Why DHT is important and should NOT be nuked to the ground

[DR. NICKGA]

Bro it wasn’t dht
It was your fucked up (micronutrients profile your high cortisol and prolactin low E2 low igf-1 low sleep high Bp and water retention)

I sincerely believe that unless your using 500 mg of test+++ you shouldn’t need to use fin (dut is a death sentence don’t talk about it) haven’t you tead that study where they use nandrolone with dut vs nandrolone alone and nandrolone alone caused less hairloss haven’t you read that study where they used Tamoxifen with finasteride and they got hairloss

My new thread gonna absolute destroy you with the power of facts and logic

THAT DHT CAUSE BALDING

YOU TAINTED ME, PREPARE TO BE DESTROYED

 

[DR. NICKGA]

so what the fuck do we do if ur going bald later on in life ? i dont wanna have to take that poison finasteride or dutasteride shit, whats the next solution?

WITHOUT FIN AND DUT

YOU NEXT SOLUTION IS TO BE BALD YOU UTTERLY SUBHUMAN

 

[Tom Jones]

Not a molecule

What this mean?

 

[iblamemandible7]

so what the fuck do we do if ur going bald later on in life ? i dont wanna have to take that poison finasteride or dutasteride shit, whats the next solution?

There is none retard quit being a pussy and just take the damn pill you scared of it or smth?

 

[chadhamed]

What this mean?

Means that im not reading a molecule

 

[DR. NICKGA]

No matter how many study i post everyone gonna think dht ks a useless hormone

Yes its fucking usseless

 

[Trisomy 21]

There is none retard quit being a pussy and just take the damn pill you scared of it or smth?

shut the fuck up faggot, theres many niggers that literally said their dick stopped working after even stopping retard.

so now u have good hair still, but ur dick doesnt work, whats the point? idiot faggot, u will have to turn into a bottom and get fucked by men, cuz u cant use ur cock

 

[JohnBaza]

I'm tired of trannies here trying to convince people that DHT is useless. This thread will cover the importance of DHT and why you should not block it by the use of 5ARIs.

My friend @20/04/2008 made a really good thread on it, so I will just TLDR it:

DHT is not only the stronger version of Testosterone (2-4 times higher affinity to the androgen receptor), but also binds differently to the AR because of its unique structure.

This means each androgen exerts unique effects, including DHT. So only using Testosterone is not the right solution.

---

Now to the other uses of the enzyme 5α-reductase: It doesn't only affect the conversion of T to DHT, here are some other reactions:

Cholestenone → 5α-Cholestanone
Progesterone → 5α-Dihydroprogesterone
3α-Dihydroprogesterone → Allopregnanolone
3β-Dihydroprogesterone → Isopregnanolone
Deoxycorticosterone → 5α-Dihydrodeoxycorticosterone
Corticosterone → 5α-Dihydrocorticosterone
Aldosterone → 5α-Dihydroaldosterone
Androstenedione → 5α-Androstanedione
Nandrolone → 5α-Dihydronandrolone

The functions of some of the 5α-reduced steroids are unknown, that's why it isn't a good idea to "just use fin bro because you'll be fine."

Allopregnanolone is an important neurosteroid made from 3α-dihydroprogesterone. Without 5α-reductase on the other hand, this important neurosteroid can't be synthesized.

Progesterone → DHP via 5α-reductase
DHP → Allopregnanolone via 3α-HSD
Allopregnanolone → Positive allosteric modulator of GABA-A receptors

TLDR: No allopregnanolone -> More Anxiety, brain fog, stress, etc.

View attachment 3391024

Not forgetting the sexual dysfunction aspect of inhibiting 5AR.

The cells of the corpus cavernosum have androgen receptors, when you agonize these receptors they produce endothelial nitric oxide. NO triggers the relaxation of smooth muscle cells, leading to vasodilation and increased blood flow, which is needed for achieving and maintaining an erection. This can't be replaced with cialis, you need the local AR activation.

The only solution to solve this, is to have high levels of testosterone especially locally in the area of the penis or to use topical DHT/testosterone gels, just to maintain the normal function of the penis.

