Dr. Bill Andrews Is Working On Curing Aging By Telomere Elongation + Here Are Some Of My Thoughts

[Youㅤ]

Anti-Aging Research | Anti-Aging Medicine – Sierra Sciences’

Our mission is to find the best cure for aging—Find it at our leading anti-aging research firm and learn more about anti-aging science.

sierrasci.com

Read through the above website. Pretty exciting stuff. Being alive in 2023 can be depressing, but don’t take it for granted; it’s an interesting time to be alive.

Here’s a LongeCity thread you should definitely read:

Hello. I am starting an open source initiative to cure aging - AgingResearch

Hello. I am starting an open source initiative to cure aging - posted in AgingResearch: Hey everyone, Excited to meet you all! I’m here to kick off an open-source project to tackle aging. Not sure if anyone’s tried this before, but I’m not talking about supplements or small-scale stuff, so...

www.longecity.org

I’ve been thinking about telomeres and DNA damage, and have been thinking about the potential of DNA damage being a significant catalyst for telomere shortening. (In the very least, DNA damage would make a telomere-lengthening anti aging approach dangerous due to cancer risk)

I’ve thought of some ideas, although I’ve yet to do research. You could help in that regard. Anyway, the idea is that (considering DNA damage accelerates telomere shortening AND increases the risk of cancer) preventing or repairing DNA should be prioritized. Considering cosmic radiation can damage DNA, perhaps encasing your room in a radiation-blocking material, such as lead, could reduce DNA damage and thus slow cellular senescence and protect from cancer. I’ve also thought about p53 (p53 activation can either induce cell growth cycle arrest and DNA repair, apoptosis, or cellular senescence) and wondered if it’s possible to augment the chances so that the path of cellular growth cycle arrest and DNA repair occurs more often relative to apoptosis and cellular senescence. The perspective I’ve been thinking in is that DNA damage is CAUSATIVE to telomere degeneration and overall organismic senescence.

@lookmaxascend @KING REIDYZ @ogu @spirili @mvp2v1 @the MOUSE @hellolol

 

[Youㅤ]

0

 

[thereallegend]

Astragulus lengthens telomeres

 

[Mr. President]

Anti-Aging Research | Anti-Aging Medicine – Sierra Sciences’

Our mission is to find the best cure for aging—Find it at our leading anti-aging research firm and learn more about anti-aging science.

sierrasci.com

Read through the above website. Pretty exciting stuff. Being alive in 2023 can be depressing, but don’t take it for granted; it’s an interesting time to be alive.

Here’s a LongeCity thread you should definitely read:

Hello. I am starting an open source initiative to cure aging - AgingResearch

Hello. I am starting an open source initiative to cure aging - posted in AgingResearch: Hey everyone, Excited to meet you all! I’m here to kick off an open-source project to tackle aging. Not sure if anyone’s tried this before, but I’m not talking about supplements or small-scale stuff, so...

www.longecity.org

I’ve been thinking about telomeres and DNA damage, and have been thinking about the potential of DNA damage being a significant catalyst for telomere shortening. (In the very least, DNA damage would make a telomere-lengthening anti aging approach dangerous due to cancer risk)

I’ve thought of some ideas, although I’ve yet to do research. You could help in that regard. Anyway, the idea is that (considering DNA damage accelerates telomere shortening AND increases the risk of cancer) preventing or repairing DNA should be prioritized. Considering cosmic radiation can damage DNA, perhaps encasing your room in a radiation-blocking material, such as lead, could reduce DNA damage and thus slow cellular senescence and protect from cancer. I’ve also thought about p53 (p53 activation can either induce cell growth cycle arrest and DNA repair, apoptosis, or cellular senescence) and wondered if it’s possible to augment the chances so that the path of cellular growth cycle arrest and DNA repair occurs more often relative to apoptosis and cellular senescence. The perspective I’ve been thinking in is that DNA damage is CAUSATIVE to telomere degeneration and overall organismic senescence.

@lookmaxascend @KING REIDYZ @ogu @spirili @mvp2v1 @the MOUSE @hellolol

gonna be to high iq for this forum boss

 

[Abhorrence]

 

[Kirby7000]

View attachment 2552452

imagine living longer as a sub5 male.

 

[Deleted member 8365]

Anti-Aging Research | Anti-Aging Medicine – Sierra Sciences’

Our mission is to find the best cure for aging—Find it at our leading anti-aging research firm and learn more about anti-aging science.

sierrasci.com

Read through the above website. Pretty exciting stuff. Being alive in 2023 can be depressing, but don’t take it for granted; it’s an interesting time to be alive.

Here’s a LongeCity thread you should definitely read:

Hello. I am starting an open source initiative to cure aging - AgingResearch

Hello. I am starting an open source initiative to cure aging - posted in AgingResearch: Hey everyone, Excited to meet you all! I’m here to kick off an open-source project to tackle aging. Not sure if anyone’s tried this before, but I’m not talking about supplements or small-scale stuff, so...

www.longecity.org

I’ve been thinking about telomeres and DNA damage, and have been thinking about the potential of DNA damage being a significant catalyst for telomere shortening. (In the very least, DNA damage would make a telomere-lengthening anti aging approach dangerous due to cancer risk)

I’ve thought of some ideas, although I’ve yet to do research. You could help in that regard. Anyway, the idea is that (considering DNA damage accelerates telomere shortening AND increases the risk of cancer) preventing or repairing DNA should be prioritized. Considering cosmic radiation can damage DNA, perhaps encasing your room in a radiation-blocking material, such as lead, could reduce DNA damage and thus slow cellular senescence and protect from cancer. I’ve also thought about p53 (p53 activation can either induce cell growth cycle arrest and DNA repair, apoptosis, or cellular senescence) and wondered if it’s possible to augment the chances so that the path of cellular growth cycle arrest and DNA repair occurs more often relative to apoptosis and cellular senescence. The perspective I’ve been thinking in is that DNA damage is CAUSATIVE to telomere degeneration and overall organismic senescence.

