Avoiding Negative Sides While Heightmaxing : Hairloss with Aromasin/Exemestane

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Wrecker

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In the heightmaxing community, many hours of research have been conducted to uncover (generally) how to increase human height. Many individuals seek to go the natural route, taking over the counter supplements combined with daily exercise and yoga. Whereas some individuals seek to go down the... not so natural route. As an example, take these regimens from - Extra Chromosome , JustTrynaGrow , Wincel , etc. If you wish to get into heightmaxing, these guides are a great place to start/revist.

As mentioned previously, the regimens above are NOT natural; They are mainly composed of peptide injections combined with oral supplements. Most of which are already being taken by many - Glucosamine, Magnesium, Chondroitin, Melatonin, etc. HOWEVER..... One of the biggest factors that "hinders" height in humans is the amount of Estrogen present. That is why an AI ( Androgen Inhibitor ) is almost always present within a regimen. Some of the most common AI's are Aromasin ( Exemestane) , Arimidex ( anastrozole ) and Femera ( letrozole ) . Exemestane/Aromasin most common. The reason that this is brought up is, this medication is originally designed for female breast cancer patients. It's ultimate goal is to block the enzyme aromatase (found in the body's muscle, skin, breast and fat), which is used to convert androgens (hormones produced by the adrenal glands) into estrogen. Heightmaxers use it to stop estrogen from closing growth plates, which estrogen plays an important role in that.

Regardless of how it works or what it does, it has some side effects. One of which is hair loss. This one is scary because no one can see that your joints hurt a little, but people can most definitely see if you have hair or not. Hair / head hair is important to generally all of the male population, and people certainly don't want to be losing their hair; especially if they are on looksmax.

There have been a multitude of clinical studies conducted on breast cancer patients that have been put on an AI's , and hair loss is most definitely a concern. If you look at this IES study , here , it can be easily seen that 15.1 % of 1058 females treated with Exemestane were found with alopecia / hair loss. It is also well known that AI's widely used in BC treatment (letrozole, anastrozole, exemestane) and tamoxifen are known to cause HL (male pattern baldness) . See this exact study, here.

It's good to note that within the study that was just mentioned, it brings up how to combat the hair loss as well. As it is known, there is no evidence based therapy in regards to this issue. However, topical 5-alpha reductase inhibitors and supplementation of vitamin C and omega 3 fatty acids in a non-toxic dose without leading to any adverse effect on the person appear to be the most appropriate options in the management of this issue.


Post your thoughts below. Do you believe there is a better way to combat MPB / male pattern baldness whilst on an AI like Aromasin? What else can be done in regards to this issue?

Thank you.
 
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Good thread
 
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aromasin for example raises dht by 198% lol
take dht blocker if u had relatives with mpb
 
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Good thread
 
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aromasin for example raises dht by 198% lol
take dht blocker if u had relatives with mpb
Goat supplement for pubertaxxers
 
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Jokes on you, AIs dont work properly when you take ARIs

Some freaky genes are in play there
 
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Please elaborate on that
Any chance you COULD elaborate on that? If you have done any research regarding this topic..
I dont remember the pathway anymore

@x30001 I dont like to tag you that much, but do you remember the gene/pathway which acts or is rendered useless when taken with AI AND Nizoral for example? or finasteride? I dont quite remember
 
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I dont remember the pathway anymore

@x30001 I dont like to tag you that much, but do you remember the gene/pathway which acts or is rendered useless when taken with AI AND Nizoral for example? or finasteride? I dont quite remember
Dou you mean that AIs dont work while on duta or fin?
 
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easy solution.
Fulvestrant, for example, is a selective estradiol receptor degrader with 100% antagonism for the estradiol receptors.
whereas the active metabolites of both clomifene and tamoxifen display both agonistic and antagonistic effects in select tissue.
 
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can not tell you for sure, I am waiting for this smartcel I tagged, otherwise just ignore and try it
easy solution.
Fulvestrant, for example, is a selective estradiol receptor degrader with 100% antagonism for the estradiol receptors.
whereas the active metabolites of both clomifene and tamoxifen display both agonistic and antagonistic effects in select tissue.

Is this the best option in your opinion? I dont know if you have seen @JustTrynaGrow 's stack for height increase, but that is the one I am most interested in at the moment. I am an openplatecel, just concerned about the sides of Aromasin tbh.
 