There is also the mental aspect of the erectile dysfunction. The main role of the sex drive of males consists of the dopamine/oxytocin signalling, which is inhibited by the use of 5ARIs. So in short, 5ARIs result in a reduction of libido.

View attachment 3391036

DHT is important for bone mass, which in the cited study even resulted in an increase of bone mass. DHT is also important for muscle strength.

"We have previously reported that DHT treatment increases bone mass in orchidectomized mice and that this effect is independent of estrogen receptors."

5AR is important for the metabolism of cortisol in the liver according to this study.

Persistent sexual side effects by the way... "Subjects reported new-onset persistent sexual dysfunction associated with the use of finasteride: 94% developed low libido, 92% developed erectile dysfunction, 92% developed decreased arousal, and 69% developed problems with orgasm."

Also ever wondered why most of the Finasteride users like Orc and Clavicular here never experience that many side effects from the 5ARI use? It's because they blast exogenous hormones to the point where the accumulated testosterone compensates for most of the side effects of the lacking DHT.

---

See? DHT is a vital male hormone, blocking it completely is harmful and has many other harms like discussed in this thread.

View attachment 3391173

Tag list: @halloweed @JohnDoe @Always Stay You @ConfusedBolivian

ayo this is facts nigga

 

[JohnBaza]

I'm tired of trannies here trying to convince people that DHT is useless. This thread will cover the importance of DHT and why you should not block it by the use of 5ARIs.

My friend @20/04/2008 made a really good thread on it, so I will just TLDR it:

DHT is not only the stronger version of Testosterone (2-4 times higher affinity to the androgen receptor), but also binds differently to the AR because of its unique structure.

This means each androgen exerts unique effects, including DHT. So only using Testosterone is not the right solution.

---

Now to the other uses of the enzyme 5α-reductase: It doesn't only affect the conversion of T to DHT, here are some other reactions:

Cholestenone → 5α-Cholestanone
Progesterone → 5α-Dihydroprogesterone
3α-Dihydroprogesterone → Allopregnanolone
3β-Dihydroprogesterone → Isopregnanolone
Deoxycorticosterone → 5α-Dihydrodeoxycorticosterone
Corticosterone → 5α-Dihydrocorticosterone
Aldosterone → 5α-Dihydroaldosterone
Androstenedione → 5α-Androstanedione
Nandrolone → 5α-Dihydronandrolone

The functions of some of the 5α-reduced steroids are unknown, that's why it isn't a good idea to "just use fin bro because you'll be fine."

Allopregnanolone is an important neurosteroid made from 3α-dihydroprogesterone. Without 5α-reductase on the other hand, this important neurosteroid can't be synthesized.

Progesterone → DHP via 5α-reductase
DHP → Allopregnanolone via 3α-HSD
Allopregnanolone → Positive allosteric modulator of GABA-A receptors

TLDR: No allopregnanolone -> More Anxiety, brain fog, stress, etc.

View attachment 3391024

Not forgetting the sexual dysfunction aspect of inhibiting 5AR.

The cells of the corpus cavernosum have androgen receptors, when you agonize these receptors they produce endothelial nitric oxide. NO triggers the relaxation of smooth muscle cells, leading to vasodilation and increased blood flow, which is needed for achieving and maintaining an erection. This can't be replaced with cialis, you need the local AR activation.

The only solution to solve this, is to have high levels of testosterone especially locally in the area of the penis or to use topical DHT/testosterone gels, just to maintain the normal function of the penis.

There is also the mental aspect of the erectile dysfunction. The main role of the sex drive of males consists of the dopamine/oxytocin signalling, which is inhibited by the use of 5ARIs. So in short, 5ARIs result in a reduction of libido.

View attachment 3391036

DHT is important for bone mass, which in the cited study even resulted in an increase of bone mass. DHT is also important for muscle strength.

"We have previously reported that DHT treatment increases bone mass in orchidectomized mice and that this effect is independent of estrogen receptors."

5AR is important for the metabolism of cortisol in the liver according to this study.

