Why DHT is fucking useless and should be NUKED TO THE GROUND

[appeal]

Their risks are much more than DHT. The androgenic compounds you have in mind will rape their follicles even more than DHT nigga u don’t make sense

yes because it’s more androgenic so you get more androgenic facial effects.

 

[halloweed]

yes because it’s more androgenic so you get more androgenic facial effects.

Side effects in comparison to DHT are extensive.

 

[ReasonableAdvice]

Why you should be actively nuking DHT to the absolute fucking ground.
Everyone talks about, needing DHT for bone mass, and says DHT is what causes bone growth of the chin and brow ridge and frame for example. or increase height etc. This is complete fucking bro science, a complete myth. Testosterone is the only androgen that induces bone growth.

The Study

5-Alpha-Reductase Deficiency - StatPearls - NCBI Bookshelf

What the studies of 5AR inhibition show is that people that are deficient in 5AR (people who have practically 0 DHT) have normal heights compared to their normal siblings (who produce DHT), and their bone mineral density is the same as any normal person.

View attachment 3390643

Left and middle (5ARD), right (normal). As you can see, their skeletal and muscle mass is not affected by DHT at all.

What does DHT effect then?
All DHT affects beard, penis, skin and hair follicles.

So, stop the DHT cope ASAP.
So stop the DHT cope, even with supraphysiological amounts of DHT, it won't induce facial bone growth to a comparable extent as testosterone.
You could have taken 2.5mg of Dutasteride since you were bone, and you would have ended up looking the exact same as you do right now, some would have looked better, since you complete retards left it too late since they wanted a better frame, or chin or something all while your hair is being actively raped. Any change you would have gotten to anything you thought DHT was going to do, was done by Testosterone.

After puberty DHT will make you look like The Rock or Jason Statham. But probably less muscular and less good looking... A lot less. So if your not hopping on at least Duta 0.5mg a day, you’re actively looksmining yourself.

DHT copers are complete brainless retards, who would rather lose their hair and skin for muh bones that was caused by Testosterone anyway, what complete fucking jesters.

TLDR: These fucking pills right here will save your fucking life, you'll keep your hair (hair is life), you'll keep your youthful skin, without inhibiting any sort of bone growth.

Spoiler

View attachment 3390659

@Clavicular @Dyorotic

DHT does have a role in muscle mass and skeletal health, and I wouldn't suggest literally nuking DHT as it has reams of negative effects, but despite our arguments over DNP, this post is actually very insightful and I agree people should stop posting pseudoscientific slop making it out as some wonder-chemical (e.g., my argument on https://looksmax.org/threads/dht-maxing-ultimate-guide.167747/page-9#post-18523302).

Influence of testosterone and dihydrotestosterone on bone-matrix induced endochondral bone formation - Calcified Tissue International

A bone matrix-induced endochondral bone development model has been used to study the effects of androgens on different stages of bone development in castrated young adult rats. Androgen treatment, especially with dihydrotestosterone (DHT) for 7 days, inhibited35SO4 incorporation by the...

link.springer.com

Dihydrotestosterone, a robust promoter of osteoblastic proliferation and differentiation: understanding of time-mannered and dose-dependent control of bone forming cells - PMC

The present study was aimed to evaluate the time-mannered and dose-dependent effects of 5α-dihydrotestosterone (5α-DHT) on the proliferation and differentiation of bone forming cells using MC3T3-E1 cells. Cell proliferation was analyzed using MTS ...

pmc.ncbi.nlm.nih.gov

https://www.sciencedirect.com/science/article/abs/pii/8756328296001354?via%3Dihub

Effects of dihydrotestosterone alone and combined with estrogen on bone mineral density, bone growth, and formation rates in ovariectomized rats - PubMed

Androgens are associated with the greater skeletal mass and size in men compared with women and have been used as anabolic agents promoting skeletal growth and mineral accretion in both sexes, but specific effects on growth and bone formation in the female skeleton are not well understood. The...

pubmed.ncbi.nlm.nih.gov

https://academic.oup.com/jcem/article-abstract/91/8/3017/2656509?redirectedFrom=fulltext (BMD = Bone Mineral Density)

 

[appeal]

DHT does have a role in muscle mass and skeletal health, and I wouldn't suggest literally nuking DHT as it has reams of negative effects, but despite our arguments over DNP, this post is actually very insightful and I agree people should stop posting pseudoscientific slop making it out as some wonder-chemical (e.g., my argument on https://looksmax.org/threads/dht-maxing-ultimate-guide.167747/page-9#post-18523302).

