TeenAscender
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This is an area of heughtmaxxing completely untouched upon even in the glory days of Dyorotic2
We all know the mechanism of growth of the long bone is through malleable cartilage on the end of the bone that then turns into new bone called the epiphyseal plate
So common heightmaxxing knowledge tells us to take an aromatase inhibitor in order to block the lead factor of growth plate senescence: estrogen.
However there is no such malleable cartilage with a tangible start and end to its growth: the vertebrae.
The vertebrae is not classified as a “long bone” only the limb bones like the arms and the legs.
The vertebrae is described to not have any stage of “epiphyseal fusion” which is very confusing to me
An example of a patient with aromatase deficiency, he is very tall (6’6) but most of that height comes from his legs
Additionally something scary that potentiates an AI’s negative effect on vertebral height:
“We conclude that AI therapy during prepuberty or early puberty may predispose to vertebral deformities, which probably reflect impaired vertebral body growth rather than impaired bone quality and compression fractures.”
So what is estradiol’s effect on vertebral height? I note that acromegalic patients have very large sitting heights, but that is from IGF 1 excess not aromatase deficiency, even though theoretically the two have similar effects
@Henry_Gandy
@Henry_Gandy
@Henry_Gandy
@Henry_Gandy
GANDY PLEASE RESPOND MY BROTHER WE NEED U AND UR KNOWLEDGE
@AscendingHero
@6foot2_17y0
@SexuallyAbusive
@Alibaba69
@FreakkForLife
@Bitchwhipper2
Tag more ppl yall
We all know the mechanism of growth of the long bone is through malleable cartilage on the end of the bone that then turns into new bone called the epiphyseal plate
So common heightmaxxing knowledge tells us to take an aromatase inhibitor in order to block the lead factor of growth plate senescence: estrogen.
However there is no such malleable cartilage with a tangible start and end to its growth: the vertebrae.
The vertebrae is not classified as a “long bone” only the limb bones like the arms and the legs.
The vertebrae is described to not have any stage of “epiphyseal fusion” which is very confusing to me
An example of a patient with aromatase deficiency, he is very tall (6’6) but most of that height comes from his legs
Additionally something scary that potentiates an AI’s negative effect on vertebral height:
“We conclude that AI therapy during prepuberty or early puberty may predispose to vertebral deformities, which probably reflect impaired vertebral body growth rather than impaired bone quality and compression fractures.”
So what is estradiol’s effect on vertebral height? I note that acromegalic patients have very large sitting heights, but that is from IGF 1 excess not aromatase deficiency, even though theoretically the two have similar effects
@Henry_Gandy
@Henry_Gandy
@Henry_Gandy
@Henry_Gandy
GANDY PLEASE RESPOND MY BROTHER WE NEED U AND UR KNOWLEDGE
@AscendingHero
@6foot2_17y0
@SexuallyAbusive
@Alibaba69
@FreakkForLife
@Bitchwhipper2
Tag more ppl yall