The older you are, the more extreme your heightmaxing should be.

[enchanted_elixir]

Folks here state otherwise.

It may work it may not. I said it may not. Theoretically it may in some cases. It certainly cannot be guaranteed.

There's a pathway capable of completely stopping and expanding growth plate. However, it's not a fairy tail. The downside is that you could lose every single strand of hair in your head and many other umpredictable side effects. So I don't make this info public just for safety. Else id have done myself already

What's the pathway?

 

[lepzerin]

5'8

Why are you so short when you know so much about heightening? Please don't get me wrong dude

 

[letsdoit]

Yeah 1295

Was the 1265 a typo or is that another compound?

 

[J o r o]

What's the pathway?

Do you have information about "growth plate never fuse completely"?

 

[J o r o]

we can make exchange

 

[Osie]

So letrozole would be too strong of an estrogen inhibitor to use?

It depends if you are using it properly and whether you have bloodwork to check where your e2 levels are.

If you are taking Letrozole 3x a week 1.25MG, your e2 might not be absolutely crushed and you can still have somewhat optimal E2 levels. But your e2 levels has to 15 pg or below for it to be helpful for heightmaxxing so if your body responds good to those levels you are in luck,

 

[enchanted_elixir]

Do you have information about "growth plate never fuse completely"?

Did I say that on some thread or something?

 

[PURE ARYAN GENETICS]

just get cancer for 2mm theory

 

[enchanted_elixir]

just get cancer for 2mm theory

Nobody will get cancer unless you already have it or your immune system is tera shit

 

[enchanted_elixir]

And even if you did, you'd die of hyperhyperhyperacromegaly wayyyyyyyy before.

 

[Saint Casanova]

Is mk-677 with CJC-1295 a good alternative to injecting pure HGH as it follows a more similar mechanism to how your body actually produces Growth hormone, just at an increased magnitude?

 

[FrenchChad]

So letrozole would be too strong of an estrogen inhibitor to use?

Of course that it would. aside from joint pains, eventually hair loss if prone to it (t will be converted in DHT as an homeostatic response, this is why aromasin increase dht by 160%) and igf-1 receptor desensitization (which can be induced by chronic elevated igf-1 levels as suggested by the stack in op) you will fuck your mental health, cognitive function and want to kill yourself.
Estrogens are involved in the synthetization of basically all of the most important neurotransmitters except glutamate, acetylcholine and epinephrine : Serotonin, Dopamine, GABA and Norepinephrine.

Serotonin is highly responsible for happiness and well-being, there is a reasons why antidepressants (SSRI's) acts on this specific neurotransmitter.
Totally crashing your estrogens with letrozole is not a good idea for this mere reason.

Also for igf-1 receptors desensitization here is some interesting studies :
Yakar S, et al. (2001). Circulating levels of IGF-1 directly regulate bone growth and density. In The Journal of Clinical Investigation, 110(6), 771-781.
In this one they show that elevated sustained igf-1 levels may reduce responsiveness of bone tissue to igf-1, so "megadosing" igf-1 lr3 aside from drastically increasing your chances of getting a cancer would probably be counterproductive.
Sandhu MS, et al. (2002). IGF-I and risk of type 2 diabetes: A study of 15,000 individuals. In Diabetes, 51(12), 12.
This study investigates the association between high levels of IGF-1 and the risk of developing type 2 diabetes. It suggests that chronically elevated IGF-1 levels may be related to a reduced sensitivity to the metabolic effects of IGF-1.

 

[FrenchChad]

And even if you did, you'd die of hyperhyperhyperacromegaly wayyyyyyyy before.

Cope jfl.
More cells division = higher risk of getting cancer, igf-1 is indirectly linked with all forms of cancers and high igf-1 levels are directly linked with breasts, colorectal and prostate cancers.
And people with acromegaly who dies from complications of this disease usually die from cardiovascular problems, respiratory problems or cancers induced by their astronomical levels of igf-1.

 

[NECK&VOICE]

Letrozole - strongest aromatase inhibitor (estrogen inhibitor)
IGF-1 LR3 megadose (4 weeks on, 2 off, repeat)
CJC-1265 with DAC
Protein ultra hyperdose

If you're in your early 20s, your stack should look something like that or else manlet forever.

You wont grow once growth plates are closed

 

[J o r o]

Cope jfl.
More cells division = higher risk of getting cancer, igf-1 is indirectly linked with all forms of cancers and high igf-1 levels are directly linked with breasts, colorectal and prostate cancers.
And people with acromegaly who dies from complications of this disease usually die from cardiovascular problems, respiratory problems or cancers induced by their astronomical levels of igf-1.

More cell division = higher risk of cancer is a 50/50 statement.

During development we have much more cell proliferation than in adulthood as organs are growing but that doesnt imply cancer.

The reason why Igf1 is upregulated in cancer is a biologically induced phemenom by cáncer cels/ and its enviroment in order to aid themselves with proliferation just like it happens during development. However, if no cancer cells are present then no cancer cells will multiply. Now, nothing can grow indifinitely in a healthy state. So what we look for is moderate alteration until desired results.

Acromegaly and all those folks have a permanent condition involving maybe many other factors beyond igf1 that i might not be aware of. Such as the methylation dysregulation for igf1 to be high all time, perhaps its affecting other pathways and lead to cancer. Its already a sign of defects in the system for sure. But back to my point what we do is for a period of time. Not permanent. Then everything goes back to normal.

 

[J o r o]

Did I say that on some thread or something?