Influence of testosterone and dihydrotestosterone on bone-matrix induced endochondral bone formation - Calcified Tissue International

A bone matrix-induced endochondral bone development model has been used to study the effects of androgens on different stages of bone development in castrated young adult rats. Androgen treatment, especially with dihydrotestosterone (DHT) for 7 days, inhibited35SO4 incorporation by the...

link.springer.com

Dihydrotestosterone, a robust promoter of osteoblastic proliferation and differentiation: understanding of time-mannered and dose-dependent control of bone forming cells - PMC

The present study was aimed to evaluate the time-mannered and dose-dependent effects of 5α-dihydrotestosterone (5α-DHT) on the proliferation and differentiation of bone forming cells using MC3T3-E1 cells. Cell proliferation was analyzed using MTS ...

pmc.ncbi.nlm.nih.gov

https://www.sciencedirect.com/science/article/abs/pii/8756328296001354?via%3Dihub

Effects of dihydrotestosterone alone and combined with estrogen on bone mineral density, bone growth, and formation rates in ovariectomized rats - PubMed

Androgens are associated with the greater skeletal mass and size in men compared with women and have been used as anabolic agents promoting skeletal growth and mineral accretion in both sexes, but specific effects on growth and bone formation in the female skeleton are not well understood. The...

pubmed.ncbi.nlm.nih.gov

https://academic.oup.com/jcem/article-abstract/91/8/3017/2656509?redirectedFrom=fulltext (BMD = Bone Mineral Density)

After puberty, which for most is past the age of 18, I believe DHT to not have any positives physically, only raping hairline, inhibiting elastin production along with body hair etc. i am aware of the positive mental effects of dht, however a raped hairline will cause much more mental trauma especially to young people than lower dht would imo, no one wants to be with a balding 18-20 yea old. whites are extremely prone to balding, unless native american, by 20 most white I know already have recession, and in my option, prevention is a much better option than waiting for supposed changes (that u don’t know will even happen) like better facial bones or frame caused by dht (which the study i linked show it’s mainly based on test, not dht) whilst having your hairline raped so even if dht did had an effect after puberty, it’s not nearly as much as test, so in my opinion the trade off is completely worth it.

i haven’t read the study’s you linked, but i will.

i made after puberty bold since some 16 year olds there think i was referring to nuking dht during puberty which yes is completely fucking retarded.

 

[appeal]

After puberty, which for most is past the age of 18, I believe DHT to not have any positives physically, only raping hairline, inhibiting elastin production along with body hair etc. i am aware of the positive mental effects of dht, however a raped hairline will cause much more mental trauma especially to young people than lower dht would imo, no one wants to be with a balding 18-20 yea old. whites are extremely prone to balding, unless native american, by 20 most white I know already have recession, and in my option, prevention is a much better option than waiting for supposed changes (that u don’t know will even happen) like better facial bones or frame caused by dht (which the study i linked show it’s mainly based on test, not dht) whilst having your hairline raped so even if dht did had an effect after puberty, it’s not nearly as much as test, so in my opinion the trade off is completely worth it.

i haven’t read the study’s you linked, but i will.

i made after puberty bold since some 16 year olds there think i was referring to nuking dht during puberty which yes is completely fucking retarded.

i am also aware their our outliers, yes, not eveyone is finished by 28, but the vast majority are. if your tanner stage 5, your good to go imo, or haven’t grown in ages, like over a year.

