgens and gh will result in much higher proliferation and final hypertrophied size of chondrocytes, as well as expanding the resting zone. NAAAS are even stronger due to not being locally inhibited by estrogen within cells.

In conclusion, using NAAAS to suppress natural hormones and e2 would lead to growth plates that dont close for many, many years. With proper methods to increase growth velocity, you could basically choose any height you want with no e2.
man just made a thread on that and i explain how to mitigate the lack e2 for brain development..
 
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NAAAS (Non-Aromatizing Androgenic Anabolic Steroids) are the second most important part of heightmaxxing.
Growth plates SOLELY close because of estrogen. People think androgens close growth plates, when they simply make them multiply more. But this only advances cells towards the hayflick limit. However, if someone reached the hayflick limit of chondrocytes without estrogen, they would be over 12 feet tall. Effectively, androgens do not close growth plates. The biggest piece of evidence in support of this is aromatase deficient men, growing into 20s and 30s despite higher testosterone.

The reason WHY estrogen closes growth plates is through 2 things:
1. Upregulating VEGF (Vascular Endothelial Growth Factor) which makes blood vessels invade the growth plate, causing it to turn into bone
2. Rapidly cycling chondrocytes through the hayflick limit WITHOUT letting them proliferate.. AND it inhibits the final hypertrophied size.

Androgens do neither of these.

Androgens enhance proliferation rate, effectively giving a permanent growth spurt in the absence of estrogen. Normally, androgens aromatize into estrogen, which is why steroids have the reputation of stunting growth.

This is where NAAAS come in. NAAAS (Such as Masteron, Trenbolone, Anavar, Primobolan, Halotestin, etc) are incapable of being converted into estrogen. This means they solely give the growth boost. On TOP of that, external steroid use shuts down your HPTA (Hypothalamic-Pituitary-Testicular Axis), which is what we want in this situation. Shutting off your own hormones with NAAAS guarantees zero estrogen (except for small amounts from adrenal glands, so an aromatase inhibitor can be helpful)

Anavar at an extremely low dose (2.5mg) was used in children with growth delay and it DOUBLED rate of height growth. Even at such a low dose it helped.

Overall, NAAAS are better than AIs at inhibiting estrogen and give a strong growth signal. Proliferation AND the final size of hypertrophied chondrocytes is increased. Hypertrophied chondrocytes are what actually add to your height. Many content creators say that androgens advance bone age, which is partially true, but only up to a certain point. If they really wanted to avoid estrogen and androgens, they would take puberty blockers. Yet in EVERY study, blocking androgens with puberty blockers vastly decreased growth rates.

Not all NAAAS are created equal, though. Anavar is mild and very hepatotoxic, Halotestin is extremely hepatotoxic, Tren can replicate progesterone and cause breast growth, and Primo is more anabolic than androgenic. You want androgenic signalling for facial and height growth. This is why Masteron is the best NAAAS available. Very androgenic, injectable, no major caveats. Something to note is that bone density is very impaired with low e2. Tamoxifen (a unique medicine that displays either estrogenic or anti estrogenic properties based on the type of tissue) can be used to improve bone density and lipid profiles because it is uniquely estrogenic in trabecular and cortical bone, and the liver, while being anti estrogenic in breast and growth plates. This improves bone density, lipid profiles, growth plates, and may help manage tren tits if you take tren.

Androgens are also synergistic with growth hormone, enhancing the receptors as well as modulating IGFBPs (IGF Binding Proteins) to make igf1 more effective. They cause paracrine (local) release of igf1 in chondrocytes. Androgens and gh will result in much higher proliferation and final hypertrophied size of chondrocytes, as well as expanding the resting zone. NAAAS are even stronger due to not being locally inhibited by estrogen within cells.

In conclusion, using NAAAS to suppress natural hormones and e2 would lead to growth plates that dont close for many, many years. With proper methods to increase growth velocity, you could basically choose any height you want with no e2.
@ihatemySOST can you debunk this
 
Why Not use Like 25mg Test as a Test base, and then add NAAAS? Or Even heg as a test base Like 500IU e3d
 
NAAAS (Non-Aromatizing Androgenic Anabolic Steroids) are the second most important part of heightmaxxing.
Growth plates SOLELY close because of estrogen. People think androgens close growth plates, when they simply make them multiply more. But this only advances cells towards the hayflick limit. However, if someone reached the hayflick limit of chondrocytes without estrogen, they would be over 12 feet tall. Effectively, androgens do not close growth plates. The biggest piece of evidence in support of this is aromatase deficient men, growing into 20s and 30s despite higher testosterone.

The reason WHY estrogen closes growth plates is through 2 things:
1. Upregulating VEGF (Vascular Endothelial Growth Factor) which makes blood vessels invade the growth plate, causing it to turn into bone
2. Rapidly cycling chondrocytes through the hayflick limit WITHOUT letting them proliferate.. AND it inhibits the final hypertrophied size.

Androgens do neither of these.

Androgens enhance proliferation rate, effectively giving a permanent growth spurt in the absence of estrogen. Normally, androgens aromatize into estrogen, which is why steroids have the reputation of stunting growth.

This is where NAAAS come in. NAAAS (Such as Masteron, Trenbolone, Anavar, Primobolan, Halotestin, etc) are incapable of being converted into estrogen. This means they solely give the growth boost. On TOP of that, external steroid use shuts down your HPTA (Hypothalamic-Pituitary-Testicular Axis), which is what we want in this situation. Shutting off your own hormones with NAAAS guarantees zero estrogen (except for small amounts from adrenal glands, so an aromatase inhibitor can be helpful)

Anavar at an extremely low dose (2.5mg) was used in children with growth delay and it DOUBLED rate of height growth. Even at such a low dose it helped.