I saw someone talk about it i dont remember. Since u were heightmaxxer i thought maybe youd be aware

 

[Deleted member 30323]

Mk677 instead of IGF 1 and its perfect

 

[enchanted_elixir]

Mk677 instead of IGF 1 and its perfect

Are you going to hyperdose protein alongside mk?

 

[enchanted_elixir]

Of course that it would. aside from joint pains, eventually hair loss if prone to it (t will be converted in DHT as an homeostatic response, this is why aromasin increase dht by 160%) and igf-1 receptor desensitization (which can be induced by chronic elevated igf-1 levels as suggested by the stack in op) you will fuck your mental health, cognitive function and want to kill yourself.
Estrogens are involved in the synthetization of basically all of the most important neurotransmitters except glutamate, acetylcholine and epinephrine : Serotonin, Dopamine, GABA and Norepinephrine.

Serotonin is highly responsible for happiness and well-being, there is a reasons why antidepressants (SSRI's) acts on this specific neurotransmitter.
Totally crashing your estrogens with letrozole is not a good idea for this mere reason.

Also for igf-1 receptors desensitization here is some interesting studies :
Yakar S, et al. (2001). Circulating levels of IGF-1 directly regulate bone growth and density. In The Journal of Clinical Investigation, 110(6), 771-781.
In this one they show that elevated sustained igf-1 levels may reduce responsiveness of bone tissue to igf-1, so "megadosing" igf-1 lr3 aside from drastically increasing your chances of getting a cancer would probably be counterproductive.
Sandhu MS, et al. (2002). IGF-I and risk of type 2 diabetes: A study of 15,000 individuals. In Diabetes, 51(12), 12.
This study investigates the association between high levels of IGF-1 and the risk of developing type 2 diabetes. It suggests that chronically elevated IGF-1 levels may be related to a reduced sensitivity to the metabolic effects of IGF-1.

That's why I recommend 4 weeks on, 2 weeks off since your IGF-1 receptors would become less responsive.

 

[enchanted_elixir]

Cope jfl.
More cells division = higher risk of getting cancer, igf-1 is indirectly linked with all forms of cancers and high igf-1 levels are directly linked with breasts, colorectal and prostate cancers.
And people with acromegaly who dies from complications of this disease usually die from cardiovascular problems, respiratory problems or cancers induced by their astronomical levels of igf-1.

If absurd levels of growth factors causes cancer, it would be a common cause of death with acromegaly ridden folks or even babies.

In adults, you're going to die of complications of decades of absurd growth before you die of cancer.

 

[enchanted_elixir]

I saw someone talk about it i dont remember. Since u were heightmaxxer i thought maybe youd be aware

I did have this thought when seeing this guy called Adam Rainer who kept growing into his thirties with severe, severe, severe acromegaly. But I can't confirm without me conducting a scientific study.

I don't think I was the one who said this though.

 

[Lorsss]

Doctors use stacks with similar mechanisms of action to help turbomanlets grow. They give HGH with aromatase inhibitors (check Pubmed for proof)
You probably say this because I am giving advice in a harsh, uncredible way because it's a quick post.

 

[J o r o]

I did have this thought when seeing this guy called Adam Rainer who kept growing into his thirties with severe severe acromegaly. But I can't confirm without me conducting a scientific study.

I don't think I was the one who said this though.

Ive done some research. The age of the growth plate would seem to indicate for them to be "fused" but we dont have a complete confirmation. From none of those people which is very weird as its basic documenting

 

[Lorsss]

@enchanted_elixir the thing is that Adam Reiner, the only person in history who suffered both dwarfism and later gigantism, he contracted pituary gland tumor in his early 20s and his high hgh level made him grow until his late 20s.

However you should consider he had dwarfism so maybe his condition may have inhibited closure of growth plates

Adam Rainer - Wikipedia

en.m.wikipedia.org

 

[enchanted_elixir]

@enchanted_elixir the thing is that Adam Reiner, the only person in history who suffered both dwarfism and later gigantism, he contracted pituary gland tumor in his early 20s and his high hgh level made him grow until his late 20s.

However you should consider he had dwarfism so maybe his condition may have inhibited closure of growth plates

Adam Rainer - Wikipedia

en.m.wikipedia.org

Well dwarfism isn't hypogonadism, so I don't think his estrogen levels were lower than average

 

[materialistic]

While incel spends thousands of dollars on substances and risks getting cancer chad eats pizza.

 

[FrenchChad]

More cell division = higher risk of cancer is a 50/50 statement.

During development we have much more cell proliferation than in adulthood as organs are growing but that doesnt imply cancer.

The reason why Igf1 is upregulated in cancer is a biologically induced phemenom by cáncer cels/ and its enviroment in order to aid themselves with proliferation just like it happens during development. However, if no cancer cells are present then no cancer cells will multiply. Now, nothing can grow indifinitely in a healthy state. So what we look for is moderate alteration until desired results.

Acromegaly and all those folks have a permanent condition involving maybe many other factors beyond igf1 that i might not be aware of. Such as the methylation dysregulation for igf1 to be high all time, perhaps its affecting other pathways and lead to cancer. Its already a sign of defects in the system for sure. But back to my point what we do is for a period of time. Not permanent. Then everything goes back to normal.

It is true that childrens and teenagers have much more cells divisions than adults and don't get cancers, however what they do also have are excellent dna repair mechanisms (by far the most important defense line against cancer) which get less efficient with age, along ideal t killer cells production for late teens. However, the older we get, the less efficient our DNA repair mechanisms become, and the less t killer cells we produce. Per se a 13 years old have much lesser chances of getting a cancer (except some rare forms) than a 24 years old dude (which himself have a much lesser chance than a 55 yo dude) for that reason.
Even temporarily you are increasing your chances of getting a cancer, especially considering that igf-1 lr3 have a very high half-life compared to DES (around 24h iirc), just ask yourself is the ride is worth it for gains that are unfortunately likely to be negligible past 20, while on the other hand LL is getting cheaper and more and more common.

