Why DHT is important and should NOT be nuked to the ground

[Jonas2k7]

I'm tired of trannies here trying to convince people that DHT is useless. This thread will cover the importance of DHT and why you should not block it by the use of 5ARIs.

My friend @20/04/2008 made a really good thread on it, so I will just TLDR it:

DHT is not only the stronger version of Testosterone (2-4 times higher affinity to the androgen receptor), but also binds differently to the AR because of its unique structure.

This means each androgen exerts unique effects, including DHT. So only using Testosterone is not the right solution.

---

Now to the other uses of the enzyme 5α-reductase: It doesn't only affect the conversion of T to DHT, here are some other reactions:

Cholestenone → 5α-Cholestanone
Progesterone → 5α-Dihydroprogesterone
3α-Dihydroprogesterone → Allopregnanolone
3β-Dihydroprogesterone → Isopregnanolone
Deoxycorticosterone → 5α-Dihydrodeoxycorticosterone
Corticosterone → 5α-Dihydrocorticosterone
Aldosterone → 5α-Dihydroaldosterone
Androstenedione → 5α-Androstanedione
Nandrolone → 5α-Dihydronandrolone

The functions of some of the 5α-reduced steroids are unknown, that's why it isn't a good idea to "just use fin bro because you'll be fine."

Allopregnanolone is an important neurosteroid made from 3α-dihydroprogesterone. Without 5α-reductase on the other hand, this important neurosteroid can't be synthesized.

Progesterone → DHP via 5α-reductase
DHP → Allopregnanolone via 3α-HSD
Allopregnanolone → Positive allosteric modulator of GABA-A receptors

TLDR: No allopregnanolone -> More Anxiety, brain fog, stress, etc.

Not forgetting the sexual dysfunction aspect of inhibiting 5AR.

The cells of the corpus cavernosum have androgen receptors, when you agonize these receptors they produce endothelial nitric oxide. NO triggers the relaxation of smooth muscle cells, leading to vasodilation and increased blood flow, which is needed for achieving and maintaining an erection. This can't be replaced with cialis, you need the local AR activation.

The only solution to solve this, is to have high levels of testosterone especially locally in the area of the penis or to use topical DHT/testosterone gels, just to maintain the normal function of the penis.

There is also the mental aspect of the erectile dysfunction. The main role of the sex drive of males consists of the dopamine/oxytocin signalling, which is inhibited by the use of 5ARIs. So in short, 5ARIs result in a reduction of libido.

DHT is important for bone mass, which in the cited study even resulted in an increase of bone mass. DHT is also important for muscle strength.

"We have previously reported that DHT treatment increases bone mass in orchidectomized mice and that this effect is independent of estrogen receptors."

5AR is important for the metabolism of cortisol in the liver according to this study.

Persistent sexual side effects by the way... "Subjects reported new-onset persistent sexual dysfunction associated with the use of finasteride: 94% developed low libido, 92% developed erectile dysfunction, 92% developed decreased arousal, and 69% developed problems with orgasm."

Also ever wondered why most of the Finasteride users like Orc and Clavicular here never experience that many side effects from the 5ARI use? It's because they blast exogenous hormones to the point where the accumulated testosterone compensates for most of the side effects of the lacking DHT.

---

See? DHT is a vital male hormone, blocking it completely is harmful and has many other harms like discussed in this thread.

Tag list: @halloweed @JohnDoe @Always Stay You @ConfusedBolivian

 

[appeal]

Cope. Pure bro science.

Why DHT is fucking useless and should be NUKED TO THE GROUND

Why you should be actively nuking DHT to the absolute fucking ground. Everyone talks about, needing DHT for bone mass, and says DHT is what causes bone growth of the chin and brow ridge and frame for example. or increase height etc. This is complete fucking bro science, a complete myth...

looksmax.org

 

[Jonas2k7]

Cope. Pure bro science.