Persistent sexual side effects by the way... "Subjects reported new-onset persistent sexual dysfunction associated with the use of finasteride: 94% developed low libido, 92% developed erectile dysfunction, 92% developed decreased arousal, and 69% developed problems with orgasm."

Also ever wondered why most of the Finasteride users like Orc and Clavicular here never experience that many side effects from the 5ARI use? It's because they blast exogenous hormones to the point where the accumulated testosterone compensates for most of the side effects of the lacking DHT.

---

See? DHT is a vital male hormone, blocking it completely is harmful and has many other harms like discussed in this thread.

View attachment 3391173

Tag list: @halloweed @JohnDoe @Always Stay You @ConfusedBolivian

W thread bookmarked

 

[idkwhattoputhere]

dnr, mpmd did a case study on two brothers one had less dht than a girl and they developed the exact same

 

[iblamemandible7]

shut the fuck up faggot, theres many niggers that literally said their dick stopped working after even stopping retard.

so now u have good hair still, but ur dick doesnt work, whats the point? idiot faggot, u will have to turn into a bottom and get fucked by men, cuz u cant use ur cock

Ok just dont take it and be bald faggot idc have fun being a sexually frustrated baldcel

 

[JcPenny]

only the dumbest of the goyim think that a hormone that your body produces is not important, the dumbest of the gentiles

 

[JcPenny]

the problem is that people make too much DHT by jerking off constantly. UNHEALTHY DHT levels. FAPPING IS WHAT MAKES TEST CONVERT INTO DHT FAPPERS

 

[Trisomy 21]

Ok just dont take it and be bald faggot idc have fun being a sexually frustrated baldcel

r u retarded nigger, so if my dick stops working, then what will u sya Nigga?

i wanna fucking kill u and lynch ur balls dumb nigger faggot i will kill u

 

[iblamemandible7]

r u retarded nigger, so if my dick stops working, then what will u sya Nigga?

i wanna fucking kill u and lynch ur balls dumb nigger faggot i will kill u

Lmaooo bros mad over fake nocebo side effects get a grip retard youre never gonna be able to hardmax if fin scares you this much jfl

 

[Timmy.]

I'm tired of trannies here trying to convince people that DHT is useless. This thread will cover the importance of DHT and why you should not block it by the use of 5ARIs.

My friend @20/04/2008 made a really good thread on it, so I will just TLDR it:

DHT is not only the stronger version of Testosterone (2-4 times higher affinity to the androgen receptor), but also binds differently to the AR because of its unique structure.

This means each androgen exerts unique effects, including DHT. So only using Testosterone is not the right solution.

---

Now to the other uses of the enzyme 5α-reductase: It doesn't only affect the conversion of T to DHT, here are some other reactions:

Cholestenone → 5α-Cholestanone
Progesterone → 5α-Dihydroprogesterone
3α-Dihydroprogesterone → Allopregnanolone
3β-Dihydroprogesterone → Isopregnanolone
Deoxycorticosterone → 5α-Dihydrodeoxycorticosterone
Corticosterone → 5α-Dihydrocorticosterone
Aldosterone → 5α-Dihydroaldosterone
Androstenedione → 5α-Androstanedione
Nandrolone → 5α-Dihydronandrolone

The functions of some of the 5α-reduced steroids are unknown, that's why it isn't a good idea to "just use fin bro because you'll be fine."

Allopregnanolone is an important neurosteroid made from 3α-dihydroprogesterone. Without 5α-reductase on the other hand, this important neurosteroid can't be synthesized.

Progesterone → DHP via 5α-reductase
DHP → Allopregnanolone via 3α-HSD
Allopregnanolone → Positive allosteric modulator of GABA-A receptors

TLDR: No allopregnanolone -> More Anxiety, brain fog, stress, etc.

View attachment 3391024

Not forgetting the sexual dysfunction aspect of inhibiting 5AR.

The cells of the corpus cavernosum have androgen receptors, when you agonize these receptors they produce endothelial nitric oxide. NO triggers the relaxation of smooth muscle cells, leading to vasodilation and increased blood flow, which is needed for achieving and maintaining an erection. This can't be replaced with cialis, you need the local AR activation.