And i wasn't talking about the upregulation of growth factors once the tumor is initiated but the linkage between the birth of the malignant tumor and high igf-1 levels, which is established for some forms of cancers (there is a reason why the production of igf-1.
Acromegaly is usually caused by a benign tumor in the pituitary gland, which is a concern in itself and the abnormally high cells division rate significantly increase your chances of getting a cancer with age because of the combination of that and the factors explained above.

If absurd levels of growth factors causes cancer, it would be a common cause of death with acromegaly ridden folks or even babies.

In adults, you're going to die of complications of decades of absurd growth before you die of cancer.

Same explanation, they contribute to it although not the sole factor and the older you get the less efficient dna repair mechanisms are while they are excellent during childhood and teens.
Here is the studies linking directly high igf-1 levels (not even absurd levels, just higher than normal) with the forms of cancer that i previously mentioned :

- Kaaks, R., Lukanova, A., Rinaldi, S., & et al. (2002). Interactions of insulin-like growth factors and insulin in colorectal cancer risk in the European Prospective Investigation into Cancer and Nutrition. Cancer Epidemiology, Biomarkers & Prevention, 11(8), 799-804. This study found that elevated IGF-1 levels were associated with an increased risk of colorectal cancer.
- Kaaks, R., Lukanova, A., Rinaldi, S., & et al. (2002). Interactions of insulin-like growth factors and insulin in colorectal cancer risk in the European Prospective Investigation into Cancer and Nutrition. Cancer Epidemiology, Biomarkers & Prevention, 11(8), 799-804. This study found that elevated IGF-1 levels were associated with an increased risk of colorectal cancer.
- Renehan, A. G., Zwahlen, M., Minder, C., & et al. (2004). Insulin-like growth factor (IGF)-I, IGF binding protein-3, and cancer risk: systematic review and meta-regression analysis. The Lancet, 363(9418), 1346-1353. This meta-analysis examined multiple studies and concluded that higher IGF-1 levels were associated with an increased risk of breast, prostate, and colorectal cancers.
- Giovannucci, E., Pollak, M., Liu, Y., & et al. (2003). Nutritional predictors of insulin-like growth factor I and their relationships to cancer in men. Cancer Epidemiology, Biomarkers & Prevention, 12(2), 84-89. This study investigated the relationship between diet, IGF-1 levels, and cancer risk in men and found that higher IGF-1 levels were associated with an increased risk of colorectal and prostate cancers.

As for acromegaly mens, cancer is one of the common causes of death among those mens (when they die of a cause related to acromegaly in the first place), respiratory and cardiac problems are just more common because heart muscles are responsive to anabolic pathways (also one of the reasons why many steroids users including zyzz himself died of heart problems).


I recall of some users trying heightmaxxing protocol in the past when heightmaxxing was a big thing here.
@Extra Chromosome tried several years ago a heightmaxxing protocol including letrozole and cjc dac, he just didn't had igf-1 lr3, he concluded after some times that heightmaxxing was cope and unironically roped, he was 18/19.
Same for Dyorotic2 (the dude who created the pubertymaxxing thread) who injected hgh but concluded that heightmaxxing was cope too and left, he was 17.
I hope that people who will try this protocol won't end up as disappointed at least by insufficient gains compared to the investment made.

 

[Deleted member 28722]

It is true that childrens and teenagers have much more cells divisions than adults and don't get cancers, however what they do also have are excellent dna repair mechanisms (by far the most important defense line against cancer) which get less efficient with age, along ideal t killer cells production for late teens. However, the older we get, the less efficient our DNA repair mechanisms become, and the less t killer cells we produce. Per se a 13 years old have much lesser chances of getting a cancer (except some rare forms) than a 24 years old dude (which himself have a much lesser chance than a 55 yo dude) for that reason.
Even temporarily you are increasing your chances of getting a cancer, especially considering that igf-1 lr3 have a very high half-life compared to DES (around 24h iirc), just ask yourself is the ride is worth it for gains that are unfortunately likely to be negligible past 20, while on the other hand LL is getting cheaper and more and more common.

And i wasn't talking about the upregulation of growth factors once the tumor is initiated but the linkage between the birth of the malignant tumor and high igf-1 levels, which is established for some forms of cancers (there is a reason why the production of igf-1.
Acromegaly is usually caused by a benign tumor in the pituitary gland, which is a concern in itself and the abnormally high cells division rate significantly increase your chances of getting a cancer with age because of the combination of that and the factors explained above.



Same explanation, they contribute to it although not the sole factor and the older you get the less efficient dna repair mechanisms are while they are excellent during childhood and teens.
Here is the studies linking directly high igf-1 levels (not even absurd levels, just higher than normal) with the forms of cancer that i previously mentioned :