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Is this the best option in your opinion? I dont know if you have seen @JustTrynaGrow 's stack for height increase, but that is the one I am most interested in at the moment. I am an openplatecel, just concerned about sides.
No, it isn't.
there is a buttload of selective estrogen receptor modulators. All in which are dangerous to mess around with, tamoxifen, for example, antagonizes the estradiol receptors in the breast tissue potently, whereas it exerts estrogenic effects within the bone and liver due to its selective receptor agonism, hence the name of the class of drugs 'selective estrogen receptor modulator'. The same thing goes for clomifene, it exerts estrogenic effects within the liver and bone tissue, yet deprives the hypothalamic estradiol receptors due to the potent antagonization. There are of course drugs that exert non-selective estradiol receptor antagonism, like enclomifene which is highly antagonistic whereas zuclomifene has a highly agonistic affinity for the estradiol receptors.

letrozole and anastrozole are still better options because they eliminate the bodies' most agonistic endogenous estradiol receptor ligands. The only downside, they upregulate 5-alpha-reductase (could be a positive for some) in response to lowered serum estradiol and aromatase.
 
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No, it isn't.
there is a buttload of selective estrogen receptor modulators. All in which are dangerous to mess around with, tamoxifen, for example, antagonizes the estradiol receptors in the breast tissue potently, whereas it exerts estrogenic effects within the bone and liver due to its selective receptor agonism, hence the name of the class of drugs 'selective estrogen receptor modulator'. The same thing goes for clomifene, it exerts estrogenic effects within the liver and bone tissue, yet deprives the hypothalamic estradiol receptors due to the potent antagonization. There are of course drugs that exert non-selective estradiol receptor antagonism, like enclomifene which is highly antagonistic whereas zuclomifene has a highly agonistic affinity for the estradiol receptors.

letrozole and anastrozole are still better options because they eliminate the bodies' most agonistic endogenous estradiol receptor ligands. The only downside, they upregulate 5-alpha-reductase (could be a positive for some) in response to lowered serum estradiol and aromatase.
High iq
 
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No, it isn't.
there is a buttload of selective estrogen receptor modulators. All in which are dangerous to mess around with, tamoxifen, for example, antagonizes the estradiol receptors in the breast tissue potently, whereas it exerts estrogenic effects within the bone and liver due to its selective receptor agonism, hence the name of the class of drugs 'selective estrogen receptor modulator'. The same thing goes for clomifene, it exerts estrogenic effects within the liver and bone tissue, yet deprives the hypothalamic estradiol receptors due to the potent antagonization. There are of course drugs that exert non-selective estradiol receptor antagonism, like enclomifene which is highly antagonistic whereas zuclomifene has a highly agonistic affinity for the estradiol receptors.

letrozole and anastrozole are still better options because they eliminate the bodies' most agonistic endogenous estradiol receptor ligands. The only downside, they upregulate 5-alpha-reductase (could be a positive for some) in response to lowered serum estradiol and aromatase.

I see. I've spent quite a lot of time on @Extra Chromosome 's heightmaxing thread. I am unsure if you know what that is. I think I've seen you in a few threads regarding heighmaxing.. Anyhow, in the comments people mentioned how Aromasin was the most safe because it was a suicidal AI, or something like that; it dident tank up your estrogen and release it all at once. I was looking into Aromasin because my budget isint all that amazing, and if I were to miss a dose it wouldent be a major issue. Am I mistaken at all? I'm looking to heightmax, but I don't know which AI to use considering the side effects. Main one being hairloss becuase that's kinda..... It's just scary to me. I'm a youngcel.
 
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I see. I've spent quite a lot of time on @Extra Chromosome 's heightmaxing thread. I am unsure if you know what that is. I think I've seen you in a few threads regarding heighmaxing.. Anyhow, in the comments people mentioned how Aromasin was the most safe because it was a suicidal AI, or something like that; it dident tank up your estrogen and release it all at once. I was looking into Aromasin because my budget isint all that amazing, and if I were to miss a dose it wouldent be a major issue. Am I mistaken at all? I'm looking to heightmax, but I don't know which AI to use considering the side effects. Main one being hairloss becuase that's kinda..... It's just scary to me. I'm a youngcel.
For me aromasin fucked me up
For me aromasin fucked me up
Try arimidex instead
 
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I see. I've spent quite a lot of time on @Extra Chromosome 's heightmaxing thread. I am unsure if you know what that is. I think I've seen you in a few threads regarding heighmaxing.. Anyhow, in the comments people mentioned how Aromasin was the most safe because it was a suicidal AI, or something like that; it dident tank up your estrogen and release it all at once. I was looking into Aromasin because my budget isint all that amazing, and if I were to miss a dose it wouldent be a major issue. Am I mistaken at all? I'm looking to heightmax, but I don't know which AI to use considering the side effects. Main one being hairloss becuase that's kinda..... It's just scary to me. I'm a youngcel.
no pain no gain.
if you're not willing to sacrifice some hair that you can always gain back later for the sake of gaining some height, then don't even bother.

