Drug Induced Blackpilled Psychosis: My Experience (High IQ Analysis)

Diyorldar

Diyorldar

It's not like the movies
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I’m posting this as a detailed mechanistic breakdown of the factors causing my psychotic break in mid February. It's both a cautionary tale and a deep dive into the soft white underbelly of the human psyche. Enjoy

I had zero psychiatric history prior to February 2026. In mid February I had a full drug-induced psychotic episode that led to 5150 hold, inpatient admission, and an initial Bipolar 1 diagnosis that has since been formally revised. The content of the psychosis was 100% blackpilled structural airway inadequacy: convinced my airway was 3.9 mm and collapsing, produced a 23-page clinical slideshow with CBCT measurements taken from the incorrect landmark, and reached out to top-tier maxillofacial surgeons (Alfaro, Raffani, Pagnoni). It was not generic disorganized thought. It was the exact looksmax structural deficit pattern-recognition system hyper-activated and directed inward under a precise neurochemical convergence.

The blackpill itself did not cause the episode. It supplied the content architecture because of pharmacological risk overlap on top of pre-existing substrate. I’m laying out the exact mechanisms, timeline, and my own lapses so others can avoid the same cascade.

Pre-existing vulnerabilities
  • Multi-year chronic sleep debt (2022 onward) from early-morning swim + AP Class schedule. Produced progressive prefrontal hypofunction, elevated amygdala reactivity, degraded executive regulation, and a significantly lowered psychosis threshold.
  • Undiagnosed ADHD (confirmed via neuropsych eval January 2026, right before the episode — objective baseline with no mood disorder flagged). Contributed hyperfocus, novelty-seeking, and impaired braking on cannabis escalation.
  • Severe cystic acne driving initial looksmax engagement and profound self-image disruption.
  • Athletic identity collapse
  • Sustained relational trauma and 2-month stalking campaign (300+ calls, multi-platform harassment). Produced acquired hypervigilance that later redirected inward.
None of these alone produced psychosis. The February 2026 pharmacological stack did.

Exact timeline and overlapping triggers (Feb 18–23 2026)
  • Feb 18: Abrupt cessation of Cannabis after months of heavy chronic use.
  • Feb 20 & 22: Multiple DMT trips (full 5-HT2A agonism with residual receptor sensitization persisting 48–72 hours).
  • Feb 23: New Adderall protocol initiated on zero sleep the preceding night due to cannabis withdrawal rebound insomnia.

Core Mechanisms
  1. Cannabis withdrawal and CB1 downregulation crash.
    Chronic high-dose THC produced extensive CB1 receptor downregulation. Abrupt stop caused acute endocannabinoid system collapse: loss of GABAergic/glutamatergic buffering that had become load-bearing for mood homeostasis, anxiety regulation, sleep architecture, and reality testing. This created the primary neurological destabilization.

  2. DMT-induced serotonergic sensitization
    I took DMT multiple times on February 20 and February 22 — the 48–72 hours immediately preceding acute psychosis onset. DMT is a full 5-HT2A agonist that produces profound serotonergic disruption. Residual receptor sensitization following repeated exposure in close temporal proximity lowered my threshold for
    psychotic symptom expression and disrupted the serotonergic regulatory systems that provide secondary reality testing.

  3. Acute + chronic sleep deprivation
    72–96 hours severely disrupted sleep layered on years of baseline debt. Prefrontal cortex hypofunction eliminated reality testing; amygdala dysregulation amplified threat scanning; dopamine sensitivity was massively heightened.

  4. Amphetamine-driven mesolimbic dopaminergic excess (the final trigger)
    Adderall (dopamine/norepinephrine reuptake inhibition + reversal transport) on a brain already in active cannabis withdrawal, DMT afterglow, and total sleep deprivation produced sustained dopaminergic overflow in the mesolimbic pathway — the established core mechanism of psychosis across etiologies.

  5. Heightened neuroplasticity + Hebbian conditioning from looksmax engagement
    The critical piece that made this blackpilled psychosis rather than generic paranoia. Stimulant + withdrawal + sleep deprivation + DMT created a high-plasticity window. The most recently and heavily trained neural pathways are preferentially amplified via Hebbian plasticity (“neurons that fire together wire together”).

The outcome was highly ordered, anatomically precise blackpilled thinking applied to a delusional premise. Standard reassurance failed because the prefrontal mechanisms required for reality testing were chemically offline.

My own lapses (full accountability)
  • Cold-turkey cannabis cessation instead of controlled taper.
  • DMT use during active withdrawal, relying on pharmacological knowledge as false security rather than risk assessment.
  • Initiating Adderall on zero sleep during the crash window.
  • Failure to recognize that my biochem background was being co-opted to rationalize continued exposure instead of enforcing stabilization.
Post-episode course

Triggers removed: 50+ days cannabis sobriety, full DMT abstinence, Adderall discontinued, sleep normalized on Trazodone. The airway delusion resolved as inflammation subsided and sleep returned. Fully stable 6+ weeks with no mood or psychotic symptoms. Currently on Lithium 300mg a temporary measure as the timeline inconsistent with primary Bipolar 1.

The Psych Ward Experience

The Psych ward is a trip, nearly everyone in there has had their brains melted. I had to go out of my way to stop them from putting me on oral Olanzapine (Zyprexa) which is probably the #1 looksmin drug of all time. I was in the psych ward for 10 days, for the first 3 or so I was still convinced I had a 3.9mm airway (the Hebbian connections and real inflammation were that firm so no progress was made. By the time my inflammation began to go down my breathing issues naturally resolved.