 

[ReasonableAdvice]

1. I agree, no significant physical changes post-puberty, although DHT is still useful so I wouldn't advise you tell people to nuke it.
2. Yes it does have strong links to AGA, elastin production reduction, and body hair. I still wouldn't suggest nuking it, because it has other bodily functions even post-puberty, but I wouldn't advise people go and artificially boost their DHT production myself for this reason.
3. Focusing on hair preservation via DHT inhibition is reasonable if you have that as a genetic predisposition, so yes fair enough.
4. DHT is less critical on skeletal changes but still useful.

What I said is still pretty accurate, but I agree that it's utility is less pronounced after puberty. I still would advise against NUKING it, as it can result in reduced libido and dysfunction (low in clinical trials but I am aware there are much higher anecdotal reports, which is still a clear factor even if you argue it is due to other variables). Some find the symptoms continue when you stop taking stuff like finasteride.

I would advise against long-term inhibition as it can cause problems with muscle mass and bone density benefits even as a secondary driver, as well as potentially chronic dry skin.

I would only "nuke" DHT if you have severe hair loss and the less extreme options haven't worked (e.g. Finasteride opposed to Dutasteride).


You don't need to look at the studies if you agree DHT is useful for muscle mass and bone growth/density, I was just substantiating my point as you asked someone else for evidence.

 

[appeal]

1. I agree, no significant physical changes post-puberty, although DHT is still useful so I wouldn't advise you tell people to nuke it.
2. Yes it does have strong links to AGA, elastin production reduction, and body hair. I still wouldn't suggest nuking it, because it has other bodily functions even post-puberty, but I wouldn't advise people go and artificially boost their DHT production myself for this reason.
3. Focusing on hair preservation via DHT inhibition is reasonable if you have that as a genetic predisposition, so yes fair enough.
4. DHT is less critical on skeletal changes but still useful.

What I said is still pretty accurate, but I agree that it's utility is less pronounced after puberty. I still would advise against NUKING it, as it can result in reduced libido and dysfunction (low in clinical trials but I am aware there are much higher anecdotal reports, which is still a clear factor even if you argue it is due to other variables). Some find the symptoms continue when you stop taking stuff like finasteride.

I would advise against long-term inhibition as it can cause problems with muscle mass and bone density benefits even as a secondary driver, as well as potentially chronic dry skin.

I would only "nuke" DHT if you have severe hair loss and the less extreme options haven't worked (e.g. Finasteride opposed to Dutasteride).


You don't need to look at the studies if you agree DHT is useful for muscle mass and bone growth/density, I was just substantiating my point as you asked someone else for evidence.

IMO it’s risk vs reward, as someone who has a bald father, and all his brothers also bald (although on my mothers side they are all perfectly fine) as someone who noticed slight temple recession, i wouldn’t have risked my hairline. I used Duta since it had benefits to skin also (Fin does not) and I don’t see a reason to use a less effective form of the medication. In regards to causing problems to bone mass and density, i use test, and test is both anabolic and androgenic, so i dont think i have a problem in that regard. (Rescission was noticed before test btw)

So for me, the only risk i see from using Duta is possible mental sides. I believe i’d still continue having changes in bone growth and density due to test since that is the primary driver.

 

[ReasonableAdvice]

IMO it’s risk vs reward, as someone who has a bald father, and all his brothers also bald (although on my mothers side they are all perfectly fine) as someone who noticed slight temple recession, i wouldn’t have risked my hairline. I used Duta since it had benefits to skin also (Fin does not) and I don’t see a reason to use a less effective form of the medication. In regards to causing problems to bone mass and density, i use test, and test is both anabolic and androgenic, so i dont think i have a problem in that regard. (Rescission was noticed before test btw)

So for me, the only risk i see from using Duta is possible mental sides. I believe i’d still continue having changes in bone growth and density due to test since that is the primary driver.

Just keep an eye on your mental health bro, you don't want any irreversible conditions. Also avoid nuking DHT to the max as it can affect prostate health (even if reducing conditions like BPH iirc), sexual function (as it has a stronger effect than testosterone I'm pr sure, at least in terms of libido and functional erections), fat distribution, and vascular health. But honestly, as none of these will give you any immediate conditions, I trust you can/have regulated it maturely and are able to reduce the dosage or stop it entirely if the adverse effects outweigh the positive ones.