The only solution to solve this, is to have high levels of testosterone especially locally in the area of the penis or to use topical DHT/testosterone gels, just to maintain the normal function of the penis.

There is also the mental aspect of the erectile dysfunction. The main role of the sex drive of males consists of the dopamine/oxytocin signalling, which is inhibited by the use of 5ARIs. So in short, 5ARIs result in a reduction of libido.

View attachment 3391036

DHT is important for bone mass, which in the cited study even resulted in an increase of bone mass. DHT is also important for muscle strength.

"We have previously reported that DHT treatment increases bone mass in orchidectomized mice and that this effect is independent of estrogen receptors."

5AR is important for the metabolism of cortisol in the liver according to this study.

Persistent sexual side effects by the way... "Subjects reported new-onset persistent sexual dysfunction associated with the use of finasteride: 94% developed low libido, 92% developed erectile dysfunction, 92% developed decreased arousal, and 69% developed problems with orgasm."

Also ever wondered why most of the Finasteride users like Orc and Clavicular here never experience that many side effects from the 5ARI use? It's because they blast exogenous hormones to the point where the accumulated testosterone compensates for most of the side effects of the lacking DHT.

---

See? DHT is a vital male hormone, blocking it completely is harmful and has many other harms like discussed in this thread.

View attachment 3391173

Tag list: @halloweed @JohnDoe @Always Stay You @ConfusedBolivian

Just eat a healthy diet jfl

 

[DR. NICKGA]

He at least sources studies. Im too lazy to pull out my folder of studies. Niggas will just insult it rather then actually watching it, and continue to take biased studies they find from PFS funded networks and cry about how good DHT is.

I GONNA MAKE A NEW THREAD , THE DHT DICKSUCKERS GONNA BE ATTACKED SEVERLY WITH ME NEW THREAD

SOON SOON

Needed to make a quick thread, this guy @appeal tried to make people in puberty block DHT completely, argumenting that it's useless for development

View attachment 3391345

YOU ATTACK ONE OF US MEMBERS , WE WILL ATTACK ALL OF YOU MEMBERS

 

[Jonas2k7]

I GONNA MAKE A NEW THREAD , THE DHT DICKSUCKERS GONNA BE ATTACKED SEVERLY WITH ME NEW THREAD

SOON SOON


YOU ATTACK ONE OF US MEMBERS , WE WILL ATTACK ALL OF YOU MEMBERS

@piec holy DHT war

 

[|Daddy_Zygos|]

I'm tired of trannies here trying to convince people that DHT is useless. This thread will cover the importance of DHT and why you should not block it by the use of 5ARIs.

My friend @20/04/2008 made a really good thread on it, so I will just TLDR it:

DHT is not only the stronger version of Testosterone (2-4 times higher affinity to the androgen receptor), but also binds differently to the AR because of its unique structure.

This means each androgen exerts unique effects, including DHT. So only using Testosterone is not the right solution.

---

Now to the other uses of the enzyme 5α-reductase: It doesn't only affect the conversion of T to DHT, here are some other reactions:

Cholestenone → 5α-Cholestanone
Progesterone → 5α-Dihydroprogesterone
3α-Dihydroprogesterone → Allopregnanolone
3β-Dihydroprogesterone → Isopregnanolone
Deoxycorticosterone → 5α-Dihydrodeoxycorticosterone
Corticosterone → 5α-Dihydrocorticosterone
Aldosterone → 5α-Dihydroaldosterone
Androstenedione → 5α-Androstanedione
Nandrolone → 5α-Dihydronandrolone

The functions of some of the 5α-reduced steroids are unknown, that's why it isn't a good idea to "just use fin bro because you'll be fine."

Allopregnanolone is an important neurosteroid made from 3α-dihydroprogesterone. Without 5α-reductase on the other hand, this important neurosteroid can't be synthesized.