If you ever find yourself there: play along, take whatever they give you (pick the least bad meds), attend every group meeting, and tell the psych exactly what they want to hear. You cannot talk your way out of a psychiatric hold. Larp. Larp. Larp. The system is not built for nuanced mechanistic explanations in acute treatment.

Next Steps

I remain fully committed to evidence-based looksmaxing. I am scheduling a bimax+genio with undereye fat grafting with Dr.Eren Pera for next July. The delusional framework is resolved, the prior measurements were erroneous, but the underlying structural optimization goal stands. I'm looking forward to staying engaged with the community, missed you guys.

TLDR

I had a full drug-induced blackpilled psychotic episode: convinced my airway was collapsing, made a detailed slideshow, and contacted top maxfax surgeons. The stack was cold-turkey cannabis withdrawal + DMT + Adderall on zero sleep, all on top of years of sleep debt, undiagnosed ADHD, acne, identity loss, and trauma. My trained looksmax structural scanning became the fixed delusion through neuroplasticity. Stabilized fast once triggers were removed — diagnosis revised to substance-induced, now on low-dose Lithium. Psych ward: had to larp hard and dodge Zyprexa. Lesson: your deepest insecurities get broadcast as fixed delusion the moment regulatory systems collapse.

@Orka @Sayori @abzz @aids @chudpiller
 
Last edited:
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Bump
 
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@Starborn
 
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I missed u

Give tldr please
 
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Wait so you went on that airway craze due to drug induced psychosis?

Also, give a life update since you were hospitalized. Did you have to hop off all androgens?
 
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If you ever find yourself there: play along, take whatever they give you (pick the least bad meds), attend every group meeting, and tell the psych exactly what they want to hear. You cannot talk your way out of a psychiatric hold. Larp. Larp. Larp. The system is not built for nuanced mechanistic explanations in acute treatment.
Thanks for this advice. I will use it when i inevitably enter the psych ward
 
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Wait so you went on that airway craze due to drug induced psychosis?

Also, give a life update since you were hospitalized. Did you have to hop off all androgens?
Yes, I had legit inflammation+psychosis making me think I was dying.

I'm currently natty too lol
 
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Reactions: chudpiller
Yes, I had legit inflammation+psychosis making me think I was dying.

I'm currently natty too lol
lmao i remembered thinking that you were buggin out when you sent me all that

how are you feeling now?
 
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lmao i remembered thinking that you were buggin out when you sent me all that

how are you feeling now?
I’m chilling now, back to normal 100%

Honestly psychosis was a good thing for me

The way I see it the brain can only take so much before it shuts off. My lifestyle was unsunstainable. It was a necessary intervention.
 
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Reactions: fazehamster and chudpiller
I’m posting this as a detailed mechanistic breakdown of the factors causing my psychotic break in mid February. It's both a cautionary tale and a deep dive into the soft white underbelly of the human psyche. Enjoy

I had zero psychiatric history prior to February 2026. In mid February I had a full drug-induced psychotic episode that led to 5150 hold, inpatient admission, and an initial Bipolar 1 diagnosis that has since been formally revised. The content of the psychosis was 100% blackpilled structural airway inadequacy: convinced my airway was 3.9 mm and collapsing, produced a 23-page clinical slideshow with CBCT measurements taken from the incorrect landmark, and reached out to top-tier maxillofacial surgeons (Alfaro, Raffani, Pagnoni). It was not generic disorganized thought. It was the exact looksmax structural deficit pattern-recognition system hyper-activated and directed inward under a precise neurochemical convergence.

The blackpill itself did not cause the episode. It supplied the content architecture because of pharmacological risk overlap on top of pre-existing substrate. I’m laying out the exact mechanisms, timeline, and my own lapses so others can avoid the same cascade.

Pre-existing vulnerabilities
  • Multi-year chronic sleep debt (2022 onward) from early-morning swim + AP Class schedule. Produced progressive prefrontal hypofunction, elevated amygdala reactivity, degraded executive regulation, and a significantly lowered psychosis threshold.
  • Undiagnosed ADHD (confirmed via neuropsych eval January 2026, right before the episode — objective baseline with no mood disorder flagged). Contributed hyperfocus, novelty-seeking, and impaired braking on cannabis escalation.
  • Severe cystic acne driving initial looksmax engagement and profound self-image disruption.
  • Athletic identity collapse
  • Sustained relational trauma and 2-month stalking campaign (300+ calls, multi-platform harassment). Produced acquired hypervigilance that later redirected inward.
None of these alone produced psychosis. The February 2026 pharmacological stack did.

Exact timeline and overlapping triggers (Feb 18–23 2026)
  • Feb 18: Abrupt cessation of Cannabis after months of heavy chronic use.
  • Feb 20 & 22: Multiple DMT trips (full 5-HT2A agonism with residual receptor sensitization persisting 48–72 hours).
  • Feb 23: New Adderall protocol initiated on zero sleep the preceding night due to cannabis withdrawal rebound insomnia.

Core Mechanisms
  1. Cannabis withdrawal and CB1 downregulation crash.
    Chronic high-dose THC produced extensive CB1 receptor downregulation. Abrupt stop caused acute endocannabinoid system collapse: loss of GABAergic/glutamatergic buffering that had become load-bearing for mood homeostasis, anxiety regulation, sleep architecture, and reality testing. This created the primary neurological destabilization.