Why DHT is fucking useless and should be NUKED TO THE GROUND

Why you should be actively nuking DHT to the absolute fucking ground. Everyone talks about, needing DHT for bone mass, and says DHT is what causes bone growth of the chin and brow ridge and frame for example. or increase height etc. This is complete fucking bro science, a complete myth...

looksmax.org

I posted the thread 1 minute ago and cited many studies. This thread also is partially a response to your thread. Now explain why this is "bro science"

 

[castizo_ascender]

I'm tired of trannies here trying to convince people that DHT is useless. This thread will cover the importance of DHT and why you should not block it by the use of 5ARIs.

My friend @20/04/2008 made a really good thread on it, so I will just TLDR it:

DHT is not only the stronger version of Testosterone (2-4 times higher affinity to the androgen receptor), but also binds differently to the AR because of its unique structure.

This means each androgen exerts unique effects, including DHT. So only using Testosterone is not the right solution.

---

Now to the other uses of the enzyme 5α-reductase: It doesn't only affect the conversion of T to DHT, here are some other reactions:

Cholestenone → 5α-Cholestanone
Progesterone → 5α-Dihydroprogesterone
3α-Dihydroprogesterone → Allopregnanolone
3β-Dihydroprogesterone → Isopregnanolone
Deoxycorticosterone → 5α-Dihydrodeoxycorticosterone
Corticosterone → 5α-Dihydrocorticosterone
Aldosterone → 5α-Dihydroaldosterone
Androstenedione → 5α-Androstanedione
Nandrolone → 5α-Dihydronandrolone

The functions of some of the 5α-reduced steroids are unknown, that's why it isn't a good idea to "just use fin bro because you'll be fine."

Allopregnanolone is an important neurosteroid made from 3α-dihydroprogesterone. Without 5α-reductase on the other hand, this important neurosteroid can't be synthesized.

Progesterone → DHP via 5α-reductase
DHP → Allopregnanolone via 3α-HSD
Allopregnanolone → Positive allosteric modulator of GABA-A receptors

TLDR: No allopregnanolone -> More Anxiety, brain fog, stress, etc.

View attachment 3391024

Not forgetting the sexual dysfunction aspect of inhibiting 5AR.

The cells of the corpus cavernosum have androgen receptors, when you agonize these receptors they produce endothelial nitric oxide. NO triggers the relaxation of smooth muscle cells, leading to vasodilation and increased blood flow, which is needed for achieving and maintaining an erection. This can't be replaced with cialis, you need the local AR activation.

The only solution to solve this, is to have high levels of testosterone especially locally in the area of the penis or to use topical DHT/testosterone gels, just to maintain the normal function of the penis.

There is also the mental aspect of the erectile dysfunction. The main role of the sex drive of males consists of the dopamine/oxytocin signalling, which is inhibited by the use of 5ARIs. So in short, 5ARIs result in a reduction of libido.

View attachment 3391036

DHT is important for bone mass, which in the cited study even resulted in an increase of bone mass. DHT is also important for muscle strength.

"We have previously reported that DHT treatment increases bone mass in orchidectomized mice and that this effect is independent of estrogen receptors."

5AR is important for the metabolism of cortisol in the liver according to this study.

Persistent sexual side effects by the way... "Subjects reported new-onset persistent sexual dysfunction associated with the use of finasteride: 94% developed low libido, 92% developed erectile dysfunction, 92% developed decreased arousal, and 69% developed problems with orgasm."

Also ever wondered why most of the Finasteride users like Orc and Clavicular here never experience that many side effects from the 5ARI use? It's because they blast exogenous hormones to the point where the accumulated testosterone compensates for most of the side effects of the lacking DHT.

---

See? DHT is a vital male hormone, blocking it completely is harmful and has many other harms like discussed in this thread.

View attachment 3391173

Tag list: @halloweed @JohnDoe @Always Stay You @ConfusedBolivian

nice high effort post saar. i hope more ppl here see this

 

[Hexmask]

 

[Jonas2k7]

Yeah, trust this guy whose whole personality consists of two medications

 

[iblamemandible7]

Persistent sexual side effects study is completely out of context, those percentages are out of people who already reported sexual side effects in the first place, most sound pro-dht argument

 

[iblamemandible7]

"We conducted standardized interviews with 71 otherwise healthy men aged 21-46 years who reported the new onset of sexual side effects associated with the temporal use of finasteride, in which the symptoms persisted for at least 3 months despite the discontinuation of finasteride."