Overall, NAAAS are better than AIs at inhibiting estrogen and give a strong growth signal. Proliferation AND the final size of hypertrophied chondrocytes is increased. Hypertrophied chondrocytes are what actually add to your height. Many content creators say that androgens advance bone age, which is partially true, but only up to a certain point. If they really wanted to avoid estrogen and androgens, they would take puberty blockers. Yet in EVERY study, blocking androgens with puberty blockers vastly decreased growth rates.

Not all NAAAS are created equal, though. Anavar is mild and very hepatotoxic, Halotestin is extremely hepatotoxic, Tren can replicate progesterone and cause breast growth, and Primo is more anabolic than androgenic. You want androgenic signalling for facial and height growth. This is why Masteron is the best NAAAS available. Very androgenic, injectable, no major caveats. Something to note is that bone density is very impaired with low e2. Tamoxifen (a unique medicine that displays either estrogenic or anti estrogenic properties based on the type of tissue) can be used to improve bone density and lipid profiles because it is uniquely estrogenic in trabecular and cortical bone, and the liver, while being anti estrogenic in breast and growth plates. This improves bone density, lipid profiles, growth plates, and may help manage tren tits if you take tren.

Androgens are also synergistic with growth hormone, enhancing the receptors as well as modulating IGFBPs (IGF Binding Proteins) to make igf1 more effective. They cause paracrine (local) release of igf1 in chondrocytes. Androgens and gh will result in much higher proliferation and final hypertrophied size of chondrocytes, as well as expanding the resting zone. NAAAS are even stronger due to not being locally inhibited by estrogen within cells.

In conclusion, using NAAAS to suppress natural hormones and e2 would lead to growth plates that dont close for many, many years. With proper methods to increase growth velocity, you could basically choose any height you want with no e2.
Your gonna get brain damage from having 0 estrogen for months on end if you really want to optimize your estrogen levels run a non aromatizing compound and microdose injectable estrogen to keep your e2 levels at the exact level you want this is pretty much the only use for inj e2 besides trannies
 
Masteron is ass. Good for people that want to be balding, ballsack wrinkly hagcels.
 
study please
The study was halotestin, not anavar

 
stop leaking
 
10iu GH, aromasin, Test P 280 mg, Tamoxifen
whats your tamoxifen dose? I have it because I was going to use it for PCT but im going to blast and cruise now. I am open to using it for the reasons you give though.
 
Honestly, this might be one of the worst posts ever created on the org. First, you're confusing two different processes. Bone aging and growth plate closure are not exactly the same thing. Bone aging is the preparatory stage for growth plate closure. Growth plate closure is the final stage of this process. So, I'll assume you mean that androgens don't directly stimulate bone maturation, and that's a big mistake. First, you said that patients with aromatase deficiency are evidence of this, but it proves the exact opposite. These patients have very advanced bone age (15-17.5) even though they literally have 0 e2. So how did their bone age reach this stage even though estrogen isn't present? Here you are contradicting yourself. There is strong evidence that androgens directly accelerate bone maturation with an effect independent of estrogen.
1. Individuals with high androgen receptor sensitivity are significantly shorter than normal individuals.
2. Administering non-aromatizable androgens to adolescents/children significantly accelerates bone maturation.
3. Patients with complete estrogen deficiency have a newly advanced bone age solely due to physiological levels of androgens.
4. Modifying mice to produce excessive androgen receptors causes severe bone shortens
5. Castrating mice (which literally leaves them with zero estrogen, and mice cannot produce E2 from the adrenal glands like humans) and administering non-aromatizable androgens results in severe calcification of the growth plate.
6. Administering a complete estrogen receptor antagonist does not prevent the bone maturation induced by androgen administration in mice.
7. There is a clear biological mechanism for how this occurs: androgens increase sensitivity. IGF-1 is present in the pre-hypertrophic region, thus accelerating the entry of chondrocytes into the terminal hyperplasia and calcification phase. Therefore, all studies without exception have found that androgens do not increase terminal bone length; in fact, they decrease it if administered for an extended period.

Therefore, there is very strong and undeniable evidence regarding the direct effect of androgens on bone maturation.

I don't understand where you got the idea that taking non-aromatizable androgens would delay the closure of growth plates for many years. If this were true, why do human studies show the opposite?
 
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Masteron is ass. Good for people that want to be balding, ballsack wrinkly hagcels.
only true if ur a clueless fag who dont know how to run it and how to prevent these side effect i truly believe that mast could be a great tool in early and even late puberty
 
only true if ur a clueless fag who dont know how to run it and how to prevent these side effect i truly believe that mast could be a great tool in early and even late puberty
Do you just "truly believe" that, or have you used it yourself?
 
For how long? Did you get any desired results, or any results in general?
No brah not any result at all beacause i took it for only one week and decided to come off and save the rest for a big blast wich i will be documenting. (I just know that many people love masteron for fatloss muscle hardening and its one of the best option for male sexual dimorphism) i would not recommend to anyone using it past puberty tho
 
10iu GH, aromasin, Test P 280 mg, Tamoxifen
Nigga talked about how estrogen is bad and how non-aromatising androgens are better, but uses 300mgs of a aromatisable-androgen

Mirin bro
 
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v bad if goal is height, lower test to 100-150 max and add NAAS , arimidex 1 mg daily , nolvadex is awful too for height
150 test is retarded keep in mind ts replaces your natural production it doesnt add on to it and 100 test only puts you in like the 550-850 ng/dl range which is probably the range he is already in if not lower
 
Bad idea, they will deplete parasympathetic potential.
There are fat better ideas like oleuropein+icariin+hiit+lactoferrin combination
 

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