- Kaaks, R., Lukanova, A., Rinaldi, S., & et al. (2002). Interactions of insulin-like growth factors and insulin in colorectal cancer risk in the European Prospective Investigation into Cancer and Nutrition. Cancer Epidemiology, Biomarkers & Prevention, 11(8), 799-804. This study found that elevated IGF-1 levels were associated with an increased risk of colorectal cancer.
- Kaaks, R., Lukanova, A., Rinaldi, S., & et al. (2002). Interactions of insulin-like growth factors and insulin in colorectal cancer risk in the European Prospective Investigation into Cancer and Nutrition. Cancer Epidemiology, Biomarkers & Prevention, 11(8), 799-804. This study found that elevated IGF-1 levels were associated with an increased risk of colorectal cancer.
- Renehan, A. G., Zwahlen, M., Minder, C., & et al. (2004). Insulin-like growth factor (IGF)-I, IGF binding protein-3, and cancer risk: systematic review and meta-regression analysis. The Lancet, 363(9418), 1346-1353. This meta-analysis examined multiple studies and concluded that higher IGF-1 levels were associated with an increased risk of breast, prostate, and colorectal cancers.
- Giovannucci, E., Pollak, M., Liu, Y., & et al. (2003). Nutritional predictors of insulin-like growth factor I and their relationships to cancer in men. Cancer Epidemiology, Biomarkers & Prevention, 12(2), 84-89. This study investigated the relationship between diet, IGF-1 levels, and cancer risk in men and found that higher IGF-1 levels were associated with an increased risk of colorectal and prostate cancers.

As for acromegaly mens, cancer is one of the common causes of death among those mens (when they die of a cause related to acromegaly in the first place), respiratory and cardiac problems are just more common because heart muscles are responsive to anabolic pathways (also one of the reasons why many steroids users including zyzz himself died of heart problems).


I recall of some users trying heightmaxxing protocol in the past when heightmaxxing was a big thing here.
@Extra Chromosome tried several years ago a heightmaxxing protocol including letrozole and cjc dac, he just didn't had igf-1 lr3, he concluded after some times that heightmaxxing was cope and unironically roped, he was 18/19.
Same for Dyorotic2 (the dude who created the pubertymaxxing thread) who injected hgh but concluded that heightmaxxing was cope too and left, he was 17.
I hope that people who will try this protocol won't end up as disappointed at least by insufficient gains compared to the investment made.

Dyrocrotic was 17, and he also moved to discord you fucking idiot. He started at 5’11 and now he’s 6’1.5. Can you please stop being a fucking retard?

 

[Deleted member 28722]

It is true that childrens and teenagers have much more cells divisions than adults and don't get cancers, however what they do also have are excellent dna repair mechanisms (by far the most important defense line against cancer) which get less efficient with age, along ideal t killer cells production for late teens. However, the older we get, the less efficient our DNA repair mechanisms become, and the less t killer cells we produce. Per se a 13 years old have much lesser chances of getting a cancer (except some rare forms) than a 24 years old dude (which himself have a much lesser chance than a 55 yo dude) for that reason.
Even temporarily you are increasing your chances of getting a cancer, especially considering that igf-1 lr3 have a very high half-life compared to DES (around 24h iirc), just ask yourself is the ride is worth it for gains that are unfortunately likely to be negligible past 20, while on the other hand LL is getting cheaper and more and more common.

And i wasn't talking about the upregulation of growth factors once the tumor is initiated but the linkage between the birth of the malignant tumor and high igf-1 levels, which is established for some forms of cancers (there is a reason why the production of igf-1.
Acromegaly is usually caused by a benign tumor in the pituitary gland, which is a concern in itself and the abnormally high cells division rate significantly increase your chances of getting a cancer with age because of the combination of that and the factors explained above.



Same explanation, they contribute to it although not the sole factor and the older you get the less efficient dna repair mechanisms are while they are excellent during childhood and teens.
Here is the studies linking directly high igf-1 levels (not even absurd levels, just higher than normal) with the forms of cancer that i previously mentioned :

- Kaaks, R., Lukanova, A., Rinaldi, S., & et al. (2002). Interactions of insulin-like growth factors and insulin in colorectal cancer risk in the European Prospective Investigation into Cancer and Nutrition. Cancer Epidemiology, Biomarkers & Prevention, 11(8), 799-804. This study found that elevated IGF-1 levels were associated with an increased risk of colorectal cancer.
- Kaaks, R., Lukanova, A., Rinaldi, S., & et al. (2002). Interactions of insulin-like growth factors and insulin in colorectal cancer risk in the European Prospective Investigation into Cancer and Nutrition. Cancer Epidemiology, Biomarkers & Prevention, 11(8), 799-804. This study found that elevated IGF-1 levels were associated with an increased risk of colorectal cancer.
- Renehan, A. G., Zwahlen, M., Minder, C., & et al. (2004). Insulin-like growth factor (IGF)-I, IGF binding protein-3, and cancer risk: systematic review and meta-regression analysis. The Lancet, 363(9418), 1346-1353. This meta-analysis examined multiple studies and concluded that higher IGF-1 levels were associated with an increased risk of breast, prostate, and colorectal cancers.
- Giovannucci, E., Pollak, M., Liu, Y., & et al. (2003). Nutritional predictors of insulin-like growth factor I and their relationships to cancer in men. Cancer Epidemiology, Biomarkers & Prevention, 12(2), 84-89. This study investigated the relationship between diet, IGF-1 levels, and cancer risk in men and found that higher IGF-1 levels were associated with an increased risk of colorectal and prostate cancers.

As for acromegaly mens, cancer is one of the common causes of death among those mens (when they die of a cause related to acromegaly in the first place), respiratory and cardiac problems are just more common because heart muscles are responsive to anabolic pathways (also one of the reasons why many steroids users including zyzz himself died of heart problems).


I recall of some users trying heightmaxxing protocol in the past when heightmaxxing was a big thing here.
@Extra Chromosome tried several years ago a heightmaxxing protocol including letrozole and cjc dac, he just didn't had igf-1 lr3, he concluded after some times that heightmaxxing was cope and unironically roped, he was 18/19.
Same for Dyorotic2 (the dude who created the pubertymaxxing thread) who injected hgh but concluded that heightmaxxing was cope too and left, he was 17.
I hope that people who will try this protocol won't end up as disappointed at least by insufficient gains compared to the investment made.