Progesterone → DHP via 5α-reductase
DHP → Allopregnanolone via 3α-HSD
Allopregnanolone → Positive allosteric modulator of GABA-A receptors

TLDR: No allopregnanolone -> More Anxiety, brain fog, stress, etc.

View attachment 3391024

Not forgetting the sexual dysfunction aspect of inhibiting 5AR.

The cells of the corpus cavernosum have androgen receptors, when you agonize these receptors they produce endothelial nitric oxide. NO triggers the relaxation of smooth muscle cells, leading to vasodilation and increased blood flow, which is needed for achieving and maintaining an erection. This can't be replaced with cialis, you need the local AR activation.

The only solution to solve this, is to have high levels of testosterone especially locally in the area of the penis or to use topical DHT/testosterone gels, just to maintain the normal function of the penis.

There is also the mental aspect of the erectile dysfunction. The main role of the sex drive of males consists of the dopamine/oxytocin signalling, which is inhibited by the use of 5ARIs. So in short, 5ARIs result in a reduction of libido.

View attachment 3391036

DHT is important for bone mass, which in the cited study even resulted in an increase of bone mass. DHT is also important for muscle strength.

"We have previously reported that DHT treatment increases bone mass in orchidectomized mice and that this effect is independent of estrogen receptors."

5AR is important for the metabolism of cortisol in the liver according to this study.

Persistent sexual side effects by the way... "Subjects reported new-onset persistent sexual dysfunction associated with the use of finasteride: 94% developed low libido, 92% developed erectile dysfunction, 92% developed decreased arousal, and 69% developed problems with orgasm."

Also ever wondered why most of the Finasteride users like Orc and Clavicular here never experience that many side effects from the 5ARI use? It's because they blast exogenous hormones to the point where the accumulated testosterone compensates for most of the side effects of the lacking DHT.

---

See? DHT is a vital male hormone, blocking it completely is harmful and has many other harms like discussed in this thread.

View attachment 3391173

Tag list: @halloweed @JohnDoe @Always Stay You @ConfusedBolivian

good thread

 

[PTOSIS]

Dnr, don't care wtf a 17 year old has to say + I take dutasteride because I would rather be dead than bald.

 

[|Daddy_Zygos|]

good thread

also androgen receptors are very important

 

[piec]

@piec holy DHT war

i am fully armed and ready to go

 

[Jonas2k7]

Dnr, don't care wtf a 17 year old has to say + I take dutasteride because I would rather be dead than bald.

Your comment didn't contribute anything to this discussion, also if you read the last thread you should know that MPB is caused my multiple factors, not even DHT primarely.

 

[DR. NICKGA]

Dnr, don't care wtf a 17 year old has to say + I take dutasteride because I would rather be dead than bald.

dont forget, they are dht lovers

the worst kind of humans, go insult them

 

[DR. NICKGA]

View attachment 3391503

Your comment didn't contribute anything to this discussion, also if you read the last thread you should know that MPB is caused my multiple factors, not even DHT primarely.

dht is the ONLY FACTOR OF BALDING

ME NEW THREAD WILL SEE ...

 

[Jonas2k7]

dht is the ONLY FACTOR OF BALDING

ME NEW THREAD WILL SEE ...

What an oversimplification.

You should start viewing the human body in a holistic perspective, not like DHT is the only cause of balding.

 

[The Dark Phoenix]

dnr full head of hair btw

 

[The Dark Phoenix]

Dnr, don't care wtf a 17 year old has to say + I take dutasteride because I would rather be dead than bald.

This

 

[Hexmask]

also androgen receptors are very important

lol what. DHT does not build muscle

 

[Jonas2k7]

 

[penaldinho]

"We have previously reported that DHT treatment increases bone mass in orchidectomized mice and that this effect is independent of estrogen receptors."

Is this with DHT gel

 

[penaldinho]

What about fin and dut effect on epigenetics because they are anti androgens?

