Aboulfz
Iron
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How AAS potentially improve maxillary devolement.
in this study:
We can see one of the few human studies investigating anabolic steriods and craniofacial morphology compared 4 AAs user with 4 matched controls.
The AAs group used nandrolone decanote (deca durabolin) + stanazolol ( winstrol). Note that the AAS users where adults in their early to mid 20s.
Using Mcnamara cephalometric analysis reseachers found the Co-A (Condylion-Point A) distance, a standard mesure of effective maxillary length, was signaifcantly greater in AAs group ( 104,8 nm vs 95 nm: p = 0,020).
Other craniofacial measurements (ANS–M, Co–Gn, A–Nperp, P–Nperp) also trended higher in AAS users but did not reach statistical significance.
One of the most interesting findings wasn't just the significant increase in Co–A (effective maxillary length). 3 out of 4 AAS users also presented Angle Class II malocclusion, while every control subject had Class I occlusion. One of those Class II cases was diagnosed as skeletal maxillary protrusion. Obviously, this doesn't prove AAS caused forward maxillary growth, but when combined with the significantly greater Co–A measurement, it raises an interesting possibility that AAS may influence maxillary morphology.
Here is a good picture indicating how different Bites can look like:
There is a few studies also on how nandrolone changes the maxilla in rats;
This studie shows how high-dose nadrolone produced overt shape changes, notably a maxillomandibular, anteroposterior jaw discrepancy due to maxillary excess.
TDLR: AAS like nandrolone decanote (deca durabolin) + stanazolol ( winstrol) promote and change the maxilla in both humans and rats.
in this study:
We can see one of the few human studies investigating anabolic steriods and craniofacial morphology compared 4 AAs user with 4 matched controls.
The AAs group used nandrolone decanote (deca durabolin) + stanazolol ( winstrol). Note that the AAS users where adults in their early to mid 20s.
Using Mcnamara cephalometric analysis reseachers found the Co-A (Condylion-Point A) distance, a standard mesure of effective maxillary length, was signaifcantly greater in AAs group ( 104,8 nm vs 95 nm: p = 0,020).
Other craniofacial measurements (ANS–M, Co–Gn, A–Nperp, P–Nperp) also trended higher in AAS users but did not reach statistical significance.
One of the most interesting findings wasn't just the significant increase in Co–A (effective maxillary length). 3 out of 4 AAS users also presented Angle Class II malocclusion, while every control subject had Class I occlusion. One of those Class II cases was diagnosed as skeletal maxillary protrusion. Obviously, this doesn't prove AAS caused forward maxillary growth, but when combined with the significantly greater Co–A measurement, it raises an interesting possibility that AAS may influence maxillary morphology.
Here is a good picture indicating how different Bites can look like:
There is a few studies also on how nandrolone changes the maxilla in rats;
The Angle Orthodontist
The Angle Orthodontist is the official publication of the Edward H. Angle Society of Orthodontists (EHASO) and is published bimonthly by The EH Angle Education and Research Foundation Inc.
angle-orthodontist.kglmeridian.com
This studie shows how high-dose nadrolone produced overt shape changes, notably a maxillomandibular, anteroposterior jaw discrepancy due to maxillary excess.
TDLR: AAS like nandrolone decanote (deca durabolin) + stanazolol ( winstrol) promote and change the maxilla in both humans and rats.