 

[Ur fav sub 5]

I'm tired of trannies here trying to convince people that DHT is useless. This thread will cover the importance of DHT and why you should not block it by the use of 5ARIs.

My friend @20/04/2008 made a really good thread on it, so I will just TLDR it:

DHT is not only the stronger version of Testosterone (2-4 times higher affinity to the androgen receptor), but also binds differently to the AR because of its unique structure.

This means each androgen exerts unique effects, including DHT. So only using Testosterone is not the right solution.

---

Now to the other uses of the enzyme 5α-reductase: It doesn't only affect the conversion of T to DHT, here are some other reactions:

Cholestenone → 5α-Cholestanone
Progesterone → 5α-Dihydroprogesterone
3α-Dihydroprogesterone → Allopregnanolone
3β-Dihydroprogesterone → Isopregnanolone
Deoxycorticosterone → 5α-Dihydrodeoxycorticosterone
Corticosterone → 5α-Dihydrocorticosterone
Aldosterone → 5α-Dihydroaldosterone
Androstenedione → 5α-Androstanedione
Nandrolone → 5α-Dihydronandrolone

The functions of some of the 5α-reduced steroids are unknown, that's why it isn't a good idea to "just use fin bro because you'll be fine."

Allopregnanolone is an important neurosteroid made from 3α-dihydroprogesterone. Without 5α-reductase on the other hand, this important neurosteroid can't be synthesized.

Progesterone → DHP via 5α-reductase
DHP → Allopregnanolone via 3α-HSD
Allopregnanolone → Positive allosteric modulator of GABA-A receptors

TLDR: No allopregnanolone -> More Anxiety, brain fog, stress, etc.

View attachment 3391024

Not forgetting the sexual dysfunction aspect of inhibiting 5AR.

The cells of the corpus cavernosum have androgen receptors, when you agonize these receptors they produce endothelial nitric oxide. NO triggers the relaxation of smooth muscle cells, leading to vasodilation and increased blood flow, which is needed for achieving and maintaining an erection. This can't be replaced with cialis, you need the local AR activation.

The only solution to solve this, is to have high levels of testosterone especially locally in the area of the penis or to use topical DHT/testosterone gels, just to maintain the normal function of the penis.

There is also the mental aspect of the erectile dysfunction. The main role of the sex drive of males consists of the dopamine/oxytocin signalling, which is inhibited by the use of 5ARIs. So in short, 5ARIs result in a reduction of libido.

View attachment 3391036

DHT is important for bone mass, which in the cited study even resulted in an increase of bone mass. DHT is also important for muscle strength.

"We have previously reported that DHT treatment increases bone mass in orchidectomized mice and that this effect is independent of estrogen receptors."

5AR is important for the metabolism of cortisol in the liver according to this study.

Persistent sexual side effects by the way... "Subjects reported new-onset persistent sexual dysfunction associated with the use of finasteride: 94% developed low libido, 92% developed erectile dysfunction, 92% developed decreased arousal, and 69% developed problems with orgasm."

Also ever wondered why most of the Finasteride users like Orc and Clavicular here never experience that many side effects from the 5ARI use? It's because they blast exogenous hormones to the point where the accumulated testosterone compensates for most of the side effects of the lacking DHT.

---

See? DHT is a vital male hormone, blocking it completely is harmful and has many other harms like discussed in this thread.

View attachment 3391173

Tag list: @halloweed @JohnDoe @Always Stay You @ConfusedBolivian

Good thread bhai

 

[Idontknowlol]

Everytime I see someone saying DHT is useless hormone I instantly know it’s some balding subhuman copeing for his broken dick

 

[T50CeI]

Filthy Jew

 

[halloweed]

Everytime I see someone saying DHT is useless hormone I instantly know it’s some balding subhuman copeing for his broken dick

It’s always someone who is over 18 as well. Always

 

[halloweed]

Nigger whether you believe in evolution or not. There is definitely a reason why this hormone is still in your body. Only now have you started to nuke this.