  2. DMT-induced serotonergic sensitization
    I took DMT multiple times on February 20 and February 22 — the 48–72 hours immediately preceding acute psychosis onset. DMT is a full 5-HT2A agonist that produces profound serotonergic disruption. Residual receptor sensitization following repeated exposure in close temporal proximity lowered my threshold for
    psychotic symptom expression and disrupted the serotonergic regulatory systems that provide secondary reality testing.

  3. Acute + chronic sleep deprivation
    72–96 hours severely disrupted sleep layered on years of baseline debt. Prefrontal cortex hypofunction eliminated reality testing; amygdala dysregulation amplified threat scanning; dopamine sensitivity was massively heightened.

  4. Amphetamine-driven mesolimbic dopaminergic excess (the final trigger)
    Adderall (dopamine/norepinephrine reuptake inhibition + reversal transport) on a brain already in active cannabis withdrawal, DMT afterglow, and total sleep deprivation produced sustained dopaminergic overflow in the mesolimbic pathway — the established core mechanism of psychosis across etiologies.

  5. Heightened neuroplasticity + Hebbian conditioning from looksmax engagement
    The critical piece that made this blackpilled psychosis rather than generic paranoia. Stimulant + withdrawal + sleep deprivation + DMT created a high-plasticity window. The most recently and heavily trained neural pathways are preferentially amplified via Hebbian plasticity (“neurons that fire together wire together”).

The outcome was highly ordered, anatomically precise blackpilled thinking applied to a delusional premise. Standard reassurance failed because the prefrontal mechanisms required for reality testing were chemically offline.

My own lapses (full accountability)
  • Cold-turkey cannabis cessation instead of controlled taper.
  • DMT use during active withdrawal, relying on pharmacological knowledge as false security rather than risk assessment.
  • Initiating Adderall on zero sleep during the crash window.
  • Failure to recognize that my biochem background was being co-opted to rationalize continued exposure instead of enforcing stabilization.
Post-episode course

Triggers removed: 50+ days cannabis sobriety, full DMT abstinence, Adderall discontinued, sleep normalized on Trazodone. The airway delusion resolved as inflammation subsided and sleep returned. Fully stable 6+ weeks with no mood or psychotic symptoms. Currently on Lithium 300mg a temporary measure as the timeline inconsistent with primary Bipolar 1.

The Psych Ward Experience

The Psych ward is a trip, nearly everyone in there has had their brains melted. I had to go out of my way to stop them from putting me on oral Olanzapine (Zyprexa) which is probably the #1 looksmin drug of all time. I was in the psych ward for 10 days, for the first 3 or so I was still convinced I had a 3.9mm airway (the Hebbian connections and real inflammation were that firm so no progress was made. By the time my inflammation began to go down my breathing issues naturally resolved.

If you ever find yourself there: play along, take whatever they give you (pick the least bad meds), attend every group meeting, and tell the psych exactly what they want to hear. You cannot talk your way out of a psychiatric hold. Larp. Larp. Larp. The system is not built for nuanced mechanistic explanations in acute treatment.

Next Steps

I remain fully committed to evidence-based looksmaxing. I am scheduling a bimax+genio with undereye fat grafting with Dr.Eren Pera for next July. The delusional framework is resolved, the prior measurements were erroneous, but the underlying structural optimization goal stands. I'm looking forward to staying engaged with the community, missed you guys.

TLDR

I had a full drug-induced blackpilled psychotic episode: convinced my airway was collapsing, made a detailed slideshow, and contacted top maxfax surgeons. The stack was cold-turkey cannabis withdrawal + DMT + Adderall on zero sleep, all on top of years of sleep debt, undiagnosed ADHD, acne, identity loss, and trauma. My trained looksmax structural scanning became the fixed delusion through neuroplasticity. Stabilized fast once triggers were removed — diagnosis revised to substance-induced, now on low-dose Lithium. Psych ward: had to larp hard and dodge Zyprexa. Lesson: your deepest insecurities get broadcast as fixed delusion the moment regulatory systems collapse.

@Orka @Sayori @abzz @aids @chudpiller
Holy shiii bros back!!! Will read this when I hop on my pc
 
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shit man

at least skywalker summer coming up
 
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Holy shit

Glad you’re better now

Am I allowed to see the slideshow you created? :unsure:
 
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Holy shit

Glad you’re better now

Am I allowed to see the slideshow you created? :unsure:
ts lowkey doxes tf out of me but basically it was explaining in clincial detail that i thought my airway was collapsing due to mandibular recession. its accurate except i don't have that issue.
 
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ts lowkey doxes tf out of me but basically it was explaining in clincial detail that i thought my airway was collapsing due to mandibular recession. its accurate except i don't have that issue.
Crazy. Psychosis is no joke my nigga

Be careful :feelscry:
 
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Crazy. Psychosis is no joke my nigga

Be careful :feelscry:
Shit was insane bro

I actually lost my mind like in requiem for a dream.

scary stuff

I'm glad im not actually bipolar or schizo

this shit can happen to anyone tho
 
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Shit was insane bro

I actually lost my mind like in requiem for a dream.

scary stuff

I'm glad im not actually bipolar or schizo

this shit can happen to anyone tho
I’m gonna be super careful with psychedelics & dopaminergic drugs

Proper sleep is a must for everyone though. Sleep deprivation is the worst feeling
 
  • +1
Reactions: Diyorldar
I’m posting this as a detailed mechanistic breakdown of the factors causing my psychotic break in mid February. It's both a cautionary tale and a deep dive into the soft white underbelly of the human psyche. Enjoy

I had zero psychiatric history prior to February 2026. In mid February I had a full drug-induced psychotic episode that led to 5150 hold, inpatient admission, and an initial Bipolar 1 diagnosis that has since been formally revised. The content of the psychosis was 100% blackpilled structural airway inadequacy: convinced my airway was 3.9 mm and collapsing, produced a 23-page clinical slideshow with CBCT measurements taken from the incorrect landmark, and reached out to top-tier maxillofacial surgeons (Alfaro, Raffani, Pagnoni). It was not generic disorganized thought. It was the exact looksmax structural deficit pattern-recognition system hyper-activated and directed inward under a precise neurochemical convergence.

