Heightmaxing Megaguide: From Height Mogged to Height Mogger (The Mechanisms Driving Height And How To Enhance Them)

[Osie]

you know damn well no one is gonna take all this

, this is the stack of the GODS!

 

[Osie]

yeah, and these heightmaxxing threads don't offer nothing new, there's at least a million more of these on .org

Most heightmaxxing threads do offer something new tbh. Each thread has its own unique individual experience involved in each one, for example, PTH and Calcitrol is rarely discussed on this forum. An analysis of the mechanisms of growth hasn't really been discussed since 2019-2020

 

[Osie]

4) What pathways in specific do you mean? RANKL/RANK/OPG? are you referring to the other phases of bone remodelling? I'm interested to hear what your thoughts are.

While I wasn't referring to those pathways, I'm referring to another major factor of bone remodeling. And that's the primary signaling pathway that's activated by HGH, which is called the JAK-STAT (signal transducer and activator of transcription) pathway.

Activation of this pathway by HGH is very important for regulating things like IGF-1 and then by a product activating the IGF-1 receptor. And activation of this factor is important to avoiding bone health related diseases.

 

[Osie]

hey man, if i run 12.5mg aromasin, dht, and hgh 5iu daily with open plates, i should grow and it would keep my plates open? is this a decent stack? i can afford a lot so lmk ab anything

Hit PM's

 

[Osie]

So are you saying igf1 lr3 with ai is better than hgh, ai and ghrp stack or hgh, igf1 lr3 and ai stack? So much different information on Heightmaxxing stacks it's rotting my brain

Listen. If you want to maximize growth to the fullest extent, you want to promote as many bone growth factors as possible as explained in the thread.

So taking IGF-LR3, HGH, and AI compared to all the options you listed would be the best. However, that type of stack also has the highest potential side effect profile.

The other two stacks are also good, they just won't lead to as much growth as the IGF-LR3,HGH, AND AI.

 

[Vista]

While I wasn't referring to those pathways, I'm referring to another major factor of bone remodeling. And that's the primary signaling pathway that's activated by HGH, which is called the JAK-STAT (signal transducer and activator of transcription) pathway.

Activation of this pathway by HGH is very important for regulating things like IGF-1 and then by a product activating the IGF-1 receptor. And activation of this factor is important to avoiding bone health related diseases.
View attachment 2424276

Thanks for your comment, yes I already know this, hence why I included it in the guide:

Growth Hormone (HGH)
Again, a very popular growth factor on this website. Growth hormone (GH) is a peptide hormone secreted from the pituitary gland under the control of the hypothalamus. GH, along with its binding protein (GHBP), regulates growth directly through the GH receptor (GHR) and indirectly by stimulating liver and skeletal IGF-1 expression.

It has been reported that GH stimulates osteoblast proliferation and collagen production either directly and/or indirectly by increasing IGF-1 and IGF binding protein (IGFBP) production

These effects make GH useful, mostly due to its signalling properties and ability to upregulate the number of Growth Hormone receptors.
While the increase in IGF-1 is also useful, we already know that synthetic IGF-1 analogues like IGF-1 LR3 are more potent, meaning that both GH and IGF-1 LR3 could be used synergistically.

 

[Osie]

Thanks for your comment, yes I already know this, hence why I included it in the guide:

Growth Hormone (HGH)
Again, a very popular growth factor on this website. Growth hormone (GH) is a peptide hormone secreted from the pituitary gland under the control of the hypothalamus. GH, along with its binding protein (GHBP), regulates growth directly through the GH receptor (GHR) and indirectly by stimulating liver and skeletal IGF-1 expression.

It has been reported that GH stimulates osteoblast proliferation and collagen production either directly and/or indirectly by increasing IGF-1 and IGF binding protein (IGFBP) production

These effects make GH useful, mostly due to its signalling properties and ability to upregulate the number of Growth Hormone receptors.
While the increase in IGF-1 is also useful, we already know that synthetic IGF-1 analogues like IGF-1 LR3 are more potent, meaning that both GH and IGF-1 LR3 could be used synergistically.

Oh, I must've misread my apologies!

 

[Vista]

why are u reccomending teenagers to inject Igf1, an actual synthetic form of igf1 is pretty hard to find. Pathways to growth mean essentially as it literally sounds. Getting the finished product is skipping all the actual steps that lead to the final product you can see how that might fuck things up yeah? Most teenagers won’t have the money for actual height inducing IGF1 doses as if u read a study the amount needed to inject to actually see growth is pretty high and not to
Mention a legit IGF1 is fucking expensive. Trash that part of your thread we have evolved past 2021. Also

The final phase of growth in height - PubMed

Among 221 boys investigated, only 54% had the same (+/- 1 cm) body height at age 27 years as they had at age 19. Longitudinal observations of those boys indicate that the increment from 18 to 27 years is strongly dependent on skeletal maturation and even on such a distant event as age at peak...

pubmed.ncbi.nlm.nih.gov

There’s many different growth charts showing different things, I can pull two numbers out of my ass 15-17.

Different plates close at different times for different people. My brothers plates stayed open til 20 and he grew an inch from 18-20. Lots of things in this thread need to be changed

I never recommended for anyone to inject anything, the point of this guide is to hopefully help those wishing to heightmax on how to do it.

IGF-1 LR3 is also affordable, from peptide sciences https://www.peptidesciences.com/igf-1-lr3-1mg

a single 1mg vial is only $110 (exl shipping, etc), if you take 50mcg every other day, the vial will last you 40 days, even as a 15- 16 year old I was spending more than that on gear and GH secretagogues.

I also never said that your plates fuse at 16, I said most peoples plates do.

 

[Vista]

1-2cm naturally is what he meant. With encouraging growth using pharmacy aid that can be doubled and at least give an inch after 17, which most people have experienced.
What was the point of sending those sources for PTH? You can see for one of the sources 0.1MG is $485 before shipping and tax, are those sources even legit. PTH isn’t some cheap chemical and the other source is $90 for 1MG Jfl. What the fuck

I posted two different PTH sources, both of them are different. the more expensive one is has 84 N-terminal residues while the other is 34. The cheaper one is the only practical one to take and a $190 10mg vial at 25mcg/ every other day will last you 800 days. Obv you cant keep a vial reconstituted for 800 days, but you get the point, def affordable

 

[Dystopian]

mirin this absolute copypasta of a thread from dyorotic

 

[Deleted member 28722]

mirin this absolute copypasta of a thread from dyorotic

What’s ur latest gains bro? Heard ur 5’11 now u used to be 5’9

 

[Deleted member 28722]

mirin this absolute copypasta of a thread from dyorotic

Bro likes my thread and doesn’t respond. Kill yourself. I hope you make it to 5’11.999 and won’t ever hit 6ft

 

[BloodClot]

So much misinformation on this thread but i’m too lazy to correct it

 

[Vista]

So much misinformation on this thread but i’m too lazy to correct it

oy vey

 

[letsdoit]

I never recommended for anyone to inject anything, the point of this guide is to hopefully help those wishing to heightmax on how to do it.

IGF-1 LR3 is also affordable, from peptide sciences https://www.peptidesciences.com/igf-1-lr3-1mg

a single 1mg vial is only $110 (exl shipping, etc), if you take 50mcg every other day, the vial will last you 40 days, even as a 15- 16 year old I was spending more than that on gear and GH secretagogues.

I also never said that your plates fuse at 16, I said most peoples plates do.

At 20, What could IGF-1LR3 do to my body?

Just looking for frame and penis growth, any height would just be a plus.

Would it increase dimorphism?

For context I've grown 1.5 inches in the past year.

 

[Vista]

At 20, What could IGF-1LR3 do to my body?

Just looking for frame and penis growth, any height would just be a plus.

Would it increase dimorphism?

For context I've grown 1.5 inches in the past year.

1.5 inches is huge. If you want to increase dimorphism I would run a medium dose of IGF-1 LR3 Only + test/ DHT + AI. couple that with DHT gel on the dick + Penis enhancement.

 

[letsdoit]

1.5 inches is huge. If you want to increase dimorphism I would run a medium dose of IGF-1 LR3 Only + test/ DHT + AI. couple that with DHT gel on the dick + Penis enhancement.

Can I throw in hgh peptides, mk + cjc in there?
No hgh?


Why the AI? My growth plates are closed.

Would test boosting herbs suffice? Or I have to inject test?

 

[Napoleon1800]

Listen. If you want to maximize growth to the fullest extent, you want to promote as many bone growth factors as possible as explained in the thread.

So taking IGF-LR3, HGH, and AI compared to all the options you listed would be the best. However, that type of stack also has the highest potential side effect profile.