 

[Jonas2k7]

"We conducted standardized interviews with 71 otherwise healthy men aged 21-46 years who reported the new onset of sexual side effects associated with the temporal use of finasteride, in which the symptoms persisted for at least 3 months despite the discontinuation of finasteride."

My bad, then look at this study. TLDR: 20% of subjects with male pattern hair loss reported persistent sexual dysfunction for ≥6 years. These subjects also reported a wide range of symptoms including changes in cognition, ejaculate quality, and genital sensation.

 

[20/04/2008]

"We conducted standardized interviews with 71 otherwise healthy men aged 21-46 years who reported the new onset of sexual side effects associated with the temporal use of finasteride, in which the symptoms persisted for at least 3 months despite the discontinuation of finasteride."

Thread 'The Obsession about blocking DHT should end'
https://looksmax.org/threads/the-obsession-about-blocking-dht-should-end.1196460/
Waiting for your response

 

[20/04/2008]

Bro stop posting the same guy as a reply

 

[Hexmask]

"We conducted standardized interviews with 71 otherwise healthy men aged 21-46 years who reported the new onset of sexual side effects associated with the temporal use of finasteride, in which the symptoms persisted for at least 3 months despite the discontinuation of finasteride."

Control group is way too small to dictate this. Millions of men are prescribed fin in the US ALONE. Not even including other countries. So why is it just the small percentage of chronically online people who report "sides"? In a study they gave older men 5mg of fin a day for BPH. Yes 5mg 5 times the normal dosage of fin for hairloss and guess what they felt fine, because they aren't wasting away on forums.

 

[iblamemandible7]

My bad, then look at this study. TLDR: 20% of subjects with male pattern hair loss reported persistent sexual dysfunction for ≥6 years. These subjects also reported a wide range of symptoms including changes in cognition, ejaculate quality, and genital sensation.

Am I just retarded or did they not cite that figure at all? Which clinical series?

 

[Hexmask]

Bro stop posting the same guy as a reply

He at least sources studies. Im too lazy to pull out my folder of studies. Niggas will just insult it rather then actually watching it, and continue to take biased studies they find from PFS funded networks and cry about how good DHT is.

 

[iblamemandible7]

Control group is way too small to dictate this. Millions of men are prescribed fin in the US ALONE. Not even including other countries. So why is it just the small percentage of chronically online people who report "sides"? In a study they gave older men 5mg of fin a day for BPH. Yes 5mg 5 times the normal dosage of fin for hairloss and guess what they felt fine, because they aren't wasting away on forums.

Exactly pfs wasnt even a thing before the internet, im pointing out how that study is flawed

 

[20/04/2008]

I'm tired of trannies here trying to convince people that DHT is useless. This thread will cover the importance of DHT and why you should not block it by the use of 5ARIs.

My friend @20/04/2008 made a really good thread on it, so I will just TLDR it:

DHT is not only the stronger version of Testosterone (2-4 times higher affinity to the androgen receptor), but also binds differently to the AR because of its unique structure.

This means each androgen exerts unique effects, including DHT. So only using Testosterone is not the right solution.

---

Now to the other uses of the enzyme 5α-reductase: It doesn't only affect the conversion of T to DHT, here are some other reactions:

Cholestenone → 5α-Cholestanone
Progesterone → 5α-Dihydroprogesterone
3α-Dihydroprogesterone → Allopregnanolone
3β-Dihydroprogesterone → Isopregnanolone
Deoxycorticosterone → 5α-Dihydrodeoxycorticosterone
Corticosterone → 5α-Dihydrocorticosterone
Aldosterone → 5α-Dihydroaldosterone
Androstenedione → 5α-Androstanedione
Nandrolone → 5α-Dihydronandrolone

The functions of some of the 5α-reduced steroids are unknown, that's why it isn't a good idea to "just use fin bro because you'll be fine."

Allopregnanolone is an important neurosteroid made from 3α-dihydroprogesterone. Without 5α-reductase on the other hand, this important neurosteroid can't be synthesized.

Progesterone → DHP via 5α-reductase
DHP → Allopregnanolone via 3α-HSD
Allopregnanolone → Positive allosteric modulator of GABA-A receptors

TLDR: No allopregnanolone -> More Anxiety, brain fog, stress, etc.