The blackpill itself did not cause the episode. It supplied the content architecture because of pharmacological risk overlap on top of pre-existing substrate. I’m laying out the exact mechanisms, timeline, and my own lapses so others can avoid the same cascade.

Pre-existing vulnerabilities
  • Multi-year chronic sleep debt (2022 onward) from early-morning swim + AP Class schedule. Produced progressive prefrontal hypofunction, elevated amygdala reactivity, degraded executive regulation, and a significantly lowered psychosis threshold.
  • Undiagnosed ADHD (confirmed via neuropsych eval January 2026, right before the episode — objective baseline with no mood disorder flagged). Contributed hyperfocus, novelty-seeking, and impaired braking on cannabis escalation.
  • Severe cystic acne driving initial looksmax engagement and profound self-image disruption.
  • Athletic identity collapse
  • Sustained relational trauma and 2-month stalking campaign (300+ calls, multi-platform harassment). Produced acquired hypervigilance that later redirected inward.
None of these alone produced psychosis. The February 2026 pharmacological stack did.

Exact timeline and overlapping triggers (Feb 18–23 2026)
  • Feb 18: Abrupt cessation of Cannabis after months of heavy chronic use.
  • Feb 20 & 22: Multiple DMT trips (full 5-HT2A agonism with residual receptor sensitization persisting 48–72 hours).
  • Feb 23: New Adderall protocol initiated on zero sleep the preceding night due to cannabis withdrawal rebound insomnia.

Core Mechanisms
  1. Cannabis withdrawal and CB1 downregulation crash.
    Chronic high-dose THC produced extensive CB1 receptor downregulation. Abrupt stop caused acute endocannabinoid system collapse: loss of GABAergic/glutamatergic buffering that had become load-bearing for mood homeostasis, anxiety regulation, sleep architecture, and reality testing. This created the primary neurological destabilization.

  2. DMT-induced serotonergic sensitization
    I took DMT multiple times on February 20 and February 22 — the 48–72 hours immediately preceding acute psychosis onset. DMT is a full 5-HT2A agonist that produces profound serotonergic disruption. Residual receptor sensitization following repeated exposure in close temporal proximity lowered my threshold for
    psychotic symptom expression and disrupted the serotonergic regulatory systems that provide secondary reality testing.

  3. Acute + chronic sleep deprivation
    72–96 hours severely disrupted sleep layered on years of baseline debt. Prefrontal cortex hypofunction eliminated reality testing; amygdala dysregulation amplified threat scanning; dopamine sensitivity was massively heightened.

  4. Amphetamine-driven mesolimbic dopaminergic excess (the final trigger)
    Adderall (dopamine/norepinephrine reuptake inhibition + reversal transport) on a brain already in active cannabis withdrawal, DMT afterglow, and total sleep deprivation produced sustained dopaminergic overflow in the mesolimbic pathway — the established core mechanism of psychosis across etiologies.

  5. Heightened neuroplasticity + Hebbian conditioning from looksmax engagement
    The critical piece that made this blackpilled psychosis rather than generic paranoia. Stimulant + withdrawal + sleep deprivation + DMT created a high-plasticity window. The most recently and heavily trained neural pathways are preferentially amplified via Hebbian plasticity (“neurons that fire together wire together”).

The outcome was highly ordered, anatomically precise blackpilled thinking applied to a delusional premise. Standard reassurance failed because the prefrontal mechanisms required for reality testing were chemically offline.

My own lapses (full accountability)
  • Cold-turkey cannabis cessation instead of controlled taper.
  • DMT use during active withdrawal, relying on pharmacological knowledge as false security rather than risk assessment.
  • Initiating Adderall on zero sleep during the crash window.
  • Failure to recognize that my biochem background was being co-opted to rationalize continued exposure instead of enforcing stabilization.
Post-episode course

Triggers removed: 50+ days cannabis sobriety, full DMT abstinence, Adderall discontinued, sleep normalized on Trazodone. The airway delusion resolved as inflammation subsided and sleep returned. Fully stable 6+ weeks with no mood or psychotic symptoms. Currently on Lithium 300mg a temporary measure as the timeline inconsistent with primary Bipolar 1.

The Psych Ward Experience

The Psych ward is a trip, nearly everyone in there has had their brains melted. I had to go out of my way to stop them from putting me on oral Olanzapine (Zyprexa) which is probably the #1 looksmin drug of all time. I was in the psych ward for 10 days, for the first 3 or so I was still convinced I had a 3.9mm airway (the Hebbian connections and real inflammation were that firm so no progress was made. By the time my inflammation began to go down my breathing issues naturally resolved.

If you ever find yourself there: play along, take whatever they give you (pick the least bad meds), attend every group meeting, and tell the psych exactly what they want to hear. You cannot talk your way out of a psychiatric hold. Larp. Larp. Larp. The system is not built for nuanced mechanistic explanations in acute treatment.