I wouldn’t be surprised if dyro is 6’3 now since he was still on the stack when he was banned and his brother is also 6’3 and dad is tall as shit + he’s Australian.

 

[Splinter901]

It is true that childrens and teenagers have much more cells divisions than adults and don't get cancers, however what they do also have are excellent dna repair mechanisms (by far the most important defense line against cancer) which get less efficient with age, along ideal t killer cells production for late teens. However, the older we get, the less efficient our DNA repair mechanisms become, and the less t killer cells we produce. Per se a 13 years old have much lesser chances of getting a cancer (except some rare forms) than a 24 years old dude (which himself have a much lesser chance than a 55 yo dude) for that reason.
Even temporarily you are increasing your chances of getting a cancer, especially considering that igf-1 lr3 have a very high half-life compared to DES (around 24h iirc), just ask yourself is the ride is worth it for gains that are unfortunately likely to be negligible past 20, while on the other hand LL is getting cheaper and more and more common.

And i wasn't talking about the upregulation of growth factors once the tumor is initiated but the linkage between the birth of the malignant tumor and high igf-1 levels, which is established for some forms of cancers (there is a reason why the production of igf-1.
Acromegaly is usually caused by a benign tumor in the pituitary gland, which is a concern in itself and the abnormally high cells division rate significantly increase your chances of getting a cancer with age because of the combination of that and the factors explained above.



Same explanation, they contribute to it although not the sole factor and the older you get the less efficient dna repair mechanisms are while they are excellent during childhood and teens.
Here is the studies linking directly high igf-1 levels (not even absurd levels, just higher than normal) with the forms of cancer that i previously mentioned :

- Kaaks, R., Lukanova, A., Rinaldi, S., & et al. (2002). Interactions of insulin-like growth factors and insulin in colorectal cancer risk in the European Prospective Investigation into Cancer and Nutrition. Cancer Epidemiology, Biomarkers & Prevention, 11(8), 799-804. This study found that elevated IGF-1 levels were associated with an increased risk of colorectal cancer.
- Kaaks, R., Lukanova, A., Rinaldi, S., & et al. (2002). Interactions of insulin-like growth factors and insulin in colorectal cancer risk in the European Prospective Investigation into Cancer and Nutrition. Cancer Epidemiology, Biomarkers & Prevention, 11(8), 799-804. This study found that elevated IGF-1 levels were associated with an increased risk of colorectal cancer.
- Renehan, A. G., Zwahlen, M., Minder, C., & et al. (2004). Insulin-like growth factor (IGF)-I, IGF binding protein-3, and cancer risk: systematic review and meta-regression analysis. The Lancet, 363(9418), 1346-1353. This meta-analysis examined multiple studies and concluded that higher IGF-1 levels were associated with an increased risk of breast, prostate, and colorectal cancers.
- Giovannucci, E., Pollak, M., Liu, Y., & et al. (2003). Nutritional predictors of insulin-like growth factor I and their relationships to cancer in men. Cancer Epidemiology, Biomarkers & Prevention, 12(2), 84-89. This study investigated the relationship between diet, IGF-1 levels, and cancer risk in men and found that higher IGF-1 levels were associated with an increased risk of colorectal and prostate cancers.

As for acromegaly mens, cancer is one of the common causes of death among those mens (when they die of a cause related to acromegaly in the first place), respiratory and cardiac problems are just more common because heart muscles are responsive to anabolic pathways (also one of the reasons why many steroids users including zyzz himself died of heart problems).


I recall of some users trying heightmaxxing protocol in the past when heightmaxxing was a big thing here.
@Extra Chromosome tried several years ago a heightmaxxing protocol including letrozole and cjc dac, he just didn't had igf-1 lr3, he concluded after some times that heightmaxxing was cope and unironically roped, he was 18/19.
Same for Dyorotic2 (the dude who created the pubertymaxxing thread) who injected hgh but concluded that heightmaxxing was cope too and left, he was 17.
I hope that people who will try this protocol won't end up as disappointed at least by insufficient gains compared to the investment made.

I based my stack off of extra chromosome stack + I remember about an user that grew 1 inch at 18-19 from peptides, he was called @JustTrynaGrow

https://looksmax.org/threads/injecting-hgh-at-23-for-any-possible-growth.594831/

 

[FrenchChad]

I based my stack off of extra chromosome stack + I remember about an user that grew 1 inch at 18-19 from peptides, he was called @JustTrynaGrow

https://looksmax.org/threads/injecting-hgh-at-23-for-any-possible-growth.594831/

Never said it wasn't possible, the message that you are quoting was mainly discussing about the health risks of chronically elevated igf-1 levels, just that some users ended up disappointed after trying those stacks, moreover we cannot really know which part can be attributed to the protocol and which part can be attributed to genetics/late puberty.

I will just post some anecdotical reports that i've read from users here (since they were mentioned in my previous message) about hgh and more widely heightmaxxing, of course they should all be taken with a grain of salt.
For Extra Chromosome here is where i've read about his case :

To those taking aromatase inhibitors to try to get taller

I don't think alot of you fully understand the risks and implications of doing this. I will enlighten you as someone who has dealt with crashed and low estrogen many many times. The longest my estrogen has been low at a time was about 2 weeks straight. This is what happened to me during that...

looksmax.org

He was taking a high dose of letrozole, getting more depressed, his dick stopped working, then one day he disappeared. It's just speculation what actually happened to him.

