Pharmacology ascension plan

n7ebnenta7ar

n7ebnenta7ar

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Hello guys I'm a Male 5’7 growth plates not fused yet and here is my ascension plan if someone would like to help me fund it dm me
(Comes with updates if someone decide to help me out)

HGH - 3IU's a day + 700mg berberine monitor blood sugar until broke
Anavar - 6mg per day + 2mg twice a day of Erdafitinib for 3 months
Exemestane - 20mg per day until no money is left
Teriparatide - 20mcg per day for 3 months
Vorinostat - 40mcg per day for 3 months
Gluthathione - 600mg per day until real results

Some of you guys May ask why pair Erdafitinib with Anavar? As we know studies show that as we know Anavar can temporarily stimulate bone growth, but it also accelerates the maturation and closure of the growth plates, which may reduce final height, the theoretical use of FGFR3 inhibitors aims to slow this process, helping to maintain growth plate activity and prolong skeletal elongation even during steroid exposure
This is why I would use FGFR3 inhibitor (Erdafitinib)

Exemestane is included in this stack to lower natural estrogen levels, which can help extend the period of growth. Since estrogen is the main hormone driving Early growth plate closure reducing it may allow for a few extra years of skeletal elongation (if used for a long period of time do not expect 1 extra year of skeletal elongation if only used for a couple of months)

Vorinostat is included to support bone mass by activating the RUNX2 gene which plays a major role in bone formation it basically helps the body produce more bone tissue, potentially enhancing overall skeletal development.

Erdafitnib part is 100% theoretical for discussion purposes only.
 
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Remove vorinostat tbh
 
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We need it otherwise without it Erdafitinib will molest your bonemass
 
The only solution is to remove completely HDAC inhibitors because theres quite literally no HDAC inhibs who doesnt inhibit at least class I or IIa they all do, the best solution would be Martinostat tbh
 
The only solution is to remove completely HDAC inhibitors because theres quite literally no HDAC inhibs who doesnt inhibit at least class I or IIa they all do, the best solution would be Martinostat tbh
Thats what im saying, HDACi aren’t realistic
 
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