The other two stacks are also good, they just won't lead to as much growth as the IGF-LR3,HGH, AND AI.

Thanks mate. Can I talk to you in PMs if I have any questions?

 

[Arditi]

omw to become shaq with this guide

 

[Methhead]

Introduction
​

As many of you know ‘heightmaxing’ is a popular topic of discussion on this website, which is no surprise given the importance of height on attractiveness as well as a plethora of other benefits associated with being taller.

I’m sure we’re all familiar with the importance of height, but there is a concerning deficit of information regarding methods and knowledge on how to engender an increase of height.

This shouldn’t come as much of a surprise, since the mechanisms behind increasing your height are very complex and we still don’t know a lot about the subject. Many factors work together to determine height and there is a lot of disagreement in the scientific community on the subject.

The purpose of this guide is to take a relatively simplistic, scientific and easy to understand approach to height maxing, which will hopefully dispel some of the confusion on the subject. The ‘science’ bits will be written in red for those wanting elaboration.

View attachment 2423234

Overview of the mechanisms involved in growing taller​

1) Activation Phase: This is the beginning of bone growth. Cells called chondrocytes (cartilage) come together and proliferate (replicate). They create a blueprint made of cartilage, like a model for the bone to follow.

2) Osteoclast Recruitment and Resorption Phase (Hypertrophy Phase): Chondrocytes grow bigger (hypertrophy) and start turning the cartilage into hard stuff. Think of them as builders laying down the foundation for the bone.

3) Reversal Phase (Osteoblast Phase): A group of cells called osteoblasts moves in. They see the blueprint created by the cartilage and start adding bone on stop.

4) Formation Phase (Calcification Phase): The material the osteoblasts add becomes hard and strong because it calcifies, which is like turning it into concrete. Picture the bone getting denser and stronger.

5) Mineralization Phase (Ossification Phase): This is when the bone really takes shape. More bone cells come in, and they keep building and strengthening the bone, kind of like construction workers finishing a house.

6) Closure Phase (Epiphyseal Plate Closure): During adolescence, special plates at the ends of your long bones, called epiphyseal plates or growth plates, are active and contribute to your height. But once you reach a certain age, usually 15- 17, these plates close up.

View attachment 2423236

During osteoclastogenesis phases 1- 5 are constantly repeating in a loop until the closure phase. ​

The phase in osteoclastogenesis (bone growth) which contributes to enlarging your longitudinal height the most is the activation phase, which starts when you are an embryo and ends with the closure phase.

Now the question becomes: how do I enhance the mechanisms of the activation phase.

Spoiler: What is cell proliferation

During the activation phase cartilage grows through proliferation and differentiation.

Cell proliferation = Cell gets bigger + Cell divides, AKA More cells

View attachment 2423237

For the purpose of heightmaxing, proliferation is one of the important mechanisms that drives height growth.

We need to do everything possible to enhance cell proliferation and cell size in the growth plate.

What we need to do to heightmax

1) Increase cartilage proliferation (replication)
2) Increase cartilage hypertrophy (size)
3) Delay the closure phase

One of the ways we can increase proliferation and hypertrophy is with growth factors (a subset of cytokines).



An Introduction To Growth Factors & Hormones

​

There are some things we can do to increase the proliferation and hypertrophy of cartilage in the growth plate. This is done through bone growth factors, which work through a variety of different mechanisms and pathways.

Known bone growth factors (outside of foetal development) include:

Spoiler: Insulin-like growth factor-1 (IGF-1)

• Insulin-like growth factor-1 (IGF-1) is the most abundant growth factor deposited in the bone matrix and stimulates cell proliferation and function, and survival of osteoblasts.
• There are several reports demonstrating the synergistic effects of IGF-1 and PTH on bone remodelling and establishing the involvement of locally produced IGF-1 in the anabolic effects of PTH

Spoiler: Parathyroid hormone-related peptide (PTHrP and PTH)

• PTH can exert both catabolic and anabolic effects on bone. It is well established that daily injections of low doses of PTH increase bone mass in animals and humans

• Transforming growth factor beta (TGF-β)
  • TGF-β1
  • TGF-β3
    • Sort of a double edged sword, can cause an increase of bone mass but also osteoporosis, it is best to use this as little as possible.

• Fibroblast growth factors (FGF)
  • FGF 1 - 10
  • FGF18
    • Endogenous FGF-2 was found to be necessary for the bone anabolic effects of PTH and BMP-2 in mice

• Platetet- Derived Growth Factor (PDGF)

• Growth Differentiation Factor (GDF)
  • GDF3
  • GDF6
  • GDF10

• Fibroblast Growth Factor (FGF)

Hormones also play a role in height and morphology, a brief overview of which is provided below: ​

• Growth Hormone:
  • Stimulates IGF-1 growth factor AND IGF-1 receptor production. Growth hormone (GH) is a peptide hormone secreted from the pituitary gland under the control of the hypothalamus. It has been reported that both GH-deficient humans and mice have reduced longitudinal bone growth

• Testosterone & DHT:
  
  • Broadens Shoulders & promotes sexual dimorphism. Androgens can also modulate growth plate maturation and closure, and thus affect longitudinal bone growth. In addition, androgens regulate trabecular and cortical bone mass, and inhibit bone loss

• Oestrogen (Estrogen):
  • Causes the hips to widen and become rounded & speed up the rate of Growth Plate fusion (more on this in part 4). Oestrogen is also needed for healthy bones.

• Thyroid Hormones: Thyroid hormones, particularly thyroxine (T4) and triiodothyronine (T3) promote chondrocyte proliferation and the synthesis of collagen and other bone matrix proteins.

Something rarely mentioned in heightmaxing is Growth Factor receptors, growth factor receptors and growth factors as two puzzle pieces, they need each other to work.​

There aren’t many ways to directly increase Growth factor receptors, but some studies suggest that increasing one growth factor can signal others to proliferate.

What this also means is that depending on your specific distribution of growth factor receptors your growth will be somewhat random. People who have more receptors in their legs/ epiphyseal plate will experience more growth in that area than someone with more receptors in their shoulders or nose.

View attachment 2423238

An overview of some of the process' involved promoting bone growth



Applying Growth Factors



​

Primary Growth Factors - Factors that lead to the most growth


IGF-1 (IGF-1 LR3)

One of the most popular growth factors on looksmax for heightmaxing is IGF-1, which granted its price and efficacy should come as no surprise . Another advantage of IGF-1 is that it binds and activates its own receptor.​

​

Among the insulin-like growth factor 1 (IGF-1) analogues, the one with the best balance between potency and half life is IGF-1 LR3. It has a half life of between 20-30 hours and a stronger binding affinity than naturally occurring IGF-1​

Growth Hormone (HGH)

Again, a very popular growth factor on this website. Growth hormone (GH) is a peptide hormone secreted from the pituitary gland under the control of the hypothalamus. GH, along with its binding protein (GHBP), regulates growth directly through the GH receptor (GHR) and indirectly by stimulating liver and skeletal IGF-1 expression.​

​

It has been reported that GH stimulates osteoblast proliferation and collagen production either directly and/or indirectly by increasing IGF-1 and IGF binding protein (IGFBP) production​

​

These effects make GH useful, mostly due to its signalling properties and ability to upregulate the number of Growth Hormone receptors. ​

​

While the increase in IGF-1 is also useful, we already know that synthetic IGF-1 analogues like IGF-1 LR3 are more potent, meaning that both GH and IGF-1 LR3 could be used synergistically. ​

PTH

A compound not typically discussed on this forum is Parathyroid hormone.​

​

Parathyroid hormone (PTH), also called parathormone or parathyrin, is a peptide hormone secreted by the parathyroid glands.​

​

As mentioned earlier, PTH and IGF-1 are known to have synergistic effects, therefore it is advantageous to be running both IGF and PTH in the same stack.​

​

View attachment 2423240

Osteogenic differentiation of BMSCs treated with vehicle (-), IGF-1 (50 ng·mL-1), PTH (100 nmol·L-1), or both as assessed by alkaline phosphatase.