 

[20/04/2008]

Bla bla bla nigga my grandpa started going bald at the exact same time and my dad is balding right now genetics are merciless we just have extremely dht sensitive follicles all the water retention in the world aint changing that

I take multivitamins 100g+ protein a day and get 6-8 hours sleep which isnt the best but I can still guarantee my micronutrients are better than my ancestors

Can guarantee E2 isnt low either cuz Ive had mild gyno since 13 jfl balding is a genetic disease and the cure is killing your dht

Bro gyno isn’t necessarily high E2
That just show how retarded you are when it coles to Roids

 

[Jonas2k7]

Is this with DHT gel

Most likely.

What about fin and dut effect on epigenetics because they are anti androgens?

They cause epigenetic changes, that's why many people experience PFS in the first place.

 

[20/04/2008]

chad eats pizza with milkshake while jeffrey investigates every hormone
@itzyaboyJJ @Kaari @MA_ascender @cobicado901 @wsada

Androgen receptor sensitivity
Even jeffreey could achieve Chad levels but he would need to compensate with enough androgens

 

[MyDreamIsToBe183CM]

I'm tired of trannies here trying to convince people that DHT is useless. This thread will cover the importance of DHT and why you should not block it by the use of 5ARIs.

My friend @20/04/2008 made a really good thread on it, so I will just TLDR it:

DHT is not only the stronger version of Testosterone (2-4 times higher affinity to the androgen receptor), but also binds differently to the AR because of its unique structure.

This means each androgen exerts unique effects, including DHT. So only using Testosterone is not the right solution.

---

Now to the other uses of the enzyme 5α-reductase: It doesn't only affect the conversion of T to DHT, here are some other reactions:

Cholestenone → 5α-Cholestanone
Progesterone → 5α-Dihydroprogesterone
3α-Dihydroprogesterone → Allopregnanolone
3β-Dihydroprogesterone → Isopregnanolone
Deoxycorticosterone → 5α-Dihydrodeoxycorticosterone
Corticosterone → 5α-Dihydrocorticosterone
Aldosterone → 5α-Dihydroaldosterone
Androstenedione → 5α-Androstanedione
Nandrolone → 5α-Dihydronandrolone

The functions of some of the 5α-reduced steroids are unknown, that's why it isn't a good idea to "just use fin bro because you'll be fine."

Allopregnanolone is an important neurosteroid made from 3α-dihydroprogesterone. Without 5α-reductase on the other hand, this important neurosteroid can't be synthesized.

Progesterone → DHP via 5α-reductase
DHP → Allopregnanolone via 3α-HSD
Allopregnanolone → Positive allosteric modulator of GABA-A receptors

TLDR: No allopregnanolone -> More Anxiety, brain fog, stress, etc.

View attachment 3391024

Not forgetting the sexual dysfunction aspect of inhibiting 5AR.

The cells of the corpus cavernosum have androgen receptors, when you agonize these receptors they produce endothelial nitric oxide. NO triggers the relaxation of smooth muscle cells, leading to vasodilation and increased blood flow, which is needed for achieving and maintaining an erection. This can't be replaced with cialis, you need the local AR activation.

The only solution to solve this, is to have high levels of testosterone especially locally in the area of the penis or to use topical DHT/testosterone gels, just to maintain the normal function of the penis.

There is also the mental aspect of the erectile dysfunction. The main role of the sex drive of males consists of the dopamine/oxytocin signalling, which is inhibited by the use of 5ARIs. So in short, 5ARIs result in a reduction of libido.

View attachment 3391036

DHT is important for bone mass, which in the cited study even resulted in an increase of bone mass. DHT is also important for muscle strength.

"We have previously reported that DHT treatment increases bone mass in orchidectomized mice and that this effect is independent of estrogen receptors."

5AR is important for the metabolism of cortisol in the liver according to this study.

Persistent sexual side effects by the way... "Subjects reported new-onset persistent sexual dysfunction associated with the use of finasteride: 94% developed low libido, 92% developed erectile dysfunction, 92% developed decreased arousal, and 69% developed problems with orgasm."

Also ever wondered why most of the Finasteride users like Orc and Clavicular here never experience that many side effects from the 5ARI use? It's because they blast exogenous hormones to the point where the accumulated testosterone compensates for most of the side effects of the lacking DHT.