Next Steps

I remain fully committed to evidence-based looksmaxing. I am scheduling a bimax+genio with undereye fat grafting with Dr.Eren Pera for next July. The delusional framework is resolved, the prior measurements were erroneous, but the underlying structural optimization goal stands. I'm looking forward to staying engaged with the community, missed you guys.

TLDR

I had a full drug-induced blackpilled psychotic episode: convinced my airway was collapsing, made a detailed slideshow, and contacted top maxfax surgeons. The stack was cold-turkey cannabis withdrawal + DMT + Adderall on zero sleep, all on top of years of sleep debt, undiagnosed ADHD, acne, identity loss, and trauma. My trained looksmax structural scanning became the fixed delusion through neuroplasticity. Stabilized fast once triggers were removed — diagnosis revised to substance-induced, now on low-dose Lithium. Psych ward: had to larp hard and dodge Zyprexa. Lesson: your deepest insecurities get broadcast as fixed delusion the moment regulatory systems collapse.

@Orka @Sayori @abzz @aids @chudpiller
brutal story bro glad ur doing better bro
 
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I’m gonna be super careful with psychedelics & dopaminergic drugs
Stims are the highest risk drug for sure. But if you use both be mindful of the neuroplasticity window
Proper sleep is a must for everyone though. Sleep deprivation is the worst feeling
100% and sleep deprivation itself can cause psychosis
 
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Stims are the highest risk drug for sure. But if you use both be mindful of the neuroplasticity window

100% and sleep deprivation itself can cause psychosis
Do you know anything about using an NMDA antagonist like memantine with an amphetamine every time the amphetamine is taken? Ive been looking into this because in theory, it should slow down how fast tolerance builds up
 
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Do you know anything about using an NMDA antagonist like memantine with an amphetamine every time the amphetamine is taken? Ive been looking into this because in theory, it should slow down how fast tolerance builds up
You could do it but memantine has a really long half life ~60 hours so it would be more of a "maintain stable blood serum levels" rather than dose every time.

Also i've heard it can blunt the effects subjectively

Try it and find out tho
 
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You could do it but memantine has a really long half life ~60 hours so it would be more of a "maintain stable blood serum levels" rather than dose every time.

Also i've heard it can blunt the effects subjectively

Try it and find out tho
Any clue if ketamine every 1-2 weeks at a low to medium dose on a non stim day could be any better?

Just curious to see if you know more about this. It’s probably not even practical to do this but niggas do anything for optimization anyway
 
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Any clue if ketamine every 1-2 weeks at a low to medium dose on a non stim day could be any better?

Just curious to see if you know more about this. It’s probably not even practical to do this but niggas do anything for optimization anyway
You should do ketamine (just for fun)

Memantine is def a cleaner way to do it but if I was using amphetamines I'd do the ketamine

btw do you have a source for ket lmfao
 
  • +1
Reactions: insignia_
I’m posting this as a detailed mechanistic breakdown of the factors causing my psychotic break in mid February. It's both a cautionary tale and a deep dive into the soft white underbelly of the human psyche. Enjoy

I had zero psychiatric history prior to February 2026. In mid February I had a full drug-induced psychotic episode that led to 5150 hold, inpatient admission, and an initial Bipolar 1 diagnosis that has since been formally revised. The content of the psychosis was 100% blackpilled structural airway inadequacy: convinced my airway was 3.9 mm and collapsing, produced a 23-page clinical slideshow with CBCT measurements taken from the incorrect landmark, and reached out to top-tier maxillofacial surgeons (Alfaro, Raffani, Pagnoni). It was not generic disorganized thought. It was the exact looksmax structural deficit pattern-recognition system hyper-activated and directed inward under a precise neurochemical convergence.

The blackpill itself did not cause the episode. It supplied the content architecture because of pharmacological risk overlap on top of pre-existing substrate. I’m laying out the exact mechanisms, timeline, and my own lapses so others can avoid the same cascade.

Pre-existing vulnerabilities
  • Multi-year chronic sleep debt (2022 onward) from early-morning swim + AP Class schedule. Produced progressive prefrontal hypofunction, elevated amygdala reactivity, degraded executive regulation, and a significantly lowered psychosis threshold.
  • Undiagnosed ADHD (confirmed via neuropsych eval January 2026, right before the episode — objective baseline with no mood disorder flagged). Contributed hyperfocus, novelty-seeking, and impaired braking on cannabis escalation.
  • Severe cystic acne driving initial looksmax engagement and profound self-image disruption.
  • Athletic identity collapse
  • Sustained relational trauma and 2-month stalking campaign (300+ calls, multi-platform harassment). Produced acquired hypervigilance that later redirected inward.
None of these alone produced psychosis. The February 2026 pharmacological stack did.

Exact timeline and overlapping triggers (Feb 18–23 2026)
  • Feb 18: Abrupt cessation of Cannabis after months of heavy chronic use.
  • Feb 20 & 22: Multiple DMT trips (full 5-HT2A agonism with residual receptor sensitization persisting 48–72 hours).
  • Feb 23: New Adderall protocol initiated on zero sleep the preceding night due to cannabis withdrawal rebound insomnia.

Core Mechanisms
  1. Cannabis withdrawal and CB1 downregulation crash.
    Chronic high-dose THC produced extensive CB1 receptor downregulation. Abrupt stop caused acute endocannabinoid system collapse: loss of GABAergic/glutamatergic buffering that had become load-bearing for mood homeostasis, anxiety regulation, sleep architecture, and reality testing. This created the primary neurological destabilization.