We can see that using both IGF and PTF gives us significantly more growth.​

Secondary Growth Factors - Factors That Support Growth

​

Calcitriol

Calcitriol is the biologically active form of vitamin D in the body. It is also known as 1,25-dihydroxyvitamin D3 or simply activated vitamin D. It is up to 1,000 times more potent than vitamin d2 and d3, making it ideal for supporting growth.​

Testosterone & DHT

By far one of the most influential androgens on height and the development of a sexually dimorphic skeleton. The androgen mechanism of action on height is not well understood, but it is known to stimulate longitudinal bone growth as well as radial bone growth, thereby increasing the cortical bone size.​

Lifestyle/ Diet

By far one of the most under looked growth factors is your lifestyle, it is often the easiest and one of the most impactful on your height.​

​

Eating enough food is vital to create the building blocks of hormones/ growth factors, ultimately increasing your height. It has been suggested that in addition to adequate intake of macro and micronutrients (vitamins, minerals, amino acids) intermittent fasting can also be beneficial for raising IGF-1 and increasing insulin sensitivity. Diets high in protein and dairy (not including cheese) were also found to elevate IGF-1.​

​

Adequate sleep, minimal stress and regular exercise are also seen to have positive effects on height.​

​

​

​

​

Delaying Growth Plate Fusion​

​

​

“Oestrogen causes maturation of the growth plate, accelerating skeletal maturation and the accumulation of minerals into the cartilage. Oestrogen also promotes the closure of the physis, stopping the axial growth of the bone. Decreases in total oestrogen or oestrogen receptor sensitivity results in longer bones and tall stature”​

What this means is that oestrogen is one of the driving forces behind signalling the closure phase. One of the ways Oestrogen can be regulated is via a class of drugs called antioestrogens.​

​

Antioestrogens I recommend:

• Exemestane (Aromasin) is relatively easy to get, has a long half life (24 hours), low side effects and decent bioavailability.

• Letrozole has also been shown to delay the fusing of the growth plates in mice. When used in combination with growth hormone, letrozole has been shown effective in one adolescent boy with a short stature. Mice administered with Letrozole were also found to have higher testosterone levels. I recomend cycling Lerozole with Aromasin.

​

Growing Taller​

​

​

​

If you are 15 and over I recommend getting an x- ray done of your epiphyseal plate to confirm whether or not it has fused. Complete fusion of the growth plate most commonly occurs between 15-17 in males (and no, you’re probably not an exception).

If your epiphyseal plate is open great, if not then you can still apply some of these methods to improve your frame. All bones fuse at different times, so even though your epiphyseal plate is sealed your other bones, such as the tibia and collarbone may still be open, allowing you to grow (more on this below).

It is also worth noting that Europeans & Asians have delayed growth compared to Africans and Hispanics, which means that Europeans and Asians undergo bone fusion later (usually 16) compared to as early as 14 in Africans and Hispanics.

If you are considering running a stack make sure that you can commit to running it for at least 6 months, ideally more than a year.

Stacks​

NBA Stack
For maximum height gains, the most expensive and riskiest

IGF-1 LR3​

30 - 50 mcg/every other day, subQ, preferably before bed​

​

HGH​

(Between 0.2 to 0.3 mcg) x bodyweight (kg) = weekly dosage/ 7 = daily dose, inject before bed SubQ​

​

AI​

Exemestane (Aromasin) 25mg/every other day or Letrozole every 4 days at 2.5mg. Depending on how long you plan to run a stack for, I would consider cycling this after more than 4-5 months.​

​

PTH​

Heightmaxing dosage is unknown, though it is clinically used between 25 mcg - 100 mcg everyday, I would recommend going no more than 25 mcg every other day. Inject subQ with IGF-1, before bed.​

While I recommend PTH for people wishing to significantly increase height, its use will require monitoring of blood Vitamin D, Phosphorus and Calcium levels during its use and possible supplementation.​

​

Calcitriol​

0.25- 0.50 mcg/ day, take caution if using with PTH. Watch out for hypercalcemia.​

​

Optional ​

Testosterone: any ester will be fine, though I recommend using Testosterone Propionate or Testosterone Enanthate. For height maxing a high dose of testosterone is not required, although those wishing to influence greater dimorphism may wish to increase the dosage. Do your own research if you plan to use Test.​

​

Run the stack for 4 weeks, take two weeks off, take 0.2mcg/kg HGH daily during off weeks. After 6 months: 4 weeks on, 6 weeks off, 0.2mcg HGH/day (this advice does not factor Test into account).​

​

​

​

Midstack
An affordable stack, best for most people, mild risk

IGF-1 LR3​

50 - 80 mcg/ every other day, subQ, preferably before bed​

​

Vitamin D3 (MK-7) ​

50K IU/ 3 times per week + 2.25mg K2/ with every D3 dose​

[1000 IU D3: 50mcg K2], I also recommend supplementing with magnesium, watch out for hypercalcemia.​

​

AI​

Exemestane (Aromasin) 25mg/every other day or Letrozole every 4 days at 2.5mg. Depending on how long you plan to run a stack for, I would consider cycling this after more than 4-5 months.​

​

​

​

Poorcel
Questionable results on a budget

MK677 ​

35 - 45 mg/day (bedtime) for 4 weeks, then every other day for two weeks at 20mg; repeat. I would also recommend mega dosing P-5-P throughout your cycle to decrease prolactin, especially if you are feeling symptoms of increased prolactin (gyno, decreased libido/mood, etc). For every 4 months on take 1 month off.​

​

AI (optional)​

Exemestane (Aromasin) 25mg/every other day or Letrozole every 4 days at 2.5mg. Depending on how long you plan to run a stack for, I would consider cycling this after more than 4-5 months.​

​

​

​

​

Stack Advise and Warnings​

​

​

For those whose epiphyseal plates are closed and you want to increase your frame, you do not need to run these stacks with the same dosage.​

​

If you are older than 24 do not run any of these stacks (it's already over), as you will probably suffer from Acromegaly.​

​

Users on looksmax also recommend running compounds like mod GRF, hexarelin, CJC, etc. These compounds are all GH secretagogues, meaning they tell your brain to produce more GH, and as we already know administration of IGF-1 analogues like LR3 are far more effective. For this reason I think secretagogues are subpar to IGF/GH and should be avoided (unless you have no choice).​

​

There is still lots of debate around whether exogenous administration of growth factors causes certain cancers, some studies have shown for there to be a correlation, while others have disproved it. Do this at your own risk.​

​

​

​

“Our results do not generally support a carcinogenic effect of r-hGH, but the unexplained trend in cancer mortality risk in relation to GH dose in patients with previous cancer, and the indication of possible effects on bone cancer, bladder cancer and Hodgkin lymphoma risks, need further investigation.” ​

Cancer risks in patients treated with growth hormone in childhood: the SAGhE European cohort study - PMC (nih.gov)​

​

​

Elevated levels of GH/IGF-1 have also been reported to have side effects, most notably swelling of the feet or hands. If this occurs, discontinue use for a few weeks and try again at a lower dosage.​

​

Prior to injecting or taking anything make sure to do your own research on the side effects. Decrease the dosages according to how you feel, everyone tolerates drugs differently.​

​

Long term administration of GH/ IGF-1 has also been found to decrease insulin sensitivity, if you have diabetes or a family history reconsider heightmaxing. Exercise caution after the 4-6 month mark of running a stack, consider taking more time off and monitor your blood sugar.​

​

Stretching exercises/ hanging are also a cope, there is no evidence of any long term height increases.​

​

​

​

​

Lifestyle Changes​

​

​

​

Some lifestyle changes that may contribute to increasing height:

• Eat a high protein diet (ideally >50% daily cal from protein)
• Ensure you get enough nutrients (use https://cronometer.com/), use supplements if you need to, make sure they are bioavailable.
• Eat in a slight surplus, 400 - 600 cal (unless you are obese, lose the weight first and then eat in surplus)
• Incorporate resistance training. For those serious about heightmaxing I would avoid exercises that compress your spine or pose a risk of damaging your joints (squats, deadlifts, OHP), instead opting for callisthenics and sprinting.
  • Bone morphogenetic protein 3 was found to be elevated in fractured tissue (this may include microfractures). BMP3 is an agonist to other BMP’s, which means that more BMP3 = less bone growth (this doesn't mean you shouldn't exercise).
• Diet high in dairy products and calcium
• Sleeping >8 hours/ night consistently
• Some herbal supplements like Tongat Ali, Ashwagnada, etc may be useful for lowering oestrogen/ increasing testosterone; promoting sexual dimorphism.
  • Depending on your situation you may also opt for using herbal supplements to control your oestrogen (delay fusion) and increase GH. The efficacy of these herbs are usually very questionable, and almost all (>90% of online vendors underdose their products).
• Decrease consumption of sugars (fructose, glucose, etc)

Conclusion


While this guide is far from perfect, hopefully I covered enough to help some of you on your heightmaxing journey. Best of luck and thanks for reading.
​

Spoiler: Sources

All this information was revealed to me in a dream

Inb4 greycel​

irrelavent for non 13year old cels

 

[Osie]

Thanks mate. Can I talk to you in PMs if I have any questions?