View attachment 3391024

Not forgetting the sexual dysfunction aspect of inhibiting 5AR.

The cells of the corpus cavernosum have androgen receptors, when you agonize these receptors they produce endothelial nitric oxide. NO triggers the relaxation of smooth muscle cells, leading to vasodilation and increased blood flow, which is needed for achieving and maintaining an erection. This can't be replaced with cialis, you need the local AR activation.

The only solution to solve this, is to have high levels of testosterone especially locally in the area of the penis or to use topical DHT/testosterone gels, just to maintain the normal function of the penis.

There is also the mental aspect of the erectile dysfunction. The main role of the sex drive of males consists of the dopamine/oxytocin signalling, which is inhibited by the use of 5ARIs. So in short, 5ARIs result in a reduction of libido.

View attachment 3391036

DHT is important for bone mass, which in the cited study even resulted in an increase of bone mass. DHT is also important for muscle strength.

"We have previously reported that DHT treatment increases bone mass in orchidectomized mice and that this effect is independent of estrogen receptors."

5AR is important for the metabolism of cortisol in the liver according to this study.

Persistent sexual side effects by the way... "Subjects reported new-onset persistent sexual dysfunction associated with the use of finasteride: 94% developed low libido, 92% developed erectile dysfunction, 92% developed decreased arousal, and 69% developed problems with orgasm."

Also ever wondered why most of the Finasteride users like Orc and Clavicular here never experience that many side effects from the 5ARI use? It's because they blast exogenous hormones to the point where the accumulated testosterone compensates for most of the side effects of the lacking DHT.

---

See? DHT is a vital male hormone, blocking it completely is harmful and has many other harms like discussed in this thread.

View attachment 3391173

Tag list: @halloweed @JohnDoe @Always Stay You @ConfusedBolivian

Good thread bhais even tho its not 100% accurate our first thread was much better

 

[Hexmask]

Exactly pfs wasnt even a thing before the internet, im pointing out how that study is flawed

Yeah if these niggas would just delete social media and stop caring and also take some anastrazole they probably would be perfectly fine.

 

[20/04/2008]

He at least sources studies. Im too lazy to pull out my folder of studies. Niggas will just insult it rather then actually watching it, and continue to take biased studies they find from PFS funded networks and cry about how good DHT is.

Bro i litteraly writen botb thread worthy with @Jonas2k7 compiling dozens of study no one give a fuck
Thread 'The Obsession about blocking DHT should end'
https://looksmax.org/threads/the-obsession-about-blocking-dht-should-end.1196460/

 

[20/04/2008]

No matter how many study i post everyone gonna think dht ks a useless hormone

 

[20/04/2008]

Not a molecule

Fuck you bro

 

[LongForgotten]

If you're not gonna link studies, idc ab ur analysis

 

[Jonas2k7]

Good thread bhais even tho its not 100% accurate our first thread was much better

Needed to make a quick thread, this guy @appeal tried to make people in puberty block DHT completely, argumenting that it's useless for development

 

[chadhamed]

Fuck you bro

Go suck his dick

 

[iblamemandible7]

No matter how many study i post everyone gonna think dht ks a useless hormone

Nigga you should see what dht did to my hairline in just one year I cant wait to obliterate that garbage hormone

 

[Hexmask]

Needed to make a quick thread, this guy @appeal tried to make people in puberty block DHT completely, argumenting that it's useless for development

View attachment 3391345

Past puberty, it affects your hair follicles, prostate, and sebaceous glands.

 

[Jonas2k7]

Nigga you should see what dht did to my hairline in just one year I cant wait to obliterate that garbage hormone

No wonder you are defending the use of 5ARIs then.

Also DHT has a lot of uses like shown in this thread and in the previous thread.

 

[iblamemandible7]

No wonder you are defending the use of 5ARIs then.

Also DHT has a lot of uses like shown in this thread and in the previous thread.