  2. DMT-induced serotonergic sensitization
    I took DMT multiple times on February 20 and February 22 — the 48–72 hours immediately preceding acute psychosis onset. DMT is a full 5-HT2A agonist that produces profound serotonergic disruption. Residual receptor sensitization following repeated exposure in close temporal proximity lowered my threshold for
    psychotic symptom expression and disrupted the serotonergic regulatory systems that provide secondary reality testing.

  3. Acute + chronic sleep deprivation
    72–96 hours severely disrupted sleep layered on years of baseline debt. Prefrontal cortex hypofunction eliminated reality testing; amygdala dysregulation amplified threat scanning; dopamine sensitivity was massively heightened.

  4. Amphetamine-driven mesolimbic dopaminergic excess (the final trigger)
    Adderall (dopamine/norepinephrine reuptake inhibition + reversal transport) on a brain already in active cannabis withdrawal, DMT afterglow, and total sleep deprivation produced sustained dopaminergic overflow in the mesolimbic pathway — the established core mechanism of psychosis across etiologies.

  5. Heightened neuroplasticity + Hebbian conditioning from looksmax engagement
    The critical piece that made this blackpilled psychosis rather than generic paranoia. Stimulant + withdrawal + sleep deprivation + DMT created a high-plasticity window. The most recently and heavily trained neural pathways are preferentially amplified via Hebbian plasticity (“neurons that fire together wire together”).

The outcome was highly ordered, anatomically precise blackpilled thinking applied to a delusional premise. Standard reassurance failed because the prefrontal mechanisms required for reality testing were chemically offline.

My own lapses (full accountability)
  • Cold-turkey cannabis cessation instead of controlled taper.
  • DMT use during active withdrawal, relying on pharmacological knowledge as false security rather than risk assessment.
  • Initiating Adderall on zero sleep during the crash window.
  • Failure to recognize that my biochem background was being co-opted to rationalize continued exposure instead of enforcing stabilization.
Post-episode course

Triggers removed: 50+ days cannabis sobriety, full DMT abstinence, Adderall discontinued, sleep normalized on Trazodone. The airway delusion resolved as inflammation subsided and sleep returned. Fully stable 6+ weeks with no mood or psychotic symptoms. Currently on Lithium 300mg a temporary measure as the timeline inconsistent with primary Bipolar 1.

The Psych Ward Experience

The Psych ward is a trip, nearly everyone in there has had their brains melted. I had to go out of my way to stop them from putting me on oral Olanzapine (Zyprexa) which is probably the #1 looksmin drug of all time. I was in the psych ward for 10 days, for the first 3 or so I was still convinced I had a 3.9mm airway (the Hebbian connections and real inflammation were that firm so no progress was made. By the time my inflammation began to go down my breathing issues naturally resolved.

If you ever find yourself there: play along, take whatever they give you (pick the least bad meds), attend every group meeting, and tell the psych exactly what they want to hear. You cannot talk your way out of a psychiatric hold. Larp. Larp. Larp. The system is not built for nuanced mechanistic explanations in acute treatment.

Next Steps

I remain fully committed to evidence-based looksmaxing. I am scheduling a bimax+genio with undereye fat grafting with Dr.Eren Pera for next July. The delusional framework is resolved, the prior measurements were erroneous, but the underlying structural optimization goal stands. I'm looking forward to staying engaged with the community, missed you guys.

TLDR

I had a full drug-induced blackpilled psychotic episode: convinced my airway was collapsing, made a detailed slideshow, and contacted top maxfax surgeons. The stack was cold-turkey cannabis withdrawal + DMT + Adderall on zero sleep, all on top of years of sleep debt, undiagnosed ADHD, acne, identity loss, and trauma. My trained looksmax structural scanning became the fixed delusion through neuroplasticity. Stabilized fast once triggers were removed — diagnosis revised to substance-induced, now on low-dose Lithium. Psych ward: had to larp hard and dodge Zyprexa. Lesson: your deepest insecurities get broadcast as fixed delusion the moment regulatory systems collapse.

@Orka @Sayori @abzz @aids @chudpiller
fuck weed
 
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You should do ketamine (just for fun)

Memantine is def a cleaner way to do it but if I was using amphetamines I'd do the ketamine

btw do you have a source for ket lmfao
I’ll probably use the ket for the purpose we’re talking about but surely I’ll already enjoy using it hahaha, nice side benefit

I do not have a source atm other than dh. Do you know a reliable source? Any seller on dh? Would appreciate it if you did
 
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I’ll probably use the ket for the purpose we’re talking about but surely I’ll already enjoy using it hahaha, nice side benefit
win win
I do not have a source atm other than dh. Do you know a reliable source? Any seller on dh? Would appreciate it if you did
I have some my boys gave me on telegram thats it. What is dh :feelswhy:
 
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win win

I have some my boys gave me on telegram thats it. What is dh :feelswhy:
I say dh just because I didn’t wanna say drughub. Its not a secret or anything. I just dont wanna look like a junkie for talking about this shit jfl
 
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I say dh just because I didn’t wanna say drughub. Its not a secret or anything. I just dont wanna look like a junkie for talking about this shit jfl
JFL

drugs have applications outside of rec abuse
 
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JFL

drugs have applications outside of rec abuse
Yes yes, I know. It’s just the way other people perceive you when they know this stuff abt you. I don’t care much on this forum since its not linked to my identity anyway
 
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Yes yes, I know. It’s just the way other people perceive you when they know this stuff abt you. I don’t care much on this forum since its not linked to my identity anyway
Yeah I know twin
 
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How does ket feel btw? I assume you’ve tried it
I actually haven't I want to hella bad lmfao

ket is a bad one to get addicted to tho
 
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I actually haven't I want to hella bad lmfao

ket is a bad one to get addicted to tho
Yeah, that’s why I’d recommend we only stick to low ish doses used infrequently
 
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Yeah, that’s why I’d recommend we only stick to low ish doses used infrequently
I have a very addictive personality thats my issue

High novelty drive from AHDH too.