Aight

 

[callisto]

Can you elaborate on what u mean by pathways?

i am not "that" educated in that field yet.
what i can say and try to explain is that growth works on multiple pathways, of which igf1 is only one, even if it is the most prevalent.
growth hormone seems to activate most or all of the pathways required for growth, unlike IGF1 which only works in its own pathway. that is why gh is still preferred over something like rIGF1 which is used as the alternative if kids on gh therapy dont react well.

 

[the_nextDavidLaid]

Introduction
​

As many of you know ‘heightmaxing’ is a popular topic of discussion on this website, which is no surprise given the importance of height on attractiveness as well as a plethora of other benefits associated with being taller.

I’m sure we’re all familiar with the importance of height, but there is a concerning deficit of information regarding methods and knowledge on how to engender an increase of height.

This shouldn’t come as much of a surprise, since the mechanisms behind increasing your height are very complex and we still don’t know a lot about the subject. Many factors work together to determine height and there is a lot of disagreement in the scientific community on the subject.

The purpose of this guide is to take a relatively simplistic, scientific and easy to understand approach to height maxing, which will hopefully dispel some of the confusion on the subject. The ‘science’ bits will be written in red for those wanting elaboration.

View attachment 2423234

Overview of the mechanisms involved in growing taller​

1) Activation Phase: This is the beginning of bone growth. Cells called chondrocytes (cartilage) come together and proliferate (replicate). They create a blueprint made of cartilage, like a model for the bone to follow.

2) Osteoclast Recruitment and Resorption Phase (Hypertrophy Phase): Chondrocytes grow bigger (hypertrophy) and start turning the cartilage into hard stuff. Think of them as builders laying down the foundation for the bone.

3) Reversal Phase (Osteoblast Phase): A group of cells called osteoblasts moves in. They see the blueprint created by the cartilage and start adding bone on stop.

4) Formation Phase (Calcification Phase): The material the osteoblasts add becomes hard and strong because it calcifies, which is like turning it into concrete. Picture the bone getting denser and stronger.

5) Mineralization Phase (Ossification Phase): This is when the bone really takes shape. More bone cells come in, and they keep building and strengthening the bone, kind of like construction workers finishing a house.

6) Closure Phase (Epiphyseal Plate Closure): During adolescence, special plates at the ends of your long bones, called epiphyseal plates or growth plates, are active and contribute to your height. But once you reach a certain age, usually 15- 17, these plates close up.

View attachment 2423236

During osteoclastogenesis phases 1- 5 are constantly repeating in a loop until the closure phase. ​

The phase in osteoclastogenesis (bone growth) which contributes to enlarging your longitudinal height the most is the activation phase, which starts when you are an embryo and ends with the closure phase.

Now the question becomes: how do I enhance the mechanisms of the activation phase.

Spoiler: What is cell proliferation

During the activation phase cartilage grows through proliferation and differentiation.

Cell proliferation = Cell gets bigger + Cell divides, AKA More cells

View attachment 2423237

For the purpose of heightmaxing, proliferation is one of the important mechanisms that drives height growth.

We need to do everything possible to enhance cell proliferation and cell size in the growth plate.

What we need to do to heightmax

1) Increase cartilage proliferation (replication)
2) Increase cartilage hypertrophy (size)
3) Delay the closure phase

One of the ways we can increase proliferation and hypertrophy is with growth factors (a subset of cytokines).



An Introduction To Growth Factors & Hormones

​

There are some things we can do to increase the proliferation and hypertrophy of cartilage in the growth plate. This is done through bone growth factors, which work through a variety of different mechanisms and pathways.

Known bone growth factors (outside of foetal development) include:

Spoiler: Insulin-like growth factor-1 (IGF-1)

• Insulin-like growth factor-1 (IGF-1) is the most abundant growth factor deposited in the bone matrix and stimulates cell proliferation and function, and survival of osteoblasts.
• There are several reports demonstrating the synergistic effects of IGF-1 and PTH on bone remodelling and establishing the involvement of locally produced IGF-1 in the anabolic effects of PTH

Spoiler: Parathyroid hormone-related peptide (PTHrP and PTH)

• PTH can exert both catabolic and anabolic effects on bone. It is well established that daily injections of low doses of PTH increase bone mass in animals and humans

• Transforming growth factor beta (TGF-β)
  • TGF-β1
  • TGF-β3
    • Sort of a double edged sword, can cause an increase of bone mass but also osteoporosis, it is best to use this as little as possible.

• Fibroblast growth factors (FGF)
  • FGF 1 - 10
  • FGF18
    • Endogenous FGF-2 was found to be necessary for the bone anabolic effects of PTH and BMP-2 in mice

• Platetet- Derived Growth Factor (PDGF)

• Growth Differentiation Factor (GDF)
  • GDF3
  • GDF6
  • GDF10

• Fibroblast Growth Factor (FGF)

Hormones also play a role in height and morphology, a brief overview of which is provided below: ​

• Growth Hormone:
  • Stimulates IGF-1 growth factor AND IGF-1 receptor production. Growth hormone (GH) is a peptide hormone secreted from the pituitary gland under the control of the hypothalamus. It has been reported that both GH-deficient humans and mice have reduced longitudinal bone growth

• Testosterone & DHT:
  
  • Broadens Shoulders & promotes sexual dimorphism. Androgens can also modulate growth plate maturation and closure, and thus affect longitudinal bone growth. In addition, androgens regulate trabecular and cortical bone mass, and inhibit bone loss

• Oestrogen (Estrogen):
  • Causes the hips to widen and become rounded & speed up the rate of Growth Plate fusion (more on this in part 4). Oestrogen is also needed for healthy bones.

• Thyroid Hormones: Thyroid hormones, particularly thyroxine (T4) and triiodothyronine (T3) promote chondrocyte proliferation and the synthesis of collagen and other bone matrix proteins.

Something rarely mentioned in heightmaxing is Growth Factor receptors, growth factor receptors and growth factors as two puzzle pieces, they need each other to work.​

There aren’t many ways to directly increase Growth factor receptors, but some studies suggest that increasing one growth factor can signal others to proliferate.

What this also means is that depending on your specific distribution of growth factor receptors your growth will be somewhat random. People who have more receptors in their legs/ epiphyseal plate will experience more growth in that area than someone with more receptors in their shoulders or nose.

View attachment 2423238

An overview of some of the process' involved promoting bone growth



Applying Growth Factors



​

Primary Growth Factors - Factors that lead to the most growth


IGF-1 (IGF-1 LR3)

One of the most popular growth factors on looksmax for heightmaxing is IGF-1, which granted its price and efficacy should come as no surprise . Another advantage of IGF-1 is that it binds and activates its own receptor.​

​

Among the insulin-like growth factor 1 (IGF-1) analogues, the one with the best balance between potency and half life is IGF-1 LR3. It has a half life of between 20-30 hours and a stronger binding affinity than naturally occurring IGF-1​

Growth Hormone (HGH)

Again, a very popular growth factor on this website. Growth hormone (GH) is a peptide hormone secreted from the pituitary gland under the control of the hypothalamus. GH, along with its binding protein (GHBP), regulates growth directly through the GH receptor (GHR) and indirectly by stimulating liver and skeletal IGF-1 expression.​

​

It has been reported that GH stimulates osteoblast proliferation and collagen production either directly and/or indirectly by increasing IGF-1 and IGF binding protein (IGFBP) production​

​

These effects make GH useful, mostly due to its signalling properties and ability to upregulate the number of Growth Hormone receptors. ​

​

While the increase in IGF-1 is also useful, we already know that synthetic IGF-1 analogues like IGF-1 LR3 are more potent, meaning that both GH and IGF-1 LR3 could be used synergistically. ​

PTH

A compound not typically discussed on this forum is Parathyroid hormone.​

​

Parathyroid hormone (PTH), also called parathormone or parathyrin, is a peptide hormone secreted by the parathyroid glands.​

​

As mentioned earlier, PTH and IGF-1 are known to have synergistic effects, therefore it is advantageous to be running both IGF and PTH in the same stack.​

​

View attachment 2423240

Osteogenic differentiation of BMSCs treated with vehicle (-), IGF-1 (50 ng·mL-1), PTH (100 nmol·L-1), or both as assessed by alkaline phosphatase.