exemestane is a steroidal based aromatase inhibitor, the drugs active metabolite binds to the aromatase enzyme at a higher affinity than other endogenous steroid hormones, therefore inhibiting it from binding to testosterone and synthesizing estradiol, it exerts the enzyme inactive until the body eventually metabolizes it, that's why it's called 'suicidal', because it essentially attaches to the enzyme inerting it 'inactive'. The discontinuation of exemestane won't cause a 'rebound' aromatase levels will have stayed the same, they will just become active again.

letrozole and anastrozole act on the aromatase enzyme by competitively binding to the heme of its cytochrome P450 unit. They are both non-steroidal in their respective pharmacology.
 
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I'm so sorry :(( . What happened if you don't mind me asking?
Bad headaches and hairloss wich i reversed since going back to arimidex
 
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no pain no gain.
if you're not willing to sacrifice some hair that you can always gain back later for the sake of gaining some height, then don't even bother.

exemestane is a steroidal based aromatase inhibitor, the drugs active metabolite binds to the aromatase enzyme at a higher affinity than other endogenous steroid hormones, therefore inhibiting it from binding to testosterone and synthesizing estradiol, it exerts the enzyme inactive until the body eventually metabolizes it, that's why it's called 'suicidal', because it essentially attaches to the enzyme inerting it 'inactive'. The discontinuation of exemestane won't cause a 'rebound' aromatase levels will have stayed the same, they will just become active again.

letrozole and anastrozole act on the aromatase enzyme by competitively binding to the heme of its cytochrome P450 unit. They are both non-steroidal in their respective pharmacology.

I really appreciate the information you're giving me. Personally I am gravitating to there not being a rebound, but if there are better AI options out there I'm definitely open to advice. You mention letrozole and anastrozole.. Are those any LESS dangerous to use than exemestane? You mention that they upregulate 5-alpha-reductase which isint great in regards to hairloss, can anything be done about that?
 
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if you're going as far as injecting hgh/peptides anyway & you're seriously worried about hairloss (I really don't think it's an issue but regardless) deca is probably the best all-around solution.

increases hgh levels in the body & plummets Testosterone levels. It's a pretty cheap & common steroid, can be used on it's own & hardly converts to estrogen at all.

main downsides imo are water retention if you don't cut down on carbs or use a bodybuilding dosage & the fact that it shuts you down pretty hard. 3 month long cycles max with this one if you don't wanna risk perma shutting yourself down.
 
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I really appreciate the information you're giving me. Personally I am gravitating to there not being a rebound, but if there are better AI options out there I'm definitely open to advice. You mention letrozole and anastrozole.. Are those any LESS dangerous to use than exemestane? I apologize for my lack of knowledge pertaining to this, but I most definitely appreciate the input.
look, aromatase inhibitors all do the same thing, inhibit aromatase, therefore, lower serum estradiol levels.
whether you decide to take exemestane or letrozole, it's up to you. Exemestane is a steroid that is structurally similar to 4-androstenedione, an endogenous androgenic metabolite, it is said to have metabolites that are up to 40x more androgenic than 5ar-dihydrotestosterone.

just pick one, it's not hard.
again, they all do the same thing, just at different dosages.
 
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if you're going as far as injecting hgh/peptides anyway & you're seriously worried about hairloss (I really don't think it's an issue but regardless) deca is probably the best all-around solution.

increases hgh levels in the body & plummets Testosterone levels. It's a pretty cheap & common steroid, can be used on it's own & hardly converts to estrogen at all.

main downsides imo are water retention if you don't cut down on carbs or use a bodybuilding dosage & the fact that it shuts you down pretty hard. 3 month long cycles max with this one if you don't wanna risk perma shutting yourself down.

I'm glad you showed up, lol. And injecting peptides... I'm really new to it all if I am honest. I came upon this forum a little under a week ago, but I am captivated to say the least. And I believe that male pettern baldness runs in my family, so that is where my concern comes from. Do you believe that growth would be comparable to the stack that you created? Includes GHRP-2 , Hexarelin.. Etc.
 