Brutal combo
 
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I have a very addictive personality thats my issue

High novelty drive from AHDH too.

Brutal combo
We in the same boat blud :feelswhy:

Shits tough

My ADHD meds are the only thing that make me not seek a ton of useless stimulation

But sometimes I gotta fight the urge to take a super high dose to see what it feels like
 
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We in the same boat blud :feelswhy:

Shits tough

My ADHD meds are the only thing that make me not seek a ton of useless stimulation
You gotta try psychedelics lmfao

If you think stims are good oh boy

And not an issue if you don't be retarded with them

I don't regret any of my psych use.

In fact it benefited me a ton
But sometimes I gotta fight the urge to take a super high dose to see what it feels like
Don't, once you abuse them you can't go back. It will never work functionally properly again tbh. Were addicts.
 
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You gotta try psychedelics lmfao

If you think stims are good oh boy

And not an issue if you don't be retarded with them

I don't regret any of my psych use.

In fact it benefited me a ton

Don't, once you abuse them you can't go back. It will never work functionally properly again tbh. Were addicts.
Are you in the US? You have good sources for psychedelics?

And yeah I definitely won’t up my dose. My meds are already extended release which I don’t like too much, but there’s no pure dextro that can be prescribed where I am. Need to wait to go to the US
 
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DNR will read later tho love you BR :owo:
 
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Are you in the US? You have good sources for psychedelics?
Grow shrooms yourself, and make DMT

I can ask friends for sources, I can def hook you up if you want.
And yeah I definitely won’t up my dose. My meds are already extended release which I don’t like too much, but there’s no pure dextro that can be prescribed where I am. Need to wait to go to the US
Gotchu, makes a lot of sense.

I'm sure snorting IR is a great high.
 
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Grow shrooms yourself, and make DMT

I can ask friends for sources, I can def hook you up if you want.

Gotchu, makes a lot of sense.

I'm sure snorting IR is a great high.
Might grow them myself, i dont see an issue. I’m not sure my future roommate will like that though :lul:

Also I did not mean snorting :feelswhy: not trying to become an addict. Id only source pure dextro powder if a dexedrine prescription was too expensive. Even then, taking it through the nose gotta fucking burn

Id dose it properly & all
 
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Might grow them myself, i dont see an issue. I’m not sure my future roommate will like that though :lul:
really easy to grow, id suggest PE
Also I did not mean snorting :feelswhy: not trying to become an addict. Id only source pure dextro powder if a dexedrine prescription was too expensive. Even then, taking it through the nose gotta fucking burn
LMFAO

what you really wanted was 100% dextroamphetamine not 75/25% mixed salts

Cortisol GIF

Id dose it properly & all
good man
 
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I’m posting this as a detailed mechanistic breakdown of the factors causing my psychotic break in mid February. It's both a cautionary tale and a deep dive into the soft white underbelly of the human psyche. Enjoy

I had zero psychiatric history prior to February 2026. In mid February I had a full drug-induced psychotic episode that led to 5150 hold, inpatient admission, and an initial Bipolar 1 diagnosis that has since been formally revised. The content of the psychosis was 100% blackpilled structural airway inadequacy: convinced my airway was 3.9 mm and collapsing, produced a 23-page clinical slideshow with CBCT measurements taken from the incorrect landmark, and reached out to top-tier maxillofacial surgeons (Alfaro, Raffani, Pagnoni). It was not generic disorganized thought. It was the exact looksmax structural deficit pattern-recognition system hyper-activated and directed inward under a precise neurochemical convergence.

The blackpill itself did not cause the episode. It supplied the content architecture because of pharmacological risk overlap on top of pre-existing substrate. I’m laying out the exact mechanisms, timeline, and my own lapses so others can avoid the same cascade.

Pre-existing vulnerabilities
  • Multi-year chronic sleep debt (2022 onward) from early-morning swim + AP Class schedule. Produced progressive prefrontal hypofunction, elevated amygdala reactivity, degraded executive regulation, and a significantly lowered psychosis threshold.
  • Undiagnosed ADHD (confirmed via neuropsych eval January 2026, right before the episode — objective baseline with no mood disorder flagged). Contributed hyperfocus, novelty-seeking, and impaired braking on cannabis escalation.
  • Severe cystic acne driving initial looksmax engagement and profound self-image disruption.
  • Athletic identity collapse
  • Sustained relational trauma and 2-month stalking campaign (300+ calls, multi-platform harassment). Produced acquired hypervigilance that later redirected inward.
None of these alone produced psychosis. The February 2026 pharmacological stack did.

Exact timeline and overlapping triggers (Feb 18–23 2026)
  • Feb 18: Abrupt cessation of Cannabis after months of heavy chronic use.
  • Feb 20 & 22: Multiple DMT trips (full 5-HT2A agonism with residual receptor sensitization persisting 48–72 hours).
  • Feb 23: New Adderall protocol initiated on zero sleep the preceding night due to cannabis withdrawal rebound insomnia.