We can see that using both IGF and PTF gives us significantly more growth.​

Secondary Growth Factors - Factors That Support Growth

​

Calcitriol

Calcitriol is the biologically active form of vitamin D in the body. It is also known as 1,25-dihydroxyvitamin D3 or simply activated vitamin D. It is up to 1,000 times more potent than vitamin d2 and d3, making it ideal for supporting growth.​

Testosterone & DHT

By far one of the most influential androgens on height and the development of a sexually dimorphic skeleton. The androgen mechanism of action on height is not well understood, but it is known to stimulate longitudinal bone growth as well as radial bone growth, thereby increasing the cortical bone size.​

Lifestyle/ Diet

By far one of the most under looked growth factors is your lifestyle, it is often the easiest and one of the most impactful on your height.​

​

Eating enough food is vital to create the building blocks of hormones/ growth factors, ultimately increasing your height. It has been suggested that in addition to adequate intake of macro and micronutrients (vitamins, minerals, amino acids) intermittent fasting can also be beneficial for raising IGF-1 and increasing insulin sensitivity. Diets high in protein and dairy (not including cheese) were also found to elevate IGF-1.​

​

Adequate sleep, minimal stress and regular exercise are also seen to have positive effects on height.​

​

​

​

​

Delaying Growth Plate Fusion​

​

​

“Oestrogen causes maturation of the growth plate, accelerating skeletal maturation and the accumulation of minerals into the cartilage. Oestrogen also promotes the closure of the physis, stopping the axial growth of the bone. Decreases in total oestrogen or oestrogen receptor sensitivity results in longer bones and tall stature”​

What this means is that oestrogen is one of the driving forces behind signalling the closure phase. One of the ways Oestrogen can be regulated is via a class of drugs called antioestrogens.​

​

Antioestrogens I recommend:

• Exemestane (Aromasin) is relatively easy to get, has a long half life (24 hours), low side effects and decent bioavailability.

• Letrozole has also been shown to delay the fusing of the growth plates in mice. When used in combination with growth hormone, letrozole has been shown effective in one adolescent boy with a short stature. Mice administered with Letrozole were also found to have higher testosterone levels. I recomend cycling Lerozole with Aromasin.

​

Growing Taller​

​

​

​

If you are 15 and over I recommend getting an x- ray done of your epiphyseal plate to confirm whether or not it has fused. Complete fusion of the growth plate most commonly occurs between 15-17 in males (and no, you’re probably not an exception).

If your epiphyseal plate is open great, if not then you can still apply some of these methods to improve your frame. All bones fuse at different times, so even though your epiphyseal plate is sealed your other bones, such as the tibia and collarbone may still be open, allowing you to grow (more on this below).

It is also worth noting that Europeans & Asians have delayed growth compared to Africans and Hispanics, which means that Europeans and Asians undergo bone fusion later (usually 16) compared to as early as 14 in Africans and Hispanics.

If you are considering running a stack make sure that you can commit to running it for at least 6 months, ideally more than a year.

Stacks​

NBA Stack
For maximum height gains, the most expensive and riskiest

IGF-1 LR3​

30 - 50 mcg/every other day, subQ, preferably before bed​

​

HGH​

(Between 0.2 to 0.3 mcg) x bodyweight (kg) = weekly dosage/ 7 = daily dose, inject before bed SubQ​

​

AI​

Exemestane (Aromasin) 25mg/every other day or Letrozole every 4 days at 2.5mg. Depending on how long you plan to run a stack for, I would consider cycling this after more than 4-5 months.​

​

PTH​

Heightmaxing dosage is unknown, though it is clinically used between 25 mcg - 100 mcg everyday, I would recommend going no more than 25 mcg every other day. Inject subQ with IGF-1, before bed.​

While I recommend PTH for people wishing to significantly increase height, its use will require monitoring of blood Vitamin D, Phosphorus and Calcium levels during its use and possible supplementation.​

​

Calcitriol​

0.25- 0.50 mcg/ day, take caution if using with PTH. Watch out for hypercalcemia.​

​

Optional ​

Testosterone: any ester will be fine, though I recommend using Testosterone Propionate or Testosterone Enanthate. For height maxing a high dose of testosterone is not required, although those wishing to influence greater dimorphism may wish to increase the dosage. Do your own research if you plan to use Test.​

​

Run the stack for 4 weeks, take two weeks off, take 0.2mcg/kg HGH daily during off weeks. After 6 months: 4 weeks on, 6 weeks off, 0.2mcg HGH/day (this advice does not factor Test into account).​

​

​

​

Midstack
An affordable stack, best for most people, mild risk

IGF-1 LR3​

50 - 80 mcg/ every other day, subQ, preferably before bed​

​

Vitamin D3 (MK-7) ​

50K IU/ 3 times per week + 2.25mg K2/ with every D3 dose​

[1000 IU D3: 50mcg K2], I also recommend supplementing with magnesium, watch out for hypercalcemia.​

​

AI​

Exemestane (Aromasin) 25mg/every other day or Letrozole every 4 days at 2.5mg. Depending on how long you plan to run a stack for, I would consider cycling this after more than 4-5 months.​

​

​

​

Poorcel
Questionable results on a budget

MK677 ​

35 - 45 mg/day (bedtime) for 4 weeks, then every other day for two weeks at 20mg; repeat. I would also recommend mega dosing P-5-P throughout your cycle to decrease prolactin, especially if you are feeling symptoms of increased prolactin (gyno, decreased libido/mood, etc). For every 4 months on take 1 month off.​

​

AI (optional)​

Exemestane (Aromasin) 25mg/every other day or Letrozole every 4 days at 2.5mg. Depending on how long you plan to run a stack for, I would consider cycling this after more than 4-5 months.​

​

​

​

​

Stack Advise and Warnings​

​

​

For those whose epiphyseal plates are closed and you want to increase your frame, you do not need to run these stacks with the same dosage.​

​

If you are older than 24 do not run any of these stacks (it's already over), as you will probably suffer from Acromegaly.​

​

Users on looksmax also recommend running compounds like mod GRF, hexarelin, CJC, etc. These compounds are all GH secretagogues, meaning they tell your brain to produce more GH, and as we already know administration of IGF-1 analogues like LR3 are far more effective. For this reason I think secretagogues are subpar to IGF/GH and should be avoided (unless you have no choice).​

​

There is still lots of debate around whether exogenous administration of growth factors causes certain cancers, some studies have shown for there to be a correlation, while others have disproved it. Do this at your own risk.​

​

​

​

“Our results do not generally support a carcinogenic effect of r-hGH, but the unexplained trend in cancer mortality risk in relation to GH dose in patients with previous cancer, and the indication of possible effects on bone cancer, bladder cancer and Hodgkin lymphoma risks, need further investigation.” ​

Cancer risks in patients treated with growth hormone in childhood: the SAGhE European cohort study - PMC (nih.gov)​

​

​

Elevated levels of GH/IGF-1 have also been reported to have side effects, most notably swelling of the feet or hands. If this occurs, discontinue use for a few weeks and try again at a lower dosage.​

​

Prior to injecting or taking anything make sure to do your own research on the side effects. Decrease the dosages according to how you feel, everyone tolerates drugs differently.​

​

Long term administration of GH/ IGF-1 has also been found to decrease insulin sensitivity, if you have diabetes or a family history reconsider heightmaxing. Exercise caution after the 4-6 month mark of running a stack, consider taking more time off and monitor your blood sugar.​

​

Stretching exercises/ hanging are also a cope, there is no evidence of any long term height increases.​

​

​

​

​

Lifestyle Changes​

​

​

​

Some lifestyle changes that may contribute to increasing height:

• Eat a high protein diet (ideally >50% daily cal from protein)
• Ensure you get enough nutrients (use https://cronometer.com/), use supplements if you need to, make sure they are bioavailable.
• Eat in a slight surplus, 400 - 600 cal (unless you are obese, lose the weight first and then eat in surplus)
• Incorporate resistance training. For those serious about heightmaxing I would avoid exercises that compress your spine or pose a risk of damaging your joints (squats, deadlifts, OHP), instead opting for callisthenics and sprinting.
  • Bone morphogenetic protein 3 was found to be elevated in fractured tissue (this may include microfractures). BMP3 is an agonist to other BMP’s, which means that more BMP3 = less bone growth (this doesn't mean you shouldn't exercise).
• Diet high in dairy products and calcium
• Sleeping >8 hours/ night consistently
• Some herbal supplements like Tongat Ali, Ashwagnada, etc may be useful for lowering oestrogen/ increasing testosterone; promoting sexual dimorphism.
  • Depending on your situation you may also opt for using herbal supplements to control your oestrogen (delay fusion) and increase GH. The efficacy of these herbs are usually very questionable, and almost all (>90% of online vendors underdose their products).
• Decrease consumption of sugars (fructose, glucose, etc)

Conclusion


While this guide is far from perfect, hopefully I covered enough to help some of you on your heightmaxing journey. Best of luck and thanks for reading.
​

Spoiler: Sources

All this information was revealed to me in a dream

Inb4 greycel​

Greycels saving the forum yet again (fuck off TikTok faggots)

 

[Deleted member 40986]

Good thread i wish i knew this when i was 4 days old

 

[thereallegend]

@BrahminBoss may I ask you about the impact of sugar in kid and teenager growth? OP advises against eat but I'm pretty sure I've seen something proving the opposite in the Peat forum.

sugar spikes insulin. insulin lowers growth hormone

 

[Methhead]

Introduction
​

As many of you know ‘heightmaxing’ is a popular topic of discussion on this website, which is no surprise given the importance of height on attractiveness as well as a plethora of other benefits associated with being taller.