(ambiguous message he could be talking about a near future where he becomes diabetic but type 1 diabetes is imprevisible and type 2 diabetes that result from insulin insensitivity takes years, more often decades to install, either way it doesn't change much).

Why did @Extra Chromosome left?

@Extra Chromosome we need you😢

looksmax.org

Anyway for that case that's just speculation so take it with a grain of salt, apparently someone could be larping as him on discord.


Another case is @PubertyMaxxer, he got HGH for almost one year and said it was cope (provided proof so more legit at least) :

Adressing Hormone Cope

Here is literal proof I didmt grow 1 fucking cm with HGH at 15 Its all COPE, HORMONES ARE COPE UNLESS YIURE DEFICIENT , GENETICS ARE EVERYTHING IF YIU WEREN'T MEANT TO BE CHAD YIULL NEVER BE ONE @Dyorotic2 can confirm

looksmax.org

In that thread Dyorotic2 who is said to have injected hgh and is the author of a famous heightmaxxing thread ("PubertyMaxxing guide" in BOTB) said that it was cope :

Full thread : https://looksmax.org/threads/hgh-for-height-growth-is-cope.277736/
Another anecdotical report here : https://looksmax.org/threads/hgh-was-cope-for-me-im-not-growing-past-58-5.410433/

The main problem is that most of the studies were done on gh or igf-1 deficient kids, so we cannot accurately translate the results to healthy persons.
That being said, if you have the money and the time keep going, i'd be happy to learn that you did grew.

 

[Splinter901]

Never said it wasn't possible, the message that you are quoting was mainly discussing about the health risks of chronically elevated igf-1 levels, just that some users ended up disappointed after trying those stacks, moreover we cannot really know which part can be attributed to the protocol and which part can be attributed to genetics/late puberty.

I will just post some anecdotical reports that i've read from users here (since they were mentioned in my previous message) about hgh and more widely heightmaxxing, of course they should all be taken with a grain of salt.
For Extra Chromosome here is where i've read about his case :

To those taking aromatase inhibitors to try to get taller

I don't think alot of you fully understand the risks and implications of doing this. I will enlighten you as someone who has dealt with crashed and low estrogen many many times. The longest my estrogen has been low at a time was about 2 weeks straight. This is what happened to me during that...

looksmax.org

View attachment 2282795
(ambiguous message he could be talking about a near future where he becomes diabetic but type 1 diabetes is imprevisible and type 2 diabetes that result from insulin insensitivity takes years, more often decades to install, either way it doesn't change much).

Why did @Extra Chromosome left?

@Extra Chromosome we need you😢

looksmax.org

View attachment 2282815View attachment 2282820

Anyway for that case that's just speculation so take it with a grain of salt, apparently someone could be larping as him on discord.


Another case is @PubertyMaxxer, he got HGH for almost one year and said it was cope (provided proof so more legit at least) :

Adressing Hormone Cope

Here is literal proof I didmt grow 1 fucking cm with HGH at 15 Its all COPE, HORMONES ARE COPE UNLESS YIURE DEFICIENT , GENETICS ARE EVERYTHING IF YIU WEREN'T MEANT TO BE CHAD YIULL NEVER BE ONE @Dyorotic2 can confirm

looksmax.org

In that thread Dyorotic2 who is said to have injected hgh and is the author of a famous heightmaxxing thread ("PubertyMaxxing guide" in BOTB) said that it was cope :
View attachment 2282776


View attachment 2282827
Full thread : https://looksmax.org/threads/hgh-for-height-growth-is-cope.277736/
Another anecdotical report here : https://looksmax.org/threads/hgh-was-cope-for-me-im-not-growing-past-58-5.410433/

The main problem is that most of the studies were done on gh or igf-1 deficient kids, so we cannot accurately translate the results to healthy persons.
That being said, if you have the money and the time keep going, i'd be happy to learn that you did grew.

I Don't see them posting proof pictures how I did, I'm currently on coversations with 3 individuals who have grown one is 23 the other 24 and the other 20, they grew only like 1-2cm but still they grew a bit, in my opinion manletism depending on how severe needs to be treated with everything, hgh stacks + glucosamine and stretching + lifts + ll surgery.

 

[FrenchChad]

I Don't see them posting proof pictures how I did, I'm currently on coversations with 3 individuals who have grown one is 23 the other 24 and the other 20, they grew only like 1-2cm but still they grew a bit, in my opinion manletism depending on how severe needs to be treated with everything, hgh stacks + glucosamine and stretching + lifts + ll surgery.

PubertyMaxxer posted proofs that he did inject, although not a precise proof of his measurements i don't see why he would lie about his lack of success especially considering that he had faith in hgh before getting injections.
1-2 cm could be attributed to fixing slight posture defect or measurements mistakes, however it's totally possible that they did grew with the help of exogenous hormones, that's just that at some point if you really want to make a significant difference (2 cm increase won't save most manlets unfortunately) you need drastic methods (LL) and can't be restricted to hasardous methods.
I agree for the last part, just have realistic expectations if you go through the hormones route in order to avoid bad surprises.

 

[Splinter901]

PubertyMaxxer posted proofs that he did inject, although not a precise proof of his measurements i don't see why he would lie about his lack of success especially considering that he had faith in hgh before getting injections.
1-2 cm could be attributed to fixing slight posture defect or measurements mistakes, however it's totally possible that they did grew with the help of exogenous hormones, that's just that at some point if you really want to make a significant difference (2 cm increase won't save most manlets unfortunately) you need drastic methods (LL) and can't be restricted to hasardous methods.
I agree for the last part, just have realistic expectations if you go through the hormones route in order to avoid bad surprises.