Core Mechanisms
  1. Cannabis withdrawal and CB1 downregulation crash.
    Chronic high-dose THC produced extensive CB1 receptor downregulation. Abrupt stop caused acute endocannabinoid system collapse: loss of GABAergic/glutamatergic buffering that had become load-bearing for mood homeostasis, anxiety regulation, sleep architecture, and reality testing. This created the primary neurological destabilization.

  2. DMT-induced serotonergic sensitization
    I took DMT multiple times on February 20 and February 22 — the 48–72 hours immediately preceding acute psychosis onset. DMT is a full 5-HT2A agonist that produces profound serotonergic disruption. Residual receptor sensitization following repeated exposure in close temporal proximity lowered my threshold for
    psychotic symptom expression and disrupted the serotonergic regulatory systems that provide secondary reality testing.

  3. Acute + chronic sleep deprivation
    72–96 hours severely disrupted sleep layered on years of baseline debt. Prefrontal cortex hypofunction eliminated reality testing; amygdala dysregulation amplified threat scanning; dopamine sensitivity was massively heightened.

  4. Amphetamine-driven mesolimbic dopaminergic excess (the final trigger)
    Adderall (dopamine/norepinephrine reuptake inhibition + reversal transport) on a brain already in active cannabis withdrawal, DMT afterglow, and total sleep deprivation produced sustained dopaminergic overflow in the mesolimbic pathway — the established core mechanism of psychosis across etiologies.

  5. Heightened neuroplasticity + Hebbian conditioning from looksmax engagement
    The critical piece that made this blackpilled psychosis rather than generic paranoia. Stimulant + withdrawal + sleep deprivation + DMT created a high-plasticity window. The most recently and heavily trained neural pathways are preferentially amplified via Hebbian plasticity (“neurons that fire together wire together”).

The outcome was highly ordered, anatomically precise blackpilled thinking applied to a delusional premise. Standard reassurance failed because the prefrontal mechanisms required for reality testing were chemically offline.

My own lapses (full accountability)
  • Cold-turkey cannabis cessation instead of controlled taper.
  • DMT use during active withdrawal, relying on pharmacological knowledge as false security rather than risk assessment.
  • Initiating Adderall on zero sleep during the crash window.
  • Failure to recognize that my biochem background was being co-opted to rationalize continued exposure instead of enforcing stabilization.
Post-episode course

Triggers removed: 50+ days cannabis sobriety, full DMT abstinence, Adderall discontinued, sleep normalized on Trazodone. The airway delusion resolved as inflammation subsided and sleep returned. Fully stable 6+ weeks with no mood or psychotic symptoms. Currently on Lithium 300mg a temporary measure as the timeline inconsistent with primary Bipolar 1.

The Psych Ward Experience

The Psych ward is a trip, nearly everyone in there has had their brains melted. I had to go out of my way to stop them from putting me on oral Olanzapine (Zyprexa) which is probably the #1 looksmin drug of all time. I was in the psych ward for 10 days, for the first 3 or so I was still convinced I had a 3.9mm airway (the Hebbian connections and real inflammation were that firm so no progress was made. By the time my inflammation began to go down my breathing issues naturally resolved.

If you ever find yourself there: play along, take whatever they give you (pick the least bad meds), attend every group meeting, and tell the psych exactly what they want to hear. You cannot talk your way out of a psychiatric hold. Larp. Larp. Larp. The system is not built for nuanced mechanistic explanations in acute treatment.

Next Steps

I remain fully committed to evidence-based looksmaxing. I am scheduling a bimax+genio with undereye fat grafting with Dr.Eren Pera for next July. The delusional framework is resolved, the prior measurements were erroneous, but the underlying structural optimization goal stands. I'm looking forward to staying engaged with the community, missed you guys.

TLDR

I had a full drug-induced blackpilled psychotic episode: convinced my airway was collapsing, made a detailed slideshow, and contacted top maxfax surgeons. The stack was cold-turkey cannabis withdrawal + DMT + Adderall on zero sleep, all on top of years of sleep debt, undiagnosed ADHD, acne, identity loss, and trauma. My trained looksmax structural scanning became the fixed delusion through neuroplasticity. Stabilized fast once triggers were removed — diagnosis revised to substance-induced, now on low-dose Lithium. Psych ward: had to larp hard and dodge Zyprexa. Lesson: your deepest insecurities get broadcast as fixed delusion the moment regulatory systems collapse.

@Orka @Sayori @abzz @aids @chudpiller
Read it all instead of just putting it in my bookmarks happy to see your here with me bro the amount of times ive been this close to a fucking mental joint is insane bro happy your back on the true goal tho bro:love:
 
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Read it all instead of just putting it in my bookmarks happy to see your here with me bro the amount of times ive been this close to a fucking mental joint is insane bro happy your back on the true goal tho bro:love:
Thanks for reading Bhai!

I'm sorry you've been through it, I hope you're doing well

Happy to be back!
 
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Thanks for reading Bhai!

I'm sorry you've been through it, I hope you're doing well

Happy to be back!
Its all my fault so its chill
 
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really easy to grow, id suggest PE
Will look for your old guides & see if you have talked about this. If not, do you know resources for info on growing them myself?

LMFAO

what you really wanted was 100% dextroamphetamine not 75/25% mixed salts
Oh yeah adderall probably feels bad. Some of my compulsions & behaviors already get amplified on vyvanse, so the levo from adderall would make it even worse lol

Did you react well to adderall?
 
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