I’m sure we’re all familiar with the importance of height, but there is a concerning deficit of information regarding methods and knowledge on how to engender an increase of height.

This shouldn’t come as much of a surprise, since the mechanisms behind increasing your height are very complex and we still don’t know a lot about the subject. Many factors work together to determine height and there is a lot of disagreement in the scientific community on the subject.

The purpose of this guide is to take a relatively simplistic, scientific and easy to understand approach to height maxing, which will hopefully dispel some of the confusion on the subject. The ‘science’ bits will be written in red for those wanting elaboration.

View attachment 2423234

Overview of the mechanisms involved in growing taller​

1) Activation Phase: This is the beginning of bone growth. Cells called chondrocytes (cartilage) come together and proliferate (replicate). They create a blueprint made of cartilage, like a model for the bone to follow.

2) Osteoclast Recruitment and Resorption Phase (Hypertrophy Phase): Chondrocytes grow bigger (hypertrophy) and start turning the cartilage into hard stuff. Think of them as builders laying down the foundation for the bone.

3) Reversal Phase (Osteoblast Phase): A group of cells called osteoblasts moves in. They see the blueprint created by the cartilage and start adding bone on stop.

4) Formation Phase (Calcification Phase): The material the osteoblasts add becomes hard and strong because it calcifies, which is like turning it into concrete. Picture the bone getting denser and stronger.

5) Mineralization Phase (Ossification Phase): This is when the bone really takes shape. More bone cells come in, and they keep building and strengthening the bone, kind of like construction workers finishing a house.

6) Closure Phase (Epiphyseal Plate Closure): During adolescence, special plates at the ends of your long bones, called epiphyseal plates or growth plates, are active and contribute to your height. But once you reach a certain age, usually 15- 17, these plates close up.

View attachment 2423236

During osteoclastogenesis phases 1- 5 are constantly repeating in a loop until the closure phase. ​

The phase in osteoclastogenesis (bone growth) which contributes to enlarging your longitudinal height the most is the activation phase, which starts when you are an embryo and ends with the closure phase.

Now the question becomes: how do I enhance the mechanisms of the activation phase.

Spoiler: What is cell proliferation

During the activation phase cartilage grows through proliferation and differentiation.

Cell proliferation = Cell gets bigger + Cell divides, AKA More cells

View attachment 2423237

For the purpose of heightmaxing, proliferation is one of the important mechanisms that drives height growth.

We need to do everything possible to enhance cell proliferation and cell size in the growth plate.

What we need to do to heightmax

1) Increase cartilage proliferation (replication)
2) Increase cartilage hypertrophy (size)
3) Delay the closure phase

One of the ways we can increase proliferation and hypertrophy is with growth factors (a subset of cytokines).



An Introduction To Growth Factors & Hormones

​

There are some things we can do to increase the proliferation and hypertrophy of cartilage in the growth plate. This is done through bone growth factors, which work through a variety of different mechanisms and pathways.

Known bone growth factors (outside of foetal development) include:

Spoiler: Insulin-like growth factor-1 (IGF-1)

• Insulin-like growth factor-1 (IGF-1) is the most abundant growth factor deposited in the bone matrix and stimulates cell proliferation and function, and survival of osteoblasts.
• There are several reports demonstrating the synergistic effects of IGF-1 and PTH on bone remodelling and establishing the involvement of locally produced IGF-1 in the anabolic effects of PTH

Spoiler: Parathyroid hormone-related peptide (PTHrP and PTH)

• PTH can exert both catabolic and anabolic effects on bone. It is well established that daily injections of low doses of PTH increase bone mass in animals and humans

• Transforming growth factor beta (TGF-β)
  • TGF-β1
  • TGF-β3
    • Sort of a double edged sword, can cause an increase of bone mass but also osteoporosis, it is best to use this as little as possible.

• Fibroblast growth factors (FGF)
  • FGF 1 - 10
  • FGF18
    • Endogenous FGF-2 was found to be necessary for the bone anabolic effects of PTH and BMP-2 in mice

• Platetet- Derived Growth Factor (PDGF)

• Growth Differentiation Factor (GDF)
  • GDF3
  • GDF6
  • GDF10

• Fibroblast Growth Factor (FGF)

Hormones also play a role in height and morphology, a brief overview of which is provided below: ​

• Growth Hormone:
  • Stimulates IGF-1 growth factor AND IGF-1 receptor production. Growth hormone (GH) is a peptide hormone secreted from the pituitary gland under the control of the hypothalamus. It has been reported that both GH-deficient humans and mice have reduced longitudinal bone growth

• Testosterone & DHT:
  
  • Broadens Shoulders & promotes sexual dimorphism. Androgens can also modulate growth plate maturation and closure, and thus affect longitudinal bone growth. In addition, androgens regulate trabecular and cortical bone mass, and inhibit bone loss

• Oestrogen (Estrogen):
  • Causes the hips to widen and become rounded & speed up the rate of Growth Plate fusion (more on this in part 4). Oestrogen is also needed for healthy bones.

• Thyroid Hormones: Thyroid hormones, particularly thyroxine (T4) and triiodothyronine (T3) promote chondrocyte proliferation and the synthesis of collagen and other bone matrix proteins.

Something rarely mentioned in heightmaxing is Growth Factor receptors, growth factor receptors and growth factors as two puzzle pieces, they need each other to work.​

There aren’t many ways to directly increase Growth factor receptors, but some studies suggest that increasing one growth factor can signal others to proliferate.

What this also means is that depending on your specific distribution of growth factor receptors your growth will be somewhat random. People who have more receptors in their legs/ epiphyseal plate will experience more growth in that area than someone with more receptors in their shoulders or nose.

View attachment 2423238

An overview of some of the process' involved promoting bone growth



Applying Growth Factors



​

Primary Growth Factors - Factors that lead to the most growth


IGF-1 (IGF-1 LR3)

One of the most popular growth factors on looksmax for heightmaxing is IGF-1, which granted its price and efficacy should come as no surprise . Another advantage of IGF-1 is that it binds and activates its own receptor.​

​

Among the insulin-like growth factor 1 (IGF-1) analogues, the one with the best balance between potency and half life is IGF-1 LR3. It has a half life of between 20-30 hours and a stronger binding affinity than naturally occurring IGF-1​

Growth Hormone (HGH)

Again, a very popular growth factor on this website. Growth hormone (GH) is a peptide hormone secreted from the pituitary gland under the control of the hypothalamus. GH, along with its binding protein (GHBP), regulates growth directly through the GH receptor (GHR) and indirectly by stimulating liver and skeletal IGF-1 expression.​

​

It has been reported that GH stimulates osteoblast proliferation and collagen production either directly and/or indirectly by increasing IGF-1 and IGF binding protein (IGFBP) production​

​

These effects make GH useful, mostly due to its signalling properties and ability to upregulate the number of Growth Hormone receptors. ​

​

While the increase in IGF-1 is also useful, we already know that synthetic IGF-1 analogues like IGF-1 LR3 are more potent, meaning that both GH and IGF-1 LR3 could be used synergistically. ​

PTH

A compound not typically discussed on this forum is Parathyroid hormone.​

​

Parathyroid hormone (PTH), also called parathormone or parathyrin, is a peptide hormone secreted by the parathyroid glands.​

​

As mentioned earlier, PTH and IGF-1 are known to have synergistic effects, therefore it is advantageous to be running both IGF and PTH in the same stack.​

​

View attachment 2423240

Osteogenic differentiation of BMSCs treated with vehicle (-), IGF-1 (50 ng·mL-1), PTH (100 nmol·L-1), or both as assessed by alkaline phosphatase.