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I'm glad you showed up, lol. And injecting peptides... I'm really new to it all if I am honest. I came upon this forum a little under a week ago, but I am captivated to say the least. And I believe that male pettern baldness runs in my family, so that is where my concern comes from. Do you believe that growth would be comparable to the stack that you created? Includes GHRP-2 , Hexarelin.. Etc.
Growth isn't consistent at all. If you're young you can expect to grow but there's no guarantee you will and anyone giving you estimated numbers is talking ass.

Also I didn't create the stack. I just used ECs stack, dropped the pricy compounds and slightly adjusted the dosages mainly based on data from datbetrues archive in order to find the best price/performance sweetspot i could
 
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look, aromatase inhibitors all do the same thing, inhibit aromatase, therefore, lower serum estradiol levels.
whether you decide to take exemestane or letrozole, it's up to you. Exemestane is a steroid that is structurally similar to 4-androstenedione, an endogenous androgenic metabolite, it is said to have metabolites that are up to 40x more androgenic than 5ar-dihydrotestosterone.

just pick one, it's not hard.
again, they all do the same thing, just at different dosages.

Well said. I appreciate the input.
 
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just remember, extra chromosome made the greatest heightmaxxing threads with sources in the history in this site,

then said it was all bullshit and that you're getting diabetes, hair loss, ball shrinkage, heart problems, and once u start injecting u cant stop or youre fucked, then he promptly left the site forever
 
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just remember, extra chromosome made the greatest heightmaxxing threads with sources in the history in this site,

then said it was all bullshit and that you're getting diabetes, hair loss, ball shrinkage, heart problems, and once u start injecting u cant stop or youre fucked, then he promptly left the site forever
It’s HGH or nothing. Probably a sham to get sales on multivitamins and sarms.
 
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It’s HGH or nothing. Probably a sham to get sales on multivitamins and sarms.
you either get testosterone and hgh from a legit endocrinologist, plus blood tests and growth plate checks, or ur a retard
 
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you either get testosterone and hgh from a legit endocrinologist, plus blood tests and growth plate checks, or ur a retard
How much is a growth plate check? I’m 15 and that’s more money and I’m 100% convinced that they’re open.
 
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How much is a growth plate check? I’m 15 and that’s more money and I’m 100% convinced that they’re open.


I got mine checked recently. My insurance covered it all. But look, they don't USUALLY "just check your growth plates" . My parents had to do some serious wordplay to get there.. Or at least in my case.
 
5ar inhibitors are good but def do not take saw palmetto or any orals. topical only
 
just remember, extra chromosome made the greatest heightmaxxing threads with sources in the history in this site,

then said it was all bullshit and that you're getting diabetes, hair loss, ball shrinkage, heart problems, and once u start injecting u cant stop or youre fucked, then he promptly left the site forever
Once you stop injecting what?
I’m current on HGH, I can stop whenever right ?
 
Once you stop injecting what?
I’m current on HGH, I can stop whenever right ?
once you stop injecting your levels will never be as high as they once were

for example

lets say your base natural t is 700 ng/dl

you roid up to 1000 ng/dl like a madman

however once youre off you base is now significantly less than 700ng/dl, and you will never regain that higher natural level again

its like minoxidil, the second you stop using it's worse than before

especially during 18-25 when your T levels are the highest they'll ever be.
 
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aromasin isn't going to do much anyways
 
no pain no gain.
if you're not willing to sacrifice some hair that you can always gain back later for the sake of gaining some height, then don't even bother.

exemestane is a steroidal based aromatase inhibitor, the drugs active metabolite binds to the aromatase enzyme at a higher affinity than other endogenous steroid hormones, therefore inhibiting it from binding to testosterone and synthesizing estradiol, it exerts the enzyme inactive until the body eventually metabolizes it, that's why it's called 'suicidal', because it essentially attaches to the enzyme inerting it 'inactive'. The discontinuation of exemestane won't cause a 'rebound' aromatase levels will have stayed the same, they will just become active again.

letrozole and anastrozole act on the aromatase enzyme by competitively binding to the heme of its cytochrome P450 unit. They are both non-steroidal in their respective pharmacology.
how do you gain the hair back? thinking of dhtmaxxing, bones>hair but ofc i still want hair if i start losing it
 
just ask deleted member theorem
 
aromasin for example raises dht by 198% lol
take dht blocker if u had relatives with mpb
It’s because of reservatrol which convertse estrogen into testosterone and testosterone into DHT, which if you don’t have estrogen then the only thing it would do is it to convert t into DHT.
 