The stuff I provided was also mostly based around BMD, which is a more specific role of DHT, so you don't want a decrease in strength or mineralisation even if you think you're replacing it with testosterone.

 

[appeal]

@ReasonableAdvice
Yes, maybe once i hop off test, i will stop the Dutasteride and see what position im in and switch to fin. I don’t intent on staying on test for life. Dutasteride half life is pretty long tho 4-5 weeks iirc.

Regarding fat distribution, not saying i’m going to, or recommending anyone to do so, but any idea if DHT derivatives have any effect on that? For example masteron. I am also aware that dutasteride has absolutely no effect on that since it doesn’t need to be converted with 5AR. It would realistically have to be stopped with something like RU.

Apart from fractures, and weaker bones, BMD doesn’t effect the look / size of bones, so it wouldn’t have an effect on your looks, but yes, still something to consider health and lifestyle wise. i’ll keep other effects in mind.

 

[larox.psl]

Take dht if below 20 so that you get big dick. Later you can take dut/fin

Muh muh big dick from dht.How can you not understand common sense pal?
Everyone has different areas sensitive to DHT. For example, someone might have high DHT, but their penis area isn’t sensitive to it,so it doesnt get bigger . Still, their hair is norwooding or they grow more body hair from high dht

 

[appeal]

Muh muh big dick from dht.How can you not understand common sense pal?
Everyone has different areas sensitive to DHT. For example, someone might have high DHT, but their penis area isn’t sensitive to it,so it doesnt get bigger . Still, their hair is norwooding or they grow more body hair from high dht

This is all a matter of risk va reward. if he is norwooding but believes he has more dick to grow he has to make a calculated decision on if he wants to save his hair from the norwood reaper, or have the potential to have a bigger dick.
the way i see it, duta WILL save you from the norwood reaper. DHT MAY NOT grow dick more, but WILL continue to rape your hair.

IMO easy call.

 

[overtier1011]

Muh muh big dick from dht.How can you not understand common sense pal?
Everyone has different areas sensitive to DHT. For example, someone might have high DHT, but their penis area isn’t sensitive to it,so it doesnt get bigger . Still, their hair is norwooding or they grow more body hair from high dht

Worth a shot though

 

[averagenormie]

Like bro think about it
Lets say you produce 5 mg of testosterone daily
Which is 35 mg (which is like 400-500 ng/dl) and you have 10% conversion of dht thats 3.5 mg a week we know dht has like lets 5 more affinity than test so 3.5 mg of dht would equal 17.5 mg of testosterone (so you’ll need 52.5 mg of bioavailable testosterone who would be like 125-150 mg of testosterone enanthate if you remove the ester)
So for you to pull that off completetely you’ll need to use fin (cause dut would crush allopregnolone which is impossible to get exogenously)
You’ll need to supplement with Dhea and pregnolone
Youll need an ai
And youll need an aldoesterone inhibitor (preferably spironolactone so you avoid water retention remember dht is dry test is not and to avoid other side effects)
You’ll also need an ai cause now that you have low dht its dht party)

Brutal how a guy on trt have more test than a natty

 

[muragoat]

I used Duta since it had benefits to skin also (Fin does not)

explain why fin doesn t

 

[appeal]

explain why fin doesn t

duta inhibits both type 1 and type 2 5AR
fin only inhibits one of them

 

[p0lishsubhuman]

duta inhibits both type 1 and type 2 5AR
fin only inhibits one of them

how does elastin work in comparison to collagen

is it possible to have good collagen and bad elastin?

 

[appeal]

how does elastin work in comparison to collagen

is it possible to have good collagen and bad elastin?

yes, inhibited elastin makes your skin sag.

 

[looksmax mogger]

@20/04/2008 debunk?

 

[20/04/2008]

@20/04/2008 debunk?

@appeal said his using test
And used Dut and got no side effect whatsoever
That mean he has outlier gaba receptors that mean its perfectly fine and his like in his 20s i guess
If i was him I’ll just throw in an ai with a cortisol and aldosterone inhibtor and i would also use
Either a progesteronic compound like tren (low dose of course like 75 mg) or use progestene gel its over the counter

 

[swt]

dnr