We can see that using both IGF and PTF gives us significantly more growth.​

Secondary Growth Factors - Factors That Support Growth

​

Calcitriol

Calcitriol is the biologically active form of vitamin D in the body. It is also known as 1,25-dihydroxyvitamin D3 or simply activated vitamin D. It is up to 1,000 times more potent than vitamin d2 and d3, making it ideal for supporting growth.​

Testosterone & DHT

By far one of the most influential androgens on height and the development of a sexually dimorphic skeleton. The androgen mechanism of action on height is not well understood, but it is known to stimulate longitudinal bone growth as well as radial bone growth, thereby increasing the cortical bone size.​

Lifestyle/ Diet

By far one of the most under looked growth factors is your lifestyle, it is often the easiest and one of the most impactful on your height.​

​

Eating enough food is vital to create the building blocks of hormones/ growth factors, ultimately increasing your height. It has been suggested that in addition to adequate intake of macro and micronutrients (vitamins, minerals, amino acids) intermittent fasting can also be beneficial for raising IGF-1 and increasing insulin sensitivity. Diets high in protein and dairy (not including cheese) were also found to elevate IGF-1.​

​

Adequate sleep, minimal stress and regular exercise are also seen to have positive effects on height.​

​

​

​

​

Delaying Growth Plate Fusion​

​

​

“Oestrogen causes maturation of the growth plate, accelerating skeletal maturation and the accumulation of minerals into the cartilage. Oestrogen also promotes the closure of the physis, stopping the axial growth of the bone. Decreases in total oestrogen or oestrogen receptor sensitivity results in longer bones and tall stature”​

What this means is that oestrogen is one of the driving forces behind signalling the closure phase. One of the ways Oestrogen can be regulated is via a class of drugs called antioestrogens.​

​

Antioestrogens I recommend:

• Exemestane (Aromasin) is relatively easy to get, has a long half life (24 hours), low side effects and decent bioavailability.

• Letrozole has also been shown to delay the fusing of the growth plates in mice. When used in combination with growth hormone, letrozole has been shown effective in one adolescent boy with a short stature. Mice administered with Letrozole were also found to have higher testosterone levels. I recomend cycling Lerozole with Aromasin.

​

Growing Taller​

​

​

​

If you are 15 and over I recommend getting an x- ray done of your epiphyseal plate to confirm whether or not it has fused. Complete fusion of the growth plate most commonly occurs between 15-17 in males (and no, you’re probably not an exception).

If your epiphyseal plate is open great, if not then you can still apply some of these methods to improve your frame. All bones fuse at different times, so even though your epiphyseal plate is sealed your other bones, such as the tibia and collarbone may still be open, allowing you to grow (more on this below).

It is also worth noting that Europeans & Asians have delayed growth compared to Africans and Hispanics, which means that Europeans and Asians undergo bone fusion later (usually 16) compared to as early as 14 in Africans and Hispanics.

If you are considering running a stack make sure that you can commit to running it for at least 6 months, ideally more than a year.

Stacks​

NBA Stack
For maximum height gains, the most expensive and riskiest

IGF-1 LR3​

30 - 50 mcg/every other day, subQ, preferably before bed​

​

HGH​

(Between 0.2 to 0.3 mcg) x bodyweight (kg) = weekly dosage/ 7 = daily dose, inject before bed SubQ​

​

AI​

Exemestane (Aromasin) 25mg/every other day or Letrozole every 4 days at 2.5mg. Depending on how long you plan to run a stack for, I would consider cycling this after more than 4-5 months.​

​

PTH​

Heightmaxing dosage is unknown, though it is clinically used between 25 mcg - 100 mcg everyday, I would recommend going no more than 25 mcg every other day. Inject subQ with IGF-1, before bed.​

While I recommend PTH for people wishing to significantly increase height, its use will require monitoring of blood Vitamin D, Phosphorus and Calcium levels during its use and possible supplementation.​

​

Calcitriol​

0.25- 0.50 mcg/ day, take caution if using with PTH. Watch out for hypercalcemia.​

​

Optional ​

Testosterone: any ester will be fine, though I recommend using Testosterone Propionate or Testosterone Enanthate. For height maxing a high dose of testosterone is not required, although those wishing to influence greater dimorphism may wish to increase the dosage. Do your own research if you plan to use Test.​

​

Run the stack for 4 weeks, take two weeks off, take 0.2mcg/kg HGH daily during off weeks. After 6 months: 4 weeks on, 6 weeks off, 0.2mcg HGH/day (this advice does not factor Test into account).​

​

​

​

Midstack
An affordable stack, best for most people, mild risk

IGF-1 LR3​

50 - 80 mcg/ every other day, subQ, preferably before bed​

​

Vitamin D3 (MK-7) ​

50K IU/ 3 times per week + 2.25mg K2/ with every D3 dose​

[1000 IU D3: 50mcg K2], I also recommend supplementing with magnesium, watch out for hypercalcemia.​

​

AI​

Exemestane (Aromasin) 25mg/every other day or Letrozole every 4 days at 2.5mg. Depending on how long you plan to run a stack for, I would consider cycling this after more than 4-5 months.​

​

​

​

Poorcel
Questionable results on a budget

MK677 ​

35 - 45 mg/day (bedtime) for 4 weeks, then every other day for two weeks at 20mg; repeat. I would also recommend mega dosing P-5-P throughout your cycle to decrease prolactin, especially if you are feeling symptoms of increased prolactin (gyno, decreased libido/mood, etc). For every 4 months on take 1 month off.​

​

AI (optional)​

Exemestane (Aromasin) 25mg/every other day or Letrozole every 4 days at 2.5mg. Depending on how long you plan to run a stack for, I would consider cycling this after more than 4-5 months.​

​

​

​

​

Stack Advise and Warnings​

​

​

For those whose epiphyseal plates are closed and you want to increase your frame, you do not need to run these stacks with the same dosage.​

​

If you are older than 24 do not run any of these stacks (it's already over), as you will probably suffer from Acromegaly.​

​

Users on looksmax also recommend running compounds like mod GRF, hexarelin, CJC, etc. These compounds are all GH secretagogues, meaning they tell your brain to produce more GH, and as we already know administration of IGF-1 analogues like LR3 are far more effective. For this reason I think secretagogues are subpar to IGF/GH and should be avoided (unless you have no choice).​

​

There is still lots of debate around whether exogenous administration of growth factors causes certain cancers, some studies have shown for there to be a correlation, while others have disproved it. Do this at your own risk.​

​

​

​

“Our results do not generally support a carcinogenic effect of r-hGH, but the unexplained trend in cancer mortality risk in relation to GH dose in patients with previous cancer, and the indication of possible effects on bone cancer, bladder cancer and Hodgkin lymphoma risks, need further investigation.” ​

Cancer risks in patients treated with growth hormone in childhood: the SAGhE European cohort study - PMC (nih.gov)​

​

​

Elevated levels of GH/IGF-1 have also been reported to have side effects, most notably swelling of the feet or hands. If this occurs, discontinue use for a few weeks and try again at a lower dosage.​

​

Prior to injecting or taking anything make sure to do your own research on the side effects. Decrease the dosages according to how you feel, everyone tolerates drugs differently.​

​

Long term administration of GH/ IGF-1 has also been found to decrease insulin sensitivity, if you have diabetes or a family history reconsider heightmaxing. Exercise caution after the 4-6 month mark of running a stack, consider taking more time off and monitor your blood sugar.​

​

Stretching exercises/ hanging are also a cope, there is no evidence of any long term height increases.​

​

​

​

​

Lifestyle Changes​

​

​

​

Some lifestyle changes that may contribute to increasing height:

• Eat a high protein diet (ideally >50% daily cal from protein)
• Ensure you get enough nutrients (use https://cronometer.com/), use supplements if you need to, make sure they are bioavailable.
• Eat in a slight surplus, 400 - 600 cal (unless you are obese, lose the weight first and then eat in surplus)
• Incorporate resistance training. For those serious about heightmaxing I would avoid exercises that compress your spine or pose a risk of damaging your joints (squats, deadlifts, OHP), instead opting for callisthenics and sprinting.
  • Bone morphogenetic protein 3 was found to be elevated in fractured tissue (this may include microfractures). BMP3 is an agonist to other BMP’s, which means that more BMP3 = less bone growth (this doesn't mean you shouldn't exercise).
• Diet high in dairy products and calcium
• Sleeping >8 hours/ night consistently
• Some herbal supplements like Tongat Ali, Ashwagnada, etc may be useful for lowering oestrogen/ increasing testosterone; promoting sexual dimorphism.
  • Depending on your situation you may also opt for using herbal supplements to control your oestrogen (delay fusion) and increase GH. The efficacy of these herbs are usually very questionable, and almost all (>90% of online vendors underdose their products).
• Decrease consumption of sugars (fructose, glucose, etc)

Conclusion


While this guide is far from perfect, hopefully I covered enough to help some of you on your heightmaxing journey. Best of luck and thanks for reading.
​

Spoiler: Sources

All this information was revealed to me in a dream

Inb4 greycel​

can you make a guide about how to tell what tanner stage you are in?