@lookmaxascend @KING REIDYZ @ogu @spirili @mvp2v1 @the MOUSE @hellolol

Super interesting bro

 

[lookmaxascend]

Anti-Aging Research | Anti-Aging Medicine – Sierra Sciences’

Our mission is to find the best cure for aging—Find it at our leading anti-aging research firm and learn more about anti-aging science.

sierrasci.com

Read through the above website. Pretty exciting stuff. Being alive in 2023 can be depressing, but don’t take it for granted; it’s an interesting time to be alive.

Here’s a LongeCity thread you should definitely read:

Hello. I am starting an open source initiative to cure aging - AgingResearch

Hello. I am starting an open source initiative to cure aging - posted in AgingResearch: Hey everyone, Excited to meet you all! I’m here to kick off an open-source project to tackle aging. Not sure if anyone’s tried this before, but I’m not talking about supplements or small-scale stuff, so...

www.longecity.org

I’ve been thinking about telomeres and DNA damage, and have been thinking about the potential of DNA damage being a significant catalyst for telomere shortening. (In the very least, DNA damage would make a telomere-lengthening anti aging approach dangerous due to cancer risk)

I’ve thought of some ideas, although I’ve yet to do research. You could help in that regard. Anyway, the idea is that (considering DNA damage accelerates telomere shortening AND increases the risk of cancer) preventing or repairing DNA should be prioritized. Considering cosmic radiation can damage DNA, perhaps encasing your room in a radiation-blocking material, such as lead, could reduce DNA damage and thus slow cellular senescence and protect from cancer. I’ve also thought about p53 (p53 activation can either induce cell growth cycle arrest and DNA repair, apoptosis, or cellular senescence) and wondered if it’s possible to augment the chances so that the path of cellular growth cycle arrest and DNA repair occurs more often relative to apoptosis and cellular senescence. The perspective I’ve been thinking in is that DNA damage is CAUSATIVE to telomere degeneration and overall organismic senescence.

@lookmaxascend @KING REIDYZ @ogu @spirili @mvp2v1 @the MOUSE @hellolol

Very interesting thread, I'll do my research.

 

[Deleted member 8365]

imagine living longer as a sub5 male.

But most looksmaxxer will old chads bro

 

[reptiles]

Anti-Aging Research | Anti-Aging Medicine – Sierra Sciences’

Our mission is to find the best cure for aging—Find it at our leading anti-aging research firm and learn more about anti-aging science.

sierrasci.com

Read through the above website. Pretty exciting stuff. Being alive in 2023 can be depressing, but don’t take it for granted; it’s an interesting time to be alive.

Here’s a LongeCity thread you should definitely read:

Hello. I am starting an open source initiative to cure aging - AgingResearch

Hello. I am starting an open source initiative to cure aging - posted in AgingResearch: Hey everyone, Excited to meet you all! I’m here to kick off an open-source project to tackle aging. Not sure if anyone’s tried this before, but I’m not talking about supplements or small-scale stuff, so...

www.longecity.org

I’ve been thinking about telomeres and DNA damage, and have been thinking about the potential of DNA damage being a significant catalyst for telomere shortening. (In the very least, DNA damage would make a telomere-lengthening anti aging approach dangerous due to cancer risk)

I’ve thought of some ideas, although I’ve yet to do research. You could help in that regard. Anyway, the idea is that (considering DNA damage accelerates telomere shortening AND increases the risk of cancer) preventing or repairing DNA should be prioritized. Considering cosmic radiation can damage DNA, perhaps encasing your room in a radiation-blocking material, such as lead, could reduce DNA damage and thus slow cellular senescence and protect from cancer. I’ve also thought about p53 (p53 activation can either induce cell growth cycle arrest and DNA repair, apoptosis, or cellular senescence) and wondered if it’s possible to augment the chances so that the path of cellular growth cycle arrest and DNA repair occurs more often relative to apoptosis and cellular senescence. The perspective I’ve been thinking in is that DNA damage is CAUSATIVE to telomere degeneration and overall organismic senescence.

@lookmaxascend @KING REIDYZ @ogu @spirili @mvp2v1 @the MOUSE @hellolol

@Youㅤyou have any other material similar to this its very interesting

 

[Youㅤ]

@Youㅤyou have any other material similar to this its very interesting

Well I haven’t been focusing on rejuvenation therapies because I’m prioritizing optimizing my diet first to slow aging, but you can look into sens.org, another aging reversal foundation

a potential rejuvenation method is prolonged fasting or fasting mimicking diet, there’s evidence for it increasing stem cell self-renewal and differentiation to rejuvenate some tissues

 

[Youㅤ]

@Youㅤyou have any other material similar to this its very interesting

I recommend reading through fightaging.org for interesting information

 

[ogu]

A very different take for this forum.

As mentioned, DNA damage is usually prevented by your body by other cells who detect DNA damage and kill those cells before they can reproduce. Most of the time, DNA damage is nothing serious as the nucleotides are usually still the same and still point to the same RNA and amino acid, but when there is a harmful mutation with nothing to stop it, tumors form.

There are multiple ways you can stop this. First, eat your veggies.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8146059/

Second, take SAM-e.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7464653/

Third, fast.

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7429999/

Fourth and MOST IMPORTANTLY:
Keep your stress levels low. Wake up and just be happy.