But there are cases of manletism that are so severe that even a 4 inch LL doesn't save them, if they get 4 inch from LL but before that they get 1 inch from hgh, 1 inch from glucosamine + stretching + 1 inch from lifts then a 7 inch increase could save them, a 5'3 midget going to 5'10 could be life saving and the best part is that this could be achieved even as a 26 year old.

 

[FrenchChad]

But there are cases of manletism that are so severe that even a 4 inch LL doesn't save them, if they get 4 inch from LL but before that they get 1 inch from hgh, 1 inch from glucosamine + stretching + 1 inch from lifts then a 7 inch increase could save them, a 5'3 midget going to 5'10 could be life saving and the best part is that this could be achieved even as a 26 year old.

Maybe not at 26 (spine is said to fuse around 25 at best and that's the only place where you can eventually grow longitudinally with exogenous hormones since femur/legs bones closes earlier) but sure, if he keeps the lift routine and don't stop taking glucosamin+chondroitin.
Or he can get double LL (could end up cripple but that's not like someone at this height had something to lose anyway).

 

[Splinter901]

Maybe not at 26 (spine is said to fuse around 25 at best and that's the only place where you can eventually grow longitudinally with exogenous hormones since femur/legs bones closes earlier) but sure, if he keeps the lift routine and don't stop taking glucosamin+chondroitin.
Or he can get double LL (could end up cripple but that's not like someone at this height had something to lose anyway).

26 is around 25, supposedly clavicles close at 30 but I remember there were soldier corpses whose clavicles where closed at 23 and others that had theirs opened at 31, so given a 2 year margin of error spine could grow until 23-27, or if the sources that say spine closes at 30 then until 28-32, other sources say they never close and that oldcels in their 60s grew an inch in their spines from hgh I linked that on my hgh thread, if ur 5'3 it's fucking ogre and u should try hgh regardless of age alongside all the other stuff.

 

[Osie]

I recall of some users trying heightmaxxing protocol in the past when heightmaxxing was a big thing here.
@Extra Chromosome tried several years ago a heightmaxxing protocol including letrozole and cjc dac, he just didn't had igf-1 lr3, he concluded after some times that heightmaxxing was cope and unironically roped, he was 18/19.
Same for Dyorotic2 (the dude who created the pubertymaxxing thread) who injected hgh but concluded that heightmaxxing was cope too and left, he was 17.
I hope that people who will try this protocol won't end up as disappointed at least by insufficient gains compared to the investment made.

Lol this is pretty stupid when there has been countless people on here recounting height growth from heightmaxxing. I will remake this into a whole thread.

Those guys were blasting a bunch of hormones and then we unhappy when they didn't grow in 6 months lol. You can check the logs, they stopped caring about heightmaxxing 6 months in, when it takes years to grow.

ALL THESE EXAMPLES took multiple years while those idiots gave up after months.

Example 1 of growth:
Example 2 of growth:
Example 3 of growth:
Example 4 of growth:
Example 5 of growth:
Example 6 of growth:
Example 7 of growth:
Example 8 of growth:
Example 9 of growth:
Example 10 of growth:
Example 11 of growth:

 

[Osie]

Never said it wasn't possible, the message that you are quoting was mainly discussing about the health risks of chronically elevated igf-1 levels, just that some users ended up disappointed after trying those stacks, moreover we cannot really know which part can be attributed to the protocol and which part can be attributed to genetics/late puberty.

I will just post some anecdotical reports that i've read from users here (since they were mentioned in my previous message) about hgh and more widely heightmaxxing, of course they should all be taken with a grain of salt.
For Extra Chromosome here is where i've read about his case :

To those taking aromatase inhibitors to try to get taller

I don't think alot of you fully understand the risks and implications of doing this. I will enlighten you as someone who has dealt with crashed and low estrogen many many times. The longest my estrogen has been low at a time was about 2 weeks straight. This is what happened to me during that...

looksmax.org

Full thread : https://looksmax.org/threads/hgh-for-height-growth-is-cope.277736/
Another anecdotical report here : https://looksmax.org/threads/hgh-was-cope-for-me-im-not-growing-past-58-5.410433/

The main problem is that most of the studies were done on gh or igf-1 deficient kids, so we cannot accurately translate the results to healthy persons.
That being said, if you have the money and the time keep going, i'd be happy to learn that you did grew.

I hope you do realize that there is way more evidence on this site for hiehgtmaxxing than against it. The guys who don't grow from heightmaxxing are the guys who think they are smarter than it. If you have any open growth plate in your body, and you increase your HGH and IGF-1 superphysiological levels for multiple years while using AI properly to keep your e2 underneath 15pg/ml, you will grow.

There has never been anything on this website or literature to prove that statement. Heightmaxxing isn't this type of of unproven concept made up on forums, it is something that is backed up by plenty of literature and thousand of human experiences. It's obviously smart to be realistic but don't have a pessimistic viewpoint about something to be proven. There have literally been multiple studies done on children with idiopathic short stature (they are just short genetically, with no health issues). You can go on google and find them yourself.

 

[FrenchChad]

26 is around 25, supposedly clavicles close at 30 but I remember there were soldier corpses whose clavicles where closed at 23 and others that had theirs opened at 31, so given a 2 year margin of error spine could grow until 23-27, or if the sources that say spine closes at 30 then until 28-32, other sources say they never close and that oldcels in their 60s grew an inch in their spines from hgh I linked that on my hgh thread, if ur 5'3 it's fucking ogre and u should try hgh regardless of age alongside all the other stuff.

