Docc
Iron
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was scrolling through wikipedia about neoteny in humans and something caught my eye:
en.wikipedia.org
I think if i've read this correctly the theory goes that early day humans would cook their food and burn it, which would in turn mutate the DNA to essentially remove gene networks that control the firing of genetic activity patterns that mark adulthood. Now as someone who semi believes in the raw meat primal diet sheit I def think this is another argument FOR the raw/primal diet.
the theory also argues this through great apes and humanoids with them having very prominent biomarkers for adulthood, as these lesser intelligent species did not develop the technology to cooking which checks out:
and the same can be said for other animals as well humans very distinctly have less prominent adulthood biomarkers (facially) when compared to other mammals.
Neotenous unideal features: narrow clavs, shorter legs, larger skull in proportion to body (but in a deformed boneless childlike kinda way) , smaller mandible maxilla chin and browridge, larger rounder eyes, less overall masculinity/dimorphism
tldr: if you've ever eaten burnt food you will be a perma deformed childlike subhuman
was scrolling through wikipedia about neoteny in humans and something caught my eye:
"Cooked food and protective genome simplification
Based on calculations that show that more complex gene networks are more vulnerable to mutations as more conditions that are necessary but not sufficient increases the risk of one of them being hit, there is a theory that mutagens in food were more likely to be formed when food was burned while being cooked by human ancestors lacking modern cooking technology or the greater intelligence of modern humans. These commonly present mutagens thus selected against complex gene networks because longer genomes present a larger target for mutation. This theory successfully predicts that the human genome is shorter than other Great Ape genomes and that there are significantly more defunct pseudogenes with functional homologs in the chimpanzee genome than vice versa. While the protein coding portion of the FOXP2 gene is identical to that in Neanderthals, there is one point mutation in the regulatory part thereof (modern humans having a T where Neanderthals and all nonhuman vertebrates have an A). The observation that the effect of that difference is that the modern human FOXP2 gene does not interact with RNA from other genes while all other vertebrate including Neanderthal varieties did agrees with the idea that modern human origin was marked by the elimination (not formation) of complex gene networks, as predicted by this model. The researchers behind the theory argue that neoteny is a side effect of the destruction of gene networks preventing the firing of genetic activity patterns that marked adulthood in prehuman ancestors."
Neoteny in humans - Wikipedia
I think if i've read this correctly the theory goes that early day humans would cook their food and burn it, which would in turn mutate the DNA to essentially remove gene networks that control the firing of genetic activity patterns that mark adulthood. Now as someone who semi believes in the raw meat primal diet sheit I def think this is another argument FOR the raw/primal diet.
the theory also argues this through great apes and humanoids with them having very prominent biomarkers for adulthood, as these lesser intelligent species did not develop the technology to cooking which checks out:
and the same can be said for other animals as well humans very distinctly have less prominent adulthood biomarkers (facially) when compared to other mammals.
Neotenous unideal features: narrow clavs, shorter legs, larger skull in proportion to body (but in a deformed boneless childlike kinda way) , smaller mandible maxilla chin and browridge, larger rounder eyes, less overall masculinity/dimorphism
tldr: if you've ever eaten burnt food you will be a perma deformed childlike subhuman
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