 

[Osie]

can you make a guide about how to tell what tanner stage you are in?

The tanner stage doesn't matter. You can be tanner stage 5 at 13 and as long you have open growth plates, you can grow until 17+. While it can help to give rough estimate of your bone age, it's not very accurate.

But if you want to know tanner stage, press this link

 

[Deleted member 29747]

hey bro great thread was wondering how to take testesterone during height maxxing as it increease estrogen and if u dont time your aromasin right it could close ur plates from the estrogen spike correct me if i am wrong. and then you would have to do pct after and hcg also heavly increases estrogen

 

[Osie]

isnt it impossible to find a legit source

You can search it up online and find it in three seconds

 

[Vista]

isnt it impossible to find a legit source

if u are too stupid to find a source you dont deserve to be taller

 

[Vista]

hey bro great thread was wondering how to take testesterone during height maxxing as it increease estrogen and if u dont time your aromasin right it could close ur plates from the estrogen spike correct me if i am wrong. and then you would have to do pct after and hcg also heavly increases estrogen

Thats why I didn't include test in the guide, it complicates things a lot, as long as you have a good AI and good pct protocol in place you should be fine

 

[Deleted member 29747]

if u are too stupid to find a source you dont deserve to be taller

is iron lion labs and peptide science good for igf1 lr3

 

[vulcan_]

Soooo as a 15 year old what you should be doing is

- 8 hours sleep consistent
-high intensity training
- diet with correct nutrition
And what else

 

[Napoleon1800]

@Vista So do you recommend taking igf-1 lr3 then alongside hgh and ai?

 

[Vista]

@Vista So do you recommend taking igf-1 lr3 then alongside hgh and ai?

Yeah, as I said in the guide LR3 is like the “building block” of height gains while HGH increases receptors, kind of like the “manager”, using both concurrently is ideal. Use an AI to delay growth plate fusion

 

[Napoleon1800]

Jfl. This is why we don’t let retards on the internet. Listen Buddy. IGF1 is the finished product but you lose gains when you don’t let it convert from HGH. Maybe read some studies on the shit before coming on the topic with a literal Wikipedia understanding. The doses to encourage height growth with straight IGF1 is something you won’t be running. 2. IGF1 without understanding how to cycle it can desensitize ur IGF1 receptors, and cause growth in places you don’t want, experts still don’t know IF IGF1-LR3 causes local growth, or it causes growth in the whole body yet…. It’s barely studied at all and there’s evidence to support both.

@Vista What are your thoughts on this?

 

[Vista]

@Vista What are your thoughts on this?

I didn't want to respond to this because it was so retarded but here I go:

You don't "lose" any gains from injecting IGF 1 as opposed to just injecting straight HGH, HGH produces IGF-1, by injecting IGF- 1 LR3 we are "bypassing the middleman" (HGH) so to speak and increasing serum blood concentration of IGF-1 more than HGH would ever be able to naturally do.

If you take HGH with IGF-1 you shouldn't need to worry about a lack of IGF-1 receptors either, since HGH will increase the amount of IGF-1 receptors for it to bind to and cause growth. As well as this, IGF-1 also activates its own receptors, so more IGF-1 = more IGF-1 receptors.

Like I said in the guide the distribution of your receptors is somewhat random and increasing receptors in particular areas (such as your legs for example) isn't really possible. Some areas of your body will have varying amounts of receptors depending on your genetics, but overall there is a somewhat equal distribution, which is why when you grow taller your face, arms, torso, etc will also grow. You can't control where you grow, it is random, you can only influence the rate at which you grow.

 

[Napoleon1800]

I didn't want to respond to this because it was so retarded but here I go:

You don't "lose" any gains from injecting IGF 1 as opposed to just injecting straight HGH, HGH produces IGF-1, by injecting IGF- 1 LR3 we are "bypassing the middleman" (HGH) so to speak and increasing serum blood concentration of IGF-1 more than HGH would ever be able to naturally do.

If you take HGH with IGF-1 you shouldn't need to worry about a lack of IGF-1 receptors either, since HGH will increase the amount of IGF-1 receptors for it to bind to and cause growth. As well as this, IGF-1 also activates its own receptors, so more IGF-1 = more IGF-1 receptors.

Like I said in the guide the distribution of your receptors is somewhat random and increasing receptors in particular areas (such as your legs for example) isn't really possible. Some areas of your body will have varying amounts of receptors depending on your genetics, but overall there is a somewhat equal distribution, which is why when you grow taller your face, arms, torso, etc will also grow. You can't control where you grow, it is random, you can only influence the rate at which you grow.

Thanks for the explanation.

 

[Deleted member 30323]

Most heightmaxxing threads do offer something new tbh. Each thread has its own unique individual experience involved in each one, for example, PTH and Calcitrol is rarely discussed on this forum. An analysis of the mechanisms of growth hasn't really been discussed since 2019-2020

Are you mentally retarded?
Calcitrol is just vitamin D on steroids, and if you take that of enough you don't need it.

Please kys immediately and do not spread misinformation here.

 

[Osie]

Are you mentally retarded?
Calcitrol is just vitamin D on steroids, and if you take that of enough you don't need it.

Please kys immediately and do not spread misinformation here.

Wha I do?

 

[Deleted member 30323]

Wha I do?

You are spreading misinformation, calcitriol will give you hypervitaminosis not that height growth you want.

 

[Deleted member 30323]

Just supplement with 60k IU vit d every week.

 

[Vista]

Just supplement with 60k IU vit d every week.

60k IU once per week is a joke if we’re talking about optimising height. Calcitriol can seem dangerous, but it really isn’t if you use it properly.

D3 already converts into calcitriol, which is just a metabolically active form of vitamin d. The problem is that vd3 has poor metabolic activity so this conversion isn’t so straight forward.

By taking Calcitriol directly we are skipping this conversion, which is actually safer compared to mega dosing vd3.

Since vd3 has poor metabolic activity it will build up more than Calcitriol (since Calcitriol will be used in metabolic process’ like height growth).

Build up is what causes health issues, and vd3 builds up more than Calcitriol, taking Calcitriol is safer and more effective

 

[Osie]

You are spreading misinformation, calcitriol will give you hypervitaminosis not that height growth you want.

I didn't even make this thread or recommend it lol

 

[MEMPSK]

Europeans and Asians undergo bone fusion later (usually 16) compared to as early as 14 in Africans and Hispanics.

Intresting, high quality thread

 

[MEMPSK]

Do you have a guide like this on testosterone? This format is great.

 

[kanderior]

If you brought this list to a doctor who knows you, you trust, been going to for a long time etc. and told him "guide me through this, advise me and help me implement this stack" would he do it or tell you it would go against medical ethics and recommend you to a psychiatrist?

 

[Osie]

If you brought this list to a doctor who knows you, you trust, been going to for a long time etc. and told him "guide me through this, advise me and help me implement this stack" would he do it or tell you it would go against medical ethics and recommend you to a psychiatrist?

Second option

 

[iammoh]

This is almost a cliche but: Diet, Sleep and exercise

Almost nobody gets these right.

1) Use Chronometer to make sure you're getting enough macro/ micronutrients, if not supplement with bioavailable micronutrients , avoid processed foods/ seed oils and avoid eating 3 hours before bed time. Almost everyone is deficent in Vit D + k2, so you should probs be supplementing them at least, and probably zinc, b complex and omega 3. Throne and Nootropics depot are generally good sources. Eat lots of protein.

2) Research sleep hygiene, wake up and go to bed at the same time every night (even weekends), only use your bed for sleep (dont sit on your phone in bed), mag l threonate is a good supplement for sleep (but sleep hygiene/ habits are more important), dont use melatonin.

3) Exercise, going to the gym for 45 minutes a few times per week isn't enough, you need to be exercising every day. Do sprint intervals, medium distance runs, calisthenics and weight training (if you like it). Also do lots of low impact exercise, things like walking, incline walking, swimming, etc.

These three things alone probably enhance you height and quality of life more than anything else.

Why should we avoid eating 3 hours before bedtime?

 

[Vista]

Why should we avoid eating 3 hours before bedtime?

to optimise natural gh release during sleep