DHT is useful for turning HTNs into LTN baldcels with hairy ass arms saar

 

[craven]

No tag no care NIGGER
jfl at anti dht NIGGERS tho

 

[Axhar1]

water
keep dht in puberty
nuke dht post

 

[20/04/2008]

Nigga you should see what dht did to my hairline in just one year I cant wait to obliterate that garbage hormone

Bro it wasn’t dht
It was your fucked up (micronutrients profile your high cortisol and prolactin low E2 low igf-1 low sleep high Bp and water retention)

I sincerely believe that unless your using 500 mg of test+++ you shouldn’t need to use fin (dut is a death sentence don’t talk about it) haven’t you tead that study where they use nandrolone with dut vs nandrolone alone and nandrolone alone caused less hairloss haven’t you read that study where they used Tamoxifen with finasteride and they got hairloss

 

[Jonas2k7]

No tag no care NIGGER
jfl at anti dht NIGGERS tho

I wanted to tag you right now nigga, then I got the notification that you repped the thread.

Tagging other users that might be interested:

@lestoa you can't escape the DHT pill boyo
@4lt.Real
@mvp2v1
@noodlelover

 

[DR. NICKGA]

Heh?

I wanted to tag you right now nigga, then I got the notification that you repped the thread.

Tagging other users that might be interested:

@lestoa you can't escape the DHT pill boyo
@4lt.Real
@mvp2v1
@noodlelover

You cannot escape facts and logic

 

[DR. NICKGA]

* put cool eye glasses on *

Heh.. time to destroy you so much with the power of facts and logic

 

[averagenormie]

 

[Jonas2k7]

Now said it in your own words instead of linking videos.

 

[DR. NICKGA]

Bro i litteraly writen botb thread worthy with @Jonas2k7 compiling dozens of study no one give a fuck
Thread 'The Obsession about blocking DHT should end'
https://looksmax.org/threads/the-obsession-about-blocking-dht-should-end.1196460/

Your so stupid

Why did you head not explode when you were born

 

[DR. NICKGA]

Nigger whether you believe in evolution or not. There is definitely a reason why this hormone is still in your body. Only now have you started to nuke this.

Dht cause baldness

 

[iblamemandible7]

Bro it wasn’t dht
It was your fucked up (micronutrients profile your high cortisol and prolactin low E2 low igf-1 low sleep high Bp and water retention)

I sincerely believe that unless your using 500 mg of test+++ you shouldn’t need to use fin (dut is a death sentence don’t talk about it) haven’t you tead that study where they use nandrolone with dut vs nandrolone alone and nandrolone alone caused less hairloss haven’t you read that study where they used Tamoxifen with finasteride and they got hairloss

Bla bla bla nigga my grandpa started going bald at the exact same time and my dad is balding right now genetics are merciless we just have extremely dht sensitive follicles all the water retention in the world aint changing that

I take multivitamins 100g+ protein a day and get 6-8 hours sleep which isnt the best but I can still guarantee my micronutrients are better than my ancestors

Can guarantee E2 isnt low either cuz Ive had mild gyno since 13 jfl balding is a genetic disease and the cure is killing your dht

 

[averagenormie]

Now said it in your own words instead of linking videos.

watch the vids and stop the cope, dht is trash in adulthood

 

[JohnBaza]

I'm tired of trannies here trying to convince people that DHT is useless. This thread will cover the importance of DHT and why you should not block it by the use of 5ARIs.

My friend @20/04/2008 made a really good thread on it, so I will just TLDR it:

DHT is not only the stronger version of Testosterone (2-4 times higher affinity to the androgen receptor), but also binds differently to the AR because of its unique structure.

This means each androgen exerts unique effects, including DHT. So only using Testosterone is not the right solution.

---

Now to the other uses of the enzyme 5α-reductase: It doesn't only affect the conversion of T to DHT, here are some other reactions:

Cholestenone → 5α-Cholestanone
Progesterone → 5α-Dihydroprogesterone
3α-Dihydroprogesterone → Allopregnanolone
3β-Dihydroprogesterone → Isopregnanolone
Deoxycorticosterone → 5α-Dihydrodeoxycorticosterone
Corticosterone → 5α-Dihydrocorticosterone
Aldosterone → 5α-Dihydroaldosterone
Androstenedione → 5α-Androstanedione
Nandrolone → 5α-Dihydronandrolone

The functions of some of the 5α-reduced steroids are unknown, that's why it isn't a good idea to "just use fin bro because you'll be fine."