I said "around 25 at best" in the sense that 25 seems to be the maximum upper boundary and for most people it fuses earlier.

Growth plate cartilage, which is located at each end of the vertebral body, plays a pivotal role in promoting longitudinal spinal growth in humans and other mammals8, 10, 11, 12. The completion of longitudinal growth of human vertebral bodies, called epiphyseal closure, usually occurs between the ages of 18 and 25 years, and then the epiphyseal ring, comprising foci of calcification in the edges of end plates, is completely fused with the adjacent vertebral body in a process called epiphyseal union1

Persistence of vertebral growth plate cartilage in aged cynomolgus monkeys

Growth plates at each end of vertebral bodies play a pivotal role in longitudinal spinal growth. Epiphyseal closures are formed in adult humans. Although monkeys are frequently employed in bone and disc research, the age of epiphyseal closure has not ...

www.ncbi.nlm.nih.gov

Could you link those studies if you find them ? I'm interested about it, 30 seems really late, anyway even if it was possible it seems to be more of exceptional cases than of a general rule.
And yes 5'3 is nightmare mode even with a decent face, i agree in doing everything you can do in those extreme cases.

 

[Deleted member 28722]

Never said it wasn't possible, the message that you are quoting was mainly discussing about the health risks of chronically elevated igf-1 levels, just that some users ended up disappointed after trying those stacks, moreover we cannot really know which part can be attributed to the protocol and which part can be attributed to genetics/late puberty.

I will just post some anecdotical reports that i've read from users here (since they were mentioned in my previous message) about hgh and more widely heightmaxxing, of course they should all be taken with a grain of salt.
For Extra Chromosome here is where i've read about his case :

To those taking aromatase inhibitors to try to get taller

I don't think alot of you fully understand the risks and implications of doing this. I will enlighten you as someone who has dealt with crashed and low estrogen many many times. The longest my estrogen has been low at a time was about 2 weeks straight. This is what happened to me during that...

looksmax.org

View attachment 2282795
(ambiguous message he could be talking about a near future where he becomes diabetic but type 1 diabetes is imprevisible and type 2 diabetes that result from insulin insensitivity takes years, more often decades to install, either way it doesn't change much).

Why did @Extra Chromosome left?

@Extra Chromosome we need you😢

looksmax.org

View attachment 2282815View attachment 2282820

Anyway for that case that's just speculation so take it with a grain of salt, apparently someone could be larping as him on discord.


Another case is @PubertyMaxxer, he got HGH for almost one year and said it was cope (provided proof so more legit at least) :

Adressing Hormone Cope

Here is literal proof I didmt grow 1 fucking cm with HGH at 15 Its all COPE, HORMONES ARE COPE UNLESS YIURE DEFICIENT , GENETICS ARE EVERYTHING IF YIU WEREN'T MEANT TO BE CHAD YIULL NEVER BE ONE @Dyorotic2 can confirm

looksmax.org

In that thread Dyorotic2 who is said to have injected hgh and is the author of a famous heightmaxxing thread ("PubertyMaxxing guide" in BOTB) said that it was cope :
View attachment 2282776


View attachment 2282827
Full thread : https://looksmax.org/threads/hgh-for-height-growth-is-cope.277736/
Another anecdotical report here : https://looksmax.org/threads/hgh-was-cope-for-me-im-not-growing-past-58-5.410433/

The main problem is that most of the studies were done on gh or igf-1 deficient kids, so we cannot accurately translate the results to healthy persons.
That being said, if you have the money and the time keep going, i'd be happy to learn that you did grew.

Bro stop. I literally talked to EC on discord

 

[ricetard]

Estradiol stimulates IGF-1 production in the liver, and in studies with aromatase-deficient people, they grew slightly after getting oral estrogen because it increased their bottomed-out IGF-1 levels. Aromatase-deficient people take so long to grow and don't really experience a growth spurt because of their low e2. If they had normal HGH levels, they would be even taller, faster.

how would one maintain good estradiol while on letrozole? It’s kind of a paradox because y you can’t have one without losing out on the other

 

[Osie]

how would one maintain good estradiol while on letrozole? It’s kind of a paradox because y you can’t have one without losing out on the other

Your E2 can still be very low, you just don't want it to the point where it's single digits. I'd say around 15pg/ml-17pg/ml should be enough. Most men are in between 30pg/ml - 50pg/ml

 

[Bonecell]

None of this works past 24, its cope. LL is the only way you can height ascend. Everything else is pseudo-scientific big pharma cope.

 

[Bonecell]

5'8

Truecel height in Evropa. Just LL at this rate.

 

[killaeast]

@enchanted_elixir whats the dose of cjc dac and igf lr3?

 

[J o r o]

Bro stop. I literally talked to EC on discord

U bunch of alts lol

 

[Deleted member 28722]

U bunch of alts lol

What?

 

[MrGentleman]

??? Your spine still has room to grow in your early 20s

Quit the reverse your bones fuse starting from your chest and outwards

 

[MrGentleman]

Do you have information about "growth plate never fuse completely"?

Bullshit, even if they don’t you won’t grow any taller just look at people who got acromegaly when got older they have huge heads hands and feet but still manlets

 

[simya]

Folks here state otherwise.

It may work it may not. I said it may not. Theoretically it may in some cases. It certainly cannot be guaranteed.

There's a pathway capable of completely stopping and expanding growth plate. However, it's not a fairy tail. The downside is that you could lose every single strand of hair in your head and many other umpredictable side effects. So I don't make this info public just for safety. Else id have done myself already

Is there somewhere you can explain this? Can you tell me privately?