M
In the heightmaxing community, many hours of research have been conducted to uncover (generally) how to increase human height. Many individuals seek to go the natural route, taking over the counter supplements combined with daily exercise and yoga. Whereas some individuals seek to go down the... not so natural route. As an example, take these regimens from - Extra Chromosome , JustTrynaGrow , Wincel , etc. If you wish to get into heightmaxing, these guides are a great place to start/revist.

As mentioned previously, the regimens above are NOT natural; They are mainly composed of peptide injections combined with oral supplements. Most of which are already being taken by many - Glucosamine, Magnesium, Chondroitin, Melatonin, etc. HOWEVER..... One of the biggest factors that "hinders" height in humans is the amount of Estrogen present. That is why an AI ( Androgen Inhibitor ) is almost always present within a regimen. Some of the most common AI's are Aromasin ( Exemestane) , Arimidex ( anastrozole ) and Femera ( letrozole ) . Exemestane/Aromasin most common. The reason that this is brought up is, this medication is originally designed for female breast cancer patients. It's ultimate goal is to block the enzyme aromatase (found in the body's muscle, skin, breast and fat), which is used to convert androgens (hormones produced by the adrenal glands) into estrogen. Heightmaxers use it to stop estrogen from closing growth plates, which estrogen plays an important role in that.

Regardless of how it works or what it does, it has some side effects. One of which is hair loss. This one is scary because no one can see that your joints hurt a little, but people can most definitely see if you have hair or not. Hair / head hair is important to generally all of the male population, and people certainly don't want to be losing their hair; especially if they are on looksmax.

There have been a multitude of clinical studies conducted on breast cancer patients that have been put on an AI's , and hair loss is most definitely a concern. If you look at this IES study , here , it can be easily seen that 15.1 % of 1058 females treated with Exemestane were found with alopecia / hair loss. It is also well known that AI's widely used in BC treatment (letrozole, anastrozole, exemestane) and tamoxifen are known to cause HL (male pattern baldness) . See this exact study, here.

It's good to note that within the study that was just mentioned, it brings up how to combat the hair loss as well. As it is known, there is no evidence based therapy in regards to this issue. However, topical 5-alpha reductase inhibitors and supplementation of vitamin C and omega 3 fatty acids in a non-toxic dose without leading to any adverse effect on the person appear to be the most appropriate options in the management of this issue.


Post your thoughts below. Do you believe there is a better way to combat MPB / male pattern baldness whilst on an AI like Aromasin? What else can be done in regards to this issue?

Thank you.
kre dimorphism during puberty😊, just massage your scalp every once in a while, also just have a non recessed skull so scalp blood supply isn’t bad, oh and lastly, most women take 25-50mg of aromasin or basically the full dose or more, while we have made it clear that we just want to slightly decrease our estrogen levels so the impact won’t be that bad, also I don’t really care my grandpa has no balding except some hair thinning in the middle of his head and he’s like almost 70 also he has a non recessed skull, and my father who has a recessed skull has severe balding, but since balding usually happens to u if your moms father had it, then I’m good since well like I said my grandpa doesn’t have any balding other than some slight hair thinning, actually blud gets haircuts every now and then☠️, oh yeah and lastly if you have less estrogen due to AIs don’t worry abt stunting growth, I asked my doctor if having abnormally low estrogen during puberty would affect the brain in any way and he said that since the brain can adapt to anything because of its neuroplasticity it will be fine and growth wouldn’t be brain growth/development won’t be stunted.
 
That
25 mg / day
thats fucking retarded, yeah man sinking estrogen levels into oblivion is smart asf:ROFLMAO:,in hieghtmxing ur supposed to start with 1/4 a pill and build up from there. nuking oestrogen levels instantly is absolutely retarded and could kill someone.
 
b
I'm glad you showed up, lol. And injecting peptides... I'm really new to it all if I am honest. I came upon this forum a little under a week ago, but I am captivated to say the least. And I believe that male pettern baldness runs in my family, so that is where my concern comes from. Do you believe that growth would be comparable to the stack that you created? Includes GHRP-2 , Hexarelin.. Etc.
bro the best stack to date is aromasin + igf1 lr3, thats it. also i have a legit source that sells phizer aromasin, not that fucking russian counterfeit thats probably just a placebo or sum
 
just remember, extra chromosome made the greatest heightmaxxing threads with sources in the history in this site,

then said it was all bullshit and that you're getting diabetes, hair loss, ball shrinkage, heart problems, and once u start injecting u cant stop or youre fucked, then he promptly left the site forever
yeah but to a quick summary aromasin + igf1 lr3 is the best stack to date for heightmaxxing, PROVE ME WRONG
 

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