Allopregnanolone is an important neurosteroid made from 3α-dihydroprogesterone. Without 5α-reductase on the other hand, this important neurosteroid can't be synthesized.

Progesterone → DHP via 5α-reductase
DHP → Allopregnanolone via 3α-HSD
Allopregnanolone → Positive allosteric modulator of GABA-A receptors

TLDR: No allopregnanolone -> More Anxiety, brain fog, stress, etc.

View attachment 3391024

Not forgetting the sexual dysfunction aspect of inhibiting 5AR.

The cells of the corpus cavernosum have androgen receptors, when you agonize these receptors they produce endothelial nitric oxide. NO triggers the relaxation of smooth muscle cells, leading to vasodilation and increased blood flow, which is needed for achieving and maintaining an erection. This can't be replaced with cialis, you need the local AR activation.

The only solution to solve this, is to have high levels of testosterone especially locally in the area of the penis or to use topical DHT/testosterone gels, just to maintain the normal function of the penis.

There is also the mental aspect of the erectile dysfunction. The main role of the sex drive of males consists of the dopamine/oxytocin signalling, which is inhibited by the use of 5ARIs. So in short, 5ARIs result in a reduction of libido.

View attachment 3391036

DHT is important for bone mass, which in the cited study even resulted in an increase of bone mass. DHT is also important for muscle strength.

"We have previously reported that DHT treatment increases bone mass in orchidectomized mice and that this effect is independent of estrogen receptors."

5AR is important for the metabolism of cortisol in the liver according to this study.

Persistent sexual side effects by the way... "Subjects reported new-onset persistent sexual dysfunction associated with the use of finasteride: 94% developed low libido, 92% developed erectile dysfunction, 92% developed decreased arousal, and 69% developed problems with orgasm."

Also ever wondered why most of the Finasteride users like Orc and Clavicular here never experience that many side effects from the 5ARI use? It's because they blast exogenous hormones to the point where the accumulated testosterone compensates for most of the side effects of the lacking DHT.

---

See? DHT is a vital male hormone, blocking it completely is harmful and has many other harms like discussed in this thread.

View attachment 3391173

Tag list: @halloweed @JohnDoe @Always Stay You @ConfusedBolivian

chad eats pizza with milkshake while jeffrey investigates every hormone
@itzyaboyJJ @Kaari @MA_ascender @cobicado901 @wsada

 

[wsada]

chad eats pizza with milkshake while jeffrey investigates every hormone
@itzyaboyJJ @Kaari @MA_ascender @cobicado901 @wsada

You made me hungry fuck u nigga

 

[DR. NICKGA]

No , barely any side effects get reported using a 5ar inhibitor (dht blocker)

How To Avoid Nocebo From Finasteride/Dutasteride

This will be a quick and simple guide to avoid nocebo effect when taking finasteride or dutasteride. What is nocebo? Per the dictionary: "when a patient's negative expectations about a medical treatment cause them to experience undesirable symptoms or illnesses, even though the treatment has no...

looksmax.org

I even sended many meta studys (the beat source of scientific proof) about it

You garbage theory dont HAPPEND in reality

 

[DR. NICKGA]

Bro it wasn’t dht
It was your fucked up (micronutrients profile your high cortisol and prolactin low E2 low igf-1 low sleep high Bp and water retention)

I sincerely believe that unless your using 500 mg of test+++ you shouldn’t need to use fin (dut is a death sentence don’t talk about it) haven’t you tead that study where they use nandrolone with dut vs nandrolone alone and nandrolone alone caused less hairloss haven’t you read that study where they used Tamoxifen with finasteride and they got hairloss

Wrong DHT MAKE YOU BALD

 

[DR. NICKGA]

I gonna make a new thread about it

 

[Trisomy 21]

so what the fuck do we do if ur going bald later on in life ? i dont wanna have to take that poison finasteride or dutasteride shit, whats the next solution?