---

See? DHT is a vital male hormone, blocking it completely is harmful and has many other harms like discussed in this thread.

View attachment 3391173

Tag list: @halloweed @JohnDoe @Always Stay You @ConfusedBolivian

so you think DHT gel is good?

planning to rub that shit on my dick when i hop on test instead of straight up injecting it because less will go systemic

also i already see some noticeable results from the far dissolvers i injected

 

[Jonas2k7]

so you think DHT gel is good?

Yes

planning to rub that shit on my dick when i hop on test instead of straight up injecting it because less will go systemic

Don't be scared of any side effects, only a tiny part will go systemical

also i already see some noticeable results from the far dissolvers i injected

Mirin bro

 

[appeal]

WITHOUT FIN AND DUT

YOU NEXT SOLUTION IS TO BE BALD YOU UTTERLY SUBHUMAN

nigga don’t argue with someone so stupid to think that dht doesn’t cause balding. literally the most retarded and autistic take known to man.

o you think DHT gel is good?

your asking literal 16 year olds what do do with hormones etc, that’s completely and utterly retarded. i recommend looking / asking elsewhere for information.

 

[PTOSIS]

View attachment 3391503

Your comment didn't contribute anything to this discussion, also if you read the last thread you should know that MPB is caused my multiple factors, not even DHT primarely.

Why do 5ar inhibitors work so well for most men then? We don't know all of the causes of MPB and you can still lose hair on finasteride, but there's strong evidence to support the claim that DHT sensitivity is the primary cause of MPB. Nothing else has been proven to work to combat MPB.

 

[Bitchwhipper2]

Ok but Im on fin for over a month and have had literally 0 sides whatsoever.

 

[20/04/2008]

nigga don’t argue with someone so stupid to think that dht doesn’t cause balding. literally the most retarded and autistic take known to man.

your asking literal 16 year olds what do do with hormones etc, that’s completely and utterly retarded. i recommend looking / asking elsewhere for information.

Nigga i provided studies you don’t

 

[appeal]

Nigga i provided studies you don’t

my thread is based on a study retard

 

[Jonas2k7]

nigga don’t argue with someone so stupid to think that dht doesn’t cause bloating. literally the most retarded and autistic take known to man.

your asking literal 16 year olds what do do with hormones etc, that’s completely and utterly retarded. i recommend looking / asking elsewhere for information.

Yeah sure, you know better right?

Hmm

Hmm
You are so knowledgeable over us right

This kid tries to act big even though he asked just a few days ago if roiding itself is worth it

@20/04/2008 @halloweed @mrmogger2882

 

[Jonas2k7]

Ok but Im on fin for over a month and have had literally 0 sides whatsoever.

Dosage?

 

[20/04/2008]

My new thread gonna absolute destroy you with the power of facts and logic

THAT DHT CAUSE BALDING

YOU TAINTED ME, PREPARE TO BE DESTROYED

Im waiting
Aslong their is studies
(Remember what i said i said high natural dht or slightly supraphysiological wouldn’t cause hairloss aslong your sleep is good you get all your micro-nutrients high igf-1 low prolactin low cortisol their is 0 chance you’ll go bald (i would say you have 0.01% chance of getting bald)

 

[20/04/2008]

Yeah sure, you know better right?
View attachment 3391800
Hmm
View attachment 3391801
Hmm
You are so knowledgeable over us right

View attachment 3391802

View attachment 3391803
This kid tries to act big even though he asked just a few days ago if roiding itself is worth it

@20/04/2008 @halloweed @mrmogger2882

Bro i have screenshots of his dum reply im just humble enough to not post them

 

[mrmogger2882]

your asking literal 16 year olds what do do with hormones etc, that’s completely and utterly retarded. i recommend looking / asking elsewhere for information.

Cope these "16 year olds" know their shit. I would pick them over my gynecologist

 

[20/04/2008]

Cope these "16 year olds" know their shit. I would pick them over my gynecologist

 

[Jonas2k7]

He comes in peace bro he is one of us

 

[Bitchwhipper2